Smoke Inhalation Flashcards

1
Q

Define inhalation injury

A

Nonspecific term respiratory tract or tissue damage from heat, smoke, or chemical irritants

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2
Q

Define burn injury

A

Traumatic injury to skin or other tissue mostly caused by thermal or other acute exposures

Occurs when skin or other tissue is destroyed

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3
Q

What causes burn injury

A

Heat

Electrical damage

Friction

Chemicals

Radiation

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4
Q

What factors influence the severity of smoke inhalation

A

Duration

Chemical

Size and diameter

Temp of gas

Age

Pre-existing health

Size and depth of burns

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5
Q

What are the two types of smoke

A

Pyrolysis

Combustion

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6
Q

What are the two types of common toxins found in smoke

A

Carbon monoxide

Hydrogen cyanide

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7
Q

Hazards of skin burns

A

Destroys the barrier from pathogens

Barrier contains water

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8
Q

What is the “rule of 9s”

A

Provides a quick estimate of the total estimate of body surface burned

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9
Q

According to the nines rule, the: head is how much

A

Nine per side

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10
Q

According to the nines rule, the: arm is how much

A

9 per arm

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11
Q

According to the nines rule, the: body is how much

A

18 front and back

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12
Q

According to the nines rule, the: genitals is how much

A

1

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13
Q

According to the nines rule, the: legs is how much

A

18 each

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14
Q

Explain the correlation between age, body surface burns and mortality

A

The older you are, less burns have higher mortality

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15
Q

Explain the characteristics of a superficial burn

A

First degree

Epidermal

No blisters

Bleach with pressure

No pain in 2-3 days

Heals in 6 with no scarring

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16
Q

What are the characteristics of partial thickness burns

A

Epidermis and some dermis

Superficial or deep classification

Second degree

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17
Q

What are the characteristics of superficial partial thickness burns

A

Blisters in 24hrs

first looks like only epidermis; dermis appears damaged after 12-24 hrs

Heals in 7-21 days

Usually doesn’t scare, but pigment change

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18
Q

What are the characteristics of deep partial thickness burns

A

Reaches deeper dermis

Damage to follicles and glandular tissue

Pain only to pressure

Cheesy white to red

Hypertrophic scarring

If it fails to heal in 2 wks it is cosmetically and functionally a full-thickness burn

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19
Q

What are the characteristics of full thickness burns

A

Through dermis to subcutaneous

Dry inelastic doesn’t blanch to pressure

Burn eschar remains intact, but once separated unhealed granulation tissue remains

May not heal on there own

If it reaches muscle or bone can be life-threatening

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20
Q

How is thermal injury caused

A

Upper AW usually absorbs heat, so it absorbs excessive heat

21
Q

What does thermal injury cause

A

Pharyngeal edema and swelling

Stridor

Hoarseness

Altered voice

Painful swallow

22
Q

Does thermal injury usually occur below larynx

A

Not unless it is steam inhalation

23
Q

What protects the lower respiratory tract from thermal injury

A

Upper AW which can cool the air/ absorb (Heat sink)

24
Q

What are the stages of smoke inhalation

A

Early

Intermediate

Late

25
Q

What happens during the early stage of smoke inhalation

A

Tracheobrachial tree is inflamed

Bronchospasm

Secretions move into AW

Smoke slows down mucosal cilliary transport

26
Q

Cardiogenic hazards associated with smoke inhalation (early stage)

A

Hypovolemia

Hypoxia

Increased systemic and pul resistance

CO poisoning

27
Q

What happens to vessels with smoke inhalation injury during early stage

A

Mediators increase permeability

Leak into third space

Much fluid but no in circulation

28
Q

When does generalization of edema peak with smoke inhalation injury

A

8-24 hours

29
Q

What is the timeline of intermediate burn inhalation injury

30
Q

What happens during intermediate smoke inhalation injury

A

Tracheobronchial tree becomes necrotic and sloughs

ARDS may develop

More mucus

31
Q

What occurs during late smoke inhalation injury

A

Infection of body surface

PNA

Pulmonary Embolism

Restrictive or obstructive disorder

32
Q

What problem accompany CO

A

SpO2 misleading

33
Q

How does cyanide alter the body

A

In the mitochondria it binds to cytochrome oxidase enzyme so it’s unable to metabolize O2

34
Q

What are signs of smoke inhalation

A

Soot

Oral or laryngeal edema

Cyanosis

Cough with sputum

35
Q

Is smoke inhalation usually restrictive or obstructive

A

Usually restrictive, but can be obstructive with airway construction

36
Q

What percentage indicates COHB intoxication

A

Greater than 20 percent

37
Q

What are the key initial assessment for patients with burn or smoke injury

A

ABC
Respiratory status
Cardiovascular status
Extent of burns
Depth of burns

38
Q

What questions do you ask patients suspected on smoke inhalation

A

Closed or open space

What type of material was burning

Was CO or cyanide produced

Was the patient conscious on arrival

Does history indicate test for blood alcohol levels, for poisoning, or O.D.

39
Q

What is a ideal ETT for a patient with smoke inhalation

A

8.0-8.5 for bronchoscopy

40
Q

Benefits of covering burn wounds

A

Prevent:

shock

Fluid loss

Heat loss

Pain

41
Q

How do we prevent infection for burn victims

A

Reverse isolation

Positive pressure room

Air filtration

Wound covering

42
Q

How do you treat CO poisoning

A

High FiO2

Hyperbaric oxygen

43
Q

What medication do you treat cyanide poisoning with

A

Hydoxocobalamin IV

(Urine looks grape)
Can’t do ABG

44
Q

What aerosols are given to burn patients

A

5k-10k heparin in 3mL Q4

Alternating with

20% NAC Q4
(For seven days)

45
Q

How does NAC and heparin aid in burn care

A

Reduces the formation of fibrin casts in the AW

Serve as scavengers for reactive O2 species and may reduce injury

46
Q

How do you treat secretions for patients with smoke inhalation injury

A

Sx
IPV
Bronchoscopy if needed

47
Q

Combustion gas expectation

A

Explodes with oxygen

48
Q

Pyrolysis

A

Smolders in low oxygen

49
Q

Why with CO poisoning is there a decrease in bicarbonate and pH with a low CO2

A

Inability to use O2 causes anaerobic metabolism and creating lactic acid