Cervicothoracic Spine: Test 2 Flashcards

1
Q

What might happen if the there is hypo mobility in an area of the cervicothoracic spine that is left untreated? Give and example

A

stiff area may not be painful, and if it is not addressed it will typically cause painful hyper mobile compensations elsewhere

Example: stiff facet leads to hyper mobile adjacent facets or stiff upper thoracic regions lead to hyper mobile lower cervical spine

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2
Q

how can you obtain more uniform/distributed motion in the presence of hypo mobility

A

mobilize stiff areas

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3
Q

what determines the direction and amount of motion at the facet joints

A

orientation

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4
Q

describe the motion/location of the facet joints between C2-C7

A

they lie between the frontal and transverse planes

these facets favor all motion equally; this is not true for other spinal segments

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5
Q

describe the location/motion of the upper thoracic facet joints

A

mostly lie/move in the frontal plane

favors SB more (because the facets are more vertically oriented) BUT ribs limit the ROM of the SB

Thoracic vertebrae actually have more rotation than they do SB because of this even though their orientation would imply otherwise

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6
Q

describe the rotary motion of the upper cervical region

A

over 50% of neck rotation happens at the upper cervical area

mostly at the AA joint

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7
Q

how would you define stabilization

A

controlled mobility

more than just strength of superficial and big muscles

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8
Q

what are the 4 variables for stabilization

A

Joint integrity (i.e. cartilage)

Passive stiffness (i.e. ligaments)

Neural input (least often cause; usually not a conduction issue)

muscle function

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9
Q

describe local muscles

A

closer to axis of motion
often deeper
used more for stabilization than rotary forces
shunt muscles
tonic/postural
more aerobic than anaerobic (longer endurance)

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10
Q

describe global muscles

A

further away from the axis of motion
often superficial
used more for rotary forces than stabilization
spurt forces
phasic muscles (not always contracting)
more anaerobic than aerobic (less endurance capacity)

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11
Q

why are global muscle better overall joint movers

A

they have a longer lever arm that allows them to generate better/more propulsive forces

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12
Q

describe how local and global muscles interact with roles

A

both muscle types can contribute to both stabilization and movement

Local muscles tend to ensure stabilization first, and global muscles are primarily movers

if stabilizing muscles are inefficient then global muscles tend to act as stabilizers; since their makeup is not as conducive to that there can be pain, stiffness, etc in those global muscles

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13
Q

what are common local muscles in the cervical region

A

longus Colli and other deep neck flexors

sub occipitals and splenius mm

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14
Q

what are the main local muscles of the thoracic region

A

rotatores and multifidus (the smaller these muscles are the higher injury rate)

pelvic floor and transversus abdominus (activation of these can increase the contraction of the multifidus)

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15
Q

describe the inhibitory effects of pain, swelling, joint laxity, and disuse

A

decreased/delayed motor activation/coordination (aka inhibition of local muscles)

inhibition is preferential to type I muscles (endurance/stabilizing)

this inhibition limits the amount of activating muscles which means that the ones that still are activating can tire much quicker

the more inhibition, the more atrophy present with those local muscles (door hinge examples; more damage close to the axis, the more continual damage will occur there)

ultimately more stress put on non-contractile structures

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16
Q

what are some of the changes that take place in the muscles as a result of swelling, joint laxity, and disuse causes

A

increased/inefficient motor activity of global muscles (too much global muscle movement)

decreased cervical proprioception (motion sense; more likely to get hurt again; less feedback = less injury prevention)

atrophy leads to fatty infiltration (harder to gain back strength later on)

fiber types shift towards type II (less able to do what the muscle was originally designed to do)

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17
Q

describe how muscle activity/activation is the crucial treatment for stabilization

A

muscle activation of 30% is sufficient tot keep stability and is suitable to improve muscular endurance so it doesn’t take a lot

normal muscle activity does not just return after pain is gone from instability; must retrain

