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Nursing Adult Health II > Resp. 1 (Exam 3) > Flashcards

Resp. 1 (Exam 3) Flashcards

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1
Q

What is the purpose of the respiratory system

A

exchange of carbon dioxide and oxygen between external environment and the blood.

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2
Q

Structures of the upper respiratory tract

A

nose, pharynx, adenoids, tonsils, epiglottis, larynx, trachea
(conducting airway)

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3
Q

Structures of the lower respiratory tract

A

Bronchi, alveoli, lungs, pleura, pleural cavity

conducting airways and gas exchange airway

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4
Q

What is tidal volume

A

total air volume inspired and expired during one breath cycle

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5
Q

normal tidal volume

A

500mL

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6
Q

What conditions may affect the tidal volume and why is this important

A

asthema, COPD leads to an insufficient air movement which causes hypoxia

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7
Q

What is the significance of the angle of louis? the carina?

A

The carina is highly sensitive and touching it during suctioning causes vigorous coughing.
- the carnia is the end of the upper respiratory tract

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8
Q

Ventilation

A
  • movement of air into and out of the lungs
  • involves inspiration expiration and the actual act of breathing
  • getting air where it needs to go
  • gas flows from an area of higher pressure to an area of lower pressure
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9
Q

What is the main control center for respiration

A

Brainstem Medulla

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10
Q

The 2 basic mechanisms for ventilation

A

Chemical and mechanical

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11
Q

Chemical mechanism

A

A change in respiratory rate and depth (tidal volume) is based on chemical changes in body fluids

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12
Q

Chemorecetors

A
  • receptors that communicate the changes in the main control center
  • there are central and peripheral chemoreceptors
  • a receptor that changes to the change in the chemical composition of the fluid around it
  • increase in H+ (acidosis) causes the medulla to increase resp. rate and tidal volume
  • decrease in H+ (alkalosis) opposite effect
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13
Q

Central chemo receptors are located

A

in the medulla

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14
Q

Peripheral chemo receptors are located

A

in the carotid bodies above and below the aortic arch

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15
Q

chemoreceptors respond mainly to

A

changes in pH, H ions, and CO2

peripheral receptors are secondarily respond to changes in O2

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16
Q

How chemoreceptors work

A
  • your body attempts to rid itself of excess CO2 and to inhale O2
  • as the CO2 elevates the body gets rid of it so respiration increase in rate and depth
  • CO2 converts to acid and H ions so the same thing happens when the pH drops or the H ions concentration increases
  • to the lesser degree as O2 levels decrease the body needs more oxygen so respirations may increase in rate and depth
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17
Q

Increased CO2, increase H, and Decrease pH (decrease O2) =

A

increased respiratory rate and tidal volume

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18
Q

Decease CO2, Decrease H, and Increase pH (Increase O2) =

A

decreased respiratory rate and tidal volume

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19
Q

Asouration is more likely to occur in which lung

A

Right lung

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20
Q

oxygen and carbon dioxide exchange takes place in

A

the respiratory bronchioles

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21
Q

Alveoli

A

are small sacs that are primary site of gas exchange in the lungs
- interconnected by pores of Kohn allow movement from alveoli to alveoli

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22
Q

Surfactant

A

is a lipoprotein that lowers the surface tension in the alveoli and decreases the tendency of the alveoli to collapse

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23
Q

How will a paper bag improve the patients respiratory status?

