RD CVS formatted Flashcards
- A patient is due for a fistula in his left arm, best place for the vascath is
a. Left internal jugular vein
b. Right internal jugular vein
c. Left subclavian vein
d. Right subclavian vein
e. Left femoral vein
b. Right internal jugular vein
- Subclavian arterial thrombosis (LAS – thoracic outlet syndrome), young painter, pain. Which is not associated.
a. Dissection (Subclavian artery dissection)
b. Aneurysm (Subclavian artery aneurysm)
c. Stenosis (Subclavian artery stenosis)
d. Venous thrombosis (Subclavian vein thrombosis)
e. Arterial mural thrombus (Subclavian artery wall haematoma)
a. Dissection (Subclavian artery dissection)
2. Subclavian arterial thrombosis (LAS – thoracic outlet syndrome), young painter, pain. Which is not associated.
a. Dissection (Subclavian artery dissection)
b. Aneurysm (Subclavian artery aneurysm) T – SCA aneurysm
c. Stenosis (Subclavian artery stenosis) T – narrowing of SCA which is positional (abduction)
d. Venous thrombosis (Subclavian vein thrombosis) T – may be occlusive or non-occlusive
e. Arterial mural thrombus (Subclavian artery wall haematoma) T – mural thrombi
StatDx:
• TO consists of interscalene triangle, costoclavicular space, and retropectoralis minor space (subcoracoid tunnel)
o Compression of neural, arterial, or venous structures crossing these tunnels → TOS
o Narrowing of costoclavicular distance may be most important abnormality in symptomatic patients
• Neuropathic TOS: Symptomatology 2° to brachial plexus compression (most symptomatic patients)
o Up to 98% symptomatic patients have plexus compression; minority 2° to arterial or venous impingement
• Vascular TOS: Compression of subclavian vessels o Repetitive arterial trauma → focal stenosis, aneurysm formation, micro-embolization, tissue loss
o Venous compression → SCV thrombosis
- A 45 yo man with intermittent claudication on walking. Angiography showed 3cm long stenosis of less than 50% in the mid SFA. What is the most appropriate treatment:
a. Exercise
b. Angioplasty
c. Bypass graft
d. Stent-graft
a. Exercise Based on AHA/ACC guidelines for peripheral arterial disease 2006:
• Incomplete information – depends on impact on life of disease & comorbidities
• Try supervised claudication exercise therapy & pharmacotherapy first, as well as risk factor modificationTASC-II 2007 guidelines:
- Young man in MVA, CT shows smooth lobulated bulge anteromedial aortic arch near isthmus. No mediastinal blood or other injury. Most likely cause of appearances
a. Ductus diverticulum
b. Pseudo-aneurysm
c. Traumatic aneurysm
d. Acute dissection
e. Injury to the mediastinal veins
Ductus
- AV fistula shows stenosis of arterial anastomosis. Best option for long term patency?
a. Bare metal stent
b. Covered stent
c. Self-expanding stent
d. Angioplasty
d. Angioplasty T angioplasty if upper forearm or upper arm; surgery if lower forearm
- Feature of HOCM: a. Diastolic dysfunction
b. Aortic level stenosis
c. Interatrial septal thickening
d. Normal mitral valve
a. Diastolic dysfunction T abnormal LV stiffness → impaired LV relaxation → poor early diastolic filling
- Feature of HOCM:
a. Diastolic dysfunction T abnormal LV stiffness → impaired LV relaxation → poor early diastolic filling
b. Aortic level stenosis F subaortic stenosis (septal thickening + SAM)
c. Interatrial septal thickening F asymmetric form (most common) has interventricular septal thickening
- In ‘Power Doppler’ the intensity of colour denotes:
a. Angle of insonation
b. Velocity
c. Amplitude
d. Direction of flow.
c. Amplitude
- Back pain in a 50yo male most likely:
e. Aortic dissection
f. Penetrating atherosclerotic ulcer
g. Aortic aneurysm
e. Aortic dissection peak age 60 years, range 13-87 years (Dahnert)
- Back pain in a 50yo male most likely:
e. Aortic dissection peak age 60 years, range 13-87 years (Dahnert)
f. Penetrating atherosclerotic ulcer elderly with HTN/lipids/atheroma (Dahnert)
g. Aortic aneurysm AAA usually age > 60 years M»F (Dahnert)
colour doppler, coding depends on …. what??
