Small Rum 3 Flashcards

1
Q

What is involved with planning for footrot eradication program

A
- Labour
○ DYO or Contractors
- Fencing
○ Eliminate source!!!!!!
- Infrastructure
○ Footbaths
○ Laneways
○ Inspection equipment
- Sheep
○ Replacement sheep
○ Stock numbers (hold back cfa rather than sell)?
- Budget
○ Cost benefit
○ Cash flow
○ Peak debt
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2
Q

In terms of control for eradication program for footrot what are the objectives and the 3 options

A
○ Objectives 
§ Minimise production losses
§ Improve welfare
§ Maximise chance of eradication
□ Less lesions
□ Quicker inspection time
□ Less salvage required
○ Options
1. Foot-bathing
3. vaccination 
3. do nothing - not a good option but most common
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3
Q

Footbathing for the control of footrot during eradication program what chemicals use for the 2 types, how respond to treatment, logistics and when to cut out

A
□ Walk through 8m+ - formalin or 10% zinc sulphate weekly 
□ Stand in - footrite, 10-20% zinc sulphate, radicate 12-16 days 
-  Permanent v portable
□ Infrastructure cost
□ Efficiency of chemical use
□ Zinc sulphate tester
- Response to treatment
□ Lambs>>chronic adult lesions
- Logistics
□ Makes frequent footbathing difficult
□ Good to do with short spread period or low virulent strains (<5- 10% severe lesions)
□ When to cut out before summer
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4
Q

Vaccination for control of footrot during eradication program when use, which recently registered and requirements for efficacy

A

ONLY DO IF HAVE 1-2 SEROTYPE ON THE FARM
□ With the more virulent strains
□ Commercial footvax vaccine not currently available
□ New vaccine recently registered
- Mono/bivalent vaccine in development/restricted release
◊ Shows high efficacy in field but expensive if >1-2 serogroups
□ Requirements
- Lab diagnosis
◊ Serogroups $1000
◊ Virulence (in greater than 2 SG) $2000
- Vaccination 2 doses per 2 strains ($2.20/dose)
- Up to 12 months protection
- Annual booster or eradication

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5
Q

Eradication for footrot when start and the 2 ways to perform the cull and where get new sheep

A
- Start once conditions have dried out
○ 3-4 weeks since last footbath – care
1. By inspection
2. Destocking
• Change over cost – depends on year
• Old sheep
• Poor genotype
• Change enterprise
• Not interested in destocking 
• Disease free source
» OJD?
» Footrot (good luck)
» Lice – not a big issue
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6
Q

footrot control and eradication timetable for a farm

A
- Winter - Spring
○ Control
○ Prepare for first inspection
- Summer (or when conditions dry out)
○ December 1st inspection
○ Late January 2nd inspection
○ February 3rd inspection
○ March – April 4th inspection
- Late Autumn
○ surveillance
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7
Q

footrot eradication objective and what is involved with the inspection process

A
- Objective
○ Remove all infected sheep from property by the autumn break
○ First inspection: 5-30% (salvage)
○ Second inspection <1%
○ Third inspection 1/1000
○ Fourth inspection 0
- Inspection 
○ Every claw 4,000 sheep = 32,000 toes!
○ 1 person 400 sheep inspect per day 
○ So many - repeat inspections - to help prevent human error 
○ Keep sheep with 4 normal feet
○ If in doubt chuck it out
○ Two options
	○ Red head infected
	○ Blue head clean
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8
Q

Footrot eradication salvage some sheep why would you do it, how and reinspection process

A
  • Salvage
    ○ Cost effective to maintain stocking rate
    ○ One-off antibiotic treatment
    ○ Oxytetracyclines ESI (export slaughter interval) 90 days
    ○ How?
    1. Shaping pare
    2. Inject
    3. Footbath
    4. Shed for 24 hours
    5. Reinspect in 4 weeks – expect 10% culls
  • Reinspection process
    ○ Continue over autumn
    ○ Cull all remaining infected sheep
    ○ Two clean inspection
    ○ Final inspection probably ok just at autumn break
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9
Q

Surveillance for footrot eradication program how perform and when inspect

A
  • Chance of break down
  • Separate paddocks to decrease chance
  • Follow the 7 day rule
  • Don’t let clean sheep walk where suspect sheep have been within 7 days
  • Crutching
  • Shearing
  • Inspect the following spring
  • Clearance after normal spring
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10
Q