A

This will increase the carbon dioxide level in the bag. As you rebreath the air you just exhaled, the increased level of carbon dioxide in the air you’re taking in will increase the level of CO2 in your blood stream and restore calcium levels

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24
Q

Mechanical mechanisms

A

change respiration based on mechanical factors

- are located in the lungs upper airways chest walls and diaphragm

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25
Q

insipration

A

active

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26
Q

diaphragm

A

lowers and causes negative pressure to pull air from atmosphere

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27
Q

intercostal muscles

A

aid in expanding the chest

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28
Q

accessory mucles

A

patients who have respiratory problems or who are in distress may have to use the accessory muscles of the neck and shoulders

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29
Q

compliance

A

refers to the strechability of lungs

- if compliance decreases the lungs are not able ot expand

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30
Q

expiration

A
  • passive
  • stretch receptors sense that the lungs have stretched to the normal limits
  • Hering - breuer reflex is stimulated (this makes sure the lungs dont overinnflate)
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31
Q

Perfusion

A
  • blood flows through the pulmonary circulation
  • blood has to be circulated to the lungs and then to the tissues
  • Getting the blood where it needs to go
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32
Q

V/Q ratio

A

V- ventilation
Q- prefusion
- normal V/Q ratio = 1:1
- this represents a comparion of how much ventilation (air) is available in relationship to the perfusion (blood supply)
- You want them to match, when they dont it is called a V/Q mismatch
* hypventilation most common cause

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33
Q

Normal Ratio

A

the amount of blood equals amount of gas ratio = 1:1

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34
Q

2 different types of mismatches

A
  1. shunt

2. dead space

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35
Q

Shunt

A

this occurs when ventilation decreases ( blood has to go or shunt to somewhere else to get adequate air) occurs when blood exits the heart without having participated in gas exchange

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36
Q

anatomic shunt

A

occurs when blood passes through an anatomic channel in the heart and bypass lungs

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37
Q

intrapulmonary shunt

A

occurs when blood flows through the pulmonary capillaries without participating in gas exchange

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38
Q

dead space

A

this occurs when perfusion decreases ( the air is dead without the proper blood supply to hook up with)

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39
Q

Nursing care for V/Q mismatches can be placed in 3 main categories

A

treat the cause of the mismatch
oxygenation therapy
positioning

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40
Q

positioning

A

when decided on a position; remember that air travels up and blood travels down (laws of gravity)

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41
Q

oxygenation

A

the presence of of oxygen within the body. Process of making sure the air and blood are good (O2 supply available)

  • remember that you can have oxygenation even if it is not getting where it needs to go
  • ventilation, perfusion and oxygenation work together but they all have to play their own part, they work on 3 different terms
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42
Q

Diffusion

A

describles the process of actual gas exchange.
What happens when air and blood meet
- movement of gas from areas of higher pressure or concentration to areas of lower pressure or concentration

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43
Q

Where does diffusion take place

A

across the alveolar capillary membrane

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44
Q

Nose inspection

A

check naris are occluded, patency, inflammation, deformities , inspect turbinales for polyps

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45
Q

nose symmetry

A

look for spetial devation, or broken nose

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46
Q

nose patency

A

can they breath out of each nostril

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47
Q

nose mucosa

A

should be pink and moist

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48
Q

nose discharge

A

seasonal allergies clear, or cerebral spinal fluid, dry or yellow infections process, or brown due to old blood, Presence of purulent and maloclerous discharge could indicate the presence of a foreign body

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49
Q

Mouth

A

pale grey or blue, if there is a lack of oxygen check color and for lesions, masses, bleeding, poor dentition, check tongue for symmetry and lesions

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50
Q

pharynx

A

check pressing tongue blade to middle back of tongue should be smooth and moist, no exudate, ulcerations, swelling, nasal drip

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51
Q

tonsils

A

do they have tonsils do they have pus pockets are they swollen

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52
Q

neck

A

is it symmetrical are they using accessory muscles to breath or due they have masses
- note any enlargements gagging normal responses

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53
Q

tracheal position

A

should be midline

  • to test use your index fingers to see if its midline, but most of the time you will see it and patient will be in resp. distress
  • it likes to deviate away from attention pneumotherectomy
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54
Q

Chest symmetry

A

movement should be symmetrical

if ribs are broken they could be passing bad air

55
Q

Anteroposterior (A/P) diameter

A

should be twice as wide as deep

- copd patients often have the barrel chest due to hyperinflation of the lungs and they cant get the CO2

