Sprint and Power events Flashcards
what is power a product of
force x velocity
why can the most force be generated at lower velocities
low velocities allow more time for cross bridge cycling, meaning more cross bridges can be utalised which increases the amount of force that can be generated
At what velocity of contraction is power greatest
30% of max
what are the two main determinants of muscle strength and what is the relationship between these variables and muscle strenght
size:
bigger muscle = more cross bridges
positive correlation between quads cross sectional area and strength
Architecture:
angle of pennation of the muscle
increases as the size of the muscle increases
optimum angle is thought to be 45
explain the difference between anatomical and physiological cross sectional area
for a given muscle which is likely to be larger and why
Anatomical = cross section of the muscles perpendicular axis
Physiological = cross section of the muscles perpendicular fibres
Physiological is likely to be larger because it takes into account the angle of pennation
explain the relationship between muscle size and muscle architecture and the significance of this
Larger muscle sizes will have a larger angle of pennation, potentially larger than the ideal angle
larger pennation angle is associated with a lower force production for a given amount of muscle
body builders > size vs weight lifters
also greater total force
but have lower force production when normalised to muscle size
Compare the characteristics of muscle fibres of body builders, power athletes and controls
size and total force = BB>PA>control
Normalised force = PA>C>BB
Peak power output (watts/kg) = BB=PA>C
normalised = PA>C>BB
describe the relationship between muscle fibre type and speed of shortening
vMax ratio of 1:4 (type I:type II)
isometric force generation capacity is the same but speed of shortening is much greater in type 2
therefore type 2 is able to generate more power
type 2 also has a higher optimum speed of shortening
Explain why a longer muscle is able to produce a faster speed of shortening
longer muscle = more sarcomeres in a line
sarcomeres are a set length and contract a set proportion of that length in a set amount of time
so more sarcomeres = more distance of contraction in the same amount of time
speed = distance/time
compare the findings of biopsies of sprinters muscle with the muscle of average people and endurance runners
sprinter was 80% type 2, half of that was 2x
average = 50% type 1, 45% type 2a, 5% type 2x
endurance = mainly type 1
sprinters have type 2a fibres which can produce more force than other people’s 2a
they also have longer muscle fibres which have a faster shortening velocity
outline evidence of how you can change the fibre type of muscle to increase the velocity of contraction
aim is to increase amount of type 2 fibres, but this is strongly linked to genetics
12 weeks of heavy load resistance training
3 months detraining period
biopsy at 0, 12 weeks and 3 months
following training period 2x was replaced with 2a
after detraining period 2x had returned and there was more than had been present at 0 weeks
give some critique of the work on changing muscle fibre type by resistance training
athletes don’t undergo a period of detraining
don’t know what happens between end of training and end of detraining
don’t know if muscle fibre change remains
describe briefly how increases in muscle size can be achieved
training causes an increase in muscle synthesis for 24 hrs ish
there is also an increase in breakdown but to a lesser extent therefore get a net gain
dose response relationship between training intensity and size increase up to around 60% 1RM
outline the stimulus for muscle hypertrophy
IGF peptide stimulates satellite cells to initiate tissue repair and hypertrophy
sets off a cascade of proteins leading to the activation of mTOR
mTOR activates ribosomes to promote protein synthesis
outline the methods of the trial challenging the guidelines for muscle hypertrophy training
three groups
90% 1RM to fatigue
30% 1RM to match work done by group 1
30% 1RM to fatigue
protein synthesis response was measured