Small Animal Medicine Exam I Flashcards

1
Q

For animals with GI signs, getting a good history is of the utmost importance to get on the correct diagnostic path. What are some questions you should be asking your clients?

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2
Q

Define dysphasia. List some common causes.

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3
Q

Neurogenic dysphagia can be categorized as ____, ____, or ____.

A

Prehensile, pharyngeal, or cricopharyngeal

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4
Q

What is prehensile neurogenic dysphagia?

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5
Q

Pharyngeal and cricopharyngal dysfunction/dysphasia usually result in _____.

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6
Q

Define halitosis. List some common causes.

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7
Q

Differentiate between ptyalism and pseudopytalism.

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8
Q

Differentiate between vomiting and regurgitation.

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9
Q

How can we use nausea, retching, the presence of bile, and pH to differentiate between vomiting and regurgitation?

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10
Q

Vomiting is most commonly associated with which other clinical manifestations?

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11
Q

Regurgitation paired with dysphagia should lead to explore for ___ disease.

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12
Q

Regurgitation without dysphagia should lead to explore for ___ disease. List some examples.

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13
Q

What is expectoration?

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14
Q

What is hematemesis?

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15
Q

What is diarrhea? List some different causes for acute and chronic diarrhea.

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16
Q

Describe small intestinal diarrhea in terms of weight loss, polyphagia, frequency of vowel movements, volume of feces, blood in feces, mucus in feces, tenesmsus, and vomiting.

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17
Q

Describe large intestinal diarrhea in terms of weight loss, polyphagia, frequency of vowel movements, volume of feces, blood in feces, mucus in feces, tenesmsus, and vomiting.

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18
Q

List some common causes of chronic large intestinal diarrhea.

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19
Q

____ is considered normal on the purina fecal score.

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20
Q

What is hematochezia? What is it associated with?

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21
Q

What is melena? What is it associated with?

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22
Q

Define tenesmus.

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23
Q

Define dyschezia.

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24
Q

What is constipation?

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25
Q

What is obstipation?

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26
Q

What is fecal incontinence? List some common causes.

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27
Q

What are the different GI related etiologies of weight loss?

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28
Q

Anorexia and hyporexia are common findings in GI disease. Differentiate between the two.

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29
Q

What does abdominal pain look like in the small animal patient?

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30
Q

What is an acute abdomen? List some causes.

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31
Q

List some different causes of abdominal enlargement in the small animal patient.

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32
Q
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33
Q

What is this?

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34
Q
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35
Q

What individual organ s should you be able top identify on abdominal palpation of the dog?

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36
Q

What individual organs should you be able top identify on abdominal palpation of the cat?

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37
Q

How can we increase our visualization of the base of the cat tongue to assess for linear foreign bodies?

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38
Q

A complete physical exam in the dog includes a rectal palpation. What structures should you be able to palpate?

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39
Q

Describe the value of a minimum database for a small animal patient with GI related clinical signs.

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40
Q

Describe the value of a fecal parasite test for a small animal patient with GI related clinical signs.

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41
Q

Describe the value of a bacterial fecal culture for a small animal patient with GI related clinical signs.

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42
Q

Describe the value of an ELISA/IFA/PCR fecal analysis for a small animal patient with GI related clinical signs.

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43
Q

Describe the use of ELISA testing to diagnose parvovirus.

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44
Q

Describe the use of ELISA-SNAP testing to diagnose giardia.

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45
Q

Describe the value of a fecal cytology for a small animal patient with GI related clinical signs.

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46
Q

Identify the pathogen found on a fecal cytology.

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47
Q

Identify the pathogen found on a fecal cytology.

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48
Q

Identify the pathogen found on a fecal cytology.

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49
Q

How do we use serum gastrin as a diagnostic test for GI disease?

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50
Q

How do we use Helicobacter testing as a diagnostic test for GI disease?

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51
Q

How do we use fecal alpha-1 protease inhibitor as a diagnostic test for GI disease?

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52
Q

We use _____ to diagnose EPI.

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53
Q

We use ____ to diagnose pancreatitis.

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54
Q

We use _____ to evaluate/diagnose bacterial overgrowth and intestinal disease.

