Session 9 Flashcards

1
Q

What are the intrinsic muscles of the tongue?

A
  • 4 paired muscles
  • Lie entirely within tongue
  • Run longitudinally, vertically and transversely
  • Blend with extrinsic muscles
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2
Q

What is the innervation of the intrinsic muscles of the tongue?

A
  • Hypoglossal nerve
  • Motor innervation
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3
Q

What are the extrinsic muscles of the tongue?

A
  • Genioglossus
  • Hyoglossus
  • Styloglossus
  • Palatoglossus
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4
Q

What is the innervation of the extrinsic muscles of the tongue?

A
  • Motor innervation from hypoglossal nerve
  • Innervates genioglossus, hyoglossus, styloglossus
  • Palatoglossus innervated by Vagus nerve
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5
Q

What is the action of the extrinsic muscles of the tongue?

A
  • Protrusion (genioglossus, used to test hypoglossal nerve)
  • Protraction
  • Retraction
  • Side-to-side move
  • Anchor tongue
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6
Q

Outline the sensory innervation of the tongue

A
  • Anterior 2/3 receives sensation from lingual nerve (trigeminal nerve) and taste from chorda tympani branch of facial nerve
  • Posterior 1/3 receives sensation and taste from glossopharyngeal nerve
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7
Q

What are the salivary glands that secrete saliva into the oral cavity?

A
  • Parotid glands
  • Submandibular glands
  • Sublingual glands
  • Secrete saliva under influence of autonomic nervous system
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8
Q

Which duct is associated with the submandibular glands?

A
  • Wharton duct
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9
Q

Which duct is associated with the parotid glands?

A
  • Stensen duct
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10
Q

Outline the properties of the sublingual glands?

A
  • Produce 3-5% of saliva
  • Smallest and most diffuse of the salivary glands
  • 8-20 excretory ducts per gland
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11
Q

Outline Sialolithiasis

A
  • Salivary gland stones
  • Most stones located in submandibular glands
  • Due to dehydration or reduced salivary flow
  • Most stones <1cm
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12
Q

What are the symptoms of sialolithiasis?

A
  • Pain in gland
  • Swelling
  • Can fluctuate in realtion to eating
  • Infection can occur due to stasis
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13
Q

How is sialolithiasis diagnosed?

A
  • History
  • X-ray
  • Sialogram
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14
Q

What are the signs and symptoms of tonsilitis?

A
  • Fever
  • Sore throat
  • Pain/difficulty swallowing
  • Cervical lymph nodes (jugulodigastric)
  • Bad breath
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15
Q

What causes tonsilitis?

A
  • Viral causes most common
  • Bacterial causes due to strep pyogenes
  • Causes inflammation of palatine tonsils
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16
Q

Where are the palatine tonsils located?

A
  • Between palatoglossal arch and palatopharyngeal arch
  • Uvula central
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17
Q

What are the causes of peritonsillar abscesses?

A
  • Follow on from untreated or partially treated tonsillitis
  • Or can arise on their own due to aerobic or anaerobic bacteria
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18
Q

What are the symptoms of peritonsillar abscesses?

A
  • Severe throat pain
  • Bad breath
  • Drooling
  • Difficulty opening mouth
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19
Q

What are the boundaries of the nasopharynx?

A
  • Base of skull to upper border of soft palate
  • Posterior border is C1 and C2
  • Anterior border is nasal cavity
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20
Q

What is found within the nasopharynx?

A
  • Orifice of eustachian tube
  • Pharyngeal tonsil
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21
Q

Where is the pharynx located?

A
  • Base of skull
  • Extends to C6
  • Forms part of digestive tract
  • Superior part lies posterior to nasal and oral cavities
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22
Q

What are the boundaries of the oropharynx?

A
  • Soft palate to epiglottis
  • Anterior border is oral cavity
  • Posterior border is C2 and C3
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23
Q

What is found within the oropharynx?

A
  • Palatine tonsils found between palatopharyngeal arch and palatoglossal arch
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24
Q

What are the boundaries of the laryngopharynx?

A
  • Oropharynx to oesophagus
  • Epiglottis to cricoid cartilage
  • Anterior border is larynx
  • Posterior border is C4, C5, C6
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25
Q

What are the muscles of the pharynx?