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18
Q

what is pain phenotyping

A

set of observable pain characteristics of an individual resulting from body and environment interaction

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19
Q

what are the 3 types of pain phenotyping

A

nociceptive
neuropathic
nociplastic

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20
Q

describe nociceptive pain and the subtypes

A

non-nervous tissue compromise

MSK pain (including spondylogenic)
viscerogenic (getting pain from organ dysfunction)

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21
Q

describe neuropathic pain and the subtypes

A

nervous system compromise

radicular, radiculopathy, and peripheral

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22
Q

describe nociplastic pain

A

qaltered pain perception without complete evidence of actual or treated tissue compromise

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23
Q

what is spondylogenic pain and some key characteristics

A

pain from the spine

common
can be local and/or referred spinal pain from noxious stimulation of spine structures
cannot cause visceral dysfunction as some providers claim (unless it is a spinal nerve itself; facet/bone/joint itself cannot)

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24
Q

symptoms of spondylogenic pain

A

non-segmental pain: means its not from a spinal nerve itself
rarely if any paraesthesias
vague, deep, achey, boring pain
referred into ill defined area that settles into a consistent location

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25
Q

what is segmental pain

A

pain distribution from spinal nerve in a dermatomal pattern

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26
Q

what are signs of spondylogenic pain

A

neuro scan WNL (because there is no spinal nerve involvement)
can’t reproduce entire symptoms pattern with motion

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27
Q

describe what somatic convergence means in reference to spondylogenic pain

A

aka referred pain; sensory afferents converge on and share the same innervation

this means that some joints in the neck are also innervated by nerves from some neck muscles

there is greater referral to proximal and deep structures rather than distal and superficial (i.e. the more midline/centered the joint is the more likely it will cause referred pain)

i.e. spinal facet joints refer pain more often than the elbow joint

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28
Q

describe viscerosomatic convergence

A

viscera and somatic (body) sensory afferents CONVERGE on and SHARE the same innervation

for example approximately C4-T4 there is a shared innervation of the heart and some neck muscles; thus heart issues can cause pain in the left shoulder, UE, neck, and jaw (all shared innervation)

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29
Q

signs and symptoms of viscerogenic pain

A

not typically able to be mechanically reproduced

neuro scan WNL

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30
Q

what is radicular pain

A

ectopic or abnormal discharge from highly inflammed spinal nerve (dorsal root)

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31
Q

symptoms of radicular pain

A

lancing
electrical shock like pain along an extremity in a narrow 2-3” band

neural test would possibly be normal because its new/acute/inflamed and hasn’t been around long enough to lose conduction

32
Q

signs of radicular pain (nDTR/myotome/derm, dural mobility)

A

dermatomes, DTRs, and myotomes are likely WNL: may be difficult to localize segment if acute/time because ti takes time for hypoactivity to show

positive dural mobility tests due to high inflammation

NOT common

imaging helpful for the involved spinal nerve

33
Q

what is radiculopathy

A

more persistent blocked conduction of spinal nerve due to compression or inflammation

34
Q

symptoms of radiculopathy

A

segmental paresthesias (dermatomal pattern w/ possible loss of DTR); constant or long duration; slow progression to ill defined area due to the dermatomal overlap

possible weakness (80% conduction loss needed before there is perceivable fatiguing weakness); thus off there is a positive myotome test there is a significant conduction deficit

35
Q

signs of radiculopathy

A

neuro scan positive for segmental hypoactivity

imaging helpful for involved spinal nerve

36
Q

describe peripheral nerve pain

A

decreased conduction of nerve branch (i.e. median nerve with carpal tunnel)