Prevention of footrot during eradication program what need to consider with stock on property and new stock

A
  • Fences - 100% effective needs to keep everything out
  • Roadways
    -> Communicate with neighbours
    -> If infected neighbours sheep down the road can then walk your sheep down 7 DAYS LATER
  • New stock (do not let any other sheep walk across where new sheep have walked in the previous 7 days)
    -> Bigger mobs
    ○ Do your home work before buying - inspect
    ○ Avoid buying from yards
    ○ Footbath – won’t guarantee freedom from footrot
    ○ inspection when they get home
    ○ Quarantine until they have been through a spring spread period
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11
Q

Foot abscess what are the main bacteria involved, risk factors and what makes treatment harder

A
  • Foot/Heel abscess: Fusobacterium necrophorum and Arcanobacter pyogenes - main ones
  • Toe abscess: often associated with shelly toe various pyogenic bacterium
  • Risk factors
  • Wet muddy conditions, heavy late pregnant ewes or heavy rams
  • Breeds merino, xb
  • Older sheep more vulnerable
  • If invade joint space then poorer prognosis as treatment harder
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12
Q

Foot abscess treatment

A
  • Drainage
  • Remove to dry ground
  • Antibiotics with individual valuable animals - will result in quicker recovery
  • Oxytetracyclines - longer duration of action
  • Pain relief: flunixin?
  • Manage pregnancy toxaemia risk - as very painful and pregnant ewes may stop eating enough
  • Cull chronic lame ewes/rams - increase susceptibility of recurrence especially when getting older
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13
Q

Foot abscess 3 prevention strategies and what wouldn’t you use

A
  1. Manage body condition of ewes
    - Avoid over fat ewes and ewes too light -> increase mortality with condition score less than 3 and over 3.5
  2. Run older/at risk ewes in dry paddock
  3. Gravel in yards not too rocky?
    - Footbathing may exacerbate condition - wouldn’t specifically get them in for Footbathing as moving and stressing them will INCREASE the rate of foot abscess
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14
Q

Scabby mouth what caused by, zoonosis?, is there immunity, location, environment present in, treatment and prevention

A
  • Orf/scabby mouth virus
  • Zoonosis
  • Generally only occurs once a generation in the herd as get acquired immunity
  • NOT THE HOOF -> just up the coronate
  • Lush conditions, young sheep and lambs
  • Mouth lesions with dry rough feed, thistles
  • Supportive treatment for individuals with secondary infection -> will recover in 3 weeks
  • Vaccination available but not always used -> in the peringuinal area
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15
Q

Strawberry footrot how common, cause, location, conditions present in and treatment

A
  • Generally quite rare
  • Dermatophilosis congolensis: same bacterium that causes lumpy wool or mycotic dermatitis
  • Swab/smear for diagnosis
  • Above the heel, NOT IN HOOF, more commonly on the hindlimb
  • Only in extremely wet humid conditions - generally in young hoggets
  • Antibiotic treatment - will help resolve
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16
Q

Laminitis or founder what is the general history, treatment and possible chronic issue

A
  • history of introduction of grain feeding/change ration
  • Need to slowly introduce the ration with impression feeding
  • usually recover with no treatment (acidosis may need treatment)
  • Will result in distortion of the hoof will growing so will see where the laminitis occurred
17
Q

Arthritis how common and causes

A
  • Really common
  • Causes
  • Chlamydia
  • Erysipelothrix rhusiopathiae
  • Purulent arthritis (such as joint ill) caused by various bacterium:
    ○ Arcanobacter pyogenes
    ○ Fusbacterium necrophorum
    ○ Histophilus ovis
    ○ Staph sp, Strep sp
18
Q

Arthritis pathogenesis, which sheep more vulnerable, diagnosis, treatment and prevention

A

Pathogenesis
- Infection via parentral, post marking or haematogenous route
- Younger lambs more vulnerable- Colostrum
- Post dipping lameness - to prevent this dip 10 days post shearing to allow shearing wounds to heal
Diagnosis: Swabs - smear culture and sensitivity - should figure out the bacteria cause
Treatment
- Penicillin preferred Erysipelothrix rhusiopathiae
- Oxytetracycline Chlamydia
- Occasional anti-inflammatory Rx (flunixin)
Prevention
- Hygiene marking, dipping, tail length, time of shearing
- Vaccination Eryvac® for Erysipelothrix rhusiopathiae - another reason for diagnosis of the bacteria involved

