quiz 9 & 10 Flashcards

1
Q

5 general types of voice diagnosis

A
  • history of disorder
  • auditory perceptual analysis
  • instrumental measures
  • descriptive scales of severity
  • laryngeal imaging
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2
Q

acoustic approaches used in a voice laboratory

A
  • fundamental frequency
  • intensity
  • perturbaation measures (shimmer and jitter)
  • harmonics-to-noise ratio
  • spectral analysis
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3
Q

aerodynamic approaches used in a voice laboratory

A
  • flow
  • subglottal air pressure
  • phonation threshold pressure
  • laryngeal resistance
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4
Q

physiologic approaches used in a voice laboratory

A
  • electroglottography
  • electromyography
  • laryngeal imaging
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5
Q

fundamental frequency

A

relates to our perceived pitch
- measured in hertz (cycles per second)
- easy to understand because if relates direclty to vocal fold vibration

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6
Q

intensity

A

relates to loudness, measured in decibels

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7
Q

perturbation measures

(shimmer and jitter)

A

cycle to cycle variability in a particular sound signal (for us sustained vowel production)
- shimmer = perturbation for intensity
- jitter = perturbation for frequency

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8
Q

harmonics-to-noise ratio

A

amount of relative periodicity in the acoustic signal generated at the vocal folds compared to the level of ‘noise’ in the signal

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9
Q

spectral analysis

A

speech overtime: sound spectrogram (frequency on y, time on x)
- fast fourier transform (fft) and linear predictive coding (lpc) for vowel productions at a single point in time
- long term average spectrum (ltas) for connected speech

the appropriate analysis depends on the aspect of the acoustic signal under consideration

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10
Q

flow

A

measures of flow volume and rate allow for the collection of average values that can reflect the degree of glottal closure
- using lung volumes available for sustained phonation, flow rate can be found by measuring volume of air in ml/sec
- higher airflow rate indicates less vocal fold closure

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11
Q

subglottal air pressure

A

driving force for vocal fold vibration
- insufficient pressure means folds don’t vibrate in a predictable reliable way
- measure by needle or commonly with intraoral pressure at /p/ closure

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12
Q

phonation threshold pressure

A

the least tracheal pressure needed to initiate vocal fold oscillation
- higher PTP to produce voice after vocal fatiguing task (systemic hydration delays the increase in PTP)

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13
Q

laryngeal resistance

A

divide peak intraoral pressure by peak flow rate
- ask patient to say pipipipipip at a mid lung volume so airflow is stable
- can indicate if phonation is hyperfuncitonal or hypofunctional

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14
Q

electroglottography

A

measures vocal fold contact area during sustained phonation (noninvasive)
- contact electrodes on thryroid laminae with a slight current–> the signal indicates the amount of contact area between the vocal folds

more current = more closure

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15
Q

electromyography

A

measures nerve function by inserting a needle electrode into the muscle and measuring electrical activity during phonation or breathing
- done by otolaryngologist or neurologist
- guides injection of botox

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16
Q

laryngeal imaging

A

otolaryngologists use rigid or flexible endoscopy to visualize the larynx
- laryngovideostroboscopy (lvs) can diagnos laryngeal function by showing vocal fold vibration
- the specific influence of a lesion can be diagnosed

17
Q

functional voice disorders

A

result from abuse, misuse, or overuse of the voice
- caused by producing voice in a way that disrupts balance