spine Flashcards

1
Q

Describe a hangman’s fracture

A

AKA traumatic spondylolysis of C2.
An unstable fracture caused by extreme hyperextension. Bilateral #s through pedicles of C2. Although unstable, SCI can be minimal b/c spinal canal has greatest diameter at this level.

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2
Q

Describe extension teardrop #

A

Stable in flexion. Unstable in extension
fracture in lower cervical vertebrae. Caused by extreme hyperextension with causes anterior longitudinal ligament to avulse anteriorinferior corner of body. Buckling of ligamentum flaavum into the canal can cause central cord lesion

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3
Q

What spinal cord injury is associated with a burst fracture

A

anterior cord syndrome

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4
Q

What differentiates a wedge fracture from a burst fracture

A

A burst fracture will have >40% compression of anterior vertebral body on imaging and has a characteristic vertical fracture on anterior films

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5
Q

Describe Jefferson fracture

A

Extremely unstable fracture through ring of C1. Lateral masses >7mm offset from pillars of C2. Associated with prevertebral hemorrhage and widened prevertebral space

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6
Q

When is wedge fractures (usually considered stable) potentially unstable?

A
  • Severe wedging – Loss of >50% height

- Multiple adjacent wedge fractures

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7
Q

Classify odontoid fractures.

A
  • I: Odontoid process above transverse ligament
  • II: Base of odontoin process
    III: Through body of C2
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8
Q

List mechanisms of primary cord damage.

A
  • Transection
  • Compression (hypertrophied or arthritically enlarged bony or ligamnetous elements)
  • Primary vascular damage
    o Compression by hematoma
    o Ischemia due to compression of vertebral arteries or artery of Adamkiewitcz
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9
Q

List pre-existing chronic conditions which predispose to cervical injury.

A

Down syndrome – atlanto-ocipital dislocation
(normal is 15y.o.

AAOD= anterior atlanto-odontoid distance

Rheumatoid arthritis – C2 transverse ligament rupture
Ankylosing spondylitis – Spine becomes like one long bone

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10
Q

Describe the spinal motor exam for upper and lower extremities: (Table 43-2).

A
C4	Spontaneous breathing
C5	Shrugging of shoulders
C6	Elbow flexion
C7	Elbow extension
C8-T1	Finger flexion
T1-T12	Intercostal and abdomen muscles (complete localization by sensory exam)
L1-L2	Hip flexion
L3	Knee extension + Hip adduction
L4	Knee extension + Hip adduction
L5	Ankle dorsiflexion + Hip abduction
S1-S2	Ankle plantar flexion
S2-S4	Rectal sphincter tone
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11
Q

Describe the spinal reflexes and their corresponding nerve roots (Table 43-3)

A

C6 Biceps
C7 Triceps
L4 Patellar
S1 Achilles

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12
Q

What is spinal shock? What is its significance?

A
  • Concussive injury to the spinal cord that causes total neurologic dysfunction distal to the site of injury
  • Mimics complete cord injury
  • Flaccid paralysis, areflexia
  • Prognosis cannot be accurately determined until spinal shock has ended (usually less then 24 hours, occasionally longer)
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13
Q

In a patient with flacid paralysis after a spinal cord injury, what is the significance of the bubocavernosis reflex?

A
  • Intact bulbocavernosus reflex (contraction of the rectal spincter with squeezing the glands penis or clitorus or pulling on the foley catheter) indicates that spinal shock is not or is no longer present
    o Absent – spinal shock likely – cannot determine prognosis (differentiate complete from partial injury)
    Bulbocavernosus Present – spinal shock not contributing – poor prognosis for recovery
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14
Q

What are the three common incomplete cord lesions?

A
  • Central cord
  • Brown-Sequard
  • Anterior cord
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15
Q

Describe the mechanism and clinical picture of each of the incomplete cord syndromes.

A

Central cord: weakness UE greater than LE

Brown-Sequard lesion: cord hemisection with ipsilateral loss of motor, position, vibration and contralateral loss of pain and temperature
Anterior cord: paralysis and hypoalgesia with preservation of posterior column (position/vibration)

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16
Q

List the clinical characteristics of acute cauda equine syndrome.

A
  • Unilateral or bilateral leg pain
  • Bowel or bladder dysfunction
  • Perianal anesthesia
  • Diminished rectal tone
  • Lower extremity weakness
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17
Q

What causes “dejeune onion skin” pattern of analgesia?

A
  • Damage to the spinal trigeminal tract (of the trigeminal nerve)
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18
Q

List the 5 nexus criteria for identifying patient at “extremely low probability of spinal injury.”