19
Q

How would you differentiate between benign and virulent footrot

A
  • Examine 100 sheep at random, footscore them and reassess them in mid spring - THE BEST
    ○ Why reassess -> if mainly early lesions -> cannot be sure if benign or virulent footrot
    § DON’T KNOW WHETHER HAD TIME FOR CLINICAL MANIFESTATION - need to allow time
20
Q

Nitrate/nitritie poisoning origin, what effect and why effect in respiratory and clinical signs

A

○ Nitrate from feed -> nitrite -> ammonia -> microbial protein
Nitrate has a direct caustic effect on the GIT
• Diarrhoea and vomiting.
• Salivation.
• Abdominal pain.
Nitrate/Nitrite poisoning
- If high levels of nitrite formed absorbed into circulation
○ Converts haemoglobin to methaemoglobin -> unable to carry enough oxygen
Clinical signs
- The signs of nitrite poisoning usually appear 6–24 hours after the toxic material is consumed.
- These include:
○ Rapid, noisy and difficult breathing.
○ Blue/chocolate-coloured mucous membranes.
○ Rapid pulse.
○ Salivation, bloat, tremors, staggering.
○ Weakness, coma, terminal convulsions, death.
○ Dark ‘chocolate-coloured’ blood.
○ Abortions – pregnant females that survive nitrate/nitrite poisoning may abort due to a lack of oxygen to the foetus.

21
Q

Ocular conditions in sheep what are the 2 main ones and describe cause, treatment

A
  1. Pink eye:
    - Sporadic, often self-limiting if just a few cases
    - Consider carefully whether it is worth yarding the mob or not
    - Organisms are typically Chlamydophila or Mycoplasma (was Rickettsia)
    ○ Not zoonotic
    - Not as strongly seasonal as cattle disease
    - Grazing around spiky plants can contribute to the condition
    - Can use parenteral oxytetracycline.
    - Farmers may use ‘Pink eye aerosol’ – contains oxytetracycline. Short duration.
  2. Foreign bodies
    Grass seeds – especially barley gras
22
Q

What are the 4 main clinical signs of respiratory disease

A
  • Nasal discharge, sneezing, coughing, open-mouth breathing
  • Dyspnoea, swelling (lymph nodes)
  • Exercise intolerance
  • Mortality - collapse and death
23
Q

What are 6 ways to diagnose respiratory disease

A
- Respiratory sound - nature, location 
○ Can be hard to evaluate 
- Haematology 
- Necropsy - important as lead to death 
○ Ocular fluid therapy 
- Bacteriology 
- Toxicology 
- Response to treatment
24
Q

What are the risk factors for respiratory disease

A
  • Intensive systems -> poor ventilation - build of gases
  • Mixing of animals and pathogens
  • Damage/irritation of respiratory tract defences - due to poor ventilation
  • Stress and depressed immunity
  • Easy transmission between individuals
  • Antibiotics resistance
  • Ineffective vaccination
  • Pathogen introduction via fomites
25
Q

List the 2 main diseases of the upper respiratory tract and lower respiratory tract

A

1) drenching disease
2) oestrus ovis - nasal bot
UPPER
1) pneumonia
2) pleural/superficial pulmonary abscess

26
Q

Drenching disease what are the 2 main forms and what leads to

A
  1. retropharyngeal trauma - when drench capsule is given into the lungs instead of rumen
    § Sheep neck needs to be elongated and lifted -> need to swallow the device then put in capsule
  2. aspiration pneumonia - liquid anthelmintic isn’t correctly given -> too far into throat -> drench given into trachea
    ○ Leads to sudden death
27
Q

Oestrus ovis clinical signs how to control at herd level and for hobby farm

A

○ Ongoing snotty noise but doesn’t affect body system
○ Controlled by ML anthelmintics -> when drenching sheep for other parasites will be control
○ Injectable Ivermectin can also be given if on hobby farm
§ Secondary bacterial infection generally occurs -> long course of oxytetracycline

28
Q

Pneumonia causes what are the 4 main ones and diagnosis

A

Causes
1. Iatrogenic - aspiration pneumonia from drenching incorrectly
2. Viral
3. Bacterial - mycoplasma ovis “summer pneumonia”
§ +/- complicated Mannheimia, Pasteurella - commercials - COMMON when chronic
4. Parasitological
§ Lungworm -> usually incidentally controlled by ML
Diagnosis
- Necropsy, history, bacteriology

