Spinal Cord Injury Flashcards

1
Q

Most Common causes for Spinal Cord Injury

A

Motor vehicle accidents
Falls
Violence (predominantly gun shot wounds)
Sports related injurie

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2
Q

Risk factors:

A

Younger age
Male gender (78% of population with spinal cord injury)
Alcohol/drug use

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3
Q

Spinal Cord Injury Pathophysiology

A

Transient concussion: client fully recovers

Contusion, laceration, compression of spinal cord tissue (alone or in combination)

Complete transection (severing of the spinal cord)

Vertebrae most commonly affect: C5, C6, & C7, T12, and L1
These vertebrae are more susceptible to injury due to the greater range of mobility

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4
Q

Types of Injuries:

A

Transient concussion

Contusion (bruising)

Laceration (deep cut)

Compression (pressure placed on cord)

Complete transection (severing of cord)

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5
Q

Spinal Cord Injury: Primary and Secondary

A

Primary Injury:
Result of initial trauma or injury, usually permanent
Secondary Injury: Result of edema and hemorrhage
Major concern for critical care nurses
Early treatment needed to prevent long-term damage or permanent damage

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6
Q

Clinical Manifestations- Depends on the type and level of the injury

A

Complete Spinal Cord leisons: Loss of both sensory and voluntary motor communication from brain to periphery
Results in paraplegia or tetraplegia
Incomplete spinal cord leison:
Ability of the spinal cord to relay messages to/from the brain is NOT completely absent
Sensory and/or motor fibers are preserved below the injury
Effects of Spinal Cord Injuries:
Central Cord Syndrome
Anterior Cord Syndrome
Lateral Cord Syndrome (Brown-Sequard Syndrome

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7
Q

Central Cord Syndrome

A

Characteristics:
motor deficits
Sensory loss more pronounced in the upper extremities
bowel/bladder dysfunction is variable
Or can be completely preserved
Cause:
Injury or edema to the central cord (typically cervical area)…hyperextension

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8
Q

Anterior Cord Syndrome

A

Characteristics:
Loss of pain, temperature, and motor function below the level of the lesion
Light touch, position, and vibration sensation remain intact
Cause:
Acute disc herniation or hyperflexion injuries with fracture/dislocation of vertebra.
Injury to the spinal artery

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9
Q

Lateral Cord Syndrome: Brown Sequard Syndrome

A

Characteristics:
Ipsilateral paralysis or paresis with ipsilateral loss of touch, pressure and vibration
Contralateral loss of pain and temperature
Cause:
Transverse hemisection of the cord (half of the cord is transected from north to south)
Knife or missile injury (GSW, shrapnel)
Fracture/dislocation of unilateral articular process
Acute ruptured disc

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10
Q

Assessments and Diagnostics

A

Full neuro exam with frequent neuro checks
Full head to toe exam
Diagnostics:
Lateral cervical spine x-rays
CT Scan
MRI (if further injury suspected)
If MRI is contraindicated, a myelogram may be used
Continuous ECG monitoring
Bradycardia and asystole are common in acute spinal cord injuries

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11
Q

Emergency Management

A

ALL the following clients must be ruled out for spinal cord injury:
MVC
Diving or contact sports injury
Fall
Direct trauma to head/neck

ANY OF THESE INJURIES … YOU MUST ASSUME THERE IS A SCI UNTIL IT IS RULED OUT

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12
Q

Emergency Management Cont:

A

At the Scene:
Immobilization of head/neck must occur
Focus upon maintaining head/neck in neutral position
to prevent flexion, rotation, or extension

Use hands, back board, or cervical immobilizing device-including head blocks working together

Client must be lifted in one movement with all team members

Trauma Center:

Referral to a regional spinal injury or trauma center
Many changes occur within the first 24 hours
It is important to have multiple disciplines working with patient

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13
Q

Medical Management

A

Respiratory therapy:
O2 to maintain paO2
Intubation may be required
In high cervical spine injuries, phrenic nerve is damaged (which stimulates the diaphragm)
Intramuscular diaphragmatic pacing is currently in clinical trial phase

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14
Q

Medical Management 2:

A

Skeletal fracture reduction and traction:
Traction (weights are applied)
Halo device (ring fixed to skull by 4 pins)

Surgical management indicated:
Compression is evident
fragmented/unstable vertebral body
wound that penetrates cord
bony fragments in spinal canal
neuro status is deteriorating

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15
Q

Non-Surgical Treatments

A

Steroid infusion
Decrease inflammation and swelling
High dose within 24-48hrs
Controversial / only slight benefits
Stabilization with cervical (neck) traction/alignment (see next slide):
Braces
Halo traction
Gardner-Wells Tongs traction

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16
Q

Surgical Treatments

A

Decompression
Releasing pressure on the spinal cord
Causes: bone, disc, blood clot, tumor

Internal fixation and instrumentation
Placing metal rods, screw, and/or hooks to prevent further injury

Bone grafting for fusion

17
Q

Spinal Shock

A

Sudden decrease of reflex activity below level of injury with:
No sensation, paralyzed, flaccid, and reflexes are absent
BP and HR may be decreased
Prolonged hypotension and bradycardia can worsen damage to the spinal cord; therefore, important to keep MAP > 85 mmHg
Bladder and bowel function affected
Paralytic ileus most often occurs within first 2-3 days after SCI and resolves within 3-7 days

18
Q

Neurogenic Shock

A

Loss of ANS function below level of lesion
Hypotension, bradycardia, decreased CO, pooling in extremities and peripheral vasodilation
Patient will not perspire in paralyzed portions of the body…watch for s/s fever
Respiratory problems: secretion retention, increased paCO2, decreased paO2, respiratory failure, and pulmonary edema

19
Q

Other Spinal Cord Complications:

A

Venous thromboembolism / PE
Respiratory failure / pneumonia
Autonomic dysreflexia
Pressure injuries
Infections
Urinary
Local infection at the pin sites

20
Q

Nursing Interventions

A

Promoting adequate breathing and airway clearance
Preventing injury
Maintaining skin integrity
Maintaining urinary elimination
Improving bowel function
Providing comfort measures
DVT/PE anticoagulant therapy/SCDs
Recognizing Autonomic Dysreflexia

21
Q

Autonomic Dysreflexia

A

Occurs as a result of exaggerated autonomic responses that have an injury above T6

Occurs after spinal shock is resolved

Triggers:
Distention of bladder/bowels
Stimulation of the skin
Pressure ulcers
infection
Signs/symptoms:
Severe, pounding headache
Profuse diaphoresis above spinal lesion
Nausea
Nasal congestion
Bradycardia
Extreme HTN

22
Q

Long Term Care for Paraplegia/Tretraplegia Patients

A

Increasing mobility:
Exercise programs
Mobilization
Preventing disuse syndrome:
ROM, repositioning, proper body alignment
Promoting skin integrity:
Frequent skin assessments and repositioning to decrease risk of pressure ulcers
Improving bladder management:
Encourage 2.5 liters of intake/day
Establishing bowel control:
Bowel training program

23
Q

Long term Care Continued

A

Counseling on sexual expression:
Assistance with erectile dysfunction

Enhancing coping mechanisms:
Managing grief and depression

Decreased life expectancy
Risk for:
Autonomic dysreflexia
Bladder/kidney infections
Orthostatic hypotension
VTE
Spasticity
Mental health issues
Pressure ulcers…leading to fistulas, osteomyelitis, and sepsis