Sports - Lower Extremity Flashcards

1
Q

What are the arthroscopic hip portals and structures at risk?

[AAOS comprehensive review 2, 2014]

A
  1. Anterior
  • Lateral femoral cutaneous nerve
  • Femoral nerve
  • Femoral artery
  1. Anterolateral
    * Superior gluteal nerve
  2. Posterolateral
    * Sciatic nerve
  3. Midanterior
    * Lateral femoral cutaneous nerves
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2
Q

During hip arthroscopy, what nerve is at risk due to traction and the perineal post?

[AAOS comprehensive review 2, 2014]

A

Pudendal nerve

  • Can result in:
    • Hypoaesthesia of the perineum, scrotum and glans penis
    • Erectile dysfunction
    • Urinary incontinence
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3
Q

What are the indications for hip arthroscopy?

[Sports Health. 2017; 9(5): 402–413.]

A
  1. Central compartment
  • Labral tears
  • Chondral pathology
  • Ligamentum teres pathology
  • Septic arthritis
  • Loose bodies
    2. Peripheral compartment
  • Femoroacetabular impingement
  • Subspine impingement
  • Synovial disorders
  • Capsular disorders
  • Psoas tendon disorders
    3. Peritrochanteric compartment
  • Greater trochanteric pain syndrome
  • External snapping hip/iliotibial band disorder
  1. Deep gluteal space
  • Ischiofemoral impingement
  • Proximal hamstring disorders
  • Sciatic nerve disorders
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4
Q

What are the contraindications to hip arthroscopy?

[Sports Health. 2017; 9(5): 402–413.]

A
  1. Advanced OA
  2. Ankylosis
  3. Acetabular and/or femoral dysplasia
  4. Severe deformity
    * Retroversion, SCFE, Perthes
  5. Obesity (relative)
  6. Neurological injuries/disorders (relative)
    * Eg. pudendal neuralgia or peroneal or sciatic nerve palsy
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5
Q

What are the most common complications following hip arthroscopy?

[Bone Joint J. 2017 Dec;99-B(12):1577-1583]

A
  1. Nerve injury (0.9%)
  • Pudendal > LFCN > sciatic > common peroneal > femoral
  • Traction injuries include sciatic, common peroneal and femoral [Muscles Ligaments Tendons J. 2016 Jul-Sep; 6(3): 402–409.]
  • Compression injuries include pudendal nerve
  • Portal placement injuries include LFCN
    2. Iatrogenic injury (0.7%)
  • Chondral > labral
    3. HO (0.6%)
    4. Adhesions (0.2%)
    5. Infection (0.2%)
  • Superficial > deep
    6. Other
  • DVT, perineal skin damage, hematoma, broken instrument, incomplete reshaping, femoral neck fracture, hip instability, iliopsoas tendinitis, AVN, ankle pain, arthrofibrosis, dislocation
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6
Q

What is the most common major complication following hip arthroscopy?

[Bone Joint J. 2017 Dec;99-B(12):1577-1583]

A

Intra-abdominal fluid extravasation

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7
Q

What is the innervation of the acetabular labrum?

[BMC Musculoskeletal Disorders 2014, 15:41]

A

Branch from nerve to quadratus femoris and obturator nerve

  • Contains:
    • Free nerve endings for nociception
    • Nerve end organs (Pacini, Golgi, Ruffini corpuscles) for proprioception
  • Higher concentration in:
    • Anterosuperior and postersuperior labrum
    • Articular side more so than the capsular side
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8
Q

What is the blood supply to the acetabular labrum?

[J Bone Joint Surg Am. 2010 Nov 3;92(15):2570-5]

A

Periacetabular vascular ring

  • Originates from
    • Superior and inferior gluteal vessels
    • Medial and lateral femoral circumflex arteries
    • Intrapelvic vascular system.
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9
Q

What is the function of the hip labrum?

[Journal of Biomechanics 33 (2000) 953-960]

A
  1. Deepens the acetabulum and extends the coverage of the femoral head
  2. Contributes to a negative pressure vacuum effect which adds stability to the hip joint
    * Greater force required to distract joint
  3. Provides a seal against fluid flow in and out of the intra-articular space enhancing lubrication mechanisms
    * Encapsulates the fluid in the joint
  4. Limits the rate of fluid expression from the cartilage during loading which enhances the cartilages ability to carry load and limit stresses on the cartilage
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10
Q

What is the Seldes classification of hip labral tears?

[Clin Orthop Relat Res. 2001 Jan;(382):232-40]

A

Type 1 – “Detachment”

  • Detachment of the labrum from the articular hyaline cartilage at the transition zone

Type 2 – “Intrasubstance”

  • One or more cleavage planes of variable depth within the substance of the labrum
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11
Q

What are the causes of hip labral tears?

[J Am Acad Orthop Surg 2017;25:e53-e62]

A
  1. Trauma
  2. FAI
  3. Dysplasia
  4. Hip hypermobility/capsular laxity
  5. Degeneration
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12
Q

where are labral tears typically located

A

anterosuperior aspect of the acetabulum

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13
Q

describe the decision making algorithm when considering labrum debridement vs repair vs reconstruction

A
  1. stable torn labrum
    1. acetabuloplasty not needed - selective debridement
    2. acetabuloplasty needed - repair
  2. unstable torn labrum
    1. viable tissue = repair
    2. nonviable tissue, young patient - reconstruction
    3. poor vascularity or advanced age - selective debridement
  3. mostly calcified torn labrum
    1. advanced age - selective debridement
    2. young - reconstruction
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14
Q

How can you classify damage to the ligamentum teres, labrum and articular cartilage during hip arthroscopy?

[J Am Acad Orthop Surg 2017;25:e53-e62]

A
  1. Domb classification of ligamentum teres tears
  • Grade 0 = No tear
  • Grade 1 = <50% tear
  • Grade 2 = >50% tear
  • Grade 3 = 100% tear
  1. Seldes Classification of labral tears
  • Grade 1 - chondrolabral junction tear
  • Grade 2 - intrasubstance tear
  1. ALAD (acetabular labrum articular disruption) Classification
  • Grade 1 - softening of the adjacent cartilage
  • Grade 2 - early peel of cartilage
    • Carpet delamination
  • Grade 3 - large flap of cartilage
  • Grade 4 - loss of cartilage
    4. Outerbridge classification
  • Grade 0 - normal cartilage
  • Grade 1 - cartilage with softening and swelling
  • Grade 2 - partial thickness defect with fissures on the surface that do not reach subchondral bone or exceed 1.5cm in diameter
  • Grade 3 - fissuring to the level of the subchondral bone in an area with a diameter larger than 1.5cm
  • Grade 4 - exposed subchondral bone
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15
Q

Describe the decision making algorithm when considering arthroscopic labral debridement vs. repair vs. reconstruction

[J Am Acad Orthop Surg 2017;25:e53-e62]

A
  1. Stable torn labrum
  • Acetabuloplasty not needed = selective debridement
  • Acetabuloplasty needed = repair
  1. Unstable torn labrum
  • Viable tissue = repair
  • Nonviable tissue, young patient = reconstruction
  • Poor vascularity or advanced age = selective debridement
  1. Mostly calcified torn labrum
  • Advanced age = selective debridement
  • Young = reconstruction
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16
Q

What are the 3 main types of FAI?

A
  1. Cam impingement – femoral based abnormality
  2. Pincer impingement – acetabular based abnormality
  3. Combined/mixed-type
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17
Q

What are the features of a cam-lesion?

A
  1. Aspherical femoral head
  2. Reduced head-neck offset
  3. Characteristic ‘bump’ at the head-neck junction
  4. Pistol grip deformity
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18
Q

Where is the typical cam-lesion located?

[J Am Acad Orthop Surg 2013; 21(suppl 1):S20-S26]

A

Anterosuperior head-neck junction

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19
Q

What are the features of the pincer-lesion?

[J Bone Joint Surg Am. 2013;95:82-92]

A
  1. Global overcoverage
    * Coxa profunda, coxa protrusio
  2. Focal overcoverage
    * Cephalad retroversion
  3. Acetabular retroversion
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20
Q

What femur orientation contributes to FAI – anteversion or retroversion?

A

Femoral retroversion

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21
Q

What radiographs and radiographic findings are important in assessing FAI?

[DeLee & Drez’s, 2015]

A
  1. Radiographic views
    1. AP pelvis
    2. cross-table latera
    3. 45 ° Dunn view
    4. False profile view
  2. Signs of pincer-lesion
    1. Crossover sign
      1. i. Normally the anterior lip of the acetebulum lies medial to the posterior lip and converge at the superolateral
        aspect of the acetabulum. With retroversion the anterior lip proximally lies lateral to the posterior lip and
        distally lies medially creating the crossover sign
    2. Prominent ischial spine sign
      1. Normally the ischial spine is hidden behind the acetabulum, if it appears more prominent it indicates acetabular retroversion
    3. Posterior wall sign
      1. Posterior rim of the acetabulum lies medial to the center of rotation of the femoral head indicating
        retroversion
    4. Lateral center edge angle
      1. The lateral center edge angle is the angle formed by a vertical line and a line connecting the femoral head center with the lateral edge of the acetabulum.
      2. LCE >40 suggests pincer-lesion. (<25 indicates dysplasia)
    5. acetabular index angle
      1. abnormal = <0 (>10 indicates dysplasia)
    6. anterior and posterior wall indices
      1. to calculate the acetabular walls index, the best fit circle to the femoral head contour is drawn.
      2. radius of the femoral head is determined, distance from the medial edge of circle to the anterior (aw) and posterior (pw) walls along the femoral neck axis line is measure. anterior wall index (awi) and posterior wall index are calculated as aw/R and pw/r respectively
        1. normal awi - 0.41 (0.3-0.51); normal pwi = 0.91(0.81-1.14)
      3. abnormal = awi increased (anterior overcoverage, pwi increased (posterior overcoverage); awi + pwi increased (global overcoverage)
    7. os acetabulum
    8. anterior femoral neck cortical reaction
    9. posteroinferior joint space narrowing
      1. evident on false profile view, occurs as a result of countercoup lesion with pincer type deformities, poor prognostic sign
    10. Signs of cam-lesion [JAAOS 2013;21(suppl 1):S20-S26]
    11. Alpha angle
      1. A circle is placed over the femoral head. The alpha angle is formed by a line along the axis of the femoral
        neck and a line from the center of the femoral head to the point where the head diverges outside the circle.
      2. An alpha angle >50 degrees is associated with femoroacetabular impingement.
    12. Head-neck offset and offset ratio
      1. Based on a lateral view, a line parallel to the long axis of the femoral neck is drawn along the anterior femoral neck and second line along the anterior aspect of the femoral head. The distance between the two is the head neck offset (<8mm likely represents cam-lesion). The offset ratio is the distance between the two lines divided by the diameter of the femoral head (<0.17 likely represents cam-lesion)
    13. herniation pits
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22
Q

What radiographic view best demonstrates the maximal CAM deformity?

[JAAOS 2013;21(suppl 1):S20-S26]

A

45° Dunn view

  • patient is supine with the pelvis in neutral rotation
  • the hip joint is flexed 90° and abducted 20° while the pelvis remains in neutral rotation
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23
Q

What radiographic view best demonstrates the anterior CAM deformity?

[JAAOS 2013;21(suppl 1):S20-S26]

A

Cross table lateral and frog leg lateral

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24
Q

What special tests should be performed during the physical exam for FAI?

[J Am Acad Orthop Surg 2013; 21(suppl 1):S16-S19]

A
  1. Impingement test (FADIR)
    * With the hip at 90° of hip flexion the hip is internally rotated and adducted
  2. Posterior impingement test
    * Hip extension combined with external rotation
  3. Log roll test
  4. Resisted hip flexion test
  5. FABER
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25
Q

Associated injuries in cam impingement include?

[Orthop Clin N Am 44 (2013) 575–589]

A
  1. Labral detachment from the acetabular rim
  2. Cartilage delamination (full and partial-thickness)
    * Deeper compared to peripheral cartilage delamination in pincer impingement
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26
Q

Associated injuries in pincer impingement include?

[Orthop Clin N Am 44 (2013) 575–589]

A
  1. Labral pathology
  2. Peripheral cartilage delamination
  3. Contracoup chondrolabral lesions in the posterior acetabulum
    * Due to anterior levering of the femur causing posterior shear
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27
Q

when is surgery indicated for FAI

A
  1. symptomatic FAI that fails conservative treatment
  2. patients with a history and physical exam consistent with FAI with radiographic evidence of focal impingement (cam, pincer or both, labral tears or chondrolabral disruptions) and minimal to no arthritic changes are the best candidates
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28
Q

advantages of open surgical hip dislocation and arthroscopy for the management of fAI

A
  1. advantages of open surgical dislocation
    1. 360 ° access to femoral head and acetabulum
    2. optimal visualization for correction of deformity
    3. ability confirm sphericity with open templates
    4. treatment of extra-articualr and intra-articular deformity
    5. optimal visualization with open dynamic assessment
    6. ability to perform relative neck lengthening
  2. advantages of hip arthroscopy
    1. minimally invasive
    2. outpatient procedure
    3. potentially reduced pain
    4. potentially faster rehab
    5. potential for reduced soft tissue injury
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29
Q

disadvantages of open surgical hip dislocation and arthroscopy for the management of FAI

A
  • disadvantages of open
    • trochanteric osteotomy and potential for symptomatic hardware/nonunion
    • increased blood loss
    • ligamentum teres disruption
    • potential for prolonged rehabilitation
    • risk of AVN
  • disadvantages of hip arthroscopy
    • traction-related complications and nerve injury
    • steep learning curve
    • incomplete access and correction of deformity
    • inability to directly confirm restoration of sphericity and offset
    • iatrogenic chondral injury
    • fluid extravasation and thigh or abdominal compartment syndrome
    • portal complications (lateral femoral cutaneous nerve injury)
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30
Q

relative indication for open surgical dislocation rather than arthroscopic

A
  1. large cam deformity with significant posterior and posterolateral extension
  2. associated femoral chondral defects
  3. confirmed or suspected extra-articular ischiofemoral or trochanteric-pelvic impingement
  4. protrusio and coxa profunda
  5. secondary acetabular overcoverage due to cirumferential labral ossification
  6. revision cases
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31
Q

what are predictors of positive outcome following hip arthroscopy for FAI

A
  • pain relief from preop intra-articular hip injections
  • lower BMI (<24.5kg/m2)
  • younger age
  • male sex
  • tonnis grade 0
  • increased joint space
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32
Q

predictors of negative outcomes following hip arthroscopy for FAI

A
  • older age (>45 years)
  • female sex
  • longer duration of preop pain symptoms (>8 months)
  • elevated BMI
  • OA changes - increased Tonnis grade ≥1), increased Kellgren-Lawrence grade (>3)
  • decreased joint space (≥2mm)
  • chondral defects
  • increased lateral center edge angle (LCEA)
  • undergoing of labral debridement rather than labral repair
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33
Q

What are the risk factors for femoral neck fracture after hip arthroscopy for FAI?

[J Hip Preserv Surg. 2017 Jan; 4(1): 9–17]

A
  1. Femoral osteochondroplasty
  2. Early WB
  3. Resection depth 10-33% the width of the femoral neck
  4. Increased age
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34
Q

how is snapping hip classified?