56
Q

Respirations normal rate

A

12-20 per min

57
Q

Respirations

A

rate depth, rhythm
depth are they breathing shallow and getting air into the base of their lungs , is their rhymth is it fast or slow or a different pattern are they inhaling faster than exhaling 2:1

58
Q

bradypnea

A

breaths less than 12 per min (sedation)

59
Q

tachypena

A

20+ breaths per min

60
Q

hypoventilation

A

decreased rate and depth of respirations

overdose

61
Q

hyperventilation

A

increases in rate and depth of respirations

62
Q

Kussmauls

A

rapid deep breathing because of too much acid

DKA, renal problems

63
Q

Agonal

A

end of life breathing long periods of apnea

64
Q

Cheyne Stokes

A

sometimes breath rapidly and then stop and breath rapidly again this is also associated with end of life

65
Q

Accessory muscles

A

abs, neck, shoulders, deep intercostals

  • use of accessory muscles is more of a late sign of hypoixa
  • should be using diaphragm and intercostals
66
Q

Nail beds

A

clubbing is when the angle of the nail flattens out, you will see this in COPD patients, may have delayed cap refill, this may be with peripheral cyanosis

67
Q

Auscultation

A

lung sounds

68
Q

normal sounds

A

bronchi, bronchovesicular, vesicular (clear lung sounds)

69
Q

Bronchial

A

high pitched and longer
loud sound
heard over main airways if you hear them in the lower lungs you may have fluid in the lower lung

70
Q

Bronchovesicular

A

medium pitched, dont last long

anterior on each side of the sternum and positeror between scapular

71
Q

Vesicular

A

high pitched
breezy
lower lobes

72
Q

Adventitous sounds

A

abnormal include crackles rhonci wheezes pleural friction rub

73
Q

Diminished

A

increased density or decreased air flow

- patients who arent taking deep breaths COPD patients

74
Q

Crackles

A

short pops high pitched increased fluid

- think fluid, either extracellular fluid leaking into the lungs or patient is receiving fluids

75
Q

Precaution when giving patients fluids

A

if you pump this patient with fluid too fast, check lung sounds and ejection fraction because this could cause them to go into acerbation of heart failure
- heart failure patients who need blood give them blood then Lasix then other bag of blood

76
Q

What type of patient would you hear crackles in?

A

patients with pulmonary edema, pneumonia, COPD patients

77
Q

What would you do if you heard crackles?

A

stop fluids if you have fluids running and compare to baseline

78
Q

Coarse crackles

A

more fluid in lungs

79
Q

Fine crackles

A

just a little fluid in lungs

80
Q

Where and when do you usually hear crackles?

A

on inspiration and in the base of the lungs

81
Q

Rhonchi

A

continuous running snoring sounds wet obstruction, think mucus
- heard on expiration

82
Q

What type of patient would you hear rhonchi in

A

COPD pneumonia cystic fibrosis

83
Q

What do you do if you hear rhonchi

A

cough deep breath incentive spirometer ambulate

84
Q

Pleural friction rub

A

bzzzzz with inspiration and expiration = inflamed pleura

- a lot of pain receptors in the pleaura it is painful to patient sounds like bee buzzing

85
Q

stridor

A

continuous crowning sound, major obstuction

they are struggling to get air

86
Q

palpation techniques

A

tactile fremitus, chest expansion , percussion

87
Q

tactile fremitus

A

chest wall vibration with vocalization of 99
- compare sides, should have some vibration
- may be increased fluid or decreased air
(air doesnt contract vibrations as well as fluid, COPD you should hear more sound, pneumonia and cystic fibrosis you will have more vibration)

88
Q

Chest expansion

A

place hands on the back with thumbs touching at midline
-with inhalation should separate approx 1 in
same indications as decreased symmetry