Colour displayed within BVs on colour Doppler function of: • Flow velocity • Doppler angle • Presence of aliasing • Colour map utilized • Phase of cardiac cycle
guy with US with thick walled aneurysm with increased ESR. likely??
a. inflammatory aneurysm
b. mycotic aneurysm
inflammatory A = idiopathic inflammatory aortic aneurysm = dense perianeurysmal fibrosis & a thickened aortic wall; accounts for 5-25% of all AAAs; CECT reveals delayed enhancement of soft tissue component; often fusiform
B = mycotic aneurysm = infected aortic aneurysm = uncommon (0.06-2.6% of all aneurysms); usually saccular rather than fusiform, with perianeurysmal gas, stranding & fluid +/- vertebral body & psoas involvement
which is not associated with azygous continuation of IVC
a. dextrocardia
b. polysplenia
c. left sided SVC
d. gonadal vein into renal vein
e. hepatic vein into RA
E = F hepatic veins drain directly into RA
**LJS - hepatic veins drain into post-hepatic IVC that is still present - into Ra
Young woman with hypertension and narrowing of renal ostia. Most likely
a. FMD
b. Wegners
c. PAN
C = T/F = aneurysms at bifurcation points (up to 1cm); renal infarcts; renal/retroperitoneal haemorrhage; however is listed in StatDx as cause of RAS- Maybe real answer was neurofibromatosis (aneurysm; narrowing of proximal RA) or Takayasu
Causes of RAS
• Atherosclerosis (most common cause, 70%; renal artery ostium or proximal 2cm; elderly)
• FMD (25%; mid-distal RA or hilar branches, may be multifocal; young adults; R>L; bilateral in 2/3)
• Congenital/inheritedo Congenital stenosis (childhood)o Neurofibromatosis (children; proximal renal artery)o Ehlers-Danlos or Williams syndrome
• Arteritiso Takayasuo PANo Buerger disease
• Other
o Abdominal aortic coarctation
o Thromboembolico Radiation therapy
o Aortic dissectiono Phaeochromocytoma
o Infrarenal AAA
o Retroperitoneal fibrosis
- Coronary artery dominance defined by the coronary artery that supplies:
a. The obtuse artery
b. The posterior interventricular artery
c. AV nodal artery
d. The conal artery
b. The posterior interventricular artery T
• Refers to the coronary artery that supplies the diaphragmatic surface of LV & the posterior diaphragmatic portion of the interventricular septum – i.e. the dominant artery gives the posterior interventricular (descending) branch (PDA) & the posterolateral branch (PLB)
• Right dominance denotes RCA origin of flow (80-85%)
• Left dominance denotes LCA origin of flow (15-20%) - in this case the PDA & PLB arise from the LCx artery
• Mixed dominance refers to an intermediate pattern, e.g. PDA comes from RCA & PLB comes from LCx; branches of both arteries run in or near the posterior interventricular groove
• Notes:o The LCA almost always supplies a greater volume of tissueo The non-dominant system is usually smaller in calibre c.f. the dominant system
- 50 female presents with headache and hypertension. Angiography reveals multiple renal aneurysms. Which is MOST likely?
e. Aortic dissection
f. Fibromuscular dysplasia
g. Polyarteritis nodosa
h. Takayasu’s arteritis
i. Wegener’s granulomatosis
c. Polyarteritis nodosa T kidneys involved in 70-80% with multiple peripheral small aneurysms; CNS involved in 10% (microaneurysms; most common systemic vasculitis to affect the CNS)
2. 50 female presents with headache and hypertension. Angiography reveals multiple renal aneurysms. Which is MOST likely?
a. Aortic dissection F no renal aneurysms
b. Fibromuscular dysplasia F can cause hypertension, can also involve craniocervical arteries, but aneuryms usually post-stenotic, although multiple aneurysms can be seen
c. Polyarteritis nodosa T kidneys involved in 70-80% with multiple peripheral small aneurysms; CNS involved in 10% (microaneurysms; most common systemic vasculitis to affect the CNS)
d. Takayasu’s arteritis predilection for aorta esp. AA & its branches (esp. SCA); RAS may occur; can involve proximal carotid arteries; may cause aneurysms
e. Wegener’s granulomatosis F? typically causes GN, but can cause microaneurysms by its small vessel vasculitis; may cause intracerebral & meningeal granulomas or vasculitis
- CXR shows upward bowing of cardiac apex, rib notching and retrosternal mass, most likely? (+ VIC – young male with HTN, indentation of left lateral aortic border)
t. Coarctation of aorta
u. TAPVR
v. Aortic stenosis
w. Dissection
a. Coarctation of aorta T = get rounded elevated apex; rib notching; prominent ascending aorta
23. Old dude with known coronary artery disease. Ovoid mass with areas of calcification adjacent to left sphenoid sinus. Most likely \:i) Fusiform basilar artery aneurysm ii) Fusiform ICA aneurysm iii) CoW berry aneurysm iv) Micotic MCA aneurysm
ii) Fusiform ICA aneurysm T on MR look for pulsation artifact!
