Resp Flashcards

1
Q

General age related changes

A
Decreased elasticity - cant take a deep breathe 
Increased rigidity 
Weaker thoracic muscles - weak cough 
Decreased # of alveoli 
Decreased cilla
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2
Q

Acute bronchitis

A

Viral , self limiting
Inflammation of lower resp track

S/s: cough ( with or without flem) cough can be dry or wet and develop throughout the days
- fevers, chills, wheezes or rhonchi that clear with cough, pain from cough

Organisms : influenza A or B, parainfluenza, coronavirus, rhinovirus

Tx: symptom control, bronchodilators ( albuterol ) 2 buffs Q 4 hours up to 12 puffs a day, dextromethorphan ( cough medication) , tyenlol for fever , increase humidity , follow up in 3-4 days if not getting better
AVOID antihistamines and mucolytics

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3
Q

Pertussis

A

Incidence related to non immunized people
- must get 3 vaccines

Progresses from cold symptoms to severe cough “ whooping sounds”

Look for this if there has been a community outbreak , report to DPH

Bacteria : bacterium bordetella pertussis

S/s: violent coughing, sob, after coughing taking a deep breathe ( whooping)
Dx: nasal swap
Tx: Azithromycin 500 mg day 1 then 250 mg day 2-5

Note: pt is infected from beginning of symptoms through the 3rd week or until days 5 after effective antimicrobial treatment

Prevention : Tdap ( pregnant women should get booster in 27th-36th week)

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4
Q

Pleurisy

A

Inflammation of the pleura due to infection

S/s : fever, trouble taking a full breath, may have EENT or other resp symptoms, GI upset

Tx: pain medication and symptomatic relief

It causes pleuritic pain

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5
Q

Pleuritic pain

A

Can be caused by: pneumonia , embolism ,MI, pneumothorax , TB, auto immune, GI

Sharp pain, stabbing burning when breathing

Rule out : cardiac problems

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6
Q

Pulmonary embolism

A

Blockage of the large pulmonary artery —> often due to a break of a DVT

S/s: may have none or will have CP, SOB coughing blood, DVT symptoms

Tx: anticoagulation for 3-6 months

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7
Q

CAP community acquired pneumonia

A

An acute infection of the lower resp tract occurring a in person living in the community and has not hospitalized in the past two weeks

Elderly may present with , alt mental status, falls, poor PO intake

Other s/s: high fever, sob, fatigue, aches, headache, hypotension , high hr

Consider recent hx of pneumonia and recent antibiotics use in the last 3 months

PE: crackles that do not clear with a cough

  • dull to percussion
  • egophany ( increased resonance sounds indicating consolidation )
  • bronchopany
  • whispered perctorloquy ( increased loudness
  • increased tactile fremitus ( normal finding is decreased fremitus as you go down )

Organism: Strep. Pneumonia ( sudden onset) , h. Influenza, mycoplasma ( slow onset )

TX: cough suppressants, albuterol, expectorants ( medication to bring flem up)

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8
Q

CAP treatment

A
Cough suppressants 
Short course abulterol
 Antibiotics:
 - uncomplicated 
        - Azithromycin
- allergy to macrolide 
      - doxycycline 
- complicated 
       - amoxicillin or Augmentin + azithroymycin 
- complicated allergy 
     - fluoroquinolones or ( azithrymcyin + augmentin )
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9
Q

COPD

A

Irreversible obstructive airway disease
Progressive decline in lung function

It can be chronic bronchitis or emphysema

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10
Q

Chronic Bronchitis

A

Persistent cough production for 3 consecutive months over two years with periodic exacerbations

Imbalance between ventilation and perfusion leading to hypoxemia and hypercarbia “ blue bloaters”
May lead to pulmonary hypertension

Cough, wheezes and course crackles

Polycythemia is common

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11
Q

Emphysema

A

Permanent and abnormal enlargement of any part of the airspace’s distal to the terminal bronchioles

Hyperinflanted increases A/P diameter 
SOB is more common than cough, 
Marked expiratory respiratory phase is prolonged 
PO2 is maintained = Pink puffers
Weight loss noted 

Hyperressont to percussion , breathe sounds are distant

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12
Q

COPD risk factors and PE

A

Risk factors : smoking, occupational hazards, outdoor air pollution

PE:

  • prolonged expiratory phase
  • wheezes on forces expiration
  • decreased rib cage movement
  • increased movement fo abdominals
  • forward sitting ( late stage)
  • increase resonance on percussion
  • early Inspiratory crackles
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13
Q

COPD dx criteria

A

CXR, CT CHEST, SPIROMETRY ( gold standard), EKG , CBC

Presence of symptoms

  • sob on exertion or rest
  • cough with or w/o sputum
  • progressively activity intolerance
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14
Q

Spirometry

A

Pulmonary function tests:

  • measures Forced vital capacity (FVC)
    - max volume of air that can be exhaled during a forced maneuver
  • Forced expiration volume in 1 second (FEV1)
    - how quickly can the lung be emptied

Ratio is then calculated and results are expressed as % of predicted BELOW .70 = COPD

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15
Q

COPD PLAN : staging

A

GOLD + Catergories

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16
Q

Category A ( GOLD 1-2)

A

Minimally symptomatic with low risk of exacerbations
ratio < .70
FEV1 >80

TX: SABA PRN alone or in combo with a short acting anticholinergic , flu vaccine
Example : albuterol + ipratropium

17
Q

category B ( GOLD 1-2)

A

More symptomatic but low risk for exacerbation ( moderate)
ratio < .70
50 % of FEV1 < 80 %

SABA PRN for rescues + LABA ( or long acting anticholinergic )

Example : salmeterol + albuterol (tiotropium)

18
Q

Category C ( Gold 3-4)

A

Minimally symptomatic but high risk for exacerbation ( severe)
Ratio < .70
30 % FEV1 < 50 %

SABA PRN + LAMA (Ellipta) ( if poor control can use with combination with LABA) also can alternate with inhaled glucorticosteriods ( Flovent)

19
Q

Category D ( GOLD 3-4)

A

Very severe
Ratio : < .70

SABA PRN + LABA or LAMA + INhaled gluctocoid steriods + long term oxygen

20
Q

Acute bacterial exacerbations of COPD

A

Fevers, increased sputum
Bacteria: s. Pneumonia *** , h.flu ** turn into pneumonia , claymdia, M . Catarrhalis ( want to cover against gram negative )

Antibiotics :

  • amoxicillin, doxy, bacterium, azithromcyin, levequin BID x 10 days
  • augmenten if PCN allergy
21
Q

Asthma

A

Recurrent attacks of breathlessness and wheezing
Condition die to inflammation of the air passages in the lungs and it affects teh sensitivity of the nerve endings

S/s: cough at night that wakes you up, recurring wheezes, recurrent sob, recurrent Che’s tightness

Either : airway flow limitation / obstruction or airway hyper responsiveness

22
Q

Asthma attack

A

Lining of the bronchioles tubes swell and produce extra mucus
Airway narrows Redding airflow in and out of the lungs

23
Q

Dx asthma

A

In the last 12 months

- colds that take a long time to get over , reoccurring symtpoms     - waking up at night
24
Q

Asthma dx test

A

Peak flow = % predicted or % of person best

Green, yellow, red based on age, gender and race

Get CBC to see eosinophils

Rule out other things

25
Q

STEP 1

A

Intermittent asthma
FEV > 80 %
Day time symptoms less than 2 days a week , nighttime awakened less than 2 a month

SABA ( albuterol )

26
Q

Step 2

A

Mild persistent asthma
Day time > 2 days a week but not daily
Night time 3-4 a month

SABA + low dose ICS ( Flovent)

27
Q

STEP 3

A

Moderate persistent asthma
Night time more that one per week

SABA + (ICS + LABA ) come as one now example is symbicort

28
Q

Step 4

A

Severe persistent asthma
Symptoms throughout the day with nocturnal awakenings nightly

SABA + ( MEDICS+ LABA) Advair

29
Q

ICS in general

A

Cornerstone of asthma treatment
Use must be consistent , takes 1-3 week to fully work

Can get, hoarseness, sore throat , oral thrush

Low dose ICS ( Flovent, plumicort, Asmanex)

Combo for persistent moderate / severe =
- Advair, symbicort

Close monitoring got glaucoma and cataracts

30
Q

Asthma follow up

A

When starting mono-therapy - every 2-6 weeks

Regular follow up
1-6 months

Ask about last 4 week, any symptoms that dont improve 15 min after inhaling SABA

PFTS 1-2 years once stable