29
Q

Pneumonia treatment/prevention

A

○ Empirical (oxytetracycline) - treat those with clinical signs and growing poorly
○ Occasional other respiratory ‘big guns” (tulathromycin, tilmicosin)
○ Vaccines used overseas

30
Q

Pleural/superficial pulmonary abscess what common in, clinical signs, diagnosis and treatment

A

○ Common/underdiagnosed in rams
§ Weight loss, mild pyrexia, hyperpnoea
○ May detect with chest ultrasound (or find at necropsy)
○ Seem to respond to long courses (6 weeks) of penicillin

31
Q

Cheesy gland prevalence, immunity, significance and transmission

A
  • Flock prevalence 30-100%
  • Good immunity against subsequent infections
  • Significance - COST
    ○ Condemned meat
    ○ Lost milk production
  • Transmission
    ○ Carrier animal with cheesy gland in the lung that rupture - cough onto other animal skin (where have cuts and is wet)
    § These conditions are met during shearing or sheep dips
    § Increase risk -> when young sheep in dip AFTER old sheep (old sheep contaminate)
    ○ Environmental contamination of ruptured abscess from post mortem etc.
32
Q

Cheesy gland vaccine, how long last and which groups of animals would you and would you not vaccinate

A
  • Only lasts a year - vaccinate with 5 in 1 (ewes - minor cost to add) ○ Wethers -> not vaccinated every year so unlikely to be a benefit of vaccinating UNLESS prevalence if very high
  • 2 initial doses than annual boosters
33
Q

Caprine arthritis and encephalitis cause and significance

A
  • Lentivirus - goat equivalent of sheep exotic diseases
  • Notifiable
  • Widespread australia and world
    ○ 82% of NSW flocks
  • Slow incubation, difficult detection, no treatment
  • Costly disease
    ○ Decreased milk production in sub-clinicals
    ○ Culled clinical animals
    ○ Surveillance and eradication needs lots of testing
34
Q

Caprine arthritis and encephalitis what are the 5 syndromes and transmission

A

Clinical signs - 5 syndromes - COMBINED AFFECT
1. Leuko-encephalomyelitis
2. Arthritis
3. Chronic wasting
4. Interstitial pneumonia - respiratory issue
○ Generally non-suppurative
5. Hard udder
- All have characteristics mononuclear-cell inflammation
Transmission
- Vertical (milk and colostrum) - NEED TO BE SNATCHED REARED
- Horizontal (Secretions) -> strictly quarantine the herd from clean to diseased - if possible cull the diseased

35
Q

Caprine arthritis and encephalitis diagnosis and certification qualifications

A
  • Clinical signs
  • Characteristic non-suppurative inflammation (mononuclear cells on joint tap or histopathology)
  • Complete serological diagnosis is difficult
    ○ Maternal Ab = offspring +ve until 2 months
    ○ Often low Ab levels
    § Not protective anyway
    § Develops late (>8monts)
    ○ Can relapse to negative
  • DON’T TRUST THE FIRST NEGATIVE TEST
    ○ Need 2 herd negative tests 6-12 months apart to be accredited free
    § Re-rest every 1-2 years for ongoing accreditation
36
Q

Caprine arthritis and encephalitis control and eradication

A
  • Surveillance
    ○ Repeated blood testing - determine whether negative
    ○ Cull reactors AND or run infected and uninfected herds
  • Prevent transmission
    ○ Vertical - snatch rearing, don’t pool colostrum
    ○ Horizontal - strict biosecurity between herds and only buying in from certified free herds
37
Q

List the 4 main exotic diseases that present with respiratory signs

A

1) other retroviruses
2) nasty mycoplasma disease
3) sheep and goat pox
4) bluetongue

38
Q

What are the 2 main retrovirueses that present with respiratory signs and what lead to

A
  1. Maedi visna/ovine progressive pleuropneumonia
    § Dyspnoea wasting
    § Ovine equivalent of CAE (no cross-species infections observed in the field)
  2. Jaagsiekte/pulmonary adenomatosis
    § Pulmonary tumours caused by another retrovirus
39
Q

Bluetongue what transmitted by and clinical signs

A

○ Transmitted by culicoides (not contagious)
○ Clinical signs
§ FEVER, FACE (oedema - due to vasculitis)), FEET (lameness - due to vasculitis), coronet swelling)
§ Nasal discharge
§ Dyspnoea