37
Q

symptoms of peripheral nerve pain

A

non-segmental paresthesias: intermittent/short duration; fast progression to well-defined area of numbness because of minimal overlap of peripheral nerves (unlike spinal nerves)

possible weakness

38
Q

signs of a peripheral nerve issue

A

dermatomes, DTRs, and myotomes WNL

non segmental hypoactivity; decreased sensation along peripheral n distribution; possible weakness of muscles innervated by peripheral nerve

positive dural mobility tests

39
Q

describe the relationship between the terms sensitization and nociplastic pain

A

sensitization was the initial term

sensitization is an underlying mechanism of nociplastic pain; symptoms of sensitization are present within nociplastic pain

patients with sensitization are labeled as having nociplastic pain

sensitization falls under the umbrella of nociplastic pain

40
Q

define nociplastic pain

A

altered pain perception without complete evidence of actual or threatened tissue compromise

41
Q

pathogenesis of nociplastic pain

A

thinning of myelin sheath in spots and the thinning causes the peripheral nociceptors (a-delta and C fibers that carry pain) to be extra sensitive and misinterpret the pain signals as more painful than it actually is

increased sensitivity and misinterpretation by central structures as well (doesn’t take as much stimulus for to fire)

loss of descending antinociceptive mechanisms (less hormone/opiate release in body to control the pain naturally)

42
Q

describe the somatic convergence portion of the pathogenesis of nociplastic pain

A

C fibers (carry pain) travel and split to at least 2 spinal segments superiorly and inferiorly

thus there are many areas of shared innervation and pain can begin to spread all throughout multiple segments of innervation

domino effect

43
Q

describe how somatic convergence can affect the homunculus

A

the brain perceives the pain as coming from even more areas with persistent symptoms

44
Q

what are some conditions associated with nociplastic pain prevalence

A

prevalence = growing

persistent fatigue syndrome
fibromyalgia
LBP
age related joint pain
lateral elbow pain
shoulder pain
migraines
neck pain: both traumatic and not traumatic

45
Q

S&S/criteria for “possible” nociplastic pain

A

more than 3 months of pain

regional or spreading symptoms

pain that can’t be entirely explained by nociceptive or neuropathic pathways

pain hypersensitivity or allogynia (non painful stimuli causing pain)

46
Q

S&S for “probable” nociplastic pain

A

**addition of any of these comorbidities along with the “possible” criteria

sensitive to light, sound, and or odor

sleep disturbances

fatigue

cognitive problems

47
Q

autonomic nervous system indicated S&S (x7)

A

pitting edema with lymph compromise
decreased sebaceous gland and hair follicle activity
sweaty hands/feet
decreased peripheral artery shunting = cold/clammy
loss of laterality (can’t differentiate sides)
decreased erector pili muscle (goose bumps)
positive graphesthesia (can’t differentiate drawn letters/numbers)

48
Q

why might joint mobilizations be a good rx for nociplastic pain

A

theoretical benefits on symptoms

most accepted = stimulates descending inhibitory pain mechanisms (i.e. more endorphins)
induces presynaptic inhibition (limit pain transmission/better overriding of P! stimuli)
reduced dorsal horn excitability
decreased inflammatory mediators

**basically flip all the signs and symptoms/work in the opposite direction

49
Q

how might MET be a good rx for nociceptive pain

A

low to moderate intensity of global aerobicc and resistance exercises
2-3x/wk
30-90 min/session

at least 7 week duration
helps patient interpret pain/motion as non-threatening
reorganizes homunculus
endogenous/opiate analgesia

50
Q

how might neuroscience education/behavioral therapy help nociplastic pain

A

not just mind over matter

explain increase sensitivity/misinterpretation to reduce stress/anxiety of misperceived tissue injury

challenges patients reasoning of fears and pain and helps ensure exercise safety

transition to adaptive pain coping

51
Q

prognosis of nociplastic pain

A

varying degrees of improvement
longer recovers
likely not a full resolution of symptoms

52
Q

describe the prevalence of cervical neck pain

A

70% people will experience neck pain
1/3 will have it for more than 6 months
almost 1/2 will be persistent (think nociplastic)