A
  • 3x longitudinal muscles
  • Stylopharyngeus
  • Palatopharyngeus
  • Salpingopharyngeus
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26
Q

What is the action of the pharyngeal muscles?

A
  • Elevate pharynx and larynx during swallowing
  • This widens aperture for food to go down oesophagus
  • Reduces distance food has to travel to enter oesophagus
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27
Q

Outline the origin and insertion for Stylopharyngeus

A
  • Styloid process - posterior border of thyroid cartilage
  • Glossopharyngeal nerve
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28
Q

Outline the origin and insertion for palatopharyngeus

A
  • Hard palate - posterior border to thyroid cartilage
  • Pharyngeal branch of Vagus nerve
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29
Q

Outline the origin and insertion for salpingopharyngeus

A
  • Cartilaginous part of Eustachian tube
  • Merges with palatopharyngeus
  • Pharyngeal branch of vagus nerve
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30
Q

What are the pharyngeal constrictors?

A
  • 3x circular muscles
  • Superior pharyngeal constrictor
  • Middle pharyngeal constrictor
  • Inferior pharyngeal constrictor (thyropharyngeal and cricopharyngeal)
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31
Q

What is the origin and insertion of the superior pharyngeal constrictor?

A
  • Origin is pterygomandibular raphe
  • Vagus nerve
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32
Q

What is the origin and insertion of the middle pharyngeal constrictor?

A
  • Origin is hyoid bone
  • Vagus nerve
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33
Q

What is the origin and insertion of the inferior pharyngeal constrictor?

A
  • Thyropharyngeal originates at thyroid cartilage
  • Cricopharyngeal originates ay cricoid cartilage
  • Vagus nerve
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34
Q

What is a pharyngeal pouch?

A
  • A posteromedial (false) diverticulum
  • Arises in weakness between 2 parts of inferior constrictor
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35
Q

What causes pharyngeal pouch?

A
  • Failure of upper oesophageal sphincter to relax
  • Abnormal timing of swallowing
  • Higher pressure in laryngopharynx
  • Weakness in inferior constrictor muscle produces outpouching
  • Symptoms related to food material collecting in pouch or disruption of swallow
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36
Q

What are the symptoms of pharyngeal pouch?

A
  • Bad breath
  • Regurgitation of food
  • Occasional choking on fluids
  • General difficulty swallowing
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37
Q

Where is the pharyngeal plexus located?

A
  • Mainly on surface of middle constrictor muscle
  • Vagus, glossopharyngeal and cervical sympathetic nerves
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38
Q

What is the motor innervation of the pharyngeal plexus?

A
  • CN X innervates all muscles
  • Except stylopharyngeus (CN IX)
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39
Q

What is the sensory innervation of the pharyngeal plexus?

A
  • Nasopharynx (maxillary nerve CN Vb)
  • Oropharynx (glossopharyngeal nerve CN IX)
  • Laryngopharynx (vagus nerve CN X)
40
Q

Outline the oral stage of swallowing

A
  • Voluntary
  • Preparatory phase makes bolus
  • Transit phase - bolus compressed against palate and pushed into oropharynx by tongue and soft palate
  • Hypoglossal nerve
41
Q

Outline the pharyngeal stage of swallowing

A
  • Involuntary
  • Tongue positioned against hard palate so food cannot re-enter mouth (CNXII)
  • Soft palate elevated sealing off nasopharynx (CN Vc and CN X)
  • Eustachian tube opened
  • Suprahyoid and longitudinal muscles shorten - pharynx widens and shortens to receive bolus
  • Larynx elevated and sealed off by vocal cords
41
Q

Outline the pharyngeal stage of swallowing

A
  • Involuntary
  • Tongue positioned against hard palate so food cannot re-enter mouth
  • Soft palate elevated sealing off nasopharynx
  • Eustachian tube opened
  • Suprahyoid and longitudinal muscles shorten - pharynx widens and shortens to receive bolus
  • Larynx elevated and sealed off by vocal cords
  • Epiglottis closes over larynx
  • Bolus moves through pharynx by contraction of constrictors
  • Relaxation of UOS
42
Q

Outline the oesophageal stage of swallowing

A
  • Involuntary
  • Upper striated muscle of oesophagus
  • Lower smooth muscle
43
Q

What might cause dysphagia?