A
  1. No Posterior midline tenderness
  2. No focal neuro DEFICIT
  3. Not DECREASED LOC
  4. No Intoxication (DRUNK)
  5. No painful, DISTRACTING injuries
    Validation:
    - n >34000
    - SN 99.6%, SP 12.9%, NPV 99.8%
    - Predicted high radiography rates at 87%
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19
Q

List the components of the Canadian C-spine Rule for identifying “clinically important” cervical spine injuries?

A
Inclusion
Ø Alert, stable trauma patients
Exclusion:
Ø Non-trauma
Ø GCS65
	b. Fall from >1m
	c. Axial loading
	d. High speed MVC (>100km/hr)
	e. Rollover
	f. Ejection
	g. Motorized recreational vehicle
	h. Bicycle
	i. parasthesia
2. Any Low risk present that could safely assess ROM?
	a. Simple rear end
	b. Sitting in ED
	c. Ambulatory at any time
	d. Delayed onset neck pain
	e. No midline tenderness
3. Can patient rotate head 45 degrees to left and right?
If low risk features and no high risk features, can ROM neck and clear C/S
Validation:
- n = 8924
- SN 99.4, SP 45.1, NPV 100%
- Predicted radiography rates at 58%
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20
Q

How is the posterior cervical line (Swischuk) used in children to differentiate pseudosubluxation from injury?

A

If the base of the spinous process of C2 lies >2mm anterior or posterior to the posterior cervical line (bases of spinous process of C1-C3), an injury should be suspected

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21
Q

How wide is a wide predental space?

A
  • Adults 3mm

Children 5mm

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22
Q

What are the normal sizes of the cervical soft tissue spaces?

A

Space Adults Children

23
Q

Differentiate “Spinal shock” from “neurogenic hypotenion.”

A
  • Spinal shock
    o Loss of neurologic function and automonic tone below the level of a spinal cord lesion due to a contusion of the cord
    o Clinically have flaccid paralysis, loss of sensation, DTR and urinary bladder incontinence
    o May also have bradycardia, hypotension, hypothermia, intestinal ileus
    o Generally lasts 24 hours
  • Neurogenic hypotenion (shock)
    o Loss of vasomotor tone and lack of reflex tachycardia due to interruption of the autonomic ganglia
    o Injuries >T6
24
Q

What is the DDX for back pain in children?

A
· Spondylolithesis
	· Scoliosis
	· Scheuermann’s disease
	· Sickle cell crisis
	· ID like diskitis
	· Neoplasm
	· UTI
25
Q

What are 4 can’t miss diagnosis in low back pain according to the Agency for Health Care Policy and Research?

A
  • Spinal fracture
    • Cauda equine syndrome
    • Spinal infection
      Malignancy
26
Q

What are characteristics of psuedoclaudication of spinal stenosis?

A
  • Lower extremity radiculopathy which occurs with walking and is relieved by rest and leaning forward
    Usually resolves over 10 minutes (vascular claudication tends to last only 5 minutes after resting)
27
Q

What is ankylosing spondylitis?

A

HLA B27 associated autoimmune arthritis of the spine and pelvis

28
Q

List causes of cauda equine syndrome.

A
  • Large central disc herniation à most common
    • Spinal epidural abscess
    • Hematoma
    • Trauma
      Malignancy
29
Q

Which patients are at risk for a spinal infection?

A
  • IVDU
    • Alcoholics
    • Immunocompromised patients
      o HIV, DM, Chronic renal failure, Long term corticosteroids
    • Elderly
    • Recent blunt trauma to back
    • Indwelling catheter
30
Q

Which patients are at risk for a spinal infection?

A
  • IVDU
    • Alcoholics
    • Immunocompromised patients
      o HIV, DM, Chronic renal failure, Long term corticosteroids
    • Elderly
    • Recent blunt trauma to back
    • Indwelling catheter
31
Q

What conditions predispose to spinal epidural abscess?

A
- Underlying disease: 
	o DM
	o Alcoholism
	o HIV
- Spinal abnormality or intervention
	o Degenerative joint disease
	o Trauma
	o Surgery
	o Drug injection
	o Placement of stimulator or catheters
- Potential local or systemic source of infection
	o Skin and soft tissue infections
	o Osteomyelitis
	o UTIs
	o Sepsis
	o Indwelling vascular access
	o Intravenous drug use
	o Nerve acupuncture
	o Tattooing
	o Epidural analgesia
	o Nerve block
32
Q

What is the established staging system for spinal epidural abscess?

A
  • Stage 1: Back pain at the level of the affected spine
  • Stage 2: Nerve-root pain radiating from the involved spinal area
  • Stage 3: Motor weakness and sensory deficit and bowel and bladder dysfunction
    Stage 4: Paralysis
33
Q

What are three most common symptoms of spinal epidural abscess (the clinical triad that is only manifest in a minorioty of patients)?