- Middle aged woman. End stage renal failure. AV fistula thrombosed and needs access. Decompensating badly with encephalopathy etc. Multiple previous Subclavian, IJ and femoral vascaths/lines. Treating team failed insertion IJ line on ward. Best option:
i) Peritoneal dialysis
ii) Doppler venous mapping
iii) Post con DSA venous mapping
iv) Post con CT venous mapping
v) Post con MR venous mapping
vi) Doppler venous mapping T with aim of doing US/fluoro guided central line insertionv
ii) Post con DSA venous mapping T if planning for new AVFIf in ESRF, further contrast would not be good (e.g. conventional venography) – can use Gadolinium for DSA (although this is against the RANZCR contrast guidelines!). No good evidence as yet for MRA.
Not sure what question means – if put in new AVF, would need to wait until matures anyway. Needs temporary central vein access & would assess with ultrasound in angio suite.If absolutely no access, could plan for peritoneal dialysis.Vascular access society guidelines for pre-op evaluation:
- 30 female left neck pain, 10 hrs of diplopia and dysarthria. Normal non contast CT head and neck. The next investigation should be:
a. Lumbar puncture
b. Contrast enhanced CT head
c. DSA
d. Carotid US
e. MRI brain
e. MRI brain T if with T1 fat sat neck and MRA neck/brain, although according to RG 08 has quite poor sens/spec for vertebral artery dissection. MRI brain to exclude other causes other than dissection.
4. 30 female left neck pain, 10 hrs of diplopia and dysarthria. Normal non contast CT head and neck. The next investigation should be:
a. Lumbar puncture F
b. Contrast enhanced CT head F would be T if CTA head/neck
c. DSA ?F invasive, but “gold standard”
d. Carotid US F
e. MRI brain T if with T1 fat sat neck and MRA neck/brain, although according to RG 08 has quite poor sens/spec for vertebral artery dissection. MRI brain to exclude other causes other than dissection.
50 yo mande, long hx of worsening claudication. 5cm long segment SFA occlusion. Best tx.
a. angioplasty
b. stent graft
c. bypass
d. exercise programme
e. cryotherapy
Still considered limited disease as < 10cm length. Infrainguinal. Best choice is endovascular revascularisation.RCT shows significantly higher patency rates of stenting over angioplasty alone for femoropopliteal artery lesions, however most reserve stent placement for acute failure of PTA. Therefore best answer probably A, angioplasty
.From TASC II 2007 guidelines (JVascSurg 2007)
• Acute limb ischaemia (ALI): Infrainguinal causes of ALI, such as embolism or thrombosis, are often treated with endovascular methods. Initial therapy with catheter-based thrombolysis should be considered in cases of acute thrombosis due to vulnerable atherosclerotic lesions or late bypass graft failures. When thrombolysis reveals underlying localized arterial disease, catheter-based revascularization becomes an attractive option. Stenoses and occlusions are rarely the sole cause of ALI or even severe chronic symptoms but these commonly lead to superimposed thrombosis and, therefore, should be treated to avoid recurrent thrombosis.
- Claudication: The initial approach to the treatment of limb symptoms should focus on structured exercise and, in selected patients, pharmacotherapy to treat the exercise limitation of claudication (risk factor modification and antiplatelet therapies are indicated to decrease the risk of cardiovascular events and improve survival). Failure to respond to exercise and/or drug therapy would lead to the next level of decision making, which is to consider limb revascularization. However, in patients in whom a proximal lesion is suspected (findings of buttocks claudication, reduced or absent femoral pulse) the patient could be considered for revascularization without initially undergoing extensive medical therapy.
