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RADIO INTERPRETATION > Radio anomolies > Flashcards

Radio anomolies Flashcards

1
Q

dental anomolies may be what in nature?

A

developmental or acquired

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2
Q

Hyperodontia

A

Teeth in excess of the normal complement (more than what there should be like having 4 of a molar)

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3
Q

Maxillary incisors hyperdontia name

A

mesiodens

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4
Q

Maxillary fourth molars and Mandibular fourth molars names

A
  • distodens,
  • paramolars
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5
Q

tooth type for hyperdontia prevalence in order:

A

max I>max M> man M> PM> C/LI

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6
Q

what can be seen here?

A

mesiodens

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7
Q

cystic change of supernumerary teeth

A

cystic change of follicle may or may not occur
increased likelyhood with exposure to inflammatory mediators
probability inversely related to age, however surgery better tolerated by younger pts

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8
Q

what can be seen here? where is it located?

A

odontoma, moves D with a D shift so this is located on palatal aspect (SLOB)

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9
Q

how could CBCT be useful for supernumerary teeth? con?

A

when there are many, the more there are the harder it becomes to locate them with simple technique (SLOB)
comes at an additonal cost

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10
Q

is there an anomoly? complications?

A

yes, a supernumary tooth is present at apex of PT tooth= inflammatory mediator present which can cause cystic change but this has yet to occur
the longer this is left the more likely ankylosis becomes too resulting in a difficult removal if problems arise

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11
Q

anomoly?

A

yes supernumerary tooth

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12
Q

anomoly, complcation?

A

distodens (3)
potential for pericoronal infection at the mandibular ones

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13
Q

impacted molar removal

A

best to remove these asap, more likely to undergo cystic degeneration

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14
Q

anomoly

A

impacted molar but not supernumerary

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15
Q

where is it most likely to have multiple hyperdontia (>1 extra tooth)

A

PM area

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16
Q

Syndromes with multiple teeth

A
  • Cleidocranial Dysplasia
  • Gardner
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17
Q

Cleidocranial Dysplasia additional signs

A
  • Frontal & parietal bossing
  • Clavicular aplasia
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18
Q

Gardner Syndrome additional signs

A
  • Autosomal dominant- family Hx
  • Multiple osteomas including the jaws
  • Colorectal polyps with malignant potential
  • Multiple supernumerary teeth
  • Lesions of skin (cutaneous epidermoid cysts
    and fibromas)
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19
Q

what discussed syndrome is lilkely present

A

gardner

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20
Q

Problems with Supernumeraries

A
  • Affect eruption of normal complement of
    teeth
  • Cystic degeneration of follicular epithelium of impacted teeth
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21
Q

what issue are supernumerary teeth causing in this child (4.5y)

A

difficulty with proper eruption

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22
Q

what issue have supernumerary teeth caused in the mandible?

A

cystic degeneration

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23
Q

Hypodontia

A

less than the normal tooth complement of an existing dentition; permanent or deciduous
less than one or more of an existing dentition; permanent or deciduous

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24
Q

Anodontia

A

congenital absence of all teeth

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25
Q

Anodontia v. Oligodontia v. Hypodontia

A
  • Anodontia – agenesis of all teeth
  • Oligodontia – congenital lack of > 6 permanent teeth, excluding third molars
  • Hypodontia – less than one or more of an existing dentition; permanent or deciduous
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26
Q

types of hypodontia

A
  1. Acquired
  2. Syndromal
  3. Reduction phenomenon
    - Third Molar Agenesis
    - Maxillary Lateral Incisors
    - Microdontia
    - Isolated Agenesis
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27
Q

most common type of hypodontia

A

acquired

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28
Q

how many syndromes have hypodontia associations

A

about 150

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29
Q

more common syndromes associated with hypodontia

A
  • ectodermal dysplasia
  • oligodontia-colorectal cancer syndrome
  • ectodermal dysplasia with oligodontia-colorectal cancer syndrome
  • otodental dysplasia – associated with deafness
  • Rieger Syndrome – associated with deafness
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30
Q

dental complications of ectodermal dysplasia

A

hypo or anodontia
malformed teeth
prone to caries (enamel defects and xerostomia)

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31
Q

malformed teeth

A

can be cone or peg-shaped

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32
Q
A

malformed teeth: cone/peg shaped

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33
Q

Reduction Phenomenon

A
  • Hypodontia not associated with other multiple system congenital syndrome
  • Seen with third molars and maxillary lateral incisors
  • Evolutionary trend ?
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34
Q

most common form of the reduction phenom

A

thrid molar agenesis, 10-25%/ up to 30% have 1-4 missing

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35
Q

comparing top to bottom

A

bottom image displays man 1 molar missing: could be hypodontia, agenesis, etc.
many other teeth are missing but would need med/tx history to know why

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36
Q

why are the man molars missing here?

A

likely extracted, note the furcal aleolar bone is still present

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37
Q

Agenesis of Maxillary Lateral Incisor
AMLI

A
  • Uni/bilateral reduction, i.e., peg lateral maxillary incisors
    AD inheritence, variable expression possible:
  • Uni/bilateral agenesis
  • a variable manifestation hypodontia
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38
Q

what is missing

A

Agenesis of Maxillary Lateral Incisor
AMLI

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39
Q

anomoly present?

