Renal Flashcards
When is surgery indicated for nephrolithiasis?
No passage after 4-6 w of medical management
Increased Cr (AKI)
Calcium stone size that indicates stone will pass spontaneously in 4-5 days
<4-5 mm (99%)
or
5-7 mm (60%) AND at the UVJ jxn (80%)
Complications seen in adult PCKD
MVP
Aortic Regurgitation
Intracranial aneurysms (with possible subarachnoid hemorrhage)
Difference in Hb and EPO in patients with: PCKD vs RCC
Polycystic kidney disease: anemia 2/2 low EPO
RCC: polycythemia 2/2 HIGH EPO
Type of AKI with following labs:
FENa <1%
Pre-renal (hypovolemia, sepsis, renal a. stenosis, drug toxicity)
FENa > 2% BUN/Cr < 20/1 Muddy brown casts Granular casts or renal tubule cells No blood Trace protein
Dx? Cause of AKI?
ATN (intrarenal AKI)
- ischemic (severe decline in RBF)
- nephrotoxic (ABX, radiocontrast, NSAIDs, rhabdo, chemo, MM light chains)
Struvite stones:
- cause?
- specific bacteria?
- radiopaque or radiolucent?
- urine pH will be….?
- how common are they?
- UTI 2/2…urease + bacteria
- Urease +: proteus and klebsiella
- Basic urine (pH) > 7.0
- 2nd most common
Urge incontinece
- Dx
- Tx
Clinical Dx (go go go 2/2 detrusor overactivity)
Tx: bladder training (timed voiding) x 3 months
then try: oxybutinin/tolteridine/trospium/solifenacin
(Anti-muscurinics)
Definitive diagnosis for urothelial carcinoma
Cystoscopy w/Bx
BPH Medication that gives rapid relief of symptoms
Alpha-1 Receptor Blockers
(Terazosin & Tamsulosin)
“Tera and Tammy Take away the Sx by relaxing the prostate.”
NSAIDs vs ACE-Is
Effect on kidneys
NSAIDs: afferent arteriolar vasoconstriction (inhibits Pg)
ACE-Is: efferent arteriolar vasodilation (blocks A-II)
Both causing ⬇️GFR
Equation for serum osmolality
Sosm= 2Na + BUN/2.8 + Glucose/18
Next step in evaluating hyponatemia: fluid status (hypo, euvolemic, hyper)