A
  • Back pain (3/4 of patients)
  • Fever (1/2)
    Neurologic deficits (1/3)
34
Q

What is the imaging modality of choice for spinal epidural abscess?

A
  • MRI: 90% sensitive (Less invasive then CT myelography)
35
Q

Pending culture results, what empiric coverage should be started?

A
  • Staphylococci (Vancomycin for MRSA)

- Gram negatives (4th generation cephalopsporin: Ceftazidime or Cefipime)

36
Q

List x-rays findings of spondylitis?

A
  • Erosion of contiguous vertebral endplates
  • Narrowing of the disc space
    Vertebral bony erosions
37
Q

Who are candidates for surgery for spinal stenosis.

A
  • Progressive neurologic deficits
    • Progressive loss of ability to walk due to claudication
    • Evidence of cauda equine
      Intractable pain
38
Q

List cancers that tend to go to spine.

A
  • Lung
  • Breast
  • Prostate
  • Thyroid
  • Kidney
  • Multiple myeloma
  • Lymphoma
    Sarcoma
39
Q

How much needs be eroded before metastasis will become apparent of plain film?

A
  • 50%
40
Q

What level of elevation of ESR suggest CA as a cause of back pain?

A

Greater than 100

41
Q

What is the blood supply to spinal cord

A

anterior 2/3: single anterior spinal artery
posterior 1/2 paired posterior spinal arteries
All get segmental contributions from radicular arteries largest of which is the artery of adamkiewitz

42
Q

What are the 3 most important spinal tracts

A

Lateral corticospinal tranct
Posterior columns
Lateral spinothalamic tracts

43
Q

Describe a Brown-sequard lesion

A

cord hemisection
Manifests with ipsilateral motor, vibration and proprioception loss
Contralateral pain/temperature/light touch

44
Q

Describe a central cord syndrome

A

Upper GT Lower extremity weakness and paraesthesias

Decreased pain and temperature sensation

45
Q

Anterior cord syndrome

A

Manifests with bilatearl paresis and loss of pain/temperature sensation

Preserved vibration, proprioception and light touch

Often from disc protrusion or low flow state

46
Q

Complete cord syndrome

A

total loss of sensory, motor , autonomic innervation distal to lesion

47
Q

Contrast cauda equina and conus medullaris

A

Cauda equina more gradual with more severe radicular pain less back pain and hyporeflexia +/- assymetry

Conus medullaris more gradual with less radicular pain but more back pain and hyperreflexia/faasiculations

48
Q

List 5 conditions that cause back pain and fever:

A
  • Vertebral Osteomyelitis
    • Diskitis
    • Transverse myelitis
    • Epidural abscess
    • Neoplasms (B-Sx)
49
Q

List 6 non-traumatic causes of acute spinal cord dysfunction:

A

Affecting cord or blood supply directly:

  • MS
  • Transverse myelitis
  • Poliomyelitis
  • Spinal AVM
  • Spinal SAH
  • Syringomyelia
  • HIV myelopathy
  • Other myelopathies
  • Cord infarction
Compressive lesions:
- Spinal Hematoma
- Spinal epidural abscess
- Diskitis
Neoplasm (Met or primary)
50
Q

What is Transverse Myelitis:

A
  • Acute / subacute spinal cord inflammation with dysfiunction
    • Etiologies: Following viral illness, infection, autoimmune, idiopathic
    • Rapidly progressing Sx, peak at 24 hrs à paraplegia, transverse sensory impairment, sphincter disturbance; may have ­reflexes, ¯tone, clonus
    • Management: Steroids controversial. Must rule out mass lesion amenable to removal.
      Prognosis range from complete recovery in 3 – 6 months to death from progressive neurologic dysfunction (1/3, 1/3, 1/3)
51
Q

What is the source of bleeding in spinal SAH, and how do they present?

A
  • AVM most common
    • Others: Tumors, cavernous angiomas, spontaneous with anticoagulation
    • Present with sudden onset of excruciating back pain at level of bleed +/- radicular distribution +/- H/A and nuchal rigidity
    • Need urgent MRI and NSx
52
Q

What is the classic presentation of a syringomyelia?

A
  • Syrinx is centrally located
    • Loss of pain and temperature in cape like distribution over shoulder and arms, sparing of proprioception and touch
    • Neck pain & H/A followed by sensory impairment, gait issues, lower CN dysfunction
53
Q

What is diskitis?

A

Primary infection of nucleus pulposus;

- S/s – ++ local pain exacerbated by spinal movement, radicular Sx 50-90%, fever in 90%; usually no neurol deficits

54
Q

What cancers metastasize to the spinal cord?

A
  • Breast, lung, lymphoma

- Hematogenous, direct extension