A
  • Stroke
  • Oesophageal cancer
  • ## Blockage
44
Q

What are the signs and symptoms of dysphagia?

A
  • Coughing and choking
  • Sialorrhoea (drooling)
  • Recurrent pneumonia
  • Change in voice/speech (wet voice)
  • Nasal regurgitation
45
Q

What cranial nerve problems can affect the pharynx?

A
  • IX, X
  • Absent gag
  • Uvula deviated away from lesion
  • Dysphagia
  • Taste impairment
  • Loss of sensation in oropharynx
  • Caused by medullary infarct, jugular foramen issue
46
Q

What are the functions of the nasal cavity?

A
  • Olfaction
  • Filtering and humidifying inspired air
  • Drainage of secretions from paranasal sinuses and nasolacrimal ducts
47
Q

Outline the structure of the external nose

A
  • Predominantly cartilaginous
  • Frontal processes of maxillae and nasal bones form root
48
Q

What can injuries to the nose cause?

A
  • Septal haematomas
  • Septal deviations
  • Nasal bone fractures
49
Q

Outline the structure of the nasal cavity?

A
  • Roof
  • Lateral wall
  • Medial wall
  • Floor
  • Nostrils anteriorly
  • Chonae posteriorly
50
Q

What are the regions of the nasal cavity?

A
  • Vestibule
  • Respiratory region
  • Olfactory region
51
Q

What are the bones that contribute to the nasal cavity?

A
  • Roof: frontal bone, ethmoid bone, sphenoid bone, nasal bones
  • Floor: palatine bone, maxilla
52
Q

What is important clinically about the nasal vestibule?

A
  • Route through which surgeons operate on pituitary gland
53
Q

What is the function of the concha?

A
  • Slow down air flow
  • Increase surface area
  • For warming and humidification
54
Q

What are the meatuses?

A
  • Superior, middle, inferior
  • Formed from lateral wall of nasal cavity
  • Drainage
  • Connection with paranasal air sinuses and nasolacrimal duct
55
Q

What is a septal haematoma?

A
  • Trauma to cartilage causes the perichondrium to be stripped off
  • Can lead to avascular necrosis
  • Saddle nose deformity
56
Q

How common are nasal bone fractures?

A
  • Account for 50% of all facial fractures
57
Q

How does a nasal bone fracture present?

A
  • Lots of swelling seen
  • Commonly see epistaxis
58
Q

How do we treat nasal bone fracture?

A
  • X-rays not generally required
  • Follow up in several days in outpatient clinic once swelling has settled
59
Q

What can nasal bone fracture result in?

A
  • Rare complications include CSF leak and anosmia
  • Can lead to a deviated septum and blocked nose
60
Q

What is the blood supply to the nasal cavity like?

A
  • Rich blood supply
  • Anterior and posterior ethmoidal arteries (branches of ophthalmic artery) supply medial wall
  • Sphenopalatine artery and greater petrosal artery (branches of maxillary artery) supply lateral wall
61
Q

Why does the nose need such a rich blood supply?

A
  • Allows it to perform two of its functions:
    1. Humidification
    2. Warming incoming air
62
Q

Which blood vessel is the ophthalmic artery a branch of?

A
  • Internal carotid artery
63
Q

Which blood vessel is the maxillary artery a branch off?

A
  • External carotid artery
64
Q

What is the nervous supply to the nasal cavity?

A
  • Trigeminal nerve - ophthalmic branch and maxillary branch
65
Q

What are nasal polyps?

A
  • Benign swellings of the nasal mucosa
66
Q

What is the epidemiology of nasal polyps?

A
  • Typically seen in those >40 years old
  • Affects more males than females
  • Normally bilateral
67
Q

What are the symptoms of nasal polyps?

A
  • Nasal congestions
  • Rhinorrhoea
  • Hypo- or anosmia
  • Snoring
  • Post nasal drip
68
Q

What are the symptoms of rhinitis?

A
  • Nasal congestion
  • Rhinorrhoea
  • Sneezing
  • Post-nasal drip
  • Nasal irritation
69
Q

What are the paranasal air sinuses?

A
  • Air filled spaces that are extensions of the nasal cavity
  • Lined with respiratory mucosa (ciliated and secrete mucus)
70
Q

What can infection in the nasal cavity lead to?