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55
Q

____ and ____ are two different ways we can use radiography to diagnose GI diseases.

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56
Q

List the contraindications for a GI barium study in a small animal patient.

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57
Q

What is your top differential for this patient?

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58
Q

Describe how we use ultrasonography to diagnose GI disease.

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59
Q

Differentiate between a rigid and flexible endoscope and their usefulness as a diagnostic test for a patient with GI disease.

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60
Q

What are the pros to using endoscopy/colonoscopy to diagnose GI disease?

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61
Q

What are the cons to using endoscopy/colonoscopy to diagnose GI disease?

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62
Q

What are the two main goals (general) of a laparotomy in a patient with GI disease?

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63
Q
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64
Q
A
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65
Q

Fluids should never be administered _____ since it can cause ____.

A

Intraperitoneal ; peritonitis (also takes 24-48hrs to absorb)

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66
Q

What distribution of total body water can be found intracellular, extracellular, interstitial and intravascular?

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67
Q

We use fluids in patients with GI disease to address ____, ____, and ____.

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68
Q

_____ such as ____ are known to correct acid-base deficits faster than acidifying fluids like _____.

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69
Q

_____ is a common pathology in chronic GIT disease. How do we address it with fluid therapy?

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70
Q

Differentiate between enteral, parenteral, and SQ fluid administration.

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71
Q

How do we determine the rate at which to administer fluids to our small animal patients?

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72
Q

Daily crystalloid fluids should be calculated based on ____, ____, and ____.

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73
Q

What does 5-6% dehydration look like clinically?

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74
Q

What does 7-8% dehydration look like clinically?

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75
Q

What does 9-10% dehydration look like clinically?

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76
Q

How do we calculate fluid deficit in L?

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77
Q
A
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78
Q

What are the fluid maintenance needs for a canine or feline patient?

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79
Q
A
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80
Q

How do we calculate ongoing losses as it relates to vomiting or diarrhea?

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81
Q
A
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82
Q

What are the appropriate rates for initial resuscitation boluses in canine and feline patients?

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83
Q

If your patient is anorexic or not eating enough, you can prevent hypokalemia by supplement KCl in their fluids. However, you should not add more than ___.

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84
Q

What are some foods that we consider bland and easily digestible for our small animal patients?

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85
Q

Describe hypoallergenic dietary management for GI patients.

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86
Q

What are the indications for an ultra low fat diet for a GI patient?

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87
Q

What are the indications for a low fat diet for a GI patient?

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88
Q

When and why would you consider fiber supplementation in a GI patient?

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89
Q

Differentiate between soluble and insoluble fiber.

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90
Q

How do we calculate basal energy requirements and maintenance energy requirements for our small animal patients?

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91
Q
  • Use 1.5 as your adjustment factor
A
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92
Q

In addition to adjusting the diet, how else can we modify the diet our of GI patients to encourage them to eat?

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93
Q

List the different therapeutics we use as appetite stimulates in our canine and feline patients.

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94
Q

Differentiate between enteral nutrition and tube feeding.

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95
Q
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96
Q
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97
Q
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98
Q

Differentiate between total parental nutrition and partial parental nutrition.

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99
Q

List some of the different peripherally acting antiemetics we use in our GI patients.

A
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100
Q

List some of the different centrally acting antiemetics we use in our GI patients.

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101
Q

Why do we use antacids? List the major categories.

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102
Q

List the different acid titrating antacid drugs we use in our GI patients.

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103
Q

List the different H2 blocker antacid drugs we use in our GI patients.

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104
Q

List the different proton pump inhibitors antacid drugs we use in our GI patients.

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105
Q

Why do we use intestinal protectants in our GI patients? List the ones we most commonly use.

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106
Q

Why do we use pancreatic enzyme supplements in our GI patients? List the ones we most commonly use.

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107
Q

Why do we use motility modifiers in our GI patients? List the ones we most commonly use.

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108
Q

Why do we use anti-inflammatory or anti-secretory drugs in our GI patients? List the ones we most commonly use.

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109
Q

Why do we use immunosuppressive therapies in our GI patients? List the ones we most commonly use.

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110
Q

Why do we use antibacterials in our GI patients? List the ones we most commonly use.

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111
Q

We use a combination of ____, ____, and _____ for symptomatic Helicobacter gastritis.