A
    • extra-articular
      • external (most common)
        • cause posterior ITB or anterior gluteus maximums over the greater trochanter
          • when the hip is extended the ITB lies posterior to the greater trochanter and moves anterior over the trochanter when the hip is flexed
      • internal
        • cause - iliopsoas tendon over the iliopectineal eminence of the pelvis or femoral head
          • when the hip is flexed, abducted and externally rotated the iliopsoas lies lateral and moves medial with hip extension, adduction and internal rotation
        • other suspected causes
          • iliopsoas tendon over the lesser trochanter
          • iliopsoas tendon over THA acetabualr component
          • iliofemoral ligament over the femoral head
          • long head of the biceps over the ischium
          • accessory iliopsoas tendon slips
    • intra-articular
      • causes- labral tears, Ligamentum tears, loose bodies, subtle instability
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35
Q

features on history may suggest an external, internal or intra-articular etiology

A
  1. external - lateral snapping sensation, sensation that the hip dislocates
  2. internal - anterior sensation of snapping, “ getting stuck” or locking often assciated with an audible snap
  3. intra-articular - sensation of intermittent clicking or catching (rather than snapping)

note - snapping may be painless and may be reproducible by the patient

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36
Q

what is the management of snapping hip

A
  • first line - non op
  • second line - open or arthroscopic release or lengthening of offending structure
    • external - open z-lengthening or release of ITB, open or arthroscopic release of the gluteus maximums tendon insertion on femur
    • internal - open or arthroscopic lengthening or release of iliopsoas tendon
    • intra-articular - arthroscopic debridement or repair of offending structure
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37
Q

what comprises the proximal hamstring

A
  • semimembranosus (arises from superolateral ischial tuberosity)
  • semitendinosus (arises from the inferomedial ischial tuberosity)
  • long head of the biceps femoris (arises from the inferomedial ischial tuberosity)
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38
Q

2 man types of proximal hamstring injuries

A
  • muscle belly tear
  • acute avlsions (tendinous or osseous from ischial tuberosity)
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39
Q

most common mechanism of proximal hamstring injuries

A

high speed running

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40
Q

indications for imaging of suspected proximal hamstring injuries

A
  1. concerning physical exam findings (ecchymosis or palpable defect)
  2. failure to improve with conservative care
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41
Q

what are the indications for nonop and op management or proximal hamstring injuries?

A
  1. nonop
    1. acute hamstring strains, partial tears, and single tendon avulsions
  2. op
    1. complete avulsion of all 3 tendons with retraction exceeding 2.5 cm
    2. avulsion fracture with displacement exceeding 1cm
    3. sciatic nerve involvement with paresthesia or pain in the posterior aspect of the thigh
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42
Q

ways to control intraop bleeding during knee scope

A

increase pump pressure, reduce outflow suciton, add epi tofluid, controled hypotension, tourniquet, drive scope to bleeding source, cautery, TXA

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43
Q

causes of post traumatic knee hemarthrosis

A
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44
Q

causes of post-traumatic knee hemarthrosis

A
  1. acl tear
  2. pcl tear
  3. mcl tear
  4. patellar dislocation
  5. OC fracture
  6. fracture of tib plateau, distal femur or patella
  7. tibial spine avulsion
  8. emniscus tear
  9. psterolateral corner injury (avulsion of pop tendon
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45
Q

rate of recurrent dislocation after first time patellar dislocation

A

33%

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46
Q

What percent chance exists to have a recurrent dislocation if a patient has had 2 prior patella dislocations?

A

50%

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47
Q

What are the factors that contribute to patella stability?

[JAAOS 2011;19:8-16]

A
  1. Osseous anatomy
  • Trochlear groove
    • Most important stabilizer at flexion >30°
    • Height and slope of the lateral trochlear facet provides the primary resistance
  1. Static stabilizers
  • MPFL and medial patellotibial ligament
    • Most important in full extension when patella is not engaged in trochlea (between 0-30)
    • Function to guide the patella into the groove from 0-20° flexion
      1. Dynamic stabilizers
  • VMO
    • More variable contribution to patella instability
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48
Q

Describe the anatomy of the MPFL?

A

MPFL

summary
- femoral attachment
- center of attachment is between the medial epicondyle and adductor tubercle
- patella attachment
- proximal third of the medial patella

  • Femoral attachment
    • Center of attachment is between the medial epicondyle and the adductor tubercle
  • Patella attachment
    • 7.4mm anterior to a tangent to the posterior patellar cortical line and 5.4mm distal to the proximal edge of the articular surface of the patella
    • Located at the junction of the proximal 1/3 and distal 2/3
    • Encompasses 33% of the total length of the patella [JAAOS 2014;22:175-182]
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49
Q

Describe the anatomy of the medial quadriceps tendon femoral ligament (MQTFL)

A
  • Femoral attachment - between the adductor tubercle and medial epicondyle
  • Insertion - deep layer of the quadriceps tendon
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50
Q

What are risk factors for patellar instability?

[JAAOS 2011;19:8-16]

A
  1. Unfavourable bone anatomy
  • Trochlea dysplasia (shallow, flattened trochlea groove)
    • Seen in <2% of the general population but in 85% of recurrent patellar instability
  • Patella alta
  • Increased TT-TG distance
  • Increased Q angle
  • Lower extremity rotational malalignment
    • femoral anteversion +/- external tibial torsion
      1. Dynamic stabilizer imbalance
  • ITB
    • Increased tension causes lateral patellar tracking
  • VMO/VLO
    • Imbalance in strength can lead to instability
  1. Incompetent static stabilizers
    * Generalized ligamentous laxity
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51
Q

Where anatomically is the MPFL typically injured?

[JBJS 2016;98:417-27]

A

Femoral origin

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52
Q

What is the classification of trochlear dysplasia?

[JAAOS 2011;19:8-16]

A

Dejour classification

  • Type A = shallow
    • Radiographic findings = crossing sign
  • Type B = flat
    • Radiographic findings = crossing sign, supratrochlear spur
  • Type C = lateral convexity/medial hypoplasia
    • Radiographic findings = crossing sign, double contour sign
  • Type D = cliff
    • Radiographic findings = crossing sign, double contour sign, supratrochlear spur
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53
Q

What is the classification of patellar dysplasia based on the asymmetry of the medial and lateral patellar facets?
(Skeletal Radiology (2019) 48:859-869][Sports Med Arthrosc Rev 2019;27:154-160]

A
  • Wiberg’s classification (modified by Baumgart] to include Type 4)
    • Type 1 - the facets are concave, symmetrical and roughly of equal size
    • Type 2 - the medial facet is distinctly smaller than the lateral facet. The lateral facet remains concave, but the medial facet is either flat or slightly convex
    • Type 3 - the medial facet is considerably smaller and convex. The angle subtended by the two facets is nearly 90°
    • Type 4 - no medial facet or median ridge
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54
Q

Why is the patella more unstable (and thus more prone to dislocation) in knee extension rather than flexion?

[J Bone Joint Surg Am. 2008;90:2751-62]

A
  1. Q-angle is greatest in knee extension
  2. Lowest quadriceps and patellar tendon tension
    * Low posterior directed force
  3. Patella disengages from the trochlear groove
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55
Q

What are the physical examination findings in patellar instability?

[JAAOS 2018;26:429-439]

A
  1. J sign
  • Knee is actively brought from flexion to extension
  • Positive = Patella demonstrates a sudden, exaggerated lateral deviation after it fully exits the trochlear groove in full extension
    • Indicates either trochlear dysplasia or patella alta
  1. Moving patellar apprehension test
  • Most sensitive and specific
  • Part 1:
    • Knee held in full extension and the patella is manually translated laterally with the thumb
    • Knee is then flexed to 90o and then brought back to full extension while the lateral force on the patella is maintained
    • Positive:
      • Patient orally expresses apprehension and may activate his or her quadriceps in response to apprehension
  • Part 2:
    • Knee is started in full extension, brought to 90o of flexion, and then back to full extension while the index finger is used to translate the patella medially
    • Positive:
      • Patient experiences no apprehension and allows free flexion and extension of the knee
  1. Lateral patellar translation
    * Normal = Lateral translation of the patella should be ¼ to ½ the width of the patella
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56
Q

What is the radiographic landmark of the femoral insertion of the MPFL called and where is it located?

[Orthop Clin N Am 46 (2015) 147–157]

A
  1. Schottle point
  • 1 mm anterior to the posterior cortex extension line
  • 2.5 mm distal to the posterior origin of the medial femoral condyle
  • Proximal to the level of the posterior point of the Blumensaat line
  1. Stephens normalized dimensions [JAAOS 2014;22:175-182]
  • If AP dimension of medial femoral condyle is 100%, MPFL attachment is:
    • 40% from posterior
    • 50% from distal
    • 60% from anterior
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57
Q

What are radiographic features to assess for in patellar instability?

[Orthop Clin N Am 46 (2015) 147–157] [J Bone Joint Surg Am. 2008;90:2751-62] [JAAOS 2011;19:8-16] )

A
  1. Patella alta
  • Insall-Salvati ratio (normal = 0.8-1.2)
  • Caton-Deschampe ratio (normal = 0.6-1.3)
  • Blackburn-Peel ratio (normal = 0.5-1.0)
  • Blumensaat’s line should extend to the inferior pole of the patella at 30° of knee flexion
    2. Trochlear dysplasia
  • Signs on lateral radiograph include:
    • Normally, floor of the trochlea should not pass anterior to a line extended along the anterior femoral cortex
    • Crossing sign:
      • A line represented by the deepest part of the trochlear groove crossing the anterior aspect of the condyles
    • Supratrochlear spur/prominence
      • Extension of the trochlear groove above the projection of the anterior cortex of the femur
      • Measured as the distance between the anterior femoral cortex and most anterior point of the trochlear floor
        • Abnormal = >4mm
    • Double contour
  • Signs on the skyline view
    • Sulcus angle
      • Measured from the highest point on the condyles to the lowest point in the intercondylar sulcus
      • Normal = 138 ± 6°
      • Abnormal = >145°
        • Indicates trochlear dysplasia
          3. Lateral patellar tilt/subluxation
  • Assess on skyline/Merchant view
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58
Q

What radiograph is superior for evaluating trochlear anatomy?

[JAAOS 2018;26:429-439]

A

Laurin radiograph

  • Knee flexed only 20°
  • Imaging beam directed from inferior to superior

***45° sunrise view does not visualize the proximal trochlear groove (may miss supratrochlear spur)

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59
Q

What is the role of CT in assessing patellar instability?

[Orthop Clin N Am 46 (2015) 147–157] [JAAOS 2018;26:429-439]

A

Used to assess the tibial tubercle-trochlear groove distance

  • Measured by taking a line perpendicular to a line tangential to the posterior femoral condyles through the deepest part of the trochlear groove and through the apex of the tibial tubercle
    • Distance between these 2 lines is defined as the TT-TG distance
  • used to assess patellar maltracking
    • multistage Ct scan at various degrees of flexion
    • imaging at positions both less than and grater than 30 ° can be used to avoid missing maltracking that might be captured at only certain degrees of flexion
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60
Q

What TT-TG distance is associated with patellar instability?

A

>20mm (average normal is 9mm)

  • 10-15mm = normal
    • MPFL reconstruction is usually sufficient
  • 15-20mm = gray zone
    • Consider bony reconstruction in addition to MPFL reconstuction
  • >20mm = abnormal
    • MPFL reconstruction unlikely to be sufficient
    • Needs bony reconstruction in addition to MPFL

***NOTE: 20mm is a guideline and not a rule; MRI values may be less

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61
Q

What is the role of MRI in assessing patellar instability? [Skeletal Radiology (2019) 48:859-869] [Insights into Imaging (2019) 10:65] |JAAOS 2018;26:429-439] |JBJS Am. 2008;90:2751-62][Sports Med Arthrose Rev 2016;24:44-49]

A
  • Used to assess tibial tubercle - posterior cruciate ligament distance
    - May more accurately describe tibial tubercle lateralization (uses tibial reference points)
    - Measured by taking a line perpendicular to the dorsal tibial condylar line through the midpoint of the tibial tubercle and the medial border of the PCL. The distance between the two lines is defined as the TT-PCL distance
    - Abnormal = >20mm
  • Detects acute soft tissue and bony injuries
    - Bone bruising - edema on lateral aspect of lateral femoral condyle and inferomedial patella
    - Cartilage damage - osteochondral fracture of the medial patellar facet or lateral trochlear ridge
    - MPFL damage
    - VMO injury
  • Used to assess patellar tilt
    - Lateral patellofemoral angle
    - Angle formed between a line drawn along the lateral patellar facet and a line along the anterior most points of the medial and lateral femoral condyles
    - Normal = angle should be >8° with the opening laterally
    - Abnormal = angle <8° or medial opening
    - Patella tilt angle
    - Angle between the posterior condylar line and the maximal patella width line
  • Used to assess trochlear dysplasia
    - Lateral trochlear inclination angle
    - Measured on the superior-most axial image showing trochlear cartilage.
    - It is the angle between the plane of the subchondral bone of the lateral trochlear facet, and the posterior femoral condylar line
    - Abnormal = angle of <11°
    - Trochlear depth
    - Trochlear depth = [a + b)/2] - c
    - Where, the maximum AP distance of the medial femoral condyle (a), lateral femoral condyle (b) and trochlear groove (c) as measured from a line parallel to the posterior femoral condyles
    - Facet asymmetry
    - Determined by calculating the percentage of the medial to the lateral femoral facet length
    - Asymmetry of < 40% suggests trochlear dysplasia
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62
Q

What measurement may more accurately describe tibial tubercle lateralization than TT-TG?

[JAAOS 2018;26:429-439]

A

Tibial tubercle – posterior cruciate ligament distance

  • Normal <24mm
  • Both are tibial reference points
    • Not affected by knee rotation like TT-TG
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63
Q

What MRI findings are consistent with patellar instability?

[J Bone Joint Surg Am.2008;90:2751-62][Sports Med Arthrosc Rev 2016;24:44–49)]

A
  1. Bone bruising
    * Edema on lateral aspect of lateral femoral condyle and inferomedial patella
  2. Cartilage damage
    * Osteochondral fracture of the medial patellar facet or lateral trochlear ridge
  3. MPFL damage
  4. VMO injury
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64
Q

What is the treatment of choice for first-time patella dislocation?

[JBJS 2016;98:417-27]

A

Nonoperative treatment

  • Bracing
  • ROM
  • Strengthening
  • Gradual return to play
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65
Q

What is the standard surgical approach in patient with chronic lateral patellar instability with at least 2 documented patellar dislocations?

[JAAOS 2014;22:175-182]

A

Anatomic MPFL reconstruction

  • Miniopen technique
  • Using a graft stronger than the native MPFL
    • Semi-T or gracilis
    • Compensates for the uncorrected predisposing patellar instability factors
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66
Q

What is the ideal indication for an MPFL reconstruction?

[JAAOS 2014;22:175-182]

A
  1. Recurrent lateral patellar dislocation
  2. TT-TG distance <20 mm
  3. Positive apprehension test until 30° of knee flexion
  4. Caton-Deschamps index of <1.2
  5. Normal trochlea or Dejour type A dysplasia
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67
Q

What are the errors in femoral tunnel position for MPFL reconstruction and their implications?

[JAAOS 2014;22:175-182]

A
  1. Too proximal
  • Graft lax in extension and tight in flexion
  • Clinically:
    • Anterior knee pain
    • Loss of flexion
    • Graft stretch and failure
  1. Too distal
  • Graft tight in extension and lax in flexion
  • Clinically:
    • Extension lag
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68
Q

Where is the MPFL graft typically passed between the patella and femur?

A

Between layer 2 (medial patellar retinaculum) and layer 3 (capsule)

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69
Q

At what knee flexion should graft tensioning be performed for MPFL reconstruction? [Sports Med Arthrosc Rev 2019;27:136-1423

A
  • ≥60 degrees of knee flexion
    • The key is setting the length at the degree of flexion at which the attachment sites are farthest apart so the graft will become more lax at other degrees of flexion
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70
Q

Is the native MPFL isometric?

[JAAOS 2014;22:175-182]

A

The MPFL is nonisometric over the complete ROM

  • Tight in extension, Lax in flexion
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71
Q

How should the MPFL graft function after reconstruction?

[JAAOS 2014;22:175-182]

A
  1. The ROM after MPFL graft fixation should be complete
  2. The MPFL graft should be isometric from 0-30°
  3. The MPFL graft should tighten in extension and be lax in flexion
  4. There should be a good endpoint to lateral patellar translation from 0-30°
  5. The MPFL should only tighten on lateral patellar translation
  6. Should permit 1cm of lateral translation or the equivalent of two quadrants of lateral deviation with a firm end point
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72
Q

What are the most common complications following MPFL reconstruction?