89
Q

Percussion

A

indicates combination of density and air

90
Q

resonance

A

normal combination of density and air

- low pitched sound

91
Q

hyperresonance or tympany

A

too much air

loud lower pitched sound

92
Q

dullness or flatness

A 
too much density
medium pitched (areas with mixed solid lung tissues)
93
Q

Patient history

A

get history if they have ever had resp. problems adn if so what is their history

94
Q

Dyspnea

A

is this new or previous rate on a scale of 1-10 ask them how its affecting their ability to take care of themselves can they eat without taking a break, if they get dyspnea on movement this can be a early sign of hypoxia

95
Q

cough and sputum

A

quality
frequency
character
productivity
effectiveness
acute vs chronic
does it have color oder and what time of day is it most productive
how much and how often do you cough, cough regularly or sudden and periodic
is this cough related to activity/talking
any changes over time
what efforts have been made to alleviate cough

96
Q

To get mucus to move

A

deep breath, cough, incentive spirometer, if your patient is horse is it prolonged or recent is it an obstruction/ tumor or something short term such as laryngitis

97
Q

chest pain

A

is your body telling you your not getting enough O2 do a differential diagnosis it is due to the coronary artery or due to the lungs not getting enough o2
- if it is lung involvment it is going to be sharp and localized and not going to breath and deep

98
Q

Sputum colors

A 
is it ever blood tinged?
normally - clear/ slightly whiteish
Cigarette- clear/ brown specks
COPD- clear white or yellow
- consistency? thick thin frothy
dehydration postnasal drip, pulmonary edema 
- no odor normal
- odor suggests infection
99
Q

hoarseness

A

irritation or obstructuion

- obstruction usually if it lasts longer than 2 weeks

100
Q

Chest Pain OLDCART

A 
onset
location
duration
charactristics
aggravating factors
radiation
treatment
101
Q

chest pain

A

need for differential diagnosis
usually sharp and more localized
results in decreased expansion
pleurisy- parietal pleura has the pain receptors

102
Q

Orthopnea

A

trouble breathing when laying flat
increase number of pillows
need to be sitting or standing to help with SOB
more cardiac and fluid status with gravity
- these patients usually get a fluid overload

103
Q

Paroxysmal noctural dyspnea (PND)

A 
awakened by shortness of breaht
usually respiratory in nature
Affects O2 exchange
can be related to heart failure
patient who ahs been standing up all day, and all the fluid in the circulation starts to move into the circulation and its an overload on the heart 
sleep in recliner or many pillows
104
Q

Wheezing

A

usually some sort of airway obstruction

with asthmea it can be muscus plugs

105
Q

Hemoptysis

A

when patient is coughing up blood

lung blood is bright red and frothy mixed with sputum

106
Q

Cyanosis

A

late sign of hypoxia or decreased cardiac output

  • pheripheral not as worry some
  • central - around lips or mouth major problem
  • bluish color comes from the hemoglobin if they have a low hemoglobin might turb blue
107
Q

pneumothorax

A

is an abnormal collection of air or gas in the pleural space that separates the lung from chest wall

108
Q

Oxygen transport and delivery to tissues

A

main goal= deliver O2 to tissues to be used

Oxyhemoglobin= O2 travels with hemoglobin Hgb is the carrier

109
Q

SpO2

A

saturation of peripheral oxygen (pulse ox will get you this #)
above 90 is great 95+ is better

110
Q

Pulse oximetry (SaO2)

A

percentage of saturation of Hgb with O2

- dont forget the importance of Hgb level this will affect your oxygenation level

111
Q

Partial pressure of oxygen (PaO2)

A

amount of O2 dissolved in plasma and available for diffusion to tissues

  • P02 80-100
  • thisis the amount of oxygen avaliable for gas exchange
112
Q

Oxyhemoglobin dissociation curve

A

represents the relationship between the SaO2 and PaO2

- oxygen should be just sticky enough to stay on the Hgb till its deliveed to the right place

