Somatosensation Part 3 Flashcards

1
Q

How will a patient present if they have damage to the primary somatosensory cortex?

A
  • Present with contralateral sensory deficits
  • May involve face, arm, trunk, or leg
  • All sensory modalities will be involved
  • Primary modalities may be spared, but patient will have cortical sensory loss (decreased extinction, decreased stereognosis, decreased grapesthesia)
  • Same presentation as damage to thalamus VPM/VPL or thalamic somatosensory radiations
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2
Q

What impairments may accompany damage to the primary somatosensory cortex?

A
  • UMN lesions
  • Changes in vision
  • Communication impairments
  • Cognitive impairments
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3
Q

How will a patient present if the have damage to thalamus VPM/VPL or thalamic somatosensory radiations?

A
  • Present with contralateral sensory loss
  • May involve face, arm, trunk, leg
  • All sensory modalities are involved
  • Same presentation as damage to primary somatosensory cortex
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4
Q

What impairments may accompany damage to the thalamus VPM/VPL or thalamic somatosensory radiations?

A
  • Hemiparesis (if damage to internal capsule too)
  • Hemianopia (if damage to optic radiations too)
  • Severe contralateral pain
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5
Q

How will a patient present if they have damage to the brainstem?

A
  • Sensory loss on contralateral body and ipsilateral face
  • May involve anterolateral pathway or only medial lemniscus pathway depending on location of the lesion
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6
Q

What impairments may accompany sensory loss to the brainstem?

A
  • Motor impairments to the contralateral side of the body
  • Cranial nerve damage on the ipsilateral side
  • Cerebellar signs
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7
Q

Describe the presentation of sensory loss in terms of distal symmetrical polyneuropathies

A
  • Bilateral sensory loss
  • All modalities effected
  • Stocking a glove distribution (distal portion of nerve effected first)
  • Many possible causes, often caused by systemic issue (DM, drugs, kidney failure)
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8
Q

Describe the presentation of sensory loss in terms of peripheral nerve or nerve root damage

A
  • Sensory loss to an isolated region
  • May be accompanied by LMN signs (atrophy, decreased reflexes, fasciculations)
  • Pain, tingling, and numbness in the region
  • Damage to nerve roots may be accompanied by radicular pain
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9
Q

List the types of spinal cord syndromes

A

Transverse lesion
Posterior cord syndrome
Anterior cord syndrome
Hemi-cord lesion (Brown-Sequard syndrome)
Central cord syndrome

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10
Q

Describe a transverse cord lesion

A
  • All sensory and motor pathways will be interrupted
  • Loss of all sensory and motor function below lesion
  • Common causes are trauma, tumors, and transverse myelitis
  • LMN signs at level of lesion due to damage at ventral horn, UMN signs at all levels below
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11
Q

What is spinal shock?

A

Acute UMN lesions may present as decreased tone and reflexes for a period of time, spasticity and hyperreflexia will emerge several weeks later

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12
Q

Describe posterior cord syndrome

A
  • Lesion to bilateral posterior columns leading to loss of vibration and proprioception below the level of the lesion
  • Common causes are posterior tumors, trauma, MS, and posterior spinal arteries
  • A larger lesion will also present with lateral corticospinal damage
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13
Q

Describe anterior cord syndrome

A
  • Bilateral damage to anterolateral pathways (pain and temp loss below lesion) and lateral corticospinal tracts (UMN weakness below lesion)
  • Common causes trauma with severe flexion of cord, anterior spinal artery infarct, cervical disc protrusion, and MS
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14
Q

Describe a hemi-cord lesion (brown-sequard syndrome)

A
  • Loss of motor function and sense of vibration and proprioception on the ipsilateral side, loss of pain and temperature sensation of the contralateral side (and ipsi side 2 levels below lesion due to lissaure’s tract)
  • Common causes are penetrating injuries and lateral compression tumors
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15
Q

Describe central cord syndrome in terms of a small lesion

A
  • Damage to spinothalamic fibers crossing the anterior commissure
  • Damage to medial tracts leading to loss of pain and temperature to UEs (cape distribution)
  • Common causes are hyperextension injury (spinal cord contusion), syringomyelia, intrinsic tumors, and narrowing of spinal cord (edema)
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16
Q

Describe central cord syndrome in terms of a large lesion

A
  • Bilateral damage to lateral corticospinal tracts, some of the DCML pathways, and anterolateral pathways
  • sacral sparing (tracts travel laterally)
  • Effects UE more than LE
  • Effects motor function more than sensory function
  • Common causes are hyperextension injuries, spinal cord contusion, syringomyelia, and intrinsic tumors
17
Q

What is conus medullaris syndrome?

A

A lesion at L1 leading to UMN and LMN signs

18
Q

What is the clinical presentation of a cauda equina injury?

A
  • Bowel and bladder issues
  • Saddle anesthesia
  • LE paralysis
19
Q

What are some issues with regeneration of cauda equina injuries?

A
  • Long distance between lesion and innervation site
  • Axonal regeneration may not occur along original distribution
  • Glial-collagen scarring block regeneration
  • Organ or muscle may no longer function
  • Rate of regeneration slows and stops after one year