[JBJS 2016;98:417-27]

A
  1. Recurrent apprehension
  2. Arthrofibrosis
  3. Pain
  4. Clinical failure
  5. Patellar fracture
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73
Q

What is the redislocation rate following MPFL reconstruction?

[JAAOS 2018;26:429-439]

A

<10%

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74
Q

What are the indications and contraindications for a tibial tubercle transfer in the setting of patellar instability?

[JAAOS 2018;26:429-439]

A

Indications:

  • TT-TG >20mm (measured on CT)
  • High TT-PCL distance (>24mm)
  • Caton-Deschamp ratio >1.2

Contraindications [JBJS 2016;98:417-27]

  • Open tibial apophysis
    • Causes growth arrest and recurvatum
  • Medial dislocations
  • Patellofemoral OA of the proximal and medial facets
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75
Q

What is the postoperative goal TT-TG after tibial tubercle transfer for patellar instability?

A
  1. 10mm [JAAOS 2018;26:429-439]
  2. 9-15mm [JBJS 2016;98:417-27]
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76
Q

What is the main complication following tibial tubercle transfer for patellar instability?

[JBJS 2016;98:417-27]

A

Symptomatic hardware

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77
Q

In the presence of trochlear dysplasia what is the recommended treatment for patellar instability?

[JAAOS 2011;19:8-16]

A
  1. First-line
  • Procedures to address associated factors rather than trochleoplasty
    • TT-TG and patella height normal = MPFL reconstruction
    • TT-TG >20mm = tibial tubercle transfer or femoral derotation osteotomy
      1. Second-line
  • Trochleoplasty
    • Involves reshaping the trochlea by deepening the groove by removing subchondral bone and impacting the overlying catilage into the defect
    • Indications:
      • High-grade trochlear dysplasia (where other options won’t provide stability) and salvage situations
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78
Q

What type of trochlea according to the Dejour classification is most likely to benefit from a trochleoplasty?

[JAAOS 2018;26:429-439]

A

Type B and D

  • Both have supratrochlear spurs – tend to displace the patella laterally
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79
Q

What are the indications and contraindications for trochleoplasty?

[JAAOS 2018;26:429-439]

A
  • Indications
    • dejour tpe B or D trochlear dysplasia
    • Supratrochlear spur height > 5mm
    • Presence of J-sign
  • Contraindications
    • Dejour type A or C trochlear dysplasia
    • Advanced patellofemoral OA
    • Open physes
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80
Q

What are the main goals of sulcus-deepening trochleoplasty? [Sports Med Arthrosc Rev 2019;27:161-168]

A
  • Eliminating the supratrochlear spur
  • Deepening the trochlear sulcus
  • Lateralizing the trochlear sulcus (effectively decreases the TT-TG distance)
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81
Q

What are the indications for tibial tubercle osteotomy (distal realignment procedures)?

[Clin Sports Med 33 (2014) 517–530]

A
  1. Recurrent patella instability if
  • Skeletally mature
    • Proximal tibia and tibial tubercle physis closed
  • TT-TG >15
  • Caton-Deschamps >1.2
  1. Unload focal patella cartilage lesions
  • Without cartilage resurfacing for lateral or distal lesions
  • With cartilage resurfacing for central or medial lesions
  1. Isolated lateral patellofemoral compression, tilt or overload in patients who fail lateral release
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82
Q

What are contraindications for TTO?

[Clin Sports Med 33 (2014) 517–530] [AJSM 2014; 42(8): 2006)

A
  1. Skeletally immature
  2. Medial patellar subluxation/dislocation
  3. Diffuse patellofemoral arthrosis
  4. Smoking
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83
Q

What are the types of TTOs?

[Clin Sports Med 33 (2014) 517–530]

A
  1. Medialization (Elmslie-Trillat procedure)
  • Reduces the TT-TG
  • Decreases pressure on lateral facet
  • Amount of medialization is limited by the amount of bony contact available
  1. Anterior transfer (Maquet)
  • Goal is to reduce patellofemoral contact pressures
  • Transfers the contact forces from distal to proximal patella
  • Requires allograft or autograft bone block
  • Not routinely done
    • Anteriorization usually combined with medialization
  • Can be indicated for unloading large distal patellar chondral lesions, bipolar kissing lesions or arthritis in setting of normal TT-TG
    3. Distalization
  • Indications:
    • Caton-Deschamps or Insall-Salvati >1.2 (usually >1.4)
  • Rarely done in isolation
    • Usually combined multiplanar osteotomy
  • Caution with overdistalization which can limit knee flexion
  1. Anteromedialization (Fulkerson)
  • The amount of anteriorization and medialization is determined by the obliquity of the osteotomy
    • Increased obliquity [A-P] = increased anteriorization
    • With a constant anteriorization of 15mm: [AJSM 2014; 42(8): 2006)
      • 60° slope creates ~9mm of medialization
      • 45° slope creates ~15mm of medialization
  • Advantages:
    • Preserves extensor mechanism
    • Large surface for bone healing
    • Ability to place multiple screws
    • Multiplanar adjustments
    • Early ROM
  • Disadvantages
    • Does not address incompetent MPFL
    • Hardware irritation
    • Potential NV injury
    • Increased medial PF contact pressures
    • Delayed union/nonunion
    • Cannot perform in skeletally immature
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84
Q

What are the complications associated with TTO?

[Clin Sports Med 33 (2014) 517–530][AJSM 2014; 42(8): 2006)

A
  1. Hardware irritation (50% require removal)
  2. Nonunion/delayed union
  3. Compartment syndrome
  4. Anterior tibial artery injury
  5. Deep peroneal injury
  6. Infection
  7. DVT
  8. Medial patellar instability
  9. Fracture of tubercle
  10. Fracture of proximal tibia
  11. Skin necrosis
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85
Q

What is the etiology of ACL tears?

[J Am Acad Orthop Surg 2013;21:41-50]

A
  1. Noncontact injuries (70%)
    * Most commonly a deceleration event and change in direction with a planted foot (cutting maneuver)
  2. Trauma (30%)
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86
Q

What are the risk factors for an ACL tear?

[J Am Acad Orthop Surg 2013;21:41-50]

A
  1. Female sex
  2. Increased friction and shoe-playing surface interface (eg. cleats)
  3. Notch stenosis
  4. Increased posterior tibial slope
  5. Increased Q-angle
  6. Smaller ACL
  7. Increased quadriceps to hamstring strength
  8. Poor landing position
    * Erect, hips adducted/internally rotated, knee relatively extended/valgus, tibia externally rotated
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87
Q

What factors make females 2-8x more likely than male athletes to sustain an ACL injury?

[JAAOS 2013;21:41-50]

A
  1. Increased Q angle
  2. Notch stenosis
  3. Smaller ACL
  4. Increased quadriceps to hamstring strength and recruitment ratio
  5. Poor landing position
  6. Others:
  • Increased medial posterior tibial slope
  • Hormonal factors
  • Increased generalized ligamentous laxit
  • Increased knee laxity
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88
Q

P/E special tests for ACL tears

A
  • anterior drawer test
    • patient supine, hip flexed at 45° knee flexed at 90°, the foot is fixed to the table, anterior force is applied to proximal tibia while palpating the joint line for anterior translation
    • positive = increased anterior tibial translation
  • lachman test
    • with the patient supine and the knee flexed 20-30°, the femur is stabilized laterally with one hand and the other hand grasps the tibia medially and translates the tibia anteriorly
    • positive = increased anterior tibial translation
    • grading
      • anterior translation of affected knee compared to contralateral knee
        • Grade I = >1-5mm
        • Grade II = 6-10mm
        • Grade III = >10mm
      • pivot shift test
        • with the patient supine one hand grasps the heel and the other hand is placed over the fibular head and applies a valgus force while taking the knee form extension into flexion
        • positive = reduction of a subluxated tibia at 30-40 ° sensed as a clunk or a glide
        • Grading
          • I = glide
          • II = clunk
          • III = gross reduction
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89
Q

What are the indications for ACL reconstruction?

[AAOS comprehensive review 2, 2014]

A
  1. Athletes who perform cutting and pivoting sports
  2. High demand occupations (eg. police or military)
  3. High risk occupations (eg. firefighter)
  4. Recurrent instability
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90
Q

What are indications for nonoperative management of ACL injuries?

[AAOS comprehensive review 2, 2014]

A
  1. Low physical demand patients
  2. Older age
  3. Advanced osteoarthritis
  4. Patients unwilling or unable to comply with postoperative rehab
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91
Q

What radiographic features would suggest a possible ACL injury (4)?

A
  1. Effusion
  2. Segond fracture
  3. Tibial tubercle avulsion
  4. Lateral femoral notch sign/deep sulcus sign
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92
Q

What are the MRI findings to support the diagnosis of an ACL tear?

A
  1. Direct
  • Fibre discontinuity
  • Increased signal intensity
  • Abnormal orientation
  • ‘Empty notch sign’ (chronic)
    2. Indirect
  • Bone bruises
  • Anterior tibial translation
  • PCL buckling (reduced PCL angle)
  • Uncovering of the posterior horn of the lateral meniscus
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93
Q

What is the collagen composition of the ACL?

[World J Orthop 2015,18;6(2):252-262]

A

Type I – 90%

Type III – 10%

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94
Q

What are the bundles of the ACL and what are their function?

[Sports Med Arthrosc Rev 2010;18:27–32] [Clin Sports Med 36 (2017) 9–23]

A

Bundle named based on tibial insertion

  • Anteromedial
    • Main contributor to AP stability
    • Taught in flexion, relaxed in extension
  • Posterolateral
    • Main contributor to rotational stability
    • Taught in extension, relaxed in flexion
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95
Q

Describe the femoral insertion of the ACL?

A

Medial wall of the lateral femoral condyle

  • Posterior to the resident’s ridge (lateral intercondylar ridge)
  • Lateral bifurcate ridge separating the AM from the PL bundle
    • AM bundle superior and PL bundle is inferior to bifurcate ridge
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96
Q

What are the arthroscopic landmarks to determine the centre of the ACL femoral footprint (single bundle technique)?

[JAAOS 2016;24:443-454]

A
  1. 8mm anterior to the posterior articular margin of the lateral femoral condyle
  2. 1.7mm proximal to the bifurcate ridge
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97
Q

How do you landmark the ACL femoral footprint using fluoroscopy?

[JAAOS 2016;24:443-454]

A

For single-bundle reconstruction:

  • Center of the femoral footprint can be referenced on a grid using the Blumensaat line
  • 28% of the distance from proximal to distal
  • 34% posterior to the Blumensaat line
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98
Q

Describe the tibial insertion of the ACL?

[Clin Sports Med 36 (2017) 9–23]

A

Broad insertion anterior and between the medial and lateral tibial spines

  • AM bundle located more anterior and medial
  • PL bundle located more posterior and lateral
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99
Q

What are the arthroscopic landmarks to determine the centre of the ACL tibial footprint (single bundle technique)?

[JAAOS 2016;24:443-454]

A
  1. 9mm posterior to the posterior edge of the intermeniscal ligament
  2. 5mm anterior to the peak of the medial tibial spine
  3. Midway between the medial and lateral tibial spines in the coronal plane

***Note: not recommended to use the lateral meniscus as a reference as the anterior horn insertion is variable

  • If used the centre of the footprint is posterior and medial to the anterior insertion
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100
Q

How do you landmark the ACL tibial footprint using fluoroscopy?

[JAAOS 2016;24:443-454]

A
  1. 43% of the distance anterior to posterior of the midsagittal tibial diameter
  2. 51% medial to lateral on the AP view
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101
Q

On a postoperative radiograph, what is the ideal ACL tibial tunnel position and femoral tunnel position?

[Skeletal Radiol (2013) 42:1489–1500]

A
  1. Lateral
  • Anterior wall of the tibial tunnel should be posterior to a line extended along Blumensaat’s line with knee in extension
  • No part of the femoral tunnel should intersect Blumensaat’s line
  1. AP
  • Angle between the femoral anatomical axis and the femoral tunnel ideally is 39°
    • ≤17° is associated with rotational instability
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102
Q

Describe the relationship of the ACL AM to the PL bundle in extension compared to flexion?

[World J Orthop 2015,18;6(2):252-262]

A
  1. Extension
  • Femoral insertions are vertical (AM above PL)
  • Bundles are parallel
  • PL bundle is tight and AM bundle is relaxed
  1. Flexion
  • Femoral insertions are horizontal (AM posterior to PL)
  • Bundles are crossed
  • PL bundle is relaxed and AM bundle is tight
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103
Q

What is the surgical terminology used when describing the ACL femoral tunnel placement?

A

With knee in flexion as occurs during surgery:

  • Shallow/deep (A-P)
  • High/low (superior-inferior)
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104
Q

Is the native ACL isometric?

[World J Orthop 2015,18;6(2):252-262]

A

No

  • “Isometry in ACL does not exist as there is no one point on femur that maintains a fixed distance from a single point on the tibia during the range of motion of the knee.”
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105
Q

What is the rational for isometric ACL graft placement?

[World J Orthop 2015,18;6(2):252-262]

A
  1. An isometric graft avoids changes in graft length and tension during knee flexion and extension which avoids graft failure by overstretching
  2. Downside is it results in a more vertical graft which is not as effective in controlling rotation
  3. Isometric point on the femoral side is high in the notch and posterior
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106
Q

What clock position is thought to be optimal for the ACL femoral tunnel to achieve graft isometry?

[World J Orthop 2015,18;6(2):252-262]

A

11 o’clock (right) and 1 o’clock (left)

  • Apex of the notch represented by 12 o’clock

***NOTE – changing the position to 10 o’clock (right) or 2 o’clock (left) improves the rotational stability

  • Graft becomes less vertical
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107
Q

What is the blood supply of the ACL?

[Clin J Sport Med 2012;22:349–355]

A
  1. Primary – middle genicular artery
  2. Secondary – inferomedial and inferolateral genicular arteries
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108
Q

What is the innervation of the ACL?

[Clin Sports Med 36 (2017) 9–23]

A

Branches of the tibial nerve

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109
Q

What is the average length of the native ACL?

[World J Orthop 2015,18;6(2):252-262]

A

33mm

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110
Q

What is a cyclop lesion (in context of ACL)?

A

Focal nodule of fibrous tissue sitting in the intercondylar notch anterior to the reconstructed ACL

  • Significance:
    • Limits complete knee extension due to intercondylar notch impingement
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111
Q

With an ACL injury, what is the typical bone bruises identified on MRI and what is their significance?

[AAOS comprehensive review 2, 2014][Sports Med Arthrosc Rev 2016;24:44–49]

A
  1. Anterolateral femoral condyle (sulcus terminalis) and posterolateral tibial plateau (“kissing lesion”)
  2. Compared to those without bone bruises:
  • Protracted clinical recovery
  • Greater effusions and pain scores
  • Slower return of motion
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112
Q

What is the most common meniscus injured in the acute vs. chronic ACL deficient knee?

[J Am Acad Orthop Surg 2013;21:204-213]

A
  1. Acute ACL injury – lateral meniscus tear
  2. Chronic ACL injury – medial meniscus tear
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113
Q

What are graft options for ACL reconstruction?

A
  1. Hamstring autograft
  2. Bone-patellar-bone autograft
  3. Quadriceps tendon autograft
  4. Allograft
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114
Q

Which ACL graft has the greatest strength and stiffness?

A

Quadrupled-strand hamstring graft

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115
Q

What ACL graft size diameter (mm) has been shown to decrease failure rates?

[Arthroscopy 2014 Jul;30(7):882-90]

A

8mm

  • “Quadrupled-strand hamstring autograft with a diameter equal to or larger than 8 mm decreases failure rates. Grafts larger than 8 mm were found to provide a protective effect in patients aged younger than 20 years, a group identified to be at increased risk of failure”
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116
Q

What is the concern with ACL grafts that are too large?

[JBJS 2017;99:438-45]

A

Impingement of the graft on the roof and PCL

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117
Q

When selecting an interference screw for ACL soft tissue grafts, how can you increase the fixation strength?

