Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

RADIO INTERPRETATION > multilocular lucencies > Flashcards

multilocular lucencies Flashcards

1
Q

potential multilocular lucencies

A
  • Ameloblastoma
  • Odontogenic Keratocyst
  • Central Giant Cell Granuloma
  • Odontogenic Myxoma
  • Vascular Lesions - Hemangioma
    . A-V aneurysm
  • Familial Fibrous Dysplasia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Ameloblastoma defined/aggression

A

Benign odontogenic neoplasm; one of very few true odontogenic neoplasms
Capable of uncontrolled, unlimited growth potential

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

classifications of ameloblastomas

A
  1. Conventional (Multicystic) Ameloblastoma
  2. Unicystic Ameloblastoma
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Conventional (Multicystic)
Ameloblastoma
Account for % of all ameloblastomas?
* sub-types?

A

Account for 85 – 90% of all ameloblastomas
* Five histologic sub-types;
- follicular - most common
- also have plexiform, acanthomatous,
granular, desmoplastic and basaloid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Ameloblastoma Conventional
* symptoms
* Small lesions only detected how?
* Larger lesions detected how?

A
  • Usually slow painless swellings
  • Small lesions only detected by radiographs
  • Larger lesions detected clinically
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Ameloblastoma
Conventional radio app

small/large lesions
effect on teeth?

A
  • Small lesions are unilocular with corticated borders
  • Large, aggressive lesions develop multilocular patterns
  • Displace and resorb teeth
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

can ameloblastomas expand?

A

yes, can displace teeth/ anatomical strucutres

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

ameloblastoma conventional age

A
  • Mainly adults – equal prevalence in 3’rd to 7’th decade
  • Uncommon in 2’nd decade
  • Rare in 1’st decade
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

ameloblastoma conventional location

A

prefers mandible (85%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

ameloblastoma conventional genders

A

no preference

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what could this be in 45 y/o male?

A

conventional ameloblastoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

ameloblastoma managment

A

Large lesions are aggressive requiring bone resection due to lesion being non-encapsulated

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

ameloblastoma recurrence

A

higher likelihood for recurrence, about 15% with proper resection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

ameloblastoma resection

A

Block or marginal resection; ie resect >1.0cm
past radiographic limits of tumor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

ameloblastoma recurrence without proper resection (% chance)

A

50-90%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

are ameloblastomas malignant?

A

RARE, but possible

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

can ameloblastomas app unilocular

A

yes, in early stages or as unicystic variant

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

when planning resection of ameloblastomas what imaging modality can be used?

A

CBCT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Unicystic Ameloblastoma
aggression/recurrence?

A
  • arise within a cyst lining; either luminal, intraluminal or mural
  • less aggressive form of ameloblastoma
  • Recurrence rates of 10-20%
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

dif dx

A

ameloblastoma
OKC
dentigerous cyst

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Ameloblastoma unicystic
Age
Site

A

Age: Mean age 23 years
Site: Mandible (90%), Maxilla (10%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

from 23 y/o male dif dx

A

OKC
unicystic ameloblastoma
dentigerous cyst

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Odontogenic Keratocyst
Pathophysiology
* commonality?
* aggression?
* arise from?

A
  • 10-12% of all odontogenic cysts; 3’rd highest oral cyst frequency
  • aggressive cysts; behave more like benign neoplasms
  • thought to arise from cell rests of dental lamina
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

most common and second most common cysts

A

1: PA
2: dentigerous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

histo of what lesion

A

OKC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Odontogenic Keratocyst clinical
* symptoms
* With increasing size what can occur

A
  • Normally asymptomatic
  • With increasing size, pain, swelling and exudate may oocur
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

OKC radio
* borders
* cortical plates
* teeth

A
  • Well-defined, smooth, corticated borders
  • Thinning and mild expansion with occasional perforation of cortical plates
  • Displacement of teeth
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

OKC root resorb

A
  • *only occasional root resorption
    (< dentigerous and < radicular cysts)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

OKC expansion directions

A

*mild B-Li expansion; but extensive antero-posterior extension

30
Q

dif dx

A

ameloblastoma
OKC

31
Q

dif dx

A

ameloblastoma
OKC
odontogenic myxoma

32
Q

sign of OKC with cortical plates

A

emphysema, can be pushed in

33
Q

OKC
age
site
gender

A

Age: Majority (i.e., 60%) in 2nd and 4th decade
Site: Majority (60-80%) affect mandible posterior to the
canines
Gender: Male predilection

34
Q

management OKC

A

Enucleation with curettage and toxic agent carnatene in some cases

35
Q

OKC recurrence

A
  • High recurrence rate; some rates reported @ 47 and 62% (probably parakeratinized variants)
36
Q
  • When multiple OKCs are found they
    may constitute part of?
A
  • When multiple OKCs are found they
    may constitute part of the basal cell
    nevus syndrome (a.k.a. nevoid basal
    cell carcinoma syndrome)
37
Q

Nevoid basal cell carcinoma syndrome signs:
* skin
* Palmar
* jaws
* ribs
* spine
* Skull anomalies:

A
  • multiple basal CA’s of skin
  • Palmar and plantar pitting (60%)
  • > 1 OKC (KOT) of jaws (75%)
  • Bifid or splayed ribs (60-75%)
  • Kyphoscoliosis (50%)
  • Skull anomalies:
  • frontal and parietal bossing
  • hypertelorism
  • intracranial calcifications; majority are of falx cerebri
38
Q

Nevoid basal cell carcinoma syndrome
inheritance

A

Autosomal dominant inheritance

39
Q

pt also presents with calcified falx cerebri, dx?

