Special Populations Flashcards

1
Q

3 causes of hypernasality

A
  • velopharyngeal insufficiency (anatomic; deficit of structures; i.e. cleft lip-palate)​
  • velopharyngeal incompetence (physiological; motor deficit; i.e. CP, TBI)
  • velopharyngeal mislearning (no anatomical or functional deficit, but error in learning)
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2
Q

which of hyponasality and hypernasality is SLP more involved in? which is more common?

A
  • hypernasality more common.

- little role for SLP in hyponasality (others do interventions to remove the obstruction)

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3
Q

4 types of clefts

A
  • labial (unilateral (L or R) or bilateral; complete or incomplete)
  • palatal (complete of incomplete)
  • labiopalatal
  • occult submucous
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4
Q

3 clinical signs of submucous cleft palate

A
  • bifid uvula
  • zona pellucida (blue transparent zone)
  • palpable V
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5
Q

3 important structures and role in velopharyngeal closure

A

velum
- moves towards posterior pharyngeal wall

lateral pharyngeal walls
- move towards midline

posterior phayngeal wall
- anterior movement toward velum

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6
Q

role of velopharyngeal mecanism is speech sound production

A
  • nasality
    (open for nasal vowels/consonants)
  • oral pressure
    (close to allow adequate oral pressure buildup for consonants and vowels)
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7
Q

distinction between pneumatic and non-pneumatic closure of velopharyngeal mecanism

A

pneumatic is for speech
- smaller closure to allow rapid movements; increased demands on system

non-pneumatic closure is for swallowing, vomiting, nausea reflex
- for some clefts, no difficulties in non-pneumatic closure but difficulties with pneumatic

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8
Q

effects of clefts on suction

A

typically, tongue pressing on nipple against alveolar ridge/hard palate generates positive pressure in nipple that causes liquid to come out
- palatal cleft renders pressure for suction difficult or impossible to generate

+
nasal regurgitation

(cleft lip, especially unilateral, has little effect)

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9
Q

4 types of articulatory errors with clefts

A
  • developmental errors
    (part of typical development)
  • phonological errors
    (same as SSD errors)
  • obligatory errors
    (consequence of anatomical or physiological deficit; cannot be corrected with therapy)
  • compensatory errors
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10
Q

4 types of obligatory errors with clefts

A
  • nasal emissions
  • nasal turbulence
  • weak oral consonants
  • distortions caused by fistula or malocclusion
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11
Q

5 types of compensatory errors (that should be addressed in therapy) with clefts

A
glottal stop (VPI)​
- substituting plosive

nasal air emission (“snort”) (VPI)​
- co-occurs with pressure sensitive sounds*?

pharyngeal fricative (VPI + fistula)

  • approximation of tongue back and pharynx
  • substituting fricative

pharyngeal plosive (VPI + fistula)

  • contact of tongue back and pharynx
  • substituting plosive (often velars)

platal mid-dorsal plosive (fistula)
- contact of tongue back with palate behind fistula

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12
Q

assessments tasks particular to clefts

3

A

“cul-de-sac” test

  • for hypernasality, production of /a/ or phrase with no nasal phonemes with and without pinching nostrils
  • for hyponasality, production of syllables with nasal phonemes with and without pinching nostrils

mirror test
- check for nasal emissions with mirror condensation

nasometer

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13
Q

with cleft, hypernasality should be consistent across oral sounds. why might nasal emission not be consistent?
(3)

A

variations of velopharyngeal closure related to certain sounds
- indicates not velopharyngeal dysfunction, but “phoneme specific nasal emissions”.

variations of velopharyngeal closure related to timing of closure
- i.e. motor dysfunction; like apraxia/CAS

variations of velopharyngeal closure related to strength of articulatory contacts
- touch closure in isolated sounds w/ effort; but difficulties in spontaneous context and at end of sentences

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14
Q

when is a fistula more symptomatic on resonance?

A
  • smaller fistula = less symptomatic
  • more posterior fistula = less symptomatic
    (most symptomatic fistulas are in alveolar region)
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15
Q

cerebral palsy definition and criteria

A

non-progressive disorder of movement/posture related to neurological condition caused by lesion to motor systems during prenatal, perinatal or before 2-3-5 years of age, can be congenital or acquired.
- differing profiles (cognition, language, sensory, motor) according to extent and location of lesions

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16
Q

2 major categories of intervention with intellectual deficiency

A
  • behavioural intervention
    (ABA, etc.)
  • intervention with neurotypical peers