[JBJS 2017;99:438-45]

A

Increase screw length or diameter

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118
Q

What is the general recommendation for notchplasty during ACL reconstruction?

[JBJS 2017;99:438-45]

A

Notchplasty is not recommended during ACL reconstruction

  • Smaller intercondylar notch dimensions are not associated with ACL graft failure
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119
Q

What are the cons of using an ACL BTB autograft?

[Clin J Sport Med 2012;22:349–355] [DeLee & Drez’s, 2015]

A
  1. Anterior knee pain
  2. Loss of sensation
  3. Patellar fracture
  4. Inferior patellar contracture
  5. Patellar tendon rupture
  6. Quadriceps weakness
  7. increaserd risk of patellofemoral OA
  8. kneeling pain
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120
Q

What is one way to increase the strength of an ACL BTB autograft?

[JBJS 2017;99:438-45]

A

Twisting the graft

  • “Twisting the graft by 90° increased the ultimate strength by 30%”
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121
Q

What portion of the ACL BTB graft is the strongest and stiffest – medial, central or lateral 1/3?

[JBJS 2017;99:438-45]

A

Central 1/3

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122
Q

What are two ways to manage graft tunnel mismatch when an ACL BTB graft is too long?

[JBJS 2017;99:438-45]

A
  1. Rotate the graft
  • Based on rotation from the proximal (patellar) end
  • External rotation has been shown to more significantly reduce graft length compared with internal rotation
    • Shortening of 25% at 630° external rotation
  1. Single bone plug
    * Proximal patellar tendon is removed from patella without a bone plug
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123
Q

What are the cons of using hamstring autograft for ACL reconstruction?

[Clin J Sport Med 2012;22:349–355]

A
  1. Decreased hamstring strength and endurance
  2. Difficult to harvest due to variable graft length and diameter
  3. higher failure rate than BTB
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124
Q

What are the advantages and disadvantages of allograft for ACL reconstruction?

[Clin J Sport Med 2012;22:349–355]

A

Advantages

  • Avoid donor-site morbidity
  • Reduced operative time
  • Availability of larger grafts
  • Superior cosmesis
  • Possibility for multiple ligament reconstructions

Disadvantages

  • Delayed graft incorporation
  • Disease transmission
  • Potential immune reactions
  • Altered mechanical properties caused by sterilization
  • Cost of the allograft
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125
Q

Which ACL graft, hamstring or BTB, shows greater migration of osseus fixation and graft stretching?

[JBJS 2017;99:438-45]

A

Hamstring

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126
Q

What are the main techniques for ACL reconstruction with respect to femoral tunnel preparation?

A
  1. Anteromedial
  2. Transtibial
  3. Outside-in
  4. all inside
127
Q

which ACL reconstruction technique often results in a nonanatomic femoral tunnel position

A

transtibial

128
Q

features of an all-inside technique for ACL reconstruction

A
  1. sockets are drilled from inside out
  2. graft is introduced into the knee from an arthroscopic portal rather than a tunnel
129
Q

advantages of an all-inside technique for ACL reconstruction

A
  • improved cosmesis
  • preserves bone
  • reduced postop pain and swelling
  • retrograde drilling of the femoral tunnel results in longer intraosseous distances
  • quadrupled semitendinosus graft can be utilized which preserves the gracilis tendon
    • flexion strength is improved postop
    • gracilis can be used for other ligament reconstruction or revision ACL
131
Q

Which ACL reconstruction technique results in a more anatomic femoral tunnel position?

[World J Orthop 2015,18;6(2):252-262]

A

Anteromedial portal technique

132
Q

What is the clinical difference between single and double bundle ACL reconstruction?

[JBJS 2017;99:438-45] [Arthroscopy. 2015 Jun;31(6):1185-96]

A

No significant difference

  • DB ACL-R provides significantly better knee stability (by KT arthrometry and pivot-shift testing) than SB ACL-R but no advantages in clinical outcomes or risk of graft failure
133
Q

What is the most common cause of primary ACL reconstruction failures?

[Sport Health 2014; 6(6):504]

A

Technical error

  • Improper tunnel placement outside the native femoral and tibial ACL footprints
134
Q

What is the consequence of improper ACL tunnel placement on the femoral and tibial side?

A
  1. Errors in femoral tunnel placement
  • Too anterior
    • Excessive graft tensioning with flexion leading to loss of knee flexion or stretching of the graft
  • Too posterior
    • Excessive graft tension in extension with laxity in flexion
  • Too vertical
    • Inadequate rotational stability
      1. Errors in tibial tunnel placement
  • Too anterior
    • Notch impingment and loss of extension
  • Too posterior
    • PCL impingement and loss of flexion
  • Too medial/lateral
    • Notch impingement and iatrogenic tibial plateau cartilage damage
135
Q

What evidence is there to support functional bracing post-ACL reconstruction?

[Knee Surg Sports Traumatol Arthrosc (2014) 22:1131–1141]

A
  1. No brace has been validated in the literature to improve patient outcomes or reduce the risk of reinjury
  2. Functional bracing improves proprioception, feelings of stability and confidence
  3. Functional bracing reduces anterior tibial translation (up to 140N) and rotational torques (up to 8Nm)
136
Q

What are potential risk factors for the development of posttraumatic OA following ACL injury?

[Clin Sports Med 32 (2013) 1–12]

A
  1. Meniscus injury
    * Status of the meniscus may be the most important factor for the development of OA regardless of nonoperative or ACL reconstruction
  2. BMI
    * Correlation between BMI and development of OA
  3. Chondral injury
    * May occur at time of injury or secondary to instability
  4. Graft choice
    * BTB graft may have an increased prevalence of OA compared to hamstring graft
  5. Timing of ACL reconstruction
    * Acute surgery may prevent secondary meniscal and chondral injuries
137
Q

At a minimum 10 year followup, what is the risk of developing posttraumatic OA after ACL injury treated nonoperative compared to ACL reconstruction?

[AJSM 2014; 42(9):2242]

A
  1. The relative risk of developing OA after an ACL injury (regardless of associated injuries or treatment) is 3.89 compared to the contralateral uninjured knee
    * i.e. The ACL injured knee is almost 4x more likely to develop OA compared to the contralateral knee
  2. The relative risk of developing OA after ACL reconstruction (regardless of meniscal injury) is 3.62 compared to the contralateral knee
  3. The relative risk of developing OA treated nonoperative (regardless of meniscal injury) is 4.98 compared to the contralateral knee
  4. Therefore, ACL reconstruction reduces the risk of developing OA compared to nonoperative treatment
138
Q

investigation recommended to assist in selecting appropriate surgical technique

A

wrist/hand xray - determines Sanders bone age

139
Q

risk of delaying ACLR or treating nonop in skeletally immature patients

A
  • meniscal and chondral injury
  • psychological effects resulting from activity restrictions
140
Q

RF for physeal injury in ACL reconstruction

A
  • proportion of physis violation
    • <5% = low risk
  • location of tunnel within physis
    • central = low risk of arrest(compared to peripheral)
  • orientation of tunnel
    • vertical tunnel = low risk due to lower proportion of physis affected
141
Q

indications for nonop and operative intervention

A
  • nonop
    • partial tear
    • normal or near normal physical exam
  • operative
    • older patients
    • instability on physical exam
142
Q

surgical ACL reconstruction options available in skeletally immature patient

A
  1. intra-articular extraphyseal
  2. all-epiphyseal
  3. partial transphyseal
  4. physeal-respecting transphyseal
  5. traditional transphyseal
143
Q

what ACL reconstruction options should be considered based on age

A
  1. bone age ≤8
    1. intra-articular extraphyseal
  2. bone age >8 and ≥ 2 years of growth remaining
    1. all-epiphyseal
  3. <2 years of growth remaining (approaching skeletal maturity)
    1. partial transphyseal
    2. physeal-respecting transphyseal
  4. skeletally mature
    1. traditional transphyseal
144
Q

intra-articular extraphyseal technique

A
  • IT band reconstruction (Kocher technique)
    1. nonanatomic
    2. midsubstance slip of the iliotibial band is looped posterolaterally over the lateral femoral condyle then passed through the intercondylar region, through the joint, and under the intermeniscal ligament to form a new ACL. A trough is placed under the intermeniscal ligament to allow for more anatomic graft placement without causing direct physeal injury
    3. proximally, autogenous graft is sutured to the periosteum of the lateral femoral condyle at the insertion of the intermuscular septum. Distal to the joint, the graft is sutured to periosteal flaps at the proximal anterior tibia with the use of heavy nonabsorbable sutures
145
Q

what is the all epiphyseal technique

A
  • tunnels are drilled in femoral and tibial epiphysis under fluoro visualization
  • hamstring autograft is fixed on the femoral side with a cortical button or interference screw
  • fixation on the tibial side can be with a cortical button or interference screw or distally over a post
146
Q

partial transphyseal (hybrid technique)

A
  1. all epiphyseal femoral tunnel
    1. avoidance is suggested due to
      1. more longitudinal growth from the distal femoral physis compared to the proximal tibial physis
      2. femoral tunnel is more peripheral - higher risk of angular deformity
  2. central transphyseal tibial tunnel
147
Q

what is transphyseal technique

A

femoral and tibial tunnels are transphyseal

148
Q

practices that can be used to avoid physeal injury in transphyseal ACL reconstruction technique

A
  1. vertical tunnel
  2. tunnel diameter ≤ 8mm
  3. central tunnels (avoid peripheral tunnels)
  4. avoid perichondral ring
  5. hardware and bone should not be placed across the physes
  6. soft tissue grafts preferred
  7. heat necrosis should be avoided by hand drilling or slow drilling speeds
149
Q

recommended graft choice for peds ACL

A
  1. all soft tissue and autograft
  2. note - if BTB graft is used avoid placement of bone at the level of the physis, which may increase risk of bony bridging across the physis
150
Q

risk of graft failure and retear in skeletally immature ACL reconstruction

151
Q

What is the etiology of tunnel widening following ACL reconstruction?

[JAAOS 2010;18:695-706]

A
  1. Mechanical factors
  • Mechanical theory suggests that graft motion within the tunnel can abrade the tunnel edge producing widening
  • Graft position
    • Malpositioned tunnels can lead to increased graft forces being transmitted to the graft-bone interface
    • Eg. anterior tibial tunnel leads to notch impingement and increased force on the graft
  • Fixation method
    • Increased distance between fixation points leads to decreased stiffness and increased graft motion
    • Bungee cord effect
      • Longitudinal graft motion with elongation and retraction of the graft in the tunnel
      • Seen with suspensory fixation compared to aperture fixation
    • Windshield wiper effect
      • Transverse graft motion within the tunnel
      • Seen with fixation not at the aperture of the tunnel
        2. Biological factors
  • Graft sterilization
    • Ethylene oxide sterilization associated with tunnel widening
  • Inflammatroy cytokines
    • May increase osteoclastic activity
  • Implant material
    • Bioabsorbable screws may have greater tunnel widening compared to metal screws
  • Graft type
    • Hamstring autograft shows greater tunnel widening compared to BTB
  • Graft donor
    • May be more widening with allograft compared to autograft
  • Synovial fluid propagation
    • Influx of synovial fluid may delay healing
152
Q

What is acceptable tunnel widening in the first 6-12 months post ACL reconstuction?

[Skeletal Radiol (2013) 42:1489–1500]

153
Q

What radiographic features suggest tunnel widening post ACL reconstruction?

[Skeletal Radiol (2013) 42:1489–1500]

A
  1. Widening >2mm
  2. Loss of parallel walls (cone-shaped)
154
Q

What is the best imaging modality for assessing tunnel widening post ACL reconstruction?

[Skeletal Radiol (2013) 42:1489–1500]

155
Q

What are the clinical implications of tunnel widening?

[JAAOS 2010;18:695-706]

A
  1. No implication to clinical outcome
  2. Significant if ACL revision surgery undertaken
156
Q

What are the suggested options for graft choice and implant during revision ACL recon in setting of tunnel widening?

[JAAOS 2010;18:695-706]

A
  1. Allograft achilles tendon
    * If autograft was used for primary
  2. Autograft BTB
    * If allograft was used for primary
  3. Implant – aperature fixation with metallic screw
157
Q

What are the options to manage tunnel widening during ACL revision surgery?

[JAAOS 2010;18:695-706]

A
  1. Single stage (<100% widening)
  • Option in good bone stock
    • Divergent tunnel (funnel) technique
  • Options in poor bone stock
    • Stacked screw
    • Matchstick (bullet) graft
    • Large bone plug graft
      1. Two-stage (>100% widening or tunnel diameter >15mm) [Skeletal Radiol (2017) 46:161–169]
  • Primary bone graft with delayed reconstruction
    • 3-4 months post-bone grafting
158
Q

What is the most common organism causing septic arthritis post-ACL reconstruction?

[J Am Acad Orthop Surg 2013;21:647-656]

A

Staph aureus (31%)

159
Q

In cases of ACL graft contamination (‘dropped graft’) how should you proceed?

[J Am Acad Orthop Surg 2013;21:647-656]

A

Cleanse with NS, followed by chlorhexidine then NS

160
Q

What is the recommended management of septic arthritis post-ACL reconstruction?

[J Am Acad Orthop Surg 2013;21:647-656]

A
  1. Perform arthrocentesis and blood culture prior to antibiotics
    * Septic arthritis suggested if cell count >100,000 (may be as low as 25,000) and cell differential >90 PMNs
  2. Start broad-spectrum antibiotics
    * Can delay until after intra-operative cultures are sent if patient is stable
  3. Immediate arthroscopic I&D
    * Assess graft integrity and viability (probe graft, pivot-shift)
    * Send intraoperative synovial fluid and synovium samples for culture
    * Extensive lavage and synovectomy
    * Graft removal is recommended in the setting of:
    • Significant intrasubstance degeneration
    • Gross evidence of infection compromising the graft
    • Nonfunctional graft as determined by inadequate graft tension or significant pivot shift performed under anesthesia
      * A closed-suction drainage system is typically used postoperatively to assist in wound drainage and is kept in place until there is minimal output
      * Immobilize knee in extension for 24 hours
  4. Second arthroscopic I&D is recommended
    * Repeat synovial fluid aspiration and synovium cultures
  5. 6 weeks of IV antibiotics tailored to culture and sensitivity
  6. When the graft and associated hardware must be removed, a minimum 6-month waiting period after infection eradication is generally recommended before proceeding with graft reimplantation
    * Inection eradication indicated by normalized inflammatory markers and/or normal joint aspirate cell count and culture
161
Q

What structures comprise the anterolateral complex (ALC) of the knee (from superficial to deep)? [Knee Surgery, Sports Traumatology, Arthroscopy (2019) 27:166-176][Am J Sports Med. 2017 Sep; 45(11):2595-2603]

A
  1. Superficial iliotibial band and iliopatellar band
  2. Deep iliotibial comprised of:
    a. Proximal Kaplan fibers - traverse from the undersurface of the ITB in an anterior and proximal orientation to its femoral insertion distal to the lateral intermuscular septum
    b. Distal Kaplan fibers - originate adjacent to the proximal bundle and insert distal to it along the supracondylar ridge of the distal femur
    c. Capsule-osseous layer - originates in close proximity to the lateral gastrocnemius tubercle and inserts near a tubercle, called the lateral tibial tubercle, on the anterolateral aspect of the proximal tibia, between the Gerdy tubercle and the fibular head
  3. Anterolateral ligament (ALL) and capsule
162
Q

What is the anatomy of the native Anterolateral Ligament (ALL) of the knee ?

[Arthroscopy 2019 35(2):670-681]

A
  1. Femoral footprint
  • Proximal and posterior to the lateral epicondyle
  • Most variable in the literature but recent trend is proximal and posterior to lateral epicondyle
  1. Tibial footprint
  • Midpoint between the fibular head and Gerdy’s tubercle
  • 4-7mm distal to tibial plateau
  1. Lateral meniscus attachment
    * Between the body and anterior horn of the meniscus
  2. Orientation
    * Fibres run anteroinferior
  3. Length = 30.41 - 59mm
  4. Thickness = 1 - 2mm
163
Q

What structures resist anterolateral rotation (internal tibial rotation) of the knee?