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112
Q

Differentiate between a probiotic and prebiotic.

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113
Q

Why do we use enema, laxatives, and cathartics in our GI patients? List the ones we most commonly use.

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114
Q

What is gingivitis/periodontitis? How do we diagnose and treat it?

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115
Q

What is a sialocele? How do we diagnose and treat it?

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116
Q

List the most common benign neoplasias of the canine oral cavity.

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117
Q

List the most common malignant neoplasias of the canine oral cavity.

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118
Q

How do we diagnose, stage, and treat oral neoplasia in the canine oral cavity?

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119
Q
A
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120
Q
A
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121
Q

What are some of the different oral neoplasms we may find in the feline oral cavity? Which is most common?

123
Q

Describe the etiology, lesions, diagnosis, and treatment of feline eosinophilic granulomas.

124
Q

When it comes to eosinophilic granuloma complex, you should always think of the cause as ____ or ____.

125
Q

What are some of the causes of primary and secondary stomatitis?

126
Q

What is the etiology of feline lymphocytic-plasmacytic stomatitis?

127
Q

How do we diagnose and treat feline lymphocytic-plasmacytic stomatitis?

128
Q

Describe masticatory muscle myositis as a potential causative agent of dysphasia.

129
Q

Describe cricopharyngeal achalasia/dysfunction as a potential causative agent of dysphasia.

130
Q

What is the etiology, clinical signs, diagnosis, and treatment for pharyngeal dysphasia?

131
Q

What is the etiology, clinical signs, and diagnostic tests for congenital megaesophagus?

132
Q

What is the treatment for congenital megaesophagus? What secondary complication is the primary cause of death for these patients?

134
Q

What are the different etiologies of acquire megaesophagus? How do we diagnose it?

135
Q

How do we treat acquire megaesophagus? What secondary complication is the leading cause of death for these patients?

136
Q

What is the etiology of esophagitis?

137
Q

What are the clinical signs of esophagitis? How do we diagnose it?

138
Q

How do we treat esophagitis?

139
Q

Esophageal stricture is a possibility for up to ___ following an anesthetic/intubation event.

140
Q

Describe a hiatal hernia as an esophageal pathology.

141
Q

Describe dysautonomia as an esophageal pathology.

142
Q

What is happening in this radiograph?

143
Q

What is a vascular ring anomaly? How do we diagnose and treat it?

144
Q

Identify the pathology in this radiograph.

145
Q

Identify the pathology.

147
Q

Describe the prevalence, clinical signs, sequelae, and diagnose of an esophageal foreign body.

148
Q

What are the most common locations for an esophageal foreign body?

149
Q

What is the treatment for an esophageal foreign body?

150
Q

What are your differentials for this patient?

151
Q

What is esophageal cicatrix? What is the most common clinical sign and how do we diagnose it?

152
Q

How do we treat esophageal cicatrix?

153
Q

What are the most common esophageal neoplasms seen in the dog?

154
Q

What are the most common esophageal neoplasms seen in the cat?

155
Q

How do we diagnose and treat esophagus neoplasms in our canine and feline patients?

156
Q

Identify the anatomy of the stomach.

157
Q

Identify the anatomy of the stomach.

158
Q

What key clinical pathology findings would you expect to find in a patient with chronic vomiting versus chronic diarrhea?

159
Q

Gastritis is a diagnosis of ____. What are the different kinds of gastritis we will encounter clinically?

160
Q

Describe the prevalence and causes of acute gastritis.

161
Q

What are the clinical signs of acute gastritis? How do we diagnose it?

162
Q

When should we consider acute gastritis more severe and pursue intestinal treatment?

163
Q

What is the treatment for acute gastritis?

164
Q

Describe the prevalence and key clinical signs/signalment relative to acute hemorrhagic diarrhea syndrome/

165
Q

How do we diagnose AHDS/HGE?

166
Q

How do we treat AHDS/HGE?

167
Q

What is chronic gastritis? What is the most common clinical sign?

168
Q

How do we diagnose chronic gastritis?

169
Q

What are your differentials for a patient with chronic gastritis?

170
Q

How do we use dietary management and supportive therapeutics to treat chronic gastritis?