- Revascularisation: Endovascular treatment of infrainguinal disease in patients with intermittent claudication is an established treatment modality. The low morbidity and mortality of endovascular techniques such as PTA makes it to the preferred choice of treatment in limited disease such as stenoses/occlusions up to 10 cm in length. For diffuse aortoiliac (suprainguinal) disease, bypass has better long-term patency, however the risks of surgery are greater and patient factors may lead towards PTA which has a 90% success rate
Left SVC drains into
coronary sinus orleft atrium
- Churg Strauss syndrome extrathoracic manifestations include?
a. Renal artery stenosis
b. Cerebral hemorrhage
c. Osteosarcoma
d. Hepatosplenomegaly
e. Mesenteric ischaemia
e. Mesenteric ischaemia T mesenteric vasculitisCSS = triad of asthma, hypereosinophilia & systemic small vessel granulomatous necrotizing vasculitis. Major involvement is of the heart, lungs & skin. (Dahnert – variant of PAN in asthmatic patients). Criteria for Dx (4 of 6) (1) asthma (wheezing, expiratory rhonchi), (2) eosinophilia of more than 10% in peripheral blood, (3) paranasal sinusitis, (4) pulmonary infiltrates (may be transient), (5) histological proof of vasculitis with extravascular eosinophils, and (6) mononeuritis multiplex or polyneuropathy.
- Strong association with allergic rhinitis, asthma, eosinophilia
- Vessels in lung, heart, spleen, peripheral nerves & skin involved by intravascular & extravascular granulomas, with infiltration of vessels & perivascular tissues by eosinophils
- Severe renal disease infrequent
- Coronary arteritis and myocarditis usual cause of morbidity and mortality
- Histologically may be identical to PAN and microscopic angiitis
- Both arteries and veins involved
- p-ANCA present in 40-70%
- Gastrointestinal symptoms (31%) - Symptoms related to GI vasculitis, eosinophilic gastritis, colitis (This includes abdominal pain [59%], diarrhea [33%], and GI bleeding [18%].)
- Which is NOT a recognised cause of Budd-Chiari syndrome
a. Chronic pancreatitis
b. Bone marrow transplantation
c. Chemotherapy
d. Systemic lupus erythematosus
e. Oral contraceptives
a. Chronic pancreatitis F (but does cause PV thrombosis!)
- Regarding tunneled haemodialysis catheters, which is LEAST correct?
a. Femoral catheters can remain in situ for several months
b. Tunneled catheters have a higher rate of infection than non-tunneled catheters
c. Internal jugular vein puncture is preferred to subclavian vein puncture
d. Fibrin sheath is best treated by thrombolysis
e. Optimal tip position is in the upper SVC.
a, c and e…
- Regarding tunneled haemodialysis catheters, which is LEAST correct? SK/Kandarpa p469
a. Femoral catheters can remain in situ for several months F (med comp femoral catheter is intended for use for < 30 days (product info)) Kandarpa – femoral catheters have higher incidence of infection, limits mobility & risk of iliac vein stenosis
b. Tunneled catheters have a higher rate of infection than non-tunneled catheters F non-tunnelled catheters have lower rates of blood flow, a higher incidence of infection & shortened survival c.f. tunneled catheters
c. Internal jugular vein puncture is preferred to subclavian vein puncture T preference is R IJV > L IJV or R EJV – avoid use of subclavian veins (if for later AVF) & avoid insertion on side of maturing AVF
d. Fibrin sheath is best treated by thrombolysis T
e. Optimal tip position is in the upper SVC. (F atriocaval junction or into the RA to ensure optimum flow); red lumen medial & distal (venous/blue) end hole lateral
- The most correct statement about colour Doppler US is?
a. Doppler colour coding depends on the amplitude of flow
b. Doppler colour coding depends on the vessel type
c. Doppler colour coding depends on the velocity of blood flow
d. Doppler colour coding depends on the angle of isonisation
c. Doppler colour coding depends on the velocity of blood flow T brighter colours (or different colour shades) are used to display mean velocities
d. Doppler colour coding depends on the angle of isonisation T in colour Doppler the format of transducer determines direction of Doppler beam. The Doppler angle may change with vessel orientation & produce colour changes related only to changes in the Doppler angle & not to changes in blood flow.Colour displayed within BVs on colour Doppler function of (Brant, US core curriculum) \:• Flow velocity • Doppler angle • Presence of aliasing • Colour map utilized • Phase of cardiac cycle