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TERM 4 > Rabbit Dz > Flashcards

Rabbit Dz Flashcards

1
Q

What is the causative agent for snuffles?

A

Pasteurella multocida

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2
Q

What is the transmission for snuffles?

A

direct contact

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3
Q

what is one of the most common diseases in rabbits?

A

snuffles

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4
Q

What are the c/s associated with snuffles?

A
  • Often subclinical***
  • Respiratory (coughing, dyspnea, nasal discharge) • Conjunctivitis
  • Otitis media and interna
  • Septicemia: acute death
  • Genital tract infections (male and female)
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5
Q

how do you diagnose snuffles?

A

RADIOGRAPHS
PCR
nasal culture

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6
Q

Pathology associated with snuffles?

A

Nasal passages
• Edematous, inflamed, congested • Turbinate atrophy

  • Cranioventral pneumonia
  • Fibrinopurulent pleuritis and pericarditis

Hepatic necrosis

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7
Q

Snuffles–management?

A

treat with ABX….treat symptoms…I think leave the abscesses alone

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8
Q

Tyzzers dans les lapins…transmitted how?

A

same as the others…butt to mouth

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9
Q

Tyzzers who’s affected and what are the clinical signs?

A

weanlings!
and the signs are mostly subclinical.
if you DO see signs that means it must be pretty severe…typically a co-infection involved.
Profuse diarrhea, listlessness, dehydration and death within 72 hours
• 90 – 95% mortality

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10
Q

Tyzzers definite mode of dx?

A

per/serology/culture

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11
Q

Tyzzers gross lesions involve which organs?

A

liver heart intestines

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12
Q

Tyzzer histopath lesions…which stain and which organs will you see lesions in ?

A

W. Starry. Ileum/cecum/colon (edema and necrosis) and liver (parenchymal necrosis)

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13
Q

How do you treat Tyzzers?

A
  • No treatment for clinically ill rabbits
  • Antibiotics are poor secondary to intracellular location
  • Minimize stress
  • Good husbandry
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14
Q

Enterotoxemia caused by what?

A

C. spiroforme

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15
Q

Enterotox and route of transmission?

A

ENVIRONMENT and changes in gut flora

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16
Q

Enterotox and c/s?

A

diarrhea and just pure nastiness around the perineum. moribund…pyrexic…cyanotic…

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17
Q

Enterotox treatment?

A

good husbandry
low stress
copper sulphate supplementation
poop transplant

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18
Q

Collibacillosis caused by?

A

e coli and seen in the young ones (sucklings and weanlings)

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19
Q

Collibacillosis 3 syndromes:

A

enteric/diarrheal
urinary
sepsis/meningitis

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20
Q

Collibacillosis—what kind of diarrhea do sucklings have?

A

severe yellow with high mortality

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21
Q

Collibacillosis—what kind of diarrhea do weanlings get?

A

profuse watery diarrhea, stunted growth, death

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22
Q

Diagnose Collibacillosis?

A

culture! blood agar and biotypying

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23
Q

Collibacillosis zoonotic risks?

A

EHEC/EPEC

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24
Q

Collibacillosis pathology?

A
  • Attaching & effacing lesions w/ pedestal formation
  • EPEC: Petechial‐ecchymotic serosal hemorrhages, thickening & edema of cecal & proximal colon serosa
  • EHEC: enterocolitis, nephropathy, thrombotic microangiopathy
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25
Q

Collibacillosis treatment

A
  • Avoid introducing rabbits of unknown status into a colony
  • Screen rabbits (culture) & characterize E. coli isolates (PCR) • Eradication: rederivation & antibiotic treatment
  • Antibiotic treatment?
  • EPEC Antimicrobial resistance
  • Enrofloxacin, chloramphenicol, neomycin • Fluid therapy for clinically affected animals
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26
Q

Trepanematosis aka…?

A

“Venereal spirochetosis, rabbit syphilis, vent disease”

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27
Q

Trepanematosis causative agent?

A

Treponema paraluiscuniculi

28
Q

Trepanematosis transmission

A
  • Direct contact during breeding

* +/‐ vertical transmission

29
Q

Trepanematosis c/s

A

Lesions on vulva/prepuce

• Swelling & erythema vesicles, papules ulceration, scaling & crusting

30
Q

Trepanematosis diagnose?

A

• Serology
• Nontreponemal antigen tests
• Microhemagglutination: optimal assay
• Dark‐field microscopic examination of wet‐mounted scrapings
from lesions
• Histology: silver stained testicular sections
• PCR

31
Q

Trepanematosis pathology:

A

Pathology:
• Erythematous macules or papules to erosions, ulcers & crusts
• Epidermal hyperkeratosis, hyperplasia and acanthosis with ulceration; macrophage & plasma cell infiltrate

32
Q

Trepanematosis treatment

A

• Quarantine & test new breeding animals prior to introduction into
the colony
• Hysterectomy derivation can eliminate
• Benzathine penicillin G‐procaine penicillin G at 7‐day intervals resolution of lesions in 2 weeks

33
Q

Myxomatosis cause and which rabbits?

