Respiratory Flashcards

1
Q

ASTHMA patho, cause, s/s, nursing management

A

patho: inflammation of airway -> obstruction -> decreased gas exchange

Cause: genetic, environmental (pollution, allergens)

s/s: wheezing, fatigue, DOB/SOB, anxiety, tight chest, cough (moist, rattling). cyanosis

nursing management:
- Assess respiratory status
- Administer charted oxygen or nebuliser
- Position patient for max. vent and airway patency
- Administer meds (steroids, bronchodilators)
- Suction airway if needed
- Comfort measures
- Reassure child and family

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2
Q

ASTHMA prevention strategies/ improving outcomes

A
  1. Smoke exposure
  2. Housing
  3. Income
  4. Health literacy
  5. Adherence
  6. Asthma action plan
  7. Access
  8. Ambulance
  9. Influenza vaccine
  10. Reletionships
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3
Q

ASTHMA medication

A
  1. Short-acting beta agonist (SABA)
    - all children with asthma
    - bronchodilators; quick relief of symptoms

a. salbutamol
b. terbutaline

  1. Inhaled corticosteroids (ICS)
    - suppress airway inflammation by activation or repression of target genes involved in inflammatory process

a. Beclomethasone diproprionate- low 200; Std 400-500
b. Beclomethasone dipropionate ultrafine- low 100; std 200
c. Budesonide- low 200; std 400
d. Fluticasone propionate- low 100; std 200-500

  1. Montelokast
    -CysLT1 receptor antagonist and acts to reduce bronchoconstriction and mucous secretion in response to allergens.
    - for poorly controlled LABA/ICS
    - dosing: 4mg under 5yo and 5mg over 5yo.
  2. Long-acting beta agonists + inhaled corticosteroids (LABA/ICS)
    - maintenance with SABA as reliever 5-11yo.
    - contraindicated: under 4yo
  3. Anti-inflammatory reliever therapy (AIRs)
    - using budesonide/formoterol as monotherapy or with regular budesonide/formeterol.
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4
Q

PNEUMONIA

A

DEFINE:
- inflammation and build up of fluid in the lower respiratory tract affecting alveoli

CAUSES: viral, bacterial, fungal

DIAGNOSTIC:
- Bronchiocospy
- Chest xray
- sputum culture

S/S:
- lung sound (crackles)
- SOB
- increased WOB
- fever
- coughing
- loss of appetite
- lethargy

TREATMENT:
- meds: antiviral, antibiotic, antifungal

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5
Q

CYSTIC FIBROSIS

A

define: an autosomal recessive gene disorder that damages your lungs, digestive tract and other organs.

effect on organs:
1. intestine
Mucus block duct tube transporting enzymes out of pancreas to small intestine => decreased food digestion, nutrient absorption; difficulty gaining weight and malnutrition. Also decreased insulin production and inflammation of pancreas

  1. Mucus blocks bile duct => scar tissue and damage to liver => poor liver function
  2. Bacteria thrives in airways => repeated bacterial chest infections

s/s: repeated chest infection

management:
NO CURE BUT
1. devices, massage and techniques to move mucus and unblock airways (e.g. nebuliser)
2. antibiotics
3. anti-inflammatory meds (e.g. prednisone)
4. meds for facilitation of digestion and increase vitamin absorption
5. vitamin supplements
6. chest physio/chest percussion

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6
Q

TENSION PNEUMOTHORAX

A

Define: a life-threatening emergency wherein a large air collection in the pleural space compromises respiration and cardiac function

Patho:
=> air enters pleural space
=> tension pneumothorax: air cant leave
=> pressure on lung, trachea, heart and other structures

Causes:
A. Spontaneous/simple: without underlying lung disease (primary) or with underlying lung disease (secondary)
B. Traumatic

s/s:
- SOB
- Acute chest pain
- Low BP
- Low SPO2
- High HR

Treatment: needle decompression, fowler, oxygen, 3-sided semi-occlusion dx

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7
Q

TUBERCULOSIS treatment, tests, issues

A

Tuberculosis is an infectious disease caused by the bacterium Mycobacterium tuberculosis, primarily affecting the lungs but can spread to other organs.

test:
- tuberculin skin test
- TB blood test

s/s:
- a cough lasting 3 weeks or more
- coughing up thick mucus or blood
- tiredness
- night sweats
- loss of appetite
- weight loss
- swollen glands, usually in your neck
- chest pain.

complication:
- meningitis
- major bleeding (haemorrhage) from the lungs or other organs
- widespread tuberculosis
nodules (disseminated tuberculosis)
death.
- renal, joint infection, pericarditis

treatment: antibiotics (combination) for 6 months

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8
Q

Oxygenation- masks, use etc

A
  1. Nasal cannula:
    - 1-6L/min.
    - 24-40%
  2. simple face mask:
    - 5-10L/min
    - 40-60%
  3. partial rebreather mask:
    - 6-10L/min
    - 60-80%
  4. non-breather mask:
    - 10-15L/min
    - up to 95%
  5. venturi mask:
    - interchangeable adapters to adjust flow
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9
Q

COPD

A

Define:
Umbrella term for complications associated decreases airflow in the lungs

Two types: Emphysema and bronchitis

Causes:
- Smoking
- Exposure to environment irritants
- Decrease in or anti-trypsin (emphysema)

S+S: coughing (productive), SOBUE, wheezing, tight chest, accessory muscle breathing, pursed lips, barrel chest, cyanosis, nostril flaring

Diagnosis
* Symptoms
* History of exposure to irritants
* Lung function test (spirometry- measures speed and airflow)

Treatments:
* Stop smoking and/or improve air quality
* Medication:
○ Bronchodilators: beta 2 agonists (salbutamol), anticholinergics (ipratropium), theophylline
○ Steroids
* Vaccination: flu, pneumonia
* Oxygen therapy
* Pulmonary rehabilitation
* Surgery: bullectomy, lung reduction, transplant

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10
Q

COPD- EMPHYSEMA

A

Definition: inflammation in alveoli.

=> Breakdown of alveolar walls and elastin leading
=> enlargement of the alveolar air spaces
=> hyperinflation and decreased gas exchange

Cause of Chronic inflammation in alveoli:
1. Increased oxidative stress
2. Increased proteases
= tissue breakdown (alveolar walls pulm. capillary walls, elastic fibres)

Enlarged air spaces result to:
1. Air retention -> hyperinflation -> affects on thoracic cavity and changes in lung volumes (increased lung capacity and residual volumes) -> increased work of breathing
2. Decreased Alveolar surface area -> decreased gas exchange -> decreased O2 in tissue (hypoxia) and increased CO2 in tissue (hypercapnia) -> acidosis and pulmonary hypertension -> respiratory failure

s/s:
- barrel chest
- use of accessory muscles
- cyanosis (from metabolic acidosis)
- respiratory acidosis
- risk of infection

management:
1. beta antagonist
- anticholinergic
- corticosteroids
- oxygen therapy

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11
Q

COPD- BRONCHITIS

A

Definition: inflammation in bronchioles/bronchi

Complication:
- Bronchial swelling/oedema
- Increased mucous production
- Impaired cilia function

narrowed airway + thick mucus
= increase airway resistance
= reduced ventilation
= increased infection susceptibility

S/S:
- productive cough for >3/12 months in 2 years

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