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Oral Pathology > Red and Blue Lesions > Flashcards

Red and Blue Lesions Flashcards

1
Q

Reasons that lesions appear red

A 
Dilation of blood vessels
Increase blood supply
Hemorrhage soft tissues
Thin epithelium
Epithelial erosion
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2
Q

Diascopy positive lesions

A

Hemangioma
Sturge-Weber syndrome
Hereditary hemorrhagic telamgiectasia
CREST syndrome

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3
Q

Diascopy negative lesions

A

Sub mucosal hemorrhage
Thrombocytopenia
Infectious mononucleosis

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4
Q

Another name for congenital hemangioma

A

Strawberry nevus

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5
Q

Causes hemangioma versus vascular malformation

A

He

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6
Q

What is the appearance of hemangioma of bone

A

Multi

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7
Q

Pathogenesis of hemangioma

A

abnormal endothelial cells

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8
Q

Pathogenesis of vascular formations

A

abnormal blood vessel development

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9
Q

T/F hemangioma has a bruit present with it

A

FALSE

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10
Q

T/F hemangiomas do not involute spontaneously

A

FALSE

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11
Q

Describe how hemangioma of bone would look

A

Multilocular, sunburst pattern
Delicate trabeculae
Root resorption
Cortical expansion

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12
Q

What areas of the body are affected by Sturge-Weber?

A

The brain, face, and intraorally

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13
Q

What nerve us affected by Sturge-Weber

A

Trigeminal nerve with facial lesions along the nerve

Usually UNILATERAL

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14
Q

Neurological effects of S-W Syndrome

A

Mental retardation, hemiparesis, and seizures

Intracranial calcification of leptomeningies

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15
Q

Intral oral lesions of S-W Syndrome

A

Very red

Ipsilateral oral mucosa involvement

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16
Q

Cause of Hereditary Hemorrhagic Telangiectasia (HHT)

A

Abnormal dilation of terminal vessels

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17
Q

Presentation of HHT

A

Intranasal EPITAXIS

Telangiectasa of vermillion, tongue, and buccal mucosa

18
Q

What are varix?

A

Abnormally dilated veins

19
Q

Where are varicosities multiple and solitary?

A

Multiple: ventral and lateral tongue
Solitary: vermilion and bucal mucosa

20
Q

Etiology of angina bullosa hemorrhagica

A

Trauma

Not immunologically related

21
Q

Pathogenesis of pyogenic granuloma

A

Reactive hyperplasia of vascularized granulation tissue

22
Q

Clinical presentation of pyogenic granuloma

A

Usually solitary, circumscribed red nodule

23
Q

Usual populations with Pyo. G

A

Women
During pregnancy
During hormonal changes (puburty)

24
Q

Locations of pyogenic granuloma

A

Gingiva
Tongue
Labial mucosa

25
Q

DD of PG

A

The “P’s”
Peripheral giant cell granuloma
Peripheral ossifying fibroma
Metestatic tumors

26
Q

Tx of PG

A

Remove the cause (local plaque or calculus)
Surgical excision
Occasionally lesions recur

27
Q

Cause of Periph. GCG

A

Trauma or irritation

28
Q

Location of Periph. GCG

A

Gingiva anterior to the first molar. Exclusively on the gingiva

29
Q

How are PyoG and Periph GCG differentiated

A

The presence of giant cells on PGCG

30
Q

How would an erythroplakia look intraorally and who would it be on?

A 
Well-defined macule or plaque on the floor of mouth, tongue or soft palate
Older men (50-70) usually have it
31
Q

DD of erythroplakia

A

Nonspecific mucositis
Candidiasis
Vascular lesion

32
Q

Histopathology of erythroplakia

A

40% will show sever dysplasia

50% will be SCC

33
Q

Tx of erythroplakia

A

Biopsy to confirm it.
Excise it.
Long term follow up

34
Q

Name one type of intravascular neoplasm

A

Kaposi’s sarcoma

35
Q

Tx of KS

A

Surgical excision, low-dose radiation

36
Q

DD of KS

A

Erythroplakia
Hemangioma
Melanoma
Pyogenic granuloma

37
Q

Type of Anemias

A

Plummer-Vinson syndrome

Pernicious anemia

38
Q

Plummer-Vinson syndrome cause

A

Iron deficiency

39
Q

Plummer-Vinson syndrome effects

A

Dysphagia
Erythema and papillary atrophy
Angular cheilitis
BALD TONGUE

40
Q

Perniciious anema cause

A

Intrinsic factor and Vitamin B12 deficiency

Oral Pathology (5 decks)

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