[Clin Sports Med 36 (2017) 135–153]

A
  1. ACL
  2. ALL
  3. Posterior horn of the lateral meniscus
  4. ITB
164
Q

What is the function of a lateral extra-articular tenodesis (LET) combined with an ACL reconstruction?

[Clin Sports Med 36 (2017) 135–153]

A
  1. Protects the ACL graft
    * Demonstrates load-sharing during anterior translation and internal rotation of the tibia
  2. Reduces tibial internal rotation
165
Q

What is a contraindication to LET (in addition to ACL recon)?

[Clin Sports Med 36 (2017) 135–153]

A

Posterolateral corner injury

  • In such cases a tenodesis may tether the tibia in a posterolateral subluxed position
166
Q

What are the relative indications for a LET (in adition to ACL reconstruction)?

[Clin Sports Med 36 (2017) 135–153]

A
  1. Revision ACL reconstruction
  2. Grade 2 or 3 pivot shift (high-grade rotational laxity)
  3. Young age of <25 years
  4. Generalized ligamentous laxity
  5. Genu recurvatum >10o
  6. Returning to pivoting sport (ie, soccer, basketball)
167
Q

Describe the LET procedure – Fowler Kennedy technique

[Clin Sports Med 36 (2017) 135–153]

A
  1. A 5-cm curvilinear incision is placed just posterior to the lateral femoral epicondyle
  2. An 8-cm-long x 1-cm-wide strip of ITB is harvested from the posterior half of the ITB
  3. It is left attached distally at the Gerdy tubercle, freed of any deep attachments to vastus lateralis, released proximally, and a #1 Vicryl whip stitch is placed in the free end of the graft
  4. The LCL is identified and small capsular incisions are made anterior and posterior to the proximal portion and Metzenbaum scissors are placed deep to the FCL to bluntly dissect out a tract for graft passage
  5. The ITB graft is then passed beneath the LCL from distal to proximal
  6. The lateral femoral supracondylar area is then cleared of soft tissue posterior and proximal to the lateral epicondyle
  7. The graft is then held taught but not overtensioned, with the knee at 60 degrees flexion and the foot in neutral rotation to avoid lateral compartment overconstraint
  8. The graft is secured using a small Richards staple and then folded back distally and sutured to itself using the #1 Vicryl whip stitch.
  9. The posterior aspect of the ITB, where the graft was harvested, to avoid overtightening the lateral patellofemoral joint
168
Q

What are causes of overconstraint in the LET procedure? [Knee Surgery, Sports Traumatology, Arthroscopy

A
  1. Fixation of the graft with the tibia in external rotation
  2. Over-tensioning the graft
169
Q

What are the results of the STABILITY study comparing ACL reconstruction alone to ACL reconstruction and
LET in young patients (14-25 years) at high risk of graft failure? [Am J Sports Med. 2020 Feb; 48(2):285-2971

A

The addition of LET to a single-bundle hamstring tendon autograft ACLR in young patients at high risk of failure results in a statistically significant, clinically relevant reduction in graft rupture and persistent rotatory laxity at 2 years after surgery

170
Q

What is the function of the PCL (knee)?

[Arch Bone Jt Surg. 2018; 6(1): 8-18.]

A

Primary restraint to posterior tibial translation at all flexion angles

171
Q

What are the bundles of the PCL?

[JAAOS 2016;24:277-289]

A
  1. Posteromedial
  2. Anterolateral – larger and stronger
172
Q

What structures are part of the PCL complex and may act as secondary restraints to posterior tibial translation?

[JAAOS 2016;24:277-289]

A
  1. Anterior meniscofemoral ligament (of Humphry)
    * From the posterior horn of the lateral meniscus and inserts on the femur anterior to the PM bundle
  2. Posterior meniscofemoral ligament (of Wrisberg)
    * From the posterior horn of the lateral meniscus and inserts on the femur posterior to the PM bundle
173
Q

What is the mechanism of PCL injuries?

[JAAOS 2016;24:277-289]

A

Posteriorly directed force on the proximal tibia with the knee flexed

  • “When the foot is dorsiflexed, force is transmitted through the patella and extensor mechanism. When the foot is plantarflexed, a posteriorly directed force is imparted to the proximal tibia.”
174
Q

Where is the most common location anatomically for the PCL to be injured in isolated PCL tears? [Skeleral Radiol. 2016 45(12): 1695-1703]

A

Midsubstance (69%) > proximal insertion (27%)

175
Q

What is the normal anterior tibial step-off and what is its significance?

[JAAOS 2016;24:277-289]

A
  1. The medial tibial plateau normally lies approximately 1cm anterior to the medial femoral condyle
  2. This position must be restored when performing a Lachman’s test to prevent false positive and when performing a posterior drawer to prevent a false negative
176
Q

What special tests should be performed when evaluating for PCL injuries?

[JAAOS 2016;24:277-289]

A
  1. Posterior drawer (most sensitive and specific)
    * Graded based on posterior translation of the tibial plateau relative to its position on the contralateral side or relative to the medial femoral condyle (MFC)
  • Grade I
    • 0-5mm posterior translation OR anterior relative to MFC
  • Grade II
    • 6-10mm posterior translation OR even relative to MFC
  • Grade III
    • >10mm posterior translation OR posterior relative to MFC
  1. Quadriceps active test
  • Patient lies supine with the hip flexed to 45° and the knee flexed to 90° and is instructed to slide his or her foot down the table
  • Positive = tibia shifts anterior
  1. Posterior sag
  • Examiner holds the patient’s lower extremity with the hip and knee each flexed to 90°
  • Positive = posterior sagging of the tibia or loss of prominence of the tibial tubercle.
  1. Dynamic posterior shift test
  • Leg is extended from start position of hip and knee flexed to 90°
  • Positive = palpable clunk from reduction of the tibiofemoral joint
177
Q

What associated injury needs to be ruled out in the evaluation of PCL injuries?

[JAAOS 2016;24:277-289]

A

Posterolateral corner injury

  • Dial test
    • With the patient prone an external rotation force is applied to the feet with the knees at 30° and 90°
    • Positive = >10° difference side to side
      • Only at 30° = PLC
      • Both at 30° and 90° = PLC and PCL

Grade 3 posterior drawer (>10mm) translation is associated with a PLC injury

178
Q

What additional radiographs can be obtained to evaluate for PCL injuries?

[JAAOS 2016;24:277-289]

A

Kneeling stress views

  • Measurement between a line parallel to the posterior cortex of the tibia and the posterior aspect of Blumensaat’s line is compared between knees
  • 0–7 mm of side-to-side difference in posterior displacement constitutes a partial PCL tear
  • 8–11 mm constitutes an isolated complete PCL tear
  • ≥12 mm of posterior translation constitutes a combined PCL and PLC or PMC knee injury
179
Q

What is the pattern of degenerative changes after a PCL injury?

[JAAOS 2016;24:277-289]

A

Patellofemoral and medial compartment OA

  • Medial compartment experiences more anterior and medial contact pressures
180
Q

What is the management of PCL injuries?

[JAAOS 2016;24:277-289]

A
  1. Nonoperative
  • Isolated grade I and II
  • Grade III with mild symptoms or low activity demands
  1. Operative
    * Acute or chronic grade III who fail nonoperative management
181
Q

What are the two main operative techniques for treating ACL injuries?

[JAAOS 2016;24:277-289] [Arch Bone Jt Surg. 2018; 6(1): 8-18.]

A
  1. Transtibial tunnel
  2. Tibial inlay
    * Involves creating a trough at the tibial attachment of PCL to match with the graft bone plug fixed with a cannulated screw
    * Traditional inlay technique requires an open posteromedial approach (Burks) between the semimembranosus tendon and the medial head of the gastrocnemius muscle
    * Potential benefits
    • Bony healing
    • Avoidance of graft abrasion at the so-called killer tibial turn
    • Large graft sizes
182
Q

Describe the Tibial inlay technique for PCL reconstruction?

[Curr Rev Musculoskelet Med. 2018 Jun; 11(2): 316–319]

A
  1. Patient is prepped and draped in the lateral decubitus position with affected leg up
  2. Outside-in femoral tunnel
  • Patient is positioned supine
  • Diagnostic arthroscopy performed and remnant PCL debrided and footprint identified
  • Medial skin incision is made medial to the patella and through the capsule to identify the guide placement
  • An outside-in arthroscopic guide is used to place the guide pin which is over-reamed (usually 10mm)
  • An 18-gauge metal wire loop is then placed through the femoral tunnel into the posterior aspect of the femoral notch for later graft passage
    3. PCL graft preparation
  • Achilles tendon allograft
    • Bone plug is shaped and predrilled and tapped for a 6.5mm cancellous screw
    • Tendinous portion is tubularized and sized (10mm)
  1. Open tibial inlay
  • Patient is positioned prone
  • Burks posteromedial approach is made between the medial head of gastrocs and semimembranosus
    • Vertical capsulotomy is made and the native PCL insertion is resected with osteotomes and shaped to accept the graft
  • The graft is fixed bone-to-bone with the 6.5mm screw
  • The tendinous portion is shuttled to the femoral tunnel with the 18-gauge wire
  • Wound is closed
    5. Graft tensioning
  • Patient is positioned supine
  • Graft is tensioned with the knee at 90 and interference screw is used for fixation
183
Q

What is the anatomy of the superficial MCL of the knee?

[J Bone Joint Surg Am. 2010;92:1266-80]

A

One femoral and two tibial attachments

  • Femoral attachment = 3.2mm proximal and 4.8mm posterior to the medial epicondyle
  • Proximal tibial attachment = 12.2mm distal to the tibial joint line
    • Primarily to soft tissue over the termination of the anterior arm of the semimembranosus tendon
  • Distal tibial attachment = 61.2mm distal to the tibial joint line
184
Q

What is the function of the superficial MCL?

[Instr Course Lect 2015;64:531–542]

A
  1. Primary restraint against valgus stress at 30o flexion
    * Secondary restraint to external rotation of the tibia
  2. Secondary restraing to tibial internal rotation (along with the posterior oblique ligament) at all flexion angles
185
Q

What is the anatomy of the deep MCL?

[J Bone Joint Surg Am. 2010;92:1266-80]

A

Thickening of the capsule with attachments to the medial meniscus

  • Divided into meniscotibial and meniscofemoral components
  • Meniscofemoral attachment = 12.6 mm distal and deep to the femoral attachment of the superficial MCL
  • Meniscotibial attachment = 3.2 mm distal to the medial joint line
    • Just distal to the tibial articular cartilage
186
Q

What is the anatomy of the posterior oblique ligament (POL)?

[J Bone Joint Surg Am. 2010;92:1266-80]

A
  1. Tibial origin
    * A reflection from the distal semimembranosus tendon insertion on the posteromedial tibia
  2. Femoral origin
    * Central arm of the posterior oblique ligament attaches on the femur 7.7 mm distal and 2.9 mm anterior to the gastrocnemius tubercle
187
Q

How are injuries to the medial knee ligaments classified?

[J Bone Joint Surg Am. 2010;92:1266-80]

A
  1. American Medical Association Standard Nomenclature of Athletic Injuries
    * Severity system
    • Grade I = Localized tenderness and no laxity
    • Grade II = Localized tenderness and partially torn medial collateral and posterior oblique fibers
      • Fibers are still opposed, and there may or may not be pathologic laxity
    • Grade III = Complete disruption and laxity with an applied valgus stress.
      * Laxity system (subjective gapping of the medial joint line)
    • Grade 1+ = 3 to 5 mm
    • Grade 2+ = 6 to 10 mm
    • Grade 3+ >10 mm
  2. Fetto and Marshall Classification [Iowa Orthop J. 2006; 26: 77–90.]
  • Grade I = no valgus laxity at both 0 and 30 degrees of flexion
  • Grade II = valgus laxity at 30 degrees of flexion but stable at 0 degrees of flexion
  • Grade III = valgus laxity at both 0 and 30 degrees of flexion
188
Q

What structures are thought to be intact and disrupted in the Fetto and Marshall Classification?

[J Bone Joint Surg Am. 2010;92:1266-80][Journal of Orthopaedics 14 (2017) 550–554]

A

Grade I

  • No valgus laxity at both 0 and 30o of flexion
  • Both superficial MCL and POL intact

Grade II

  • Valgus laxity at 30o of flexion but stable at 0o of flexion
  • Superficial MCL disrupted and POL intact

Grade III

  • Valgus laxity at both 0 and 30o of flexion
  • Both superficial MCL and POL disrupted
  • Often associated with an ACL injury
189
Q

Where anatomically is the superficial MCL typically injured?

[Sports Med Arthrosc Rev 2015;23:e1–e6)]

A

Femoral insertion > tibial insertion > midsubstance

190
Q

What are the radiographic features of a MCL injury?

A
  1. Stress radiographs [Am J Sports Med. 2010;38:330-8]
  • Isolated superficial MCL
    • Medial joint gapping of 1.7mm at 0° and 3.2mm at 20° of flexion
  • Superficial MCL, deep MCL and POL
    • Medial joint gapping 6.5mm at 0° and 9.8 mm at 20° of flexion
      1. Pellegrini-Stieda syndrome
  • Intraligamentous calcification in the region of the femoral attachment of the MCL caused by the chronic tear of the ligament
191
Q

What are the indications for an MRI with a clinically suspected MCL injury?

[Sports Med Arthrosc Rev 2015;23:e1–e6)]

A
  1. Grade 1 or 2 injuries with concern for associated injuries
  • Relative indications:
    • Effusion
    • Suspected cruciate injury
    • Patellar instability
    • Lateral joint line pain
  1. Grade 3 injuries
  • Assess for concomitant injuries
    • Eg. cruciate injury, stener-like lesion of the knee, etc
192
Q

What are the MRI features of a MCL injury?

[Sports Med Arthrosc Rev 2015;23:e1–e6)]

A

Grade I

  • Ligament swelling and edema
  • Partial tearing with all structures overall intact

Grade II

  • Tearing of some medial structures with others intact
    • Eg. superficial MCL is torn with deep MCL intact, or vice versa

Grade III

  • Complete disruption of the superficial and deep layers
  • Often with significant soft tissue fluid and edema from intra-articular fluid leak

Bone bruises [J Bone Joint Surg Am. 2010;92:1266-80]

  • Lateral tibial plateau and lateral femoral condyle

***Best assessed on the coronal images

193
Q

What is a stener-like lesion of the knee?

[AJR Am J Roentgenol. 2019 Aug 28:1-5.][Clin Orthop Relat Res (2010) 468:289–293]

A

Defined as a distal tear of the superficial MCL with proximal retraction and interposition of osseous or soft-tissue structures between the ligament and its tibial attachment

  • Prevents anatomic healing and with potential for secondary dynamic valgus instability
  • Most commonly stener-like lesions represent interposition of the pes anserinus (83%)
    • May also involve entrapment of the distal tibial portion of the superficial MCL in the tibiofemoral joint or in a reverse Segond fracture (17%)
194
Q

What is the significance of stener-like lesion of the knee?

[AJR Am J Roentgenol. 2019 Aug 28:1-5.]

A
  1. High association with multi-ligament knee injuries
    * Most common associated ligament injury being ACL
  2. Prevents anatomic reduction and healing resulting in valgus instability
195
Q

What is the management of a stener-like lesion of the knee?

[AJR Am J Roentgenol. 2019 Aug 28:1-5.]

A

Acute open reduction and repair

196
Q

What are the indications for nonoperative management of acute medial sided knee injuries?

[Sports Med Arthrosc Rev 2015;23:71–76]

A

Isolated Grade I, II and III MCL injuries

197
Q

What are the indications for operative management of acute medial sided knee injuries?

[Sports Med Arthrosc Rev 2015;23:71–76] [Iowa Orthop J. 2006; 26: 77–90.]

