Respiratory - Lecture 4 Flashcards

1
Q

What are the 4 common presentations for respiratory problems?

A

wheezing
coughing
hemoptysis
dyspnoea - HVS

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2
Q

4 things that can cause dyspnoea

A

Increased ventilatory demand - exertion
Decreased ventilatory capacity - pleural effusion, pneumothrax
Increased airway resistance - asthma
Decreased pulmonary compliance - intersitial fibrosis

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3
Q
Pneumothorax....compared to pul. embol.  on other side 
MUST KNOW
occurs spontaneusly
abrupt sharp chest pain & tachypnea
diminished breath sounds 
hyperresonance
A
Pulmonary embolism
MUST KNOW
occurs spontaneously, diff risk factors
abrupt sharp chest pain & tachypnea
tachycardia
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4
Q

Common asthma symptoms

A

wheezing
chest tightness
coughing

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5
Q

Hyperventilation syndrome - acute, 6 things

A
severe dyspnoea
agitation
fear
chest pain
paraesthesia
pre syncope/syncope
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6
Q

Hyperventialtion syndrome - chronic, 4 things

A

increased sighing
hard to detect
associated with depression
musculoskeletal change

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7
Q
Pneumonia....compared to COPD on other side
MUST KNOW
acute cough
look unwell
fever
productive cough
occasionally pleuritic
chest pain
areas of consolidation on lung
A
COPD
MUST KNOW
acute cough
high effort on breathing
accessory muscle use
cough, may or may not be productive
wheezing
areas of hyperresonance
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8
Q

Chronic dyspnoea - think of lungs & heart

A
Restrictive lung disease - 
pneumonia
TB
rib fracture
scoliosis

Obstructive lung disease -
COPD
chronic bronchitis - haemoptysis, fever, wheezing
asthma

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9
Q

Cough red flags

A

dyspnoea
haemoptysis
weight loss
risk factors for TB & HIV

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10
Q

Chronic bronchitis symptoms

A
haemoptysis
a productive cough on most days
dyspnoea
fever
wheezing - note: asthma is just wheezing, no other sym.
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11
Q

Upper respiratory tract infection & ace inhibitors both produce what symptoms

A

cough that is dry & non productive

can last for weeks or months

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12
Q

Haemoptysis - 3 sources & how much before it is considered a red flag?

A

True - from the lungs, bright red
Pseudo - from the nasopharynx
haematemesis - stomach/GI, coffee coloured

more than 600 mls in 24 hours…..red flag!

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13
Q

Chronic Obstructive Pulmonary Disease (COPD)

MUST KNOW

A

Epi - 3rd highest cause of death
Aet - smoking, genetic factors, child resp. disorders, pollutants
Pathogenesis - airflow limitation by airway obstruction, respiratory infection or inhalation exposure
Signs & Symptoms - cough, with or without mucus
fatigue, shortness of breath, wheezing, increased expiration, ,barrell chest, accessory muscle use

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14
Q

Asthma

MUST KNOW

A

Chronic inflammatory disease, narrowing airways and increased mucus production.
Epi - 1 in 6 adults, 1 in 4 NZ children
Aet - environmental allergens, resp. tract infections, NSAID sensitivity, GERD, emotional factors
agg: -Exercise (cold/dry air), -Occupation (air irritant)
-Allergy (pets, pollen)
Patho - airway inflamm, intermittent airflow obstruction, bronchial hyperresponsiveness
Signs/symp - Dyspnea (SOB), wheezing W exhale, coughing, chest tighness, tachypnea, tachycardia, access. muscle use, prolonged exp phase breathing.
-nocturnal SOB, cough, wheeze
Tx: brown inhaler: Blue inhaler: Bronchodilator to relieve inhaler symptoms breathless, wheezy or tight-chested.
Brown:Prevention steroid inhaler

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15
Q

Bronchitis

MUST KNOW

A

Epi - males more than females, one of top 5 reasons for a doctor visit
Aet - Acute: viral 90% & bacterial infections Chronic: smoking, air pollutants
Patho - acute: inflammation airway= narrowing- bronchial lining irritated, mucous membrane becomes swollen- air passages become clogged/irritated & lots of muscous forms, hence classic bronchial cough
chronic - Irreversible damage narrowing airway + obstruction- smoking impairs cilliary movement, inhibits function of alveolar, leads to hypertrophy & hyperplasia
Signs/symptoms - Dry cough turns to productive cough W sputum, chest pain, sore/runny nose, wheezy, finger clubbing, HA, extreme fatigue
Chronic: productive cough, wheeze, HA, Cyanosis

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16
Q

Emphysema

A

Classified as COPD
Abnormal permanent enlargement of air spaces distal to the terminal bronchioles, accompanied by the destruction of alveolar walls and without obvious fibrosis.
Aet: Smoking, air pollutants
Path: Aveoli collapse, chest space incase but decrease fusion of O2
S+S: yrs W out symp. SOB increases and difficulty exhaling. decrease in activities until SOB occurs at rest