Skin Changes in CardioPulm Diseases Flashcards
6 P’s of arterial occlusion
Parensthesia Perishing Cold Pulselessness Pain Paralysis Pallor
two signs of poor venous return
edema and varicosities
stasis dermatitis
reddish/purplish/brownish discoloration in skin develops over time
this is due to hemosiderin deposits staining the skin from red cell breakdown
occurs with decrease flow or stasis on venous side
advanced stasis dermititis
skin thickened
firm to touch
brawny edema is term used to describe if swollen
4 major signs of venous insufficiency
edema
varicose veins
skin changes/discoloration
skin ulceration
cellulitis is most often
infectious
cellulitis signs and symptoms
marked erythema
increased warmth
increased swelling
sometimes skin weeping without apparent sores
janeway lesions are
bacterial endocarditis
irregular on soles and palms
non-tender
days to weeks
osler nodes are
bacterial endocarditis
nodules 1mm to over 1cm
fingers and toes and thenar/hypothenar eminence
tender
hours to days
splinter hemorrhages are
bacterial endocarditis
microemboli produced from valvular pathology
periphery of nail bed
more likely due to nail trauma if isolated or minimal involvement
eruptive xanthoma
not seen in all pts with hyperlipidemia
clubbing
chronic pulmonary edema
may be caused by platelet and endothelial GF
peripheral hypoxia may be part of trigger
tell vascular from nerves walking
nerves increased weakness
vascular no change
tell vascular from nerves palliative factors
nerves can be improved by bending over
stopping activity in vascular
tell vascular from nerves provocative factors
nerves walking downhill and increased lordosis make worse
vascular walking uphill and increased metabolic demands make worse
