Shock 2 Flashcards

1
Q

stages of shock

A

1) initial
2) compensatory
3) intermediate
4) irreversible

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2
Q

describe the initial stage of shock

A
  • first stage
  • slight decrease in MAP <10
  • flow to vital organs is maintained
  • lose 1 L of fluid, body can compensate
  • little cold and clammy
  • vascular constriction and increase in HR to maintain C.O.
  • increase in RR and diastolic BP d/t shunting blood
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3
Q

why does diastolic BP initially increase

A

more blood is going to the heart so diastolic increases about 10

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4
Q

describe compensatory stage of shock

A
  • nonprogressive
  • MAP decreases 10-15
  • kidneys and hormones activated (renin, angiotensisn, aldosterone to conserves salt with conserves H2O and increases BP)
  • HR is still elevated
  • acidodic and hyperkalemia
  • can be reversed if stabilized
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5
Q

why does acidosis and hyperkalemia occur in compensatory

A
  • build up of lactic acid (kidneys NOT functioning)
  • RR attempting to blow off but cannot
  • the more acidodic, less Na pump can work, which increases K
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6
Q

when K reaches ____, heart stops working

A

8

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7
Q

describe intermediate stage of shock

A
  • progressive
  • decrease in MAP >20
  • less vital organs become anoxic
  • ischemia occurs
  • life threatening
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8
Q

if nothing is done within how long of intermediate, pt with die

A

1 hr

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9
Q

what to do for pt in intermediate

A

1) start 2 large IV lines (1 L of NSS)
2) give oxygen (8-10 L high flow rate) *protect airway
3) put them on monitor (EKG)
4) put foley in them

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10
Q

why is NSS hung

A

can hand blood with NSS
any drug is compatible with NSS
eventually give O negative blood

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11
Q

describe irreversible stage of shock

A

refractory

  • too much cell death and tissue damage has occurred
  • therapy is not effective, eve in MAP returns to normal
  • nothing can be done to fix/prevent death
  • warm pt up to see if dead, check for brain wave activity, check ECHO to see if heart is moving
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12
Q

how does the process of MODS occur

A

1) injury
2) release toxins into plasma/bloodstream to fix area of damage
3) cytokines to stop bleeding in area and identify infection (cells fight infection and cause dilation)
4) vasodilation and decreased BP
5) kidney liver lungs and brain
6) shock
7) MODS/death

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13
Q

how to differentiate if MODS or shock

A

if renal affected, MODS

if decrease in MAP or BP, shock

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14
Q

broadly describe hypovolemic shock

A
  • too little circulation blood volume
  • decrease in MAP and O2
  • *low volume
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15
Q

what to do for hypovolemic shock

A

1) 2 large IV with saline
2) oxygen (8-10 L)
3) blood if necessary
4) EKG monitor
5) foley
6) draw ABGs, labs
7) protect airway
8) start line (EJ or IJ)

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16
Q

what does flail chest indicate

A

multiple ribs broken

lungs and chest collapse (need chest tube)

17
Q

describe cardiogenic shock

A
  • heart and muscle is unhealthy and pump is impaired
  • heart not working
  • MI can cause this
18
Q

describe CABG

A

coronary artery bypass surgery

  • very common in pts >65
  • blockage is removed and the pt own venous or arterial blood vessel is used (internal mammary* artery or saphenous vein)
  • vessels must typically be occluded >70%
  • ejection fraction is >40% for effectiveness
19
Q

how long do we want OHS to last

A

< 4 hrs

longer the surgery, greater the risk for clots

20
Q

what is the end product of CABG

A

take blood from aorta directly to coronary arteries

21
Q

describe the steps in the CABG procedure

A

1) heart is stopped with K solution and then grafting begins
2) heart is rewarmed and observed for patency
3) epicardial wires are placed and attached to an external pacemaker
4) mediastinal chest tubes placed (usually 4)

22
Q

what do you tell pt ahead of open heart surgery

A
  • it will hurt after surgery
  • chest tube (medial stinal)
  • may come back intubated (depends on stability)
  • get them UP asap after surgery as long as vital are stable
  • recovery time is ICU for about 2 days
23
Q

why is OHS performed

A

CABG, valve repair, tumors, cardiogenic shock requiring revascularization, VAD placement