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Father of Microbiology
Virchow
It is a mechanism or sequence. progressive development of a disease process from the time it is initiated to its conclusion in recovery or death.
Pathogenesis
It is most commonly seen as a replacement from a specialized tissue type to a less specialized one but more resistant cell type, e.g., from columnar or transitional epithelia to squamous epithelia
Metaplasia
An increase in organ size or tissue mass caused by an increase in the number of constituent cells.
Hyperplasia
Organs are increased in size due to an increase in cell size without cellular proliferation
Hypertrophy
Excess of fluid in the interstitial tissue/serous cavities.
Edema
Leucocytes begin to appear in the marginal plasma stream of the venule.
Margination
It may occur accumulation of excessive amounts of collagen giving rise to a protruding tumor-like scar tissue.
Keloid
An inflammation in vein.
Phlebitis
Secretes histamine
Basophil
Presence of parasites
Eosinophils
Monocyte in tissue
Macrophage
Circulating form of macrophage (blood)
Monocyte
It is sometimes called exudative inflammation because of the numerous tissue and plasma factors that pours into the inflammatory site.
Acute inflammation
Proliferative inflammation
Chronic Inflammatory
It attract the leukocytes to migrate to the injured site are chemical mediators of inflammation.
Chemotaxis
It is a substance that can combine with a specific antibody but lacks antigenicity of its own to evaluate the properties of specific epitopes and antibodies.
Haptens
Complete atrophy
Involution
If there is less workload. Limb kept in a cast. Due to inactivity it results to reduction in size of the organ.
Disuse atrophy
It is the most commonly reported genetic defects in domestic animal species.
Autosomal Recessive inheritance