A

AMLI, peg/cone shape

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40
Q
A

AMLI

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41
Q

microdontia

A

teeth smaller than usual, can affect one (microdont) to many teeth

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42
Q

how would you classify the man 3rd molars

A

microdonts

43
Q
A

microdont

44
Q

what complication arises with microdonts

A

diastemas form=food trap and caries

45
Q
A

microdont

46
Q

how common is isolated agenesis (non-third molar)

A

non-thrid molar hypodontia: 5-10% typical

47
Q

what issue arises with isolated agenesis

A

primary teeth may be retained, can be maintained and not require removal in some cases
may also lead to compromised arch form and/or occ discrepancies

48
Q
A

isolated agenesis, 1 molar retained

49
Q
A

isolated agenesis, 1 canine retained

50
Q

macrodont

A

enlarged tooth

51
Q

marcodont issues

A

may not fit arch space
sus to caires and perio dx with increased grooves

52
Q
A

macrodont

53
Q

Bulky Incisor or Double tooth
phenomenon

etiologies

A

form of macrodontia, hard to prove absolute etiology

54
Q
A

macrodont likely fusion

55
Q
A

macrodont

56
Q

what could this be classified as?

A

Fusion, Twinning or
Supernumerary

57
Q
A

macrodont

58
Q
A

macrodont

59
Q

dilacerations

A
  • Abnormal angulation or bend in the root (and occasionally the crown) of a tooth
  • Some related to trauma during odontogenesis
  • idiopathic
60
Q
A

dilaceration

61
Q

Supernumerary Roots

A
  • Extra roots above the complement of roots classically
    described for the dentition
62
Q

1st molar anomoly

A

3rd root

63
Q

Supernumerary Roots
* Significant for what considerations?
* Appear to be excellent for>?

A
  • Significant for endodontic or exodontic considerations
  • Appear to be excellent prosthodontic abutments
64
Q
A

3rd root 1st molar

65
Q

Li Root of Mandibular First
Molars

A
  • 20% incidence in Asians in a Chinese population (Hong Kong and Taiwan)
  • 10-12% in native North American populations
66
Q
A

supernumerary roots

67
Q

Non–carious Loss of Tooth Structure forms

A

Abrasion – mechanical wear of teeth
Attrition – physiologic wear of teeth
Erosion – chemical wear of teeth

68
Q
A

attrition

69
Q
A

cervical abrasion

70
Q

what is seen in both of these?

A

cervical abrasion

71
Q
A

erosion

72
Q
A

likely lingual erosion= bulemia

73
Q

taurodont

A

enlarged pulp chambers/ smaller roots

74
Q

what anomoly can be noticed

A

taurodonts

75
Q

taurodont etiology

A
  • Idiopathic developmental disorder of odonogenesis causing elongation of the pulp chamber (aka apical
    displacement of the pulpal floor) and shorter root appendages
  • failure of Hertwig’s epithelial sheath diaphragm to invaginate at the proper horizontal level, resulting in a tooth with short root, enlarged body, an enlarged pulp and normal dentin
76
Q
A

taurodont

77
Q
A

taurodont

78
Q

when are taurodonts complicated?

A

endo/extractions

79
Q

Dens Invaginatus Dens in Dente
incidence rate?
mostly seen where?

A
  • 0.04 – 10% incidence
  • Primarily seen in lateral incisors but also centrals, premoalrs, centrals, canines…..
80
Q
A

dens in dente

81
Q

Dens Invaginatus
Dens in Dente types

A
  • Type I – coronal
  • Type II – extends apical to CEJ
  • Type III – extends through root perforating to apex
82
Q

dens in dente complication

A

can act as a food trap= caries

83
Q
A

two dens in dente type I

84
Q
A

dens in dente type 2

85
Q
A

type 3 dens in dente

86
Q

Dens Invaginatus
Dens in DenteTreatment

A

restore the coronal pit

87
Q
A

talon cusps

88
Q

drift vs migration

A

drift: movement of erupted teeth
migration: movement of unerupted teeth

89
Q

impacted teeth

A

I. Full impaction vs. partial impaction
II. Orientation
- Vertical
- Inverted
- Mesioangular
- Distoangular
- Horizontal
- Inverted Mesioangular
- Inverted Distoangular

90
Q

what is the orientation of impacted teeth needed for?

A

evaluate prognosis and surgical management

91
Q

name this

A

inverted mesioangular impacted tooth

92
Q
A

vertical partial impaction

93
Q
A

vertical full impaction

94
Q

Describe the impacted tooth

A

vertical distoangular full impaction

95
Q
A

inverted mesioangular full impaction

96
Q
A

horizontal full impaction

97
Q

Problems with Supernumeraries
and Impacted Teeth

A
  • Affect eruption of normal complement of teeth
  • Cystic degeneration of follicular epithelium
98
Q

enameloma

A

ectopic globule of enamel formation at the root surface
AKA enamel pearl

99
Q
A

enameloma

100
Q

dentinoma etiology

A
  • may result from proliferation of conn tissue and hertwig’s epithelial root sheath cells
  • epithelial remnants induce undifferentiated conn tissue cells to transform into odontoblasts and produce dentin
101
Q
A

dentinoma

102
Q
A

dentinoma

103
Q
A

dentinoma

RADIO INTERPRETATION (18 decks)

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