A

nevoid basal cell carcinoma syndrome

40
Q

when enucleating OKC of NBCCa syndrome what can be done to prevent recurrence

A

Gauze+ caratene

41
Q

Central Giant Cell Granuloma
Clinical
* symptoms
* aggression

A
  • Asymptomatic swelling
  • Can be aggressive
42
Q

CGCG histology

A

solid tumor of granuloma tissue: collagen and giant cells

43
Q

Central Giant Cell Granuloma
Radiographic app
* borders?
* locular?
* cortical plates?
* Displacement?

A
  • Well-defined borders
  • Can be multilocular
  • Thinning and expansion of cortical plates
  • Displacement of teeth and occasional root resorption
44
Q

what could this be

A

OKC
ameloblastoma
Central giant cell granuloma

45
Q

when imaging lesions how far should you go?

A

until border of lesion captured

46
Q

Central Giant Cell Granuloma
age, site, gender

A

Age: Usually < 30 years (60%)
Site: mandible (70%) & frequently between the molars
Gender: Female > male (2:1)

47
Q

Central Giant Cell Granuloma
Management

A

Enucleation with aggressive curretage

48
Q

CGCG is histologically similar to what lesion

A

Lesion is histologically similar to the Brown
tumor of primary hyperparathyroidism

49
Q

pts presenting with CGCG should be tested to rule out what?

A

hyperparathyroidism
* Screening test in the appropriate blood studies:
– serum calcium
– alkaline phosphatase
– serum phosphorus

50
Q

Odontogenic Myxoma Clinical
* Primarily a lesion of?
* Basically a?

A
  • Primarily a lesion of alveolar bone
  • Basically a fibrous lesion
51
Q

odontogenic myxoma radio

A

Scalloped and
multilocular

52
Q

which multilocular luceny presents like this

A

myxoma

53
Q
A

odontogenic myxoma

54
Q

Odontogenic Myxoma
age and site

A

Age: Young to adults (25 – 30 years)
Site: Greater prevalence in mandible

55
Q

Odontogenic Myxoma
Management

A

Excision

56
Q

odontogenic myxoma recurrence

A

Recurrence up to 25% because the lesions are not encapsulated.

57
Q

VASCULAR LESIONS

A

Central Hemangioma
Aneurysmal Bone Cyst
A-V Malformation

58
Q

Central Hemangioma
Clinical
* common sites?
* expansion?
* mucosal app?
* gingival sign?
* possible detections
* Many require what to assist diagnosis

A
  • Jaws are next most common site after skull and
    vertebrae
  • Firm, slow-growing asymmetric expansion
  • Overlying mucosa is more erythematous and warm to
    touch
  • Spontaneous gingival bleeding
  • Bruit on diascopy and pulsatile sensation may be
    detected
  • Many require needle aspiration to assist diagnosis
59
Q

Central Hemangioma
Radiographic

A
  • Variable pattern ranging from cyst-like radiolucencies
  • May have multilocular “soap-bubble” or spoke-like appearance
60
Q

diagnosis of central hermangiomas

A

usually superficial so tell signs such as redness and heat are present
may use aspiration to help diagnose, NO BIOPSY

61
Q

superficial erythmatous lesion:

A

central hermangioma, very superficial

62
Q

Central Hemangioma
age, site, gender?

A

Age: Teens and young adults
Site: Posterior mandible
Sex: Female:male 2:1

63
Q

lesion from 14 y/o female

A

central hermangioma

64
Q

Central Hemangioma
Management

A
  • Sclerosing agents, radiation, enucleation
  • Embolization of major arteries necessary prior to surgery as hemorrhage is a significant and life threatening complication
65
Q

potential etiologies of central hermangiomas

A

Etiology is either traumatic/developmental
or benign neoplasm

66
Q

Cherubism (familial fibrous dysplasia)
Clinical
* face?
* symptoms?
* Bone lesions are more active when?

A
  • Cherubic looking face by 5 years of age due to bilateral bony expansion
  • Asymptomatic
  • Bone lesions are more active in younger patients. After age 12, activity usually diminishes and finally lesions become inactive with residual deformity by age 30
67
Q

likely dx?

A

cherubism

68
Q

Cherubism Radiographic app

A
  • Bilateral multilocular cyst-like, expansile lesion, usually
    affecting the mandible and sometimes the maxilla
  • Pathologic fracture is not a feature
69
Q

Cherubism
age, site, gender

A

Age: Usually detected by age 5
Site: Bilateral mandible, may affect maxilla
Sex: Male:female 2:1

70
Q

Cherubism Management

A
  • Cosmetic osseous contouring at age 12 and later
  • Benign self-limiting condition

RADIO INTERPRETATION (18 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions