Shock Flashcards
What is shock
Inadequate circulating oxygen to meet metabolic demands
Imbalance between cellular oxygen supply and demand
Lead to Tissue hypoxia
Initiation phase of shock
Subclinical Hypoperfusion. There is inadequate DELIVERY
of O2 to the cells
Compensatory stage
Initiation of compensatory mechanisms
to maintain blood flow to vital organs
Progressive stage what happens
Compensatory measures begin to fail; vasoconstriction and shunting of blood, peripheral ischemia, lactic acidosis, electrolyte imbalances, respiratory acidosis
Refractory stage
Irreversible; loss of aerobic metabolism,
inefficient anaerobic metabolism, multisystem failure
What happens in Neural compensation
Baroreceptors in carotid sinus and aortic bodies sensitive to
changes in pressure
• Stimulation of SNS
• Release of catecholamines – Increased heart rate – Increased contractility – Systemic vasoconstriction \+Increased BP \+Redistribution of blood flow
What happens in Endocrine compensation
RAAS
Stimulation of anterior pituitary gland->
Adrenal cortex
• Glucocorticoids
– Increase circulating glucose levels
• Mineralocorticoid (Aldosterone)
– Increase circulating volume
►Stimulation of posterior pituitary gland
• ADH
What happens in Chemical compensation
Chemoreceptors in carotid and aortic bodies react to low oxygen tension • Respiratory rate and depth increase – Increased oxygenation • Respiratory alkalosis occurs – Constriction of carotid arteries
What happens to the body in the progressive stage
Compensatory mechanisms not reversing shock
• Systemic vasoconstriction/shunting of blood to vital organs
– Peripheral ischemia
• Failure of the sodium potassium pump
– Electrolyte Imbalances
• Metabolic/Respiratory acidosis
What happens to the body in the refractory stage
►Prolonged hypoperfusion ►Loss of aerobic metabolism ►Cell death ►Multiple organ dysfunction ►Death
Causes of Hypovolemic shock
External loss -Hemorrhage – Trauma/GI bleeds • Excessive urination • Burns • GI tract – Vomiting – Diarrhea
Internal loss
• Internal hemorrhage
– Fractures
– Ruptured aneurysms
Treatment for hypovolemic shock
Fluid resuscitation
Hypovolemic shock
CVP/PAOP/PAWP
CO
SVR
dec dec inc
Cardiogenic shock
CVP/PAOP/PAWP
CO
SVR
Inc dec inc
Obstructive shock causes for Impaired ventricular filling
Tension pneumothorax
Cardiac tamponade
Obstructive shock causes for Impaired ventricular emptying
• Increased Pulmonary Vascular Resistance (PVR)
– MASSIVE pulmonary embolism
• Increased Systemic Vascular Resistance (SVR)
– Severe valvular disease
Clinical manifestations of Obstructive shock
Decreased cardiac output/impaired peripheral perfusion: • Decreased level of consciousness • Hypotension • Tachycardia • Tachypnea • Decreased urine output • Weak pulses, cold, cyanotic, and mottled skin ►Chest pain, SOB, N/V ►Muffled heart sounds ►R heart failure • JVD
Obstructive shock
CVP/PAOP/PAWP
CO
SVR
Inc/dec
dec
inc
How do you treat obstructive shock
O2 management Definitive treatment: • Tension pneumothorax – Needle decompression • Cardiac tamponade - Pericardiocentesis MASSIVE pulmonary embolism – – Anticoagulants/Thrombolytics-tPA – CDT thrombolysis – Suction thrombectomy – Surgery- Pulmonary embolectomy
If obstructive shock is suspected, how would you assess
►Neurological status ►Vital signs/Respiratory status • SpO2/ABGs ►Hemodynamic parameters • CO/Filling pressures/ScvO2 /SvO2 ►Urine output ►Skin color and temperature
If obstructive shock what are your nursing actions
►Apply a 100% non-rebreather oxygen mask
►Prepare for intubation and mechanical ventilation
►Administer medications as ordered
• Vasoactive medications
• Anticoagulation via heparin administration
• Thrombolytic therapy
prep for definitive treatment
Distributive shock definition
A physiological problem that causes massive
vasodilation and poor vascular tone (decreased SVR,
increased vascular capacity, venous pooling) that creates a
RELATIVE hypovolemia.