2nd only to LBP in workers comp costs

more common in older and in women

53
Q

how much of spine pathology is related to the thoracic region

A

15%

54
Q

strongest risk factors for neck pain

A

history of neck pain and being a female

55
Q

what is neck pain typically classified as

A

mechanical neck disorder (MND) or nerve root compromise

56
Q

functional ROM for neck

A

full (40-50 degree) extension to look up

60-70 degrees needed to rotate while driving (don’t need the full 80-90 functionally)

57
Q

S&S of neck pain

A

varied in cervical spine and possible into the upper extremity

impaired scapular mechanics

58
Q

how well does imaging contribute for neck pain conditions dx

A

often fails to identify related structures

often asymptomatic MRI findings in the neck (i.e. bulging/herniated discs, annular tears, or cord compression); don’t necessarily mean they are the cause for the original pain

59
Q

what structures are involved with neck pain

A

often varied and unknown

most patients don’t have a pathoanatomical cause or known tissue producing symptoms

60
Q

how well do tests and measures help diagnose neck pain

A

nearly all clinical tests are poor screening tools and/or lack string diagnostic accuracy measures necessary for clinical decision making

61
Q

how might a cervical JM help neck pain

A

modulate neck muscle function

increase deep muscle recruitment and reduce recruitment of superficial muscles

more effective and greater cost savings vs PCM or non manual Rx

62
Q

what are the criteria that are indicators of cervical manipulation success

A

neck disability index <11.5

bilateral involvement

sedentary work less than 5 hours a day

feeling better with movement

ext doesn’t increase symptoms

OA without radiculopathy

63
Q

what are the MOST RECENT predictors of success for cervical manipulation

A

symptoms for more than 38 days

expectation that manipulation will help

greater than 10 degree difference in rotation

pain with PA springs

64
Q

criteria for success for thoracic manipulation

A

symptoms for more than 30 days
no symptoms distal to shoulder
ext doesn’t increase symptoms
fear avoidance beliefs questionnaire PA scale less than 12
diminished T3-5 kyphosis (flatter back between shoulder blades)
cervical extension less than 30 degrees

65
Q

when is met most effective for neck pain

A

with acute and persistent neck pain

additive benefit to manual therapy

66
Q

if neck pain is nociplastic, what criteria should MET follow

A

motor control and exercises for stabilization
30-60 minute sessions
2-3x/wk
7-12 wks

67
Q

what other regions of the body should be included in MET when treating neck pain

A

cervical, thoracic, scapular, and shoulder regions

work on stabilization and strength/endurance parameters

68
Q

describe how you would prescribe MET for local and global muscle training with neck pain

A

low load endurance (6 weeks)

include:
isotonic and isometric forward nodding
isometric cervical rotation
isotonic and isometric scapular exercises

69
Q

how might you incorporate local muscle training for neck muscles into functional activities

A

maintain forward head nod during balance training and with external loads

integration with function

70
Q

describe proprioceptive training for neck pain

A

eye fixation without head movement

71
Q

describe how continual exercise/MET even after therapy can affect the outcome for neck pain patients

A

those who did it 3x/wk for a year were still good at 3 years

8-12 visits along led to benefits that lasted out to 12 months

2x/wk for 6 wks led to benefits that lessened by 6 months

targeting the issue with exercise for at least 6 weeks for lasting effects, but even better results would occur from longer

72
Q

how beneficial is stretching for neck pain

A

not beneficial in isolation

needs to be combined with MET

73
Q

describe the benefits of mechanical traction

A

no support for static Tx

intermittent is not beneficial in isolation; some support for short term radiculopathy symptoms… more beneficial following CPRs or with other added interventions

74
Q

describe the support for modalities in research

A

current evidence is lacking, limited ,or conflicting and NOT recommended

75
Q

describe the benefits of education/counseling (for neck pain)

A

early movement without provocation

reassures of good prognosis and full recovery in most cases