A
  1. Acute repair
  • Large bony avulsion
  • Stener-like lesion of the knee
    • Pes anserine entrapment
    • Intra-articular entrapment
  • MRI finding of complete tibial sided avulsion in athletes
  • Presence of AMRI
  • Presence of valgus instability in 0 degrees of flexion in an underlying valgus knee alignment
  • Associated tibial plateau fracture
    2. Delayed repair
  • Combined ACL or other ligament reconstruction if the EUA shows valgus laxity in 0 degrees of flexion
    3. Augmentation
  • Combined with any repair if local tissue is deficient
    4. Reconstruction
  • Symptomatic chronic valgus laxity/medial instability
    5. Distal femoral varus osteotomy
  • Chronic valgus laxity and valgus bony alignment
198
Q

What are options for acute repair of the superficial MCL and POL?

[Instr Course Lect 2015;64:531–542][Sports Med Arthrosc Rev 2015;23:71–76]

A
  1. Sutures alone
  2. Suture plus suture anchor
  3. Staple
  4. Screw and washer
199
Q

What technique can be used to augment the superficial MCL repair?

[Instr Course Lect 2015;64:531–542]

A

Semitendinosus autograft

  • Released proximally at the musculotendinous junction leaving the distal insertion intact
  • Distal tibial fixation 6 cm from the proximal joint line with two double-loaded suture anchors
  • Graft is then passed deep to the sartorius fascia
  • Femoral fixation in a closed socket tunnel 3.2 mm proximal and 4.8 mm posterior to the medial epicondyle and fixed with an interference screw
  • Proximal tibial attachment is then secured 12 mm distal from the proximal joint line using a double loaded suture anchor
200
Q

What are the key features of a LaPrade superficial MCL and POL reconstruction?

[J Bone Joint Surg Am. 2010;92:1266-80] [Instr Course Lect 2015;64:531–542]

A
  1. Single anteromedial incision
  2. Two separate grafts
  3. 4 reconstruction tunnels at anatomic femoral and tibial insertions of the superficial MCL and POL
  4. Proximal tibial insertion of the superficial MCL is recreated with suture anchors
  5. Superficial MCL is tensioned at 30o flexion
  6. POL is tensioned at 0o flexion
201
Q

What is the anatomy of the lateral collateral ligament of the knee?

[Instr Course Lect 2015;64:531–542]

A
  1. Femoral origin
    * 1.4 mm proximal and 3.1 mm posterior to the lateral epicondyle
  2. Fibula origin
    * 28.4 mm distal to the tip of the fibular styloid
202
Q

What is the function of the LCL (knee)?

[Instr Course Lect 2015;64:531–542]

A
  1. Primary varus stabilizer at 0° and 30° of flexion
  2. Secondary restraint against tibial internal and external rotation
203
Q

What percentage of sporting-related LCL injuries are isolated (no associated pathology)?

[JAAOS 2018 26(6):e120-e127]

204
Q

What physical examination special test is most relevant for assessing the LCL?

[JAAOS 2017;25:280-287]

A

Varus stress test

  • Isolated LCL injury
    • Laxity at 30° and stable at 0° of flexion
  • LCL injury combined with PLC +/- cruciates
    • Laxity at both 30° and 0° of flexion
205
Q

What radiographic findings may be associated with LCL injuries?

[Sports Med Arthrosc Rev 2015;23:10–16] [Instr Course Lect 2015;64:531–542]

A
  1. Arcuate fracture
    * Avulsion of the fibular head or portion of
  2. Varus stress radiograph
    * Isolated LCL injury = 2.7mm lateral gapping
    * LCL and grade III PLC injury = 4.0mm lateral gapping
206
Q

What is the MRI grading system for LCL injuries?

[JAAOS 2018 26(6):e120-e127]

A

Grade I

  • Subcutaneous fluid surrounding the midsubstance of the ligament at one or both insertions

Grade II

  • Partial tearing of ligament fibers at either the midsubstance or one of the insertions, with increased edema in the area

Grade III

  • Complete tearing of ligament fibers at either the midsubstance or one of the insertions
  • Associated with increased edema
207
Q

What are the indications for nonoperative management of LCL injuries?

[JAAOS 2018;26:e120-e127]

A

Grade I and II injury

208
Q

What are the indications for repair of a LCL injury?

[Instr Course Lect 2015;64:531–542]

A

Acute avulsion injury (within 3 weeks)

209
Q

What are the indications for reconstruction of LCL injury?

[Instr Course Lect 2015;64:531–542]

A
  1. Acute Grade III midsubstance tears or attenuation
  2. Chronic LCL injury with symptomatic instability
210
Q

What are the indications for valgus producing HTO in context of LCL tear?

[Instr Course Lect 2015;64:531–542]

A

Chronic symptomatic LCL injury with varus deformity

  • HTO should precede reconstruction
    • May eliminate need for reconstruction in some cases
211
Q

What are the 3 key anatomical structures that make up the posterolateral corner (PLC) of the knee?

[Rev Bras Ortop. 2015 50(4): 363–370]

A
  1. Lateral collateral ligament
  2. Popliteus tendon
  3. Popliteofibular ligament
212
Q

Other than the primary components of the PLC, what other structures stabilize the posterolateral corner of the knee?

[JAAOS 2008;16:506-518] [Sports Med Arthrosc Rev 2015;23:2–9)]

A
  1. Posterolateral capsule
  2. Lateral meniscus
  3. Iliotibial band
  4. Fabellofibular ligament
  5. Long and short heads of the biceps femoris
  6. Lateral head of gastrocnemius muscle
213
Q

Describe the anatomy of the LCL, popliteus tendon and the popliteofibular ligament?

[Sports Med Arthrosc Rev 2015;23:2–9)]

A
  1. Lateral collateral ligament
  • Proximal attachment
    • Proximal and posterior to the lateral epicondyle of the femur
  • Distal attachment
    • Lateral aspect of the fibular head
      • 8.2mm posterior to the anterior margin of the fibular head and 28.4mm distal to the fibular styloid tip
        2. Popliteus tendon
  • Proximal attachment
    • Anterior and distal to the lateral epicondyle
      • At the anterior 1/5 of the popliteal sulcus, just posterior to the lateral femoral condyle articular cartilage
      • Attachment is 18.5mm oblique from the LCL femoral attachment
  • Distal attachment
    • Proximal posteromedial aspect of the tibia
  • ***Note: The tendon passes from intra-articular through the popliteal hiatus to become extra-articular
    • Gives off 3 popliteomeniscal fascicles to anchor to the lateral meniscus
      1. Popliteofibular ligament
  • Proximal attachment
    • Musculotendinous junction of the popliteus
  • Distal attachment
    • Posteromedial downslope of the fibular styloid process
214
Q

What is the consequence of a missed PLC injury?

[Rev Bras Ortop. 2015 50(4): 363–370]

A
  1. Failure of cruciate reconstructions
  2. Recurrent instability
215
Q

What are the special tests for the PLC?

[JAAOS 2017;25:280-287]

A
  1. External rotation recurvatum test
  • Lower leg is picked up by the great toe
  • Relative hyperextension, tibial external rotation and knee varus compared to the contralateral side
  1. Posterolateral rotary drawer test
  • With the knee flexed to 90°, the hip flexed to 45°, and the foot fixed in slight external rotation (usually best at 15°), a posteriorly directed force is applied through the tibial tuberosity
    • The PCL is relaxed in this position, allowing rotary and translator laxity
  • With an isolated PLC injury
    • More rotatory instability with slight ER than with neutral rotation because the PCL provides more translational stability with neutral rotation
  • With an isolated PCL injury
    • More translatory instability than rotary instability will be present
  1. Dial test
  • Patient prone, external rotation applied to foot and thigh foot angle compared side to side at 30 and 90 of knee flexion (Positive = 10o difference)
    • Increased at 30° = isolated PLC
    • Increased at 30° and 90° = PLC and PCL
      1. Standing apprehension test
  • Patient stands with the knee slightly bent and internally rotates the torso away from the leg, producing an internal rotation of the femur on the tibia
  • Positive = Apprehension or instability
216
Q

What classifications have been proposed to grade PLC injuries?

[JAAOS 2008;16:506-518]

A
  1. Hughston classification
  • Severity graded on varus laxity
  • Grade I = 0 to 5mm
    • Sprains without tensile failure of any capsule-ligamentous structures
  • Grade II = 6-10mm
    • Partial injuries with minimal abnormal laxity
  • Grade III = >10mm
    • Complete disruptions with significant laxity
      1. Fanelli classification
  • Severity graded on external rotation and varus laxity
  • Type A
    • Isolated rotational injury to the PFL and popliteus tendon complex
  • Type B
    • Rotational injury with a mild varus component
    • Represents an injury to the PFL and popliteus tendon complex, as well as attenuation of the LCL
  • Type C
    • Posterolateral instability with significant rotational and varus component secondary to complete disruption of the PFL, popliteus tendon complex, LCL, lateral capsule, and cruciate ligament or ligaments
      1. Modified Hughston classification
  • Severity graded on ER and varus laxity
  • Grade I
    • Minimal instability
    • Either varus or rotational instability of 0 to 5mm or 0° to 5°
  • Grade II
    • Moderate instability (6 to 10mm or 6° to 10°)
  • Grade III
    • Significant instability (>10 mm or >10°)
217
Q

What are the indications for nonoperative management of PLC injuries?

[Rev Bras Ortop. 2015 50(4): 363–370]

A

Acute, isolated Grade I and II PLC injuries

218
Q

What are the indications for operative management of PLC injuries?

[Rev Bras Ortop. 2015 50(4): 363–370]

A
  1. Isolated grade III PLC injuries
  2. Combined PLC injuries
  3. Chronic symptomatic PLC injury failing nonoperative management
219
Q

What are the indications for repair (vs. reconstruction) of PLC injuries?

[Rev Bras Ortop. 2015 50(4): 363–370]

A

Acute (<3 weeks) LCL and popliteus tendon avulsions, without midsubstance injury

220
Q

In general, in a PLC injury what type of reconstruction should be performed and of what structures?

[Knee Surgery, Sports Traumatology, Arthroscopy (2019) 27:2520–2529]

A

Anatomical reconstruction

  • Reconstruction of primary PLC components
    • LCL, popliteus tendon and PFL
  • Repair of secondary structures (hybrid construct)

***Note: Valgus producing HTO should precede or occur concurrently with reconstruction in varus knees

221
Q

What are 3 described PLC reconstructions and their distinguishing features?

A
  1. Larson [Arthroscopy. 2002 Feb; 18(2 Suppl 1): 1-8]
    1. Main features
      1. Single femoral tunnel at the lateral epicondyle (isometric point) ii. Fibular tunnel directed from anterior to posterior ili. Graft limbs reconstruct the LCL and popliteofibular ligament
  2. Arciero [Arthroscopy: 2005 Sep;21(9): 1147]
    1. Main features
      1. 2 femoral tunnels near the anatomic footprint of the LCL and popliteus
      2. Fibular tunnel oriented anterolateral to posteromedial starting just distal to the LCL fibular insertion
      3. 2 separate graft limbs reconstruct the LCL and popliteus tendon in a more anatomic orientation
  3. Laprade /Arthrosc Tech. 2016 Jun; 5(3): e563-e572.]
    1. Main features
      1. 2 femoral tunnels at the anatomic footprint of the LCL and popliteus
      2. Tibial tunnel located from posterolateral proximal tibia (location of popliteus musculotendinous junction) to anterior tibia between tibial tubercle and Gerdy’s tubercle
      3. Fibular tunnel from LCL attachment to the posteromedial down slope of the fibular styloid
      4. Graft limbs reconstruct the LCL, popliteus and popliteofibular ligament
222
Q

what are the anatomical features of the native quadriceps tendon to consider when harvesting

A
  • length = 7.5-8cm (myotendinous junction to superior pole of patella)
    • patient heightis the strongest predictor of length
  • width = 2.5-3cm
    • widest point is 3cm proximal to the superior pole of the patella
  • thickness = 18 +/- 3mm for males, 16 +/- 2mm for females
223
Q

2 forms of quadriceps autograft that can be harvested

A
  • all soft tissue
  • quadriceps tendon with proximal patellar bone block
224
Q

characteristics of the quadriceps tendon graft that are advantageous compared to BTB graft

A
  • thicker
    • patellar tendon thickness is less than 6mm
  • greater intra-articular volume
    • 87.5% greater compared to BTB
  • more native quadriceps tendon remaining after harvest compared to patellar tendon
225
Q

advantages of the quadriceps autograft

A
  1. predictability of graft size and volume
    1. compared to BTB autograft, there is a greater intra-articular volume and greater residual tendon remaining
  2. no studies demonstrating tunnel-widening
  3. decreased anterior knee pain and kneeling (compared to BTB)
  4. less risk to the infrapatellar branch of the saphenous nerve (compared to BTB)
  5. more flexion strength and hanstring/quad isokinetic ratio compared to hamstring
  6. similar funcitonal outcomes compared to BTB and hanstring
    1. including stability, patient reported outcomes, ROM, strength and failure/rupture rates
226
Q

complications associated with quad tendon autograft

A
  • hematoma formation (Due to hypervascular region)
  • cosmetic defects
    • popeye like sign has been associated with too proximal harvest into rectus myofascia funciton
  • patellar fractures
    • recommended that harvest is less than 30% thickness of patella
227
Q

overall failure rate of primary ACL reconstruction

A
  1. <5%
    1. failure rate as high as 28% in young active males
228
Q

RF for ACL failure

A
  1. technical errors
  2. younger age
  3. higher activity level
  4. irradiated allograft
  5. lower limb malalignment
  6. increased posterior tibial slope
229
Q

most common cause of primary ACL reconstruction failure

A

technical error - improper tunnel placement outside the native femoral and tibial ACL footprints

230
Q

technical errors leading to ACL re-rupture

A
  1. non anatomic tunnel placement
  2. improper graft tensioning
  3. inadequate graft fixation
  4. insufficient graft material
231
Q

consequence of improper tunnel placement on the femoral and tibial side

A
  1. Errors in femoral tunnel placement

a. Too anterior = excessive graft tensioning with flexion leading to loss of knee flexion or stretching of the graft
b. Too posterior = excessive graft tension in extension with laxity in flexion
c. To vertical = inadequate rotational stability

  1. Errors in tibial tunnel placement

a. Too anterior = notch impingment and loss of extension
b. Too posterior = PCL impingement and loss of flexion
c. Too medial/lateral = notch impingement and iatrogenic tibial plateau cartilage damage

232
Q

indications for 2-stage ACL revision reconstruction

A

tunnel widening >15mm or >100% original diameter
substantial tunnel overlap
infection
arthrofibrosis

233
Q

indications for single-stage acl revision reconstruction

A
  1. appropriately placed tunnels
  2. good bone stock
  3. hardware that can be removed or will not obstruct revision
  4. inappropriately placed tunnels that do not interfere with anatomically placed tunnels
    - if previous femoral tunnes violate blumensaat’s line then an anatomically placed tunnel can be placed while avoiding the previous tunnel
234
Q

recommended interval of time between 1st and 2nd stage revision ACL when bone grafting is utilized

235
Q

concomitant pathology that can be addressed to minimize risk of ACL revision failure

A
  • increased posterior tibial slope
    • procedure tibial deflexion osteotomy (anterior closing wedge)
      • failed revision ACL reconstruction with posterior tibial slop >12°
  • varus malalignment
    • procedure - high tibial osteotomy
      • indications
        • medial compartment arthritis
        • varus thrust
        • instability in double and triple varus
  • anterolateral rotatory instability
    • procedure - anterolateral ligament reconstruction or lateral extra-articular tenodesis
      • indications
        • grade 3 pivot shifts
        • ACL reconstruction failure with no other identifiable cause
  • meniscal deficiency
    • procedure = meniscal repair
      • indications
        • RAMP lesions
        • repairable meniscal lesions
    • procedure = meniscal allograft transplantation
      • indications = irreparable meniscal tears or previous total or near-total meniscectomies
236
Q

What are the anterior, middle and posterior thirds of the medial side of the knee as described by Robinson?

