Shoulder Complex V: Test 3 Flashcards

1
Q

prevalence of dislocation

A

GH is most commonly dislocated joint

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2
Q

cause of anterior GH dislocation

A

most common

mechanism = ER and ABD with FOOSH

anterior inferior direction

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3
Q

cause of posterior GH dislocation

A

less common (2-4%)

mechanism = 90 degrees of flexion with FOOSH

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4
Q

structures involved with dislocation

A

stretch/tear capsule/ligaments

possibly labrum (bankart lesion)

possibly SLAP

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5
Q

describe fibrocartilage

A

thicker and concave vs. articular cartilage

outer = thick

inner = thin

widens and deepens joint surface

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6
Q

locations of fibrocartilage

A

shoulder and hip labrum

SC, tibiofemoral, AC, ulnotriquetral, intervertebral, and pubic symphysis joints

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7
Q

cells of fibrocartilage

A

fibro and chondrocytes

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8
Q

what collagen makes up fibrocartilage

A

outer = mainly type I
-resist tension for stabilization
-majority type in all fibrocartilage including labrum

inner = lesser type II, III, and IV
-resist compression for shock absorption

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9
Q

vascularity of fibrocartilage

A

outer = vascular and neural; attributes to proprioception/kinesthesia like ligaments/annulus for stabilization

inner = hypo vascular/hypo neural/alymphatic

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10
Q

etiology of fibrocartilage damage

A

tears including possibly with RTC/dislocations

gradual/repetitive stresses including impingement

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11
Q

describe the healing of fibrocartilage

A

better in periphery

tensile strength initially improves at 3-5 weeks

even greater strength when dense fibrous tissue fills in at 8-12 weeks `

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12
Q

MET focus for distinctive fibrocartilage

A

tissue integrity/proliferation with vascularity issues

stabilization due to stabilizing role of fibrocartilage

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13
Q

other possible damages/involved structures with dislocation

A

fx aka hill Sachs lesion = compression fracture of humeral head

RC tears

neuromuscular structure compromise

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14
Q

symptoms fo dislocation

A

trauma in characteristic position

acute presentation

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15
Q

signs fo dislocation

A

ROM = limit/pain in most directions

RST/MMT = weak/pain in most directions

stress levels = likely + depending on structure involved

possibly + special tests for labrum

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16
Q

what are the special tests for the labrum

A

anterior instability (+ apprehension, + relocation)

anterior labrum (speeds test)

posterior inferior labrum (jerk test)

SLAP

17
Q

special tests for fx

A

olecranon-manubrium percussion test

bony apprehension test

18
Q

PT Rx for dislocation

A

immobilization up to 6 weeks

improve RC activation with contralateral UE use and ips hand squeezing activities

POLICED

19
Q

shorter immobilization periods favor what with dislocation

A

muscle integrity
proprioception
peripheral and central neural activity
dynamic stability

20
Q

MET focus for dislocation

A

stabilization
tissue integrity and proliferation

for anterior dislocation ER, FLX, and ABD are initially contraindicated so do isometrics/isotonics into opposite directions

21
Q

prognosis for shoulder dislocation

A

recurrent dislocations are highly likely of over 30 years old

remember healing of all potentially involved tissue

22
Q

MD Rx for dislocation

A

arthroscopic or open procedure

typically 3-6 month prognosis

Full ROM under anesthesia

follow protocols

23
Q

what is a coracoid transfer

A

reposition of coracoid process and coracobrahcialis and short biceps head to GH neck

24
Q

what is a capsular shift

A

aka capsuloraphy

most commonly done

overlap of torn portions of capsule folds

25
prevalence and etiology of proximal humeral fx
common in elderly FOOSH involves surgical humeral neck
26
complications present with proximal humeral fracture
axillary artery damage = cold/blanche; EMERGENCY; possibly avascular necrosis adhesive capsulitis from prolonged immobilization
27
etiology of clavicular fracture
compression mechanism thru long axis of clavicle
28
location of clavicular fx
weak spot at S curve
28
location of clavicular fx
weak spot at S curve
29
complications with clavicular fx
large displacement may require sx epiphyseal plate injury as it is the last bone to ossify at 18-25 years of age
30
PT Rx for fx
start after clinical union between 4-8 weeks pain usually not from bone PT focus on consequences fo prolonged immobilization
31
what is proximal humeral apophysitis
aka little league shoulder rare most commonly in males/overhead throwers growth + high activity
32
pathomechanics of proximal humeral apophysitis
bone growth > RC lengthening increased tendon tension Growth plate = weak spot most often inflammation complications = avulsion and/or premature closure
33
symptoms of proximal humeral apophysitis
gradual onset of shoulder pain with overuse "pop" may indicate trauma/avulsion
34
signs of proximal humeral apophysitis
impingement like RST/MMT and lower ER:IR strength ratio in adolescent athletes with GIRD >1 + impingement test up to 30% with GIRD > 1 TTP over antero- and posterolateral aspects of proximal humerus (MOST COMMON SIGN)
35
PT Rx for Proximal humeral apophysitis cues
pt edu -soreness rul -load management -mvmt (i.e. throwing cues) POLICED normalize motion (i.e. improve GIRD ratio) careful with prolonged stretch return to play program
36
MET for proximal humeral apophysistis
cuff and also trial, scapular, and LE impairments caution with muscle/tendons attached to growth plate
37
prognosis for proximal humeral apophysitis
most return to preinjury levels and as early as 2 months but possibly up to 2-8 months about 4.5 months to return to competition with an avulsion growth plate typically closes between 16-20 years old can become a recurrent/persistent problem