113
Q

Alkalosis

A

decreased CO2 ,H ions, and temp; increased PH
- have greater difference between PaO2 and SpO2
shift to the left (not the right way to go)
“left loves”

114
Q

Acidosis

A

increased CO2, H Ions, and temp, decreased pH
- shift to the right ( if you have to go one way this is the right way to go)
Right releases
* COPD patients are usually in acidosis

115
Q

ABG

A

Are measured to determine oxygenation status and acid base balance

  • can be obtained by arterial puncture or from an arterial catheter
  • tells us about alveolar ventilation, oxygenation, and acid base balance
116
Q

Bicarbonate (HCO3)

A

carbonic acid (H2CO3) buffer system
-CO2 combines with H20
controlled by lungs and fluid status
lungs attempt to compensate quickly to acid/base changes

117
Q

increased CO2 (increased RR)

A

to blow off CO2

118
Q

Decreased CO2 (decreased RR)

A

slow deep breaths to retain CO2
- CO2 dissolved in H2O
Normal Co2 levels= 32-48
if they have a low level of CO2 (28) then we have less acid AKA an alklotic value
if they have high of CO2 (56) then we have a lot of acid

119
Q

Bicarbonate (base)

A 
excretion and reabsorption 
metabolic component of ABG's = HCO3
controlled by kidneys
ability to respond affected by renal funtion
often in exhcange for H ions
slower response to acid base changes
120
Q

allens test

A

prefored before using an artery to ensure adequate circulation to extremity
-occlude both the ulnar an radial arteries then release one

121
Q

peak flow meter

A

measures how well air moves out of the lungs
maximum outflow during forced expiration measured in liters per min
shows degree of airway obstruction
- often used in asthema therapy
at home used around 3x
- higher # better the control
- use the highest of the 3

122
Q

Pulmonary function tests

A

measure lung volumes and airflow

  • used in many obstructive disorders
  • its a spirmetery
  • helps determine treatment
  • show mechanical function of the lungs
  • measures volume or flow rate
  • evaluates degree of airflow obstruction
123
Q

Tidal volume (VT)

A

normal breath in and out

500 ml

124
Q

inspiratory reserve volume

A

forced inhalation beyond VT

125
Q

expiratory reserve volume

A

forced exhalation beyond VT

126
Q

Residual volume

A

amount air left in lungs after forced expiration

- common in COPD

127
Q

Vital capacity

A

total capacity of lungs

128
Q

Sputum collection

A 
sterile
1st morning specimen
extract with suctioning or bronchoscopy
- if there are bubbles in it its no good
- do oral care first
129
Q

Bronchoscopy

A

direct visualization of bronchi with fiberoptic scope
-lighted scope into lungs
- can be done surgical outpatient , ICU beside
consent must be given, moderate sudation, numb gag reflex, check gag, NPO 6-8 hrs before
- can be done laying down or seated
- biopsy, tissue samples, remove objects, used for treatments of removal of muscous plugs or foreign objects

130
Q

Thoracentesis

A

needle inserted into the pleural space to obtain specimen for diagnosis, removal pleural fluid instill medication

131
Q

Purpose of thorcentesis

A

we need to remove fluid from the pleura space in the lungs, what position should the patient be placed in so that all of the fluid may be removed

132
Q

pulmonary angiogram/angiography

A

assess pulmonary blood vessels

  • catheter inserted into groin or arm and threaded to right side of heart into pulmonary artery - dye injected
  • look for clots/ blockages
  • catheter is inserted into the groin or arm, what nursing assessments would be priority for this patient specific to the catheter placement
133
Q

VQ scan

A

find out if its perfusion scan an ventilation problem

  • inhale radioactive gas & radioactive isotope through IV
  • pulmonary embolism affect perfusion but not ventilation
134
Q

CT scan

A

diagnose lesions difficult to assessby conventional x-ray studies Helical or spiral CT
- can be done with or without contrast

Nursing Adult Health II (6 decks)

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