Shock Flashcards
Differentiating Cardiogenic Shock
● History of cardiac abnormalities
● Physical exam findings: Enlarged liver, gallop, murmur.
● Chest X ray: Enlarged heart, pulmonary edema
Cardiogenic Shock
n Etiology:
Etiology: – – – – – – – Dysrhythmias Infection (myocarditis) Metabolic Obstructive Drug intoxication Congenital heart disease Trauma
Warm Shock
Warm Shock
• Early compensated hyperdynamic state of septic shock • Warm extremities, bounding pulses, tachycardia, wide pulse
pressure, decreased systemic vascular resistance and increased cardiac output
• Often with hyperglycemia
Cold Shock
Late uncompensated stage of septic shock with drop in cardiac
output
• Cold and clammy skin, rapid thready pulses, shallow breathing • Associated metabolic acidosis, hypoxia, coagulopathy,
hypoglycemia, capillary leak
Septic Shock
Fluid boluses. ( 20 cc/kg) up to 60cc/kg • Correct hypoglycemia • Administer 1st dose of antibiotics • Consider vasopressor drip and stress dose
hydrocortisone
• IF NOT FLUID RESPONSIVE Normotensive-Start Dopamine Hypotensive vasodilated(warm shock)- Norepinephrine Hypotensive vasoconstricted(cold shock)- Epinephrine
Shock Definition
Shock Definition: is a life-threatening state that occurs when oxygen
and nutrient delivery are insufficient to meet tissue metabolic demands
Sequelae of shock
Sequelae of shock are:
1- Metabolic acidosis,
2- Organ dysfunction
3- Death
Oxygen Delivery
Oxygen Delivery
Oxygen delivery=CO X Arterial oxygen content • CO=Heart rate X Stroke volume • Stroke volume depends on preload, afterload and contractility
Clinical Presentation
Early diagnosis requires a high index of suspicion
Diagnosis is made through the physical examination focused on tissue perfusion
• Hypotension is a late sign
Physical Exam Findings
• Neurological: Fluctuating mental status, sunken
fontanel
• Skin and extremities: Cool, pallor, mottling, cyanosis,
poor cap refill, weak pulses, poor muscle tone.
• Cardio-pulmonary:Tachypnea tachycardia. • Renal: reduce urine output
Directed History
● Past medical history – Heart disease – Surgeries – Steroid use – Past medical Medical problems ● Brief history of present illnes – Onset and progression of symptoms – Exposure n Laboratory studies: – – – – – – – ABG Blood sugar Electrolytes CBC PT/PTT Type and cross Cultures
Mangment كلها
Management-Volume Expansion
• Optimize preload • Normal saline (NS) or lactated ringer’s (RL) • Except for myocardial failure use 10-ml/kg
every 15 minutes. Reasses after every bolus.
• At 60ml/kg consider: ongoing losses, adrenal
insufficiency, intestinal ischemia, obstructive
shock. Get CXR. May need inotropes.
Hypovolemic Shock
Etiology: Hemorrhage, renal and/or GI fluid
losses, capillary leak syndromes
n Clinically, history of vomiting/diarrhea or
trauma/blood loss n Signs of dehydration: dry mucous
membranes, absent tears, decreased skin
turgor n Hypotension, tachycardia without signs of
congestive heart failure
Hemorrhagic Shock
Hemorrhagic Shock Management • Normal Saline 20 cc/kg over 15 min • Repeat up to 60 cc/kg • If due to bleeding: Packed Red blood cells 15 cc/kg
Bilateral rales
Bilateral rales= pulmonary edema