A 
Leporipoxvirus like rabbit fibroma virus
brush rabbits (wild)
34
Q

Myxomatosis transmission

A

Direct contact

• Arthropod vectors

35
Q

What stain is best used to id clostridium?

A

war. starry stain

36
Q

Which rabbit type is susceptible to myxomatosis?

A

Oryctolagus new zealand

37
Q

lesions for myxomatosis

A

Mucinous skin lesions, tumors, edema around mouth, nose, anus & genitals, progressive conjunctivitis with serous & mucopurulent secretions from eyes & nose

38
Q

myxomatosis diagnostics

A

Definitive: culture

39
Q

myxomatosis treatment

A

Prevent contact with arthropods

• Quarantine infected rabbits

40
Q

Rabbit Fibroma Virus cause?

A

Leporipoxvirus

41
Q

Rabbit Fibroma Virus lesions?

A

skin tumors on legs/face

42
Q

Leporid Herpesviruses

A

LH-4 Alpha

43
Q

Leporid Herpesviruses LH-4 Alpha

in what rabbits and causes what?

A

domestic
Severe disease in preweanlings: weakness, anorexia, conjunctivitis, keratitis, periocular swelling, & skin ulcers in some animals

44
Q

Cottontail (Shope) Papillomavirus transmission

A

+/-arthropods like myxomatosis

45
Q

c/s Cottontail (Shope) Papillomavirus

A

• Papillomas on the neck, shoulders, abdomen, eyelids, ears

46
Q

special c/s sign Cottontail (Shope) Papillomavirus

A

• Up to 25% of infected rabbits develop squamous cell carcinomas

47
Q

Rabbit Hemorrhagic Disease caused by

A

calicivirus

48
Q

Rabbit Hemorrhagic Disease transmission

A

Fecal‐oral

49
Q

Rabbit Hemorrhagic Disease C/S

A

peracute-death
acute-lymphopenia and coagulopathy
subacute- mild with survival

50
Q

Rabbit Hemorrhagic Disease Treatment:

A
  • Quarantine & depopulate

* Thoroughly cleanse & disinfect the environment

51
Q

ear mites that don’t burrow-Psoroptes

A

Pruritic lesions in the inner
surfaces of the external ear
• Scratching, head shaking, pain, self‐mutilation
• Tan, crusty exudate with moist, erythematous underlying skin

52
Q

Cheyletiella spp. important??

A

ZOONOTIC

Walking dandruff

53
Q

Cheyletiella spp. differentiating c/s compared to Psoroptes

A

no pruritis/no skin lesions

54
Q

Sarcoptic Mange does it burrow and cause extreme itchiness?

A

yes. will see secondary hair loss and most lesions seen on the head.
can sometimes have anemia, leukopenia and secondary bacterial infectoins

55
Q

what’s an unusual pathology associated with sarcastic?

A

• In severe infestations, amyloidosis of liver & glomerulus

56
Q

Tricophyton mentagrophytes…zoonotic?

A

yes

57
Q

how to treat/deal with Tricophyton mentagrophytes?

A

self limiting in healthy animals
vaccine available
may have to cull

58
Q

Hepatic Coccidiosis cause?

A

Eimeria stiedae

59
Q

Hepatic Coccidiosis c/s

A

• Hepatomegaly (common), icterus, anorexia

can also see diarrhea and weight loss

60
Q

Hepatic Coccidiosis interesting way to dx?

A

impression smears with cut surface of the liver

61
Q

Hepatic Coccidiosis pathology

A

Enlarged, discolored liver with multifocal yellowish‐white lesions of varying size, exudate in biliary tree, dilatation of bile ducts
• Papillomatous hyperplasia of ducts with multiple life cycle stages of the organism present in the biliary epithelium

62
Q

Encephalitozoonosis cause?

A

Encephalitozoon cuniculi

63
Q

Encephalitozoonosis big thing about this one…?

A

zoonotic!

64
Q

Encephalitozoonosis best place to look for dz?

A

kidneys and brain with Giemsa stain, Gram stain, Goodpasture’s carbol fuschin stain

65
Q

Encephalitozoonosis c/s

A

Convulsions, tremors, torticollis, paresis‐paralysis, coma, signs of kidney failure

66
Q

Encephalitozoonosis transmission

A
  • Direct contact, environmental contamination
  • Urine
  • Vertical transmission possible as well
67
Q

Encephalitozoonosis pathology

A

Kidney: multiple white, pinpoint areas or gray, indented areas on
renal cortical surface
• Granulomatous inflammation ± interstitial infiltration of lymphocytes & plasma cells, tubular degeneration
• Organism may be free‐floating in renal tubules
• Granulomatous encephalitis (characteristic), spinal cord lesions
• Organism often not observed histologically

TERM 4 (9 decks)

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