A
  • Sinusitis
  • Maxillary sinus most commonly affected
71
Q

What are the various functions of the paranasal air sinuses?

A
  • Humidify and warn inspired air
  • Reduce weight of the skull
72
Q

Where do the paranasal air sinuses drain to?

A
  • Drain into the nasal cavity via small channels (ostia) into a meatus
73
Q

What does the middle meatus drain?

A
  • Frontal sinus
  • Maxillary sinus
  • Anterior ethmoid sinus
74
Q

What does the middle meatus drain?

A
  • Frontal sinus
  • Maxillary sinus
  • Anterior ethmoid sinus
75
Q

What does the inferior meatus drain?

A
  • Nasolacrimal duct
76
Q

What does the superior meatus drain?

A
  • Sphenoid sinus
77
Q

What is the general sensory innervation of the sinuses?

A
  • Va supplies frontal, ethmoidal and sphenoid sinuses
  • Vb supplies the maxillary sinuses
78
Q

What usually causes sinusitis?

A
  • Viral upper respiratory tract infection
  • Spreads to sinuses
79
Q

What is the pathophysiology of sinusitis?

A
  • Inflammation of respiratory mucosa causes swelling, reduced cilia movement and increased secretions
  • Ostia can become blocked
  • Fluid builds up in sinus
  • Can develop a secondary bacterial infection
80
Q

How is sinusitis diagnosed?

A
  • Recent URTI
  • Blocked nose and rhinorrhoea +/- green/yellow discharge
  • Pyrexia
  • Headache/facial pain (in area of affected sinus) - worse on leaning forward
81
Q

How is sinusitis managed?

A
  • Conservatively
82
Q

What makes acute bacterial sinusitis more likely?

A
  • Symptoms particularly severe at onset
  • Symptoms >10 days without improvement but < 4 weeks
  • Symptoms that worsen after an initial improvement (suggesting secondary bacterial infection)
83
Q

Which organisms cause bacterial sinusitis?

A
  • Streptococcus pneumonia
  • Haemophilus influenzae
  • Moraxella catarrhalis are most common bacteria
84
Q

How can most nosebleeds be stopped?

A
  • Pinching in front of (not on) the bony bridge of the nose and holding it for 15-20 minutes
85
Q

Which vessels coalesce at Kiesselbach’s area?

A
  • Greater palatine
  • Sphenopalatine
  • Superior labial arteries
  • Anterior and posterior ethmoidal arteries
86
Q

What is the site of origin of most nosebleeds?

A
  • The cartilaginous part of the septum known as Little’s area
87
Q

Why can bleeds from the sphenopalatine artery be particularly problematic?

A
  • Blood in this vessel tends to be at higher pressure
  • And as it is posteriorly located in the nasal cavity, it is harder to reach to stop the bleed
88
Q

What can cause nosebleeds?

A
  • Spontaneous
  • Or with very minor trauma to the nose
  • May be due to underlying systemic causes e.g. abnormal coagulation and connective tissue disorders
89
Q

Who is most commonly affected by nosebleeds?

A
  • Very young people (2-10 years old)
  • Old people (50-60 years old)
90
Q

Why are serious nosebleeds so dangerous?

A
  • Significant blood loss
  • Rarely can cause death
91
Q

How is epistaxis managed?

A
  • Applying simple compression
  • Leaning forward
  • If these steps fail, attempts should be made to cauterise a visible bleeding point using silver nitrate
92
Q

What do we do if bleeding is significant or the bleeding point cannot be identified?

A
  • May mean that cauterisation is difficult
  • Anterior packing with nasal tampons can be used instead
  • These packs expand within the nasal cavity and tamponade the area of bleeding
93
Q

What do we do if bleeding is significant or the bleeding point cannot be identified?

A
  • May mean that cauterisation is difficult
  • Anterior packing with nasal tampons can be used instead
  • These packs expand within the nasal cavity and tamponade the area of bleeding
94
Q

What do we do if anterior packing of the nasal cavity fails?

A
  • Posterior packing
  • Surgical intervention as a last resort e.g. embolization, ligation of blood vessels
95
Q

What needs to be monitored in severe epistaxis?

A
  • ABCDE approach
  • Closely monitor patient
  • Blood tests to check Hb levels and clotting
  • Any underlying systemic causes for the bleeding should be sought and treated e.g. coagulopathies