171
Q

How do we diagnose and treat Helicobacter-associated disease?

172
Q

What are the different causes of gastric outflow obstructions?

173
Q

Describe the causes, risk factors, and clinical signs of pyloric stenosis.

174
Q

How do we diagnose and treat pyloric stenosis?

175
Q

Describe the prevalence and most common clinical sign of gastric foreign bodies.

176
Q

How do we diagnose gastric foreign bodies?

177
Q

What is the treatment for gastric foreign bodies?

178
Q

What are the risk factors for a GDV?

179
Q

Describe the pathophysiology of a GDV.

180
Q

Identify the anatomy.

181
Q

Identify the anatomy.

182
Q

Identify the radiographic pathology.

183
Q

How do we diagnose a GDV?

184
Q

A ___ rotations (GDV) is the most common.

185
Q

What is the treatment for a GDV?

186
Q

What should we monitor for in our GDV patients post-operatively?

187
Q

Describe the cause, clinical signs, diagnosis, and treatment for bilious vomiting syndrome.

188
Q

What are the different etiologies of gastrointestinal ulceration/erosion?

189
Q

What are the clinical signs of GI ulcers/erosions?

190
Q

How do we diagnose gastrointestinal ulceration/erosion?

191
Q

What would you expect to find on the abdominal ultrasound of a patient with GI ulcers?

192
Q

What is the treatment for GI ulcers?

193
Q

How do we prevent GI ulcers?

194
Q

What is the most common gastric neoplasia in the dog? What about the cat?

195
Q

What are the clinical signs of gastric neoplasias?

196
Q

How do we diagnose gastric neoplasia?

197
Q

What would you expect to find on the CBC of a patient with gastric neoplasia?

198
Q

What is the treatment for gastric neoplasia?

202
Q

What is the etiology of acute enteritis?

203
Q

What are the clinical signs of acute enteritis?

204
Q

How do we diagnose acute enteritis?

205
Q

What are the general treatment options for acute enteritis?

206
Q

How do we modify diet to treat acute enteritis?

207
Q

What is the etiology of dietary induced acute diarrhea?

208
Q

What are the clinical signs of dietary induced acute diarrhea?

209
Q

How do we diagnose and treat dietary induced acute diarrhea?

210
Q

What is the etiology of canine parvovirus?

211
Q

What is the most common cause of canine parvovirus infections? What breeds are most susceptible?

212
Q

What are the clinical signs of canine parvovirus?

213
Q

How do we diagnose canine parvovirus?

215
Q

What is the canine parvovirus monoclonal antibody?

216
Q

What are some common therapeutic mistakes when it comes to treating GI disease?

217
Q

In addition to canine parvovirus, what are some other causative agents of viral diarrhea?

218
Q

List three bacterial causes of diarrhea.

219
Q

List two fungal causes of diarrhea.

220
Q

Describe Campylobacter as a causative agent of bacterial diarrhea. What are the clinical signs? How is it diagnosed and treated?

221
Q

Describe salmonella as a causative agent of bacterial diarrhea. How is it spread? What are the clinical signs and treatment?

222
Q

Describe clostridium disease as a causative agent of diarrhea. What are the clinical signs? How do we diagnose it?

223
Q

How do we treat clostridial diarrhea?

224
Q

Describe histoplasmosis as it affects dogs and cats. What systems does it primarily attack?

225
Q

How do we diagnose and treat diarrhea secondary to histoplasmosis?

226
Q

Identify the organism (common and scientific name).

227
Q

What are the clinical signs of whipworms? How do we diagnose and treat them?

228
Q

Identify the organism (common and scientific name).

229
Q

What are the clinical signs of roundworms? How do we diagnose and treat them?

230
Q

Describe the public health concern relative to roundworms.

231
Q

Identify the organism (common and scientific name).

232
Q

What are the clinical signs of hookworms? How do we diagnose and treat them?

233
Q

Describe the public health concern when it coms to hookworms.

234
Q

Identify the organism (common and scientific name).

235
Q

What are the clinical signs of tapeworms? How do we diagnose and treat them?

236
Q

Identify the organism (common and scientific name).

237
Q

What are the clinical signs of coccidiosis? How do we diagnose and treat it?

238
Q

Identify the organism (common and scientific name).

239
Q

What are the clinical signs of cryptosporidia? How do we diagnose and treat it?

240
Q

Identify the parasites.

241
Q

Identify the parasites.

242
Q

Identify the organism (common and scientific name).

243
Q

What are the clinical signs of giardiasis? How do we diagnose and treat it?

244
Q

Identify the organism (common and scientific name).

245
Q

What are the clinical signs of trichomoniasis? How do we diagnose and treat it?

246
Q

Identify the parasites.

247
Q

Describe the etiology and clinical signs of EPI.

248
Q

How do we diagnose EPI?

249
Q

Describe the etiology, clinical signs, and diagnosis of dietary responsive diarrhea.

250
Q

What is the treatment for dietary responsive diarrhea?

251
Q

What is the etiology of ARE/dysbiosis?

252
Q

What are the clinical signs of ARE?dysbiosis? How do we diagnose it?

253
Q

What is the treatment for ARE/dysbiosis?

254
Q

Describe the relationship between dietary responsive diarrhea and ARE.

255
Q

What is the definitive sequence fr therapeutic trails for patients with chronic enteropathies?

256
Q

What is the etiology of IBD?

257
Q

What are the clinical signs of IBD? How do we diagnose it?

258
Q

How do we treat IBD in dogs?

259
Q

How do we treat IBD in cats?

260
Q

What is the etiology of intestinal lymphangectasia? What breeds are most susceptible?

261
Q

What are the clinical signs of intestinal lymphangectasia? How do we diagnose and treat it?

262
Q

List some of the different kinds of intestinal obstructions we may find in our small animal patients.

263
Q

How do we determine in the small intestine is distended in the radiograph of a canine abdomen?

264
Q

How do we determine if the small intestine is distended in the radiograph of a feline abdomen?

265
Q

Where is an intussusception most likely to occur in the GIT? What patients is it most likely to occur in a what are the clinical signs?

266
Q

Identify the different layers of the intestine.

267
Q

If you see this sign on ultrasound, it is indicative of an ______.

A

Intusussception

268
Q

What are some different causes of large intestinal inflammation in our small animal patients?

269
Q

What are the different causes, clinical signs, diagnosis, and treatments for acute colitis?

270
Q

What are the etiologies, diagnostic tools, and treatments for chronic colitis?

271
Q

What is this and why does it happen?

272
Q

What is the treatment for a rectal prolapse?

273
Q

What is this?

275
Q

What is this?

276
Q

What is anal sacculitis? How do we treat it?

277
Q

What are the different types of neoplasias we commonly find in the small intestines?

278
Q

What are the different types of neoplasias we commonly find in the large intestines?

279
Q

What are some different causes of constipation?

280
Q

Identify the main pathology in this radiograph.

281
Q

Identify the main pathology in this radiograph.

282
Q

What is the treatment for constipation?

283
Q

Septic peritonitis is usually caused by _____.

284
Q

What are the most common causes of septic peritonitis in the dog?

285
Q

List some of the most common organisms associated with secondary septic peritonitis.

286
Q

What are the most common causes of septic peritonitis in the cat?

287
Q

What are the causes/etiologies of primary (spontaneous) bacterial peritonitis in dogs and cats?

288
Q

When would you expect to see post operative peritonitis clinically? What are some risk factors?

289
Q

What are the clinical signs of septic peritonitis?

290
Q

How do we diagnose septic peritonitis?

291
Q

Identify the main pathology in this radiograph.

292
Q

Identify the main pathology in this radiograph.

293
Q

Identify the main pathology.

294
Q

What would you expect to see on cytology of abdominal fluid from a patient with septic peritonitis?

295
Q

What is the treatment for primary bacterial peritonitis (PBP)?

296
Q

What is the treatment for secondary (septic) peritonitis?

297
Q

What is a hemoabdomen? What are some causes?

298
Q

The number one cause of nontraumatic hemoabdomens is _____.

A

Neoplasia (hemangiosarcomas)

299
Q

Describe hemangiosarcomas as a causative agent of hemoabdomens.

300
Q

What is abdominal carcinomatosis? What is the primary complaint/clinical sign and cause?

301
Q

What is the treatment and prognosis for abdominal carcinomatosis?