[JAAOS 2017;25:752-761]

A

Anterior 1/3

  • Medial border of the patellar tendon to the anterior border of the superficial MCL

Middle 1/3

  • Width of the superficial MCL

Posterior 1/3

  • Posterior border of the superficial MCL to the medial border of the medial head of gastrocnemius
  • Posterior 1/3 = posteromedial corner
237
Q

What are the 5 components of the PMC?

[JAAOS 2017;25:752-761]

A
  1. Posterior oblique ligament
  2. Semimembranosus tendon and its expansions
  3. Oblique popliteal ligament
  4. Posteromedial joint capsule
  5. Posterior horn of the medial meniscus
238
Q

Describe the anatomy of the posterior oblique ligament (POL)?

[JAAOS 2017;25:752-761]

A
  1. Proximal attachment
    * Distal and posterior to the adductor tubercle
  2. Runs distally and posterior at 25° to the superficial MCL fibres
  3. Distally has 3 arms
  • Superficial
  • Central (tibial)
    • Largest and thickest
    • Reinforces the posteromedial capsule
    • Attaches to the posteromedial aspect of the medial meniscus and adjacent tibia
    • Main structure requiring repair or reconstruction
  • Capsular
239
Q

What are the 5 major arms or expansions of the semimembranosus tendon?

[JAAOS 2017;25:752-761]

A
  1. Pars reflecta (anterior arm)
    * Inserts into medial tibia
  2. Direct posteromedial tibial insertion (primary attachment)
    * Inserts into posteromedial tibia (tuberculum tendinis)
  3. OPL insertion
  4. POL insertion
  5. Popliteus aponeurosis expansion
240
Q

Describe the anatomy of the oblique popliteal ligament?

[JAAOS 2017;25:752-761]

A

Extends from the main semimembranosus tendon passing laterally and proximal to attach to the posterior capsule (meniscofemoral portion), fabella, and plantaris muscle

  • Difficult to distinguish from the posterior capsule
241
Q

What attachments are present on the posterior horn of the medial meniscus?

[JAAOS 2017;25:752-761]

A
  1. Posteromedial capsule (deep MCL)
  2. POL
  3. Semimembranosus expansion
242
Q

What is the most frequently injured structure in PMC injuries?

[JAAOS 2017;25:752-761]

243
Q

What are the 3 major patterns of PMC injury?

[JAAOS 2017;25:752-761]

A
  1. POL + semimembranosus tendon [capsular arm] (70%)
  2. POL + complete peripheral meniscus detachment (30%)
  3. POL + semimembranosus tendon + peripheral meniscus detachment (19%)

NOTE: isolated PMC injury is rare

244
Q

What are the findings on physical examination associated with PMC injury?

[JAAOS 2017;25:752-761]

A
  1. AMRI (anteromedial rotatory instability)
  • Hallmark of PMC injury
  • Characterized by anterior subluxation of the anteromedial tibia on the femoral condyle
  • Valgus stress at 30° flexion with foot externally rotated
    • Positive = medial joint gapping and anterior subluxation of the medial tibial plateau relative to the femur
      • Correlates with combined MCL and PMC
        2. Anterior drawer with foot 15° externally rotated
  • Positive = anteromedial tibial plateau subluxation
    • Indicates PMC injury
      1. Valgus stress at 0°
  • Positive = gapping of medial joint line
    • Suggests MCL, cruciate and PMC injury
  1. Posterior drawer with tibia in neutral and internal rotation
  • Positive = equal posterior translation
    • PCL and PMC injury
  • Decreased posterior translation with internal rotation suggests isolated PCL injury
245
Q

What are the indications for surgical repair or reconstruction of the PMC?

[JAAOS 2017;25:752-761]

A
  1. Multiligamentous injuries with AMRI
  2. Medial gapping or instability with valgus stress at full extension
  3. Positive posterior drawer with internal rotation
246
Q

When can PMC repair be considered?

[JAAOS 2017;25:752-761]

A

Acute injury

  • Particularly with distal tibial sided MCL and PMC tears
247
Q

When should PMC reconstruction be considered?

[JAAOS 2017;25:752-761]

A
  1. PMC tissue is not favourable (attenuated, midsubstance)
  2. Chronic, symptomatic PMC injuries
248
Q

What is the anatomic reconstruction of the PMC?

[JAAOS 2017;25:752-761]

A

2 grafts to reconstruct

  • Superficial MCL
    • Distal tunnel 6cm below joint line
    • Proximal tunnel just proximal and anterior to the medial epicondyle
    • Tensioned in 20° flexion and neutral rotation
    • Proximal tibial attachment is made with a suture anchor 12-13mm distal to joint line
  • POL
    • Distal tunnel just anterior to the direct arm attachment of the semimembranosus
    • Proximal tunnel 8mm distal and 3mm anterior to the gastrocnemius tubercle
    • Tensioned and fixed in full extension and neutral rotation
249
Q

What are the consequences of an unrecognized PMC injury?

[JAAOS 2017;25:752-761]

A
  1. Chronic valgus laxity
  2. Late graft failure after ACL reconstruction
250
Q

Where is the qudriceps tendon most commonly ruptured anatomically? [JBJS REVIEWS 2018;610):el]

A

Quadriceps tendon can be divided into 3 zones relative to the superior pole of the patella
a. Zone 1 (0 to 1cm)
b. Zone 2 (>1 to 2cm) - most common (relatively avascular)
c. Zone 3 (>2cm)

251
Q

What are the risk factors for quadriceps tendon tear? [JBJS REVIEWS 2018;6(10):el]

A
  1. Long standing tendinopathy
  2. Diabetes
  3. Thyroid disorders
  4. Renal disease
  5. Hyperlipidemia
  6. Systemic inflammatory disease
  7. Medications (statins, fluoroquinolones)
  8. Age (>40)
  9. Men
252
Q

When should quadriceps tendon repair be performed?

A

Within 2 weeks of injury

253
Q

What is considered the gold standard treatment of quadriceps tendon tears? [JBJS REVIEWS 2018;6(10):el]

A

Heavy non-absorbable suture in locking fashion grasping the quadriceps tendon, sutures are passed through drill holes in the patella and tied over bone bridge (transosseous tunnel fixation)

254
Q

What are reported advantages of suture anchor type constructs in the fixation of quadriceps tendon tears? [JB.JS Apr;23(4): 1039-45]
REVIEWS 20186(1) el][Anthroscopy. 2016 Jun;32(6):1117-24][Knee Surg Sports Traumatol Arthrosc. 2015

A

Smaller incision, less dissection, reduced operative time, decreased gap formation, increased load to failure

255
Q

What is the blood supply to the menisci?

[Am J Sports Med 1982; 10(2):90]

A
  1. Primary = medial and lateral geniculate arteries (both superior and inferior)
    * Gives rise to a perimeniscal capillary plexus in the synovial and capsular tissue supplying the peripheral meniscus (10-30%)
  2. Secondary - middle geniculate artery
  • Within the synovial sheath of the cruciate ligaments
  • Supplies the anterior and posterior horns for short distance
  1. Relative avascular area of the lateral meniscus at the posterolateral aspect adjacent to the popliteus tendon
  2. Three vascular zones [AAOS comprehensive review 2, 2014]
  • Red-red – peripheral 1/3 (vascular)
  • Red-white – middle 1/3 (avascular)
  • White-white – central 1/3 (avascular)
256
Q

what are clinical differences between transosseous suture and suture anchor quadriceps tendon repair? PLoS one 2018 Mar 19;13(3)

A

no differences in patient reported outcomes, strength, ROM or re-tear rate

257
Q

What is the innervation of the menisci?

[Clinical Anatomy 28:269–287 (2015)]

A
  1. Recurrent peroneal branch of the common peroneal nerve
  2. Mechanoreceptors and free nerve endings located in the anterior and posterior horns and peripheral 1/3-2/3
258
Q

What are the stabilizing ligaments of the medial and lateral meniscus? [Sports Med Arthrosc Rev 2017;25:219-226]

A
  • Transverse/intermeniscal ligament
  • Coronary ligament
  • Anterior meniscofemoral ligament (of Humphrey)
  • Posterior meniscofemoral ligament (of Wrisberg)
  • Deep MCL (meniscotibial and meniscofemoral ligaments)
  • (meniscal root attachment)
259
Q

What is a meniscal flounce and what is its significance? [Sports Med Arthrosc Rev 2017;25:219-226]

A
  1. Meniscal flounce = a buckle in the meniscus due to knee position
  2. Significance = normal finding, the absence of a flounce should prompt the surgeon to probe the meniscus for an occult tear
260
Q

What are 4 MRI features that would suggest a bucket-handle tear? [Knee Surg Sports Traumatol Arthrosc (2006) 14: 343-349]

A
  1. Double-PCL sign (sagittal)
  2. Fragment in the intercondylar notch (coronal)
  3. Double anterior horn sign/Double delta sign (sagittal)
  4. Absent bow-tie sign (sagittal)
261
Q

What is a recommended algorithm for the evaluation and management of meniscal tears? [Bone Joint J 2019;101-B: 652-659]

A
  • Based on history, physical and imaging there are five main meniscal tear scenarios:
    • Locked knee
      • Treatment = urgent arthroscopic surgery
    • Acute injury + repairable meniscus tear
      • Treatment = consider arthroscopic surgery if patient is a suitable candidate
        • No recommended timeline for repair - shared decision between surgeon and patient
    • Non-Acute injury + meniscus tear is treatable
      • Treatment = at least 3 months of non-operative treatment
    • Consider non-urgent arthroscopic surgery if symptoms persist >3 months
    • Non-Acute injury + meniscus tear is possibly treatable
      • Treatment = at least 6 months of nonoperative treatment
        • Consider non-urgent arthroscopic surgery in selected cases if symptoms persist >6 months
    • Advanced arthritis + meniscus tear
      • Treatment = nonoperative
        • Arthroscopic surgery is not appropriate
262
Q

What is the function of the meniscus?

[Clinical Anatomy 28:269–287 (2015)]

A
  1. Load transmission
    * Converts compressive forces into concentric forces
    • Hoop stresses transmitted to bony anchors of meniscal horns by circumferential fibres of meniscus
    • Diminishes forces on articular cartilage
      * ​40-60% of the load in extension and up to 90% in flexion
  2. Shock absorption
  3. Stability (limits motion in all directions)
  4. Proprioception
  5. Joint lubrication and nutrition (?theoretical)
263
Q

What are the indications for meniscal repair?

[J Am Acad Orthop Surg 2013;21:204-213]

A
  1. Tear > 1cm but <4cm in length
  2. Red-red zone tears (that tears within 2 mm of the meniscal vascular rim)
  3. Vertical/longitudinal tears
  4. Age <40 years
  5. Acute tears (<6 weeks)
  6. Concurrent ACL reconstruction
  7. No mechanical malalignment
  8. Radial tears that extend entire width of meniscus
  9. Meniscal root tears
264
Q

What are the 3 main options for meniscal repair?

[J Am Acad Orthop Surg 2013;21:204-213]

A
  1. Inside-out (gold standard)
  2. Outside-in
  3. All inside
265
Q

For the inside out technique of meniscal repair describe the medial and lateral approach?

[Arthrosc Tech. 2017 Aug; 6(4): e1221–e1227.]

A
  1. Medial approach
  • 4cm vertical incision posterior to MCL
    • 1/3 above joint and 2/3 below joint
  • Sartorial fascia is opened inline
  • Pes anserinus tendons are taken distal
  • Interval between the medial head of gastrocs and capsule is developed staying proximal to semimembranosus tendon
    • Spoon is inserted in this interval
      1. Lateral approach
  • 4cm oblique incision starting at Gerdy’s extending proximal and anterior
  • ITB is split in line with its fibres
  • Staying posterior to LCL and anterior to the biceps femoris the interval between the lateral head of gastrocs and the capsule is developed
    • Spoon is inserted in to this interval
266
Q

What is the function of the meniscus root?

[JBJS REVIEWS 2016;4(8):e2] [AJSM 2014; 42(12): 3016]

A

Dissipate axial loads by conversion to hoop stresses

  • Axial load causes circumferential fibres to stretch and meniscus to extrude
  • Tensile ‘hoop stress’ resists this extrusion in the presence of intact meniscal roots
  • Distribution of hoop stresses by the circumferential fibers helps to transmit relatively even axial loads across the joint surfaces
267
Q

What is the consequence of meniscal root tears?

[JBJS REVIEWS 2016;4(8):e2]

A
  1. Meniscal extrusion
  2. Loss of hoop stress
  3. Increased tibiofemoral contact pressures
    * Equivalent to total meniscectomy
  4. Accelerated degenerative changes
268
Q

What are the typical clinical symptoms following a meniscal root tear?

[JBJS REVIEWS 2016;4(8):e2] [AJSM 2014; 42(12): 3016]

A
  1. Joint line tenderness
  2. Pain in deep knee flexion
  3. Positive McMurray test
  4. Effusion
  5. Locking
  6. Giving way
269
Q

What are the MRI signs indicative of a meniscal root tear?

[JBJS REVIEWS 2016;4(8):e2]

A
  1. Radial tear of the meniscal root on axial imaging
  2. Truncation sign
    * Vertical linear defect in the meniscal root on coronal imaging
  3. Ghost sign
    * Increased signal in the meniscal root on sagittal sequences
  4. Meniscal extrusion >3 mm outside the peripheral margin of the joint on coronal imaging
  5. Ipsilateral tibiofemoral compartment bone marrow edema and insufficiency fractures are commonly noted in the presence of posterior meniscal tears
270
Q

What meniscal root has the highest incidence of tears?

[JBJS REVIEWS 2016;4(8):e2]

A

Posterior root of the medial meniscus

  • Least mobile of all roots
271
Q

What is the classification of meniscal root tears?

[Arch Bone Jt Surg. 2018; 6(4): 250-259.]

A

LaPrade classification

  • Type I
    • Partial root tear
  • Type II
    • Complete radial root tear
  • Type III
    • Complete root tear + bucket handle tear
  • Type IV
    • Oblique tear into root attachment
  • Type V
    • Root avulsion fracture
272
Q

What are the indications for nonoperative and operative management of meniscal root tears?

[JBJS REVIEWS 2016;4(8):e2]

A

Nonoperative

  • Sedentary lifestyle
  • Extensive medical comorbidities
  • Advanced signs of osteoarthritis
    • Outerbridge grade 3 or 4
  • Joint-space narrowing
  • Marked varus malalignment (>5°)
  • Chronic, degenerative, irreparable tears

Operative

  • Healthy individuals
  • Minimal or no degenerative changes
    • Outerbridge grade 1 or 2
  • Minimal to no joint-space narrowing
  • Normal mechanical alignment
  • Intact cruciate ligaments
273
Q

What are the indications for meniscal repair vs. partial meniscectomy in context of a root tear?

[JBJS REVIEWS 2016;4(8):e2]

A
  1. Partial meniscectomy
  • Chronic tears with advanced degenerative changes
    • Outerbridge grade 3 or 4
  1. Meniscal root repair
  • Acute tears
    • Goal of preventing arthritis
  • Chronic tears with no or little articular cartilage wear
    • Outerbridge grade 1 or 2
    • Goal of symptomatic relief
274
Q

What is the goal of operative treatment for a meniscal root tear?

[JBJS REVIEWS 2016;4(8):e2]

A

Restore an anatomical attachment of the meniscal root to bone that is capable of converting axial weight-bearing loads into hoop stresses

  • Thereby improving symptoms and function and ultimately preventing or delaying onset of degenerative changes
275
Q

What are the two main meniscal root repair techniques?

