Slide set 3 Flashcards

1
Q

Lack of sympathetic tone or heart failure would do what to a CO curve?

A

Right shift and decreased plateau

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2
Q

Increase in sympathetic stimulation or increased pumping effectiveness will cause what in a CO curve?

A

Left shift and increased plateau

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3
Q

An increase in SVR also causes an increase in

A

RVR

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4
Q

SVR is also

A

Afterload

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5
Q

An increased preload will show what variables on the PV loop

A

Increased SV and EDV; ESV stays the same

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6
Q

A decreased preload will show what variables on the PV loop

A

Decreased SV and EDV; ESV stays the same

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7
Q

Increased afterload will show what variables on the PV loop

A

Decreased ejection time and SV; increased time spent in phase II; increased ESV; EDV remains the same

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8
Q

Decrease afterload will show what variables on the PV loop

A

More time spent in ejection; Increased SV and reduced ESV

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9
Q

Contractility is denoted by what on the PV loop

A

Upper left hand corner

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10
Q

Increased contractility will show what on the PV loop

A

Steeper slope with leftward loop shift; increased SV and decreased ESV

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11
Q

Decreased contractility will show what on the PV loop

A

Reduced slope; reduced SV and increased ESV

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12
Q

What is happening during mitral regurgitation

A

Any time the pressure is higher in the ventricle than the left atria, blood is backflowing

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13
Q

Why is EF not a good diagnostic for MR

A

SV is increased

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14
Q

A temporary enhanced filling will eventually cause ______ in MR

A

eccentric hypertrophy over time

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15
Q

Mild MR

A

<30 mL backflow

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16
Q

Moderate MR

A

30-60 mL regurg flow

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17
Q

Severe MR

A

> 60 mL regurg flow

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18
Q

IABP inflates during

A

Diastole to help push blood down the atria and perfuse the CAs

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19
Q

IABP deflates during

A

Systole to create an artificially low afterload

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20
Q

What medications might we give to someone with MR and what considerations should we have

A

Afterload reducers; caution in patients with CAD and they may have trouble perfusing the CAs

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21
Q

We want to decrease the ________ to reduce regurgitant flow in MR

A

Delta P

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22
Q

Time spent in ______ can increase mitral regurgitant flow. To combat this we would allow

A

Systole; permissive slight tacycardia

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23
Q

Mitral regurgitation will show what on a PV loop

A

Increased SV and EDV; decreased ESV; longer ejection time

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24
Q

Mitral stenosis will show what on a PV loop

A

Lower SV, EDV, and ESV; left shift - similar to decreased preload

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25
The body compensates for mitral stenosis by
Increasing filing pressures and CVP
26
Increasing filling pressures of the right heart for MS can cause
Increase in pulmonary pressures, prolonged P wave and a right axis deviation
27
Mitral stenosis does not have a big effect on
the left ventricle
28
We want to avoid what intraoperatively with mitral stenosis? Why?
Tachycardia; need as much time in diastole as possible
29
MS was attributed to rheumatic fever years ago - what might cause it now?
A bad strep infection
30
Why do we want to avoid a fib in patients with MS
Loss of atrial kick to help filling
31
What happens during aortic regurgitation
Blood backflows in LV anytime the pressure is higher in the aorta
32
Compensation for aortic regurg
Fill the heart with more blood
33
What compensation for aortic regurg may make the backflow even worse
Constricting of vessels to maintain ABP; increased SVR and afterload will further increase delta P
34
What will you see on a PV loop in AR
Increased SV, EDV, and ESV
35
What kind of hypertrophy would you find in someone with AR
Eccentric
36
What medications would we consider in someone with AR
afterload reducers
37
Pulse pressure is _____ in aortic stenosis
Reduced
38
What is happening during aortic stenosis
the heart is having to pump against a non compliant valve
39
Compensation for AS
Increase in filling pressures or an increase in HR
40
What would see on a PV loop for aortic stenosis
Decreased SV; increased preload and ejection pressures; increased EDV and ESV
41
Concentric hypertrophy is
Thickening of the heart wall via addition of parallel sarcomeres
42
Why is preload increased in AS
Thicker heart wall is less compliant to filling
43
Tachycardia is bad for someone with AS because
Less time spent in diastole; therefore less time perfusing CA
44
Why is aortic stenosis resistant to CPR
The sequence has been taken out of the contraction; pushing on everything at once
45
In someone with bad CO and contractility how might we help them out?
Afterload reducer
46
During early inspiration, what occurs
R heart CO increases 2 fold, L heart CO decreases, PAP decreases due to pulm vessels being pulled open, MAP decreases
47
PPV affects thoracic pressure and hemodynamics by
Increasing thoracic pressure causing a temporary increase in R and L CO, MAP, and PAP; over time, this decreases VR
48
Coronary blood flow is equivalent to
70mL/min/100 grams of heart muscle
49
Normal coronary blood flow rate
225 mL/min
50
Delta P of aortic pressure and ventricular wall pressure tells us
Coronary blood flow
51
Temporary retrograde flow of the LCA is caused by
the beginning of systole
52
X descent
occurs after A wave; drop in pressure during atrial relaxation of mid systole
53
Y descent
occurs after V wave; drop in pressure d/t ventricular filling at the start of diastole
54
H plateau
Immediately before A wave; occurs during the middle third of diastole
55
Overdampening of an A line may be due to
Occlusion or partial occlusion
56
Underdampening of an A line may be due to
Artifact
57
When we place a spinal block we are at risk for causing
sympathetic block and knocking out the nerves that pace the heart
58
The "shortcut" from an AV fistula will cause a decrease in
RVR
59
At what percentage of blood loss is our CO affected
15%
60
At what percent of blood loss is our ABP affected
20%
61
At percent of blood loss would cause fatality if the SNS was knocked out
10-15%