[JBJS REVIEWS 2016;4(8):e2]

A
  1. Transtibial pullout repair
  • Anatomical position of the meniscal root is identified, debrided, and prepared for repair
  • An ACL tibial drill guide is utilized to drill a guide pin or a retro-cutting reamer into position through a small incision at the anteromedial aspect of the proximal aspect of the tibia
  • # 2 nonabsorbable sutures are passed through the substance of the torn meniscal root with a suture-passage device
  • Suture limbs are shuttled through the transtibial tunnel, tensioned, and fixed with use of whatever fixation construct the surgeon prefers with the knee in 30° of flexion
    • Typically a cortical button or screw and washer
      1. Suture anchor repair
  • Utilizes accessory posteromedial and posterolateral portals
  • Under arthroscopic visualization, the anatomical position of the meniscal root is identified, debrided, and prepared for repair
  • A double-loaded suture anchor is inserted at the meniscal root site and suture passage is performed with use of a suture lasso through the accessory portal or with use of a rotator cuff-type suture-passage device through the anteromedial or anterolateral portal
  • Once passed, the suture limbs are tied arthroscopically with the knee in 30° of flexion, with the arthroscopic knots being kept posterior, away from the articular surfaces of the affected compartment
276
Q

What are the complications associated with meniscal root repair techniques?

[AJSM 2014; 42(12): 3016]

A
  1. Transosseous fixation
  • Bone tunnels may interfere with concomitant ligament reconstruction
  • Risk of suture abrasion within bony tunnel
  • Risk of creep in the suture
    • Decreases strength of repair
      1. Suture anchor repair
  • Anchor pullout
  • Technically difficult
  1. Other
  • Retear and progression of arthritis
  • Infection
  • Arthrofibrosis
  • DVT
  • Iatrogenic injury to cruciates
  • Iatrogenic injury to posterior neurovascular structures
277
Q

What features differ between a discoid meniscus and a normal meniscus?

A
  1. Thicker
  2. Less peripheral vascularity
  3. Cover a larger surface area of the tibial plateau (may cover entire plateau - “true disc”)
  4. Decreased collagen fibers in a more disorganized circumferential arrangement
  5. Intrameniscal mucoid degeneration
278
Q

What is the most common classification of discoid menisci? [JAAOS 2017;25:736-743]

A
  1. Watanabe
  2. Type I - stable, complete discoid meniscus (most common)
  3. Type II - stable, incomplete discoid meniscus (covers ≤80% of plateau)
  4. Type III - unstable Wrisberg variant (lacks posterior meniscotibial attachment; only has the meniscofemoral ligament of Wrisberg)
279
Q

What are the classic symptoms of a torn or unstable discoid meniscus?

A
  1. Knee pain, popping, snapping, and a lack of terminal extension
280
Q

what are the radiographic features of discoid meniscus? [JAAOS 2017;25:736-743] [Knee Surg Relat Res 2016;28(4):255-262]

A
  1. Squaring of the lateral femoral condyle
  2. Cupping of the lateral tibial plateau
  3. Widening of the lateral joint space up to 11 mm
  4. Hypoplastic lateral tibial spine
281
Q

What are the MRI features of discoid meniscus? [Orthop Clin N Am 46 (2015) 533-540] [JAAOS 2017;25:736-143]

A
  1. Transverse meniscal diameter > 15 mm between the free margin and the periphery of the body on a coronal view
  2. Continuity between the anterior and posterior horns of the menisci (i.., bow tie sign) noted on at leas three consecutive 5-mm-thick sagittal slices
  3. Enlarged “bow-tie” appearance on a single sagittal slice
  4. Ratio of the minimal meniscal width to maximal tibial width (on the coronal MRI slice) of more than 20%
282
Q

What are the indications for nonoperative and operative management of discoid menisci? (JA AOS 2017.25.736-743)

A
  1. Nonoperativea. Asymptomaticb. Discoid menisci identified incidentally at arthroscopy should not be treated
  2. Operative
    1. Symptomatic
283
Q

what is the surgical technique for managing a discoid menisci

A
  1. arthroscopic saucerization with rim preservation +/- stabilization
    1. preserve meniscus with intact 6-8mm peripheral rim of meniscal tissue is recommended
    2. stabilization is added for watanabe type III
284
Q

What are the most common locations for OCD lesions in the knee?

[Sports Med Arthrosc Rev 2016;24:44–49]

A
  1. 70% - posterolateral aspect of the medial femoral condyle
  2. 15% - inferior central aspect of the lateral femoral condyle
  3. 5-10% - inferior medial aspect of the patella
  4. <1% - trochlea
285
Q

what is the incidence of bilateral OCD of the knee?

286
Q

What other pathology is associated with an OCD lesion of the lateral femoral condyle?

A

Discoid lateral meniscus

287
Q

What are the two broad categories of OCD?

[Curr Rev Musculoskelet Med (2015) 8:467–475]

A
  1. Juvenile (open physis)
  2. Adult (closed physis)
288
Q

What is Wilson’s sign (Knee OCD)?

[Curr Rev Musculoskelet Med (2015) 8:467–475]

A

The knee is extended from 90-30 while internally rotating the tibia

  • Positive = reproduction of symptoms with internal rotation and relief with external rotation
289
Q

What are MRI features of an unstable OCD lesion?

[Curr Rev Musculoskelet Med (2015) 8:467–475]

A
  1. High-signal intensity behind the lesion
  2. Cystic area beneath the lesion
  3. High-signal intensity through the articular cartilage
  4. Focal articular defect
290
Q

what are radiographic, mri and arthroscopic classifications of OCD of the knee

A

radiograhpic - berndt and harty
- stage 1 - small area, compression of subchondral bone
- stage 2 - partially detached OCD fragment
- stage 3 - fully detached OCD fragment, still in underlying crater
- stage 4 - compete detachment/loose body

mri = di paola
type I - thickening of articular cartilage but no break
type II - breached articular cartilage, low signal rim behind fragment indicating attachment
type III - breached articular cartilage, with high signal T2 changes behind fragment suggesting fluid around lesions
type IV - loose body and defect of articular surface

arthroscopic - rock studh group
6 arthroscopic categories
3 immobiles types
1. cue ball - no abnormality
2. shadow - cartilage is intact and suptly demarcated
3. wrinkle in the rug - cartilage is demarcated with a fissure, buckle and/or wrinkle

3 mobile types
1. locked door = cartilage fissuring at periphery unable to hinge open
2. trapdoor = cartilage fissuring at periphery able to hinge open
3. crater = exposed subchondral bone defect

291
Q

What factors are predictive of poor prognosis in OCD lesions in the knee?

[Sports Med Arthrosc Rev 2016;24:44–49]

A
  1. High signal line between the OCD lesion and underlying bone on T2-MRI (unstable)
  2. Skeletal maturity (older age)
  3. Larger lesion
  4. Patellar and lateral femoral condyle OCD lesions
  5. Cyst ≥1.3mm behind the lesion
  6. abnormal surface cartilage
  7. viability of the fragment
    1. can be determine on MRI post-gad T1 image with fat saturation - low signal compared epiphyseal bone suggests poor viability
292
Q

What is the classification of OCD lesion of the knee?

[Orthobullets]

A

Clanton Classification

  • Type I - depressed osteochondral fracture
  • Type II - fragment attached by osseous bridge
  • Type III - detached non-displaced fragment
  • Type IV - displaced fragment
293
Q

What are the indications for nonoperative management of an OCD lesion in the knee?

[Curr Rev Musculoskelet Med (2015) 8:467–475]

A

Stable lesion

294
Q

What is the protocol for nonoperative management?

[Bone Joint J 2016;98-B:723–9]

A

***No consensus

  1. TTWB 4 weeks
  2. Activity restriction (no running and sport) for 8 weeks
  3. MRI at 12 weeks
    * If no improvement in symptoms = consider surgery
    * If symptoms resolved and MRI improved = progress activity with full return after 4 weeks
    * If symptoms improved but MRI is unchanged = continue activity restriction and repeat MRI in 12 months
    • If no improvement = surgery
295
Q

What are the indications for operative management of an OCD lesion in the knee?

[Curr Rev Musculoskelet Med (2015) 8:467–475]

A
  1. Unstable lesion
  2. Displaced lesion
  3. Failure of nonoperative management
    * 6 months for juvenile, 3-6 months for adult
  4. Symptomatic loose bodies
296
Q

What are the surgical options for OCD of the knee?

[Bone Joint J 2016;98-B:723–9] [Curr Rev Musculoskelet Med (2015) 8:467–475]

A
  1. Stable symptomatic lesion
  • Retrograde or transarticular drilling
    • Equivalent effectiveness
  1. Unstable, nondisplaced
    * Internal fixation with bioabsorbable or metal pins, nails or screws
  2. Unstable, nondisplaced with cyst
    * bone grafting and internal fixation
  3. Unstable, displaced
  • internal fixation of fragment
    • Fragment trimming due to hypertrophy
  1. Unsalvageable fragment
    * Excision = small fragments (<2cm)
    * Microfracture
    * ACI/MACI
    * Osteochondral autograft or allograft
  2. Consider osteotomy or patellar realignment to offload the lesion
297
Q

What are the indications and contraindications for articular cartilage repair and restoration procedures?

[JAAOS 2017;25:321-329]

A

Indications

  • Outerbridge or International Cartilage Repair Society grade III or IV focal chondral or osteochondral defects

Contraindications

  • Inflammatory arthritis
  • Lower grade lesions
  • Inability to comply with postoperative protocol
298
Q

What are the general surgical recommendations for articular cartilage lesions based on size of defect?

[Sports Health 2015; 8(2):153]

A

<2cm2 – microfracture OR OATs

  • OATS better in higher demand patient

2-4cm2 - OATs or ACI

>4cm2 – ACI or osteochondral allograft (better in defects with bone loss or deformity)

299
Q

What is the technique for microfracture of articular cartilage lesions?

[JBJS 2010;92:994-1009] [JAAOS 2017;25:321-329]

A
  1. Create a contained lesion with stable shoulders
  • Remove the calcified cartilage layer at the base of the lesion
  • Prepare the channels by perforating the subchondral bone with a 45° awl approx. 3-4mm in depth
    • Fat droplets from the marrow should be visualized, blood should be visualized with water off
    • Space the channels 3-4mm apart
  • Allows for clot formation and migration of marrow-derived mesenchymal stem cells promoting fibrocartilage repair
  1. Postoperative
  • nonWB for 6 weeks with crutches
  • CPM from 0-60° for 6 weeks, 6-8 hours per day
  • Full ROM and closed chain exercises at 2 months
  • Full return to activity at 3 months
300
Q

What are the advantages and disadvantages of microfracture for articular cartilage lesions?

[JBJS 2010;92:994-1009]

A

Advantages

  • Technically straightforward
  • Low cost
  • Minimal morbidity

Disadvantages

  • Fibrocartilage rather than hyaline cartilage
    • Results deteriorate over time
301
Q

What is the technique for autologous osteochondral transplantation (aka. Mosaicplasty, osteoarticular transfer system – OATS) for articular cartilage lesions?

[JAAOS 2017;25:321-329]

A

Multiple small autogenous osteochondral plugs are harvested from less weightbearing areas and transferred to fill a defect

  • Plugs are approx. 10mm in depth, impacted in donor site to match the height of the adjacent cartilage
  • Periphery of femoral condyles and intercondylar notch are donor sites
302
Q

What are the advantages and disadvantages of autologous osteochondral transplantation (OATS) for articular cartilage lesions?

[JBJS 2010;92:994-1009] [JAAOS 2017;25:321-329]

A

Advantages

  • Hyaline cartilage transferred
  • Less postoperative restrictions
  • Single procedure

Disadvantages

  • Limited availability of graft
  • Donor site morbidity
  • Different thickness and mechanical properties of cartilage from donor to recipient site
  • Graft subsidence
  • Dead space between grafts
  • May require arthrotomy
303
Q

What is the technique for osteochondral allograft transplantation for articular cartilage lesions?

[JBJS 2010;92:994-1009]

A

Cadaver graft consisting of intact, viable articular cartilage and subchondral bone is transferred in the defect

304
Q

What are the advantages and disadvantages of osteochondral allograft transplantation?

[JBJS 2010;92:994-1009]

A

Advantages

  • No donor site morbidity
  • Can fill large defects
  • Hyaline cartilage transferred
  • Single stage
  • Can achieve precise surface architecture
  • Eliminates dead space

Disadvantages

  • Limited graft availability
  • High cost
  • Risk of immunological reaction and disease transmission
  • Possible incomplete graft incorporation
  • Technically demanding machining and sizing the allograft
305
Q

What are the indications for osteochondral allograft of the knee?

A
  • Young active patient
  • Posttraumatic osteochondral defects
  • Osteonecrosis
  • Osteochondritis dissecans
  • Large (≥2 cm2) focal defects
  • Previous cartilage repair failure
  • Patellofemoral joint cartilage lesion
306
Q

What are the contraindications for osteochondral allograft of the knee?

A
  • Relative
    • BMI >35
    • Concomitant ligament or meniscal injury
    • Uncorrectable malalignment of the knee joint
    • Inflammatory arthritis
    • Smoking or corticosteroid use
  • Absolute
    • Advanced osteoarthritis (bipolar lesions)
    • Poor surgical candidate
    • Chronic posttraumatic defect
307
Q

what imaging is required for preop planning of osteochondral allograft of the knee?

A
  • radiographs
    • routine WB with sizing markers
    • full length alignment films
  • MRI
    • assess for concomitant pathology
    • assess lesion size and location
308
Q

What is the technique for autologous chondrocyte implantation (ACI)?

[JBJS 2010;92:994-1009] [JAAOS 2017;25:321-329]

A

Two stage procedure

  • First for tissue harvest
    • 100-300mg of articular cartilage from nonWB surface
    • Sent for culture and expansion of donor chondrocytes
  • Second for cell implantation into the prepared recipient site
    • 3-8 weeks after the first stage
    • Cells are injected beneath a collagen membrane or periosteal patch sutured over the defect
309
Q

What are the advantages and disadvantages of ACI?

[JBJS 2010;92:994-1009] [Sports Health 2015; 8(2):153]

A

Advantages

  • Hyaline cartilage produced

Disadvantages

  • Two stage procedure
  • Technically demanding
  • High cost
  • Requires arthrotomy
310
Q

What is difference between ACI and matrix-associated chondrocyte implantation (MACI)?

[JBJS 2010;92:994-1009]

A

Chondrocytes are incorporated into a collagen membrane eliminating the need for periosteal harvest and makes for a more even cell distribution on the membrane

311
Q

What are the standard and accessory portals for knee arthroscopy?

A
  1. Standard
  • Anterolateral
  • Anteromedial
  1. Accessory [JBJS 2006; 88(4):110]
  • Posteromedial
    • Location
      • Soft spot between MCL, medial head of gastrocs and semitendinosus tendon
    • Technique
      • Perform with knee at 90
      • Knee joint distended
      • Direct visualization with transcondylar notch view
      • Soft spot is localized with a spinal needle
      • Superficial incision
      • Blunt dissection with hemostat and joint capsule is penetrated
    • Risk
      • Saphenous nerve (sartorial branch)
  • Posterolateral
    • Location
      • Soft spot between LCL, lateral head of gastrocs, posterolateral tibial plateau and biceps femoris
    • Technique
      • Perform with knee at 90
      • Knee joint distended
      • Direct visualization with transcondylar notch view
      • Soft spot is localized with a spinal needle
      • Superficial incision
      • Blunt dissection with hemostat and joint capsule is penetrated
    • Risk
      • Common peroneal nerve
  • Posterior transeptal portal
    • Location
      • Involves resection of the posterior septum from the posterolateral portal to enter the posteromedial compartment under visualization from the posteromedial portal
        • Posterior septum is a triangle-shaped two-layer synovial reflection that divides the posterior aspect of the knee into posteromedial and posterolateral compartments
        • Bounded by the posterior cruciate ligament anteriorly, the posterior portion of the femoral intercondylar notch superiorly, and the posterior aspect of the capsule posteriorly
    • Risk
      • Popliteal artery (inferiorly) and middle geniculate artery (superiorly)
312
Q

What are ways to control intraoperative bleeding during arthroscopy?

[CORR course]

A
  1. Increased pump pressure
  2. Reduce outflow suction
  3. Asd epinephrine to fluid
  4. Controlled hypotension (anaesthesia)
  5. Tourniquet
  6. Drive arthroscope to bleeding source
  7. Cautery
  8. TXA
313
Q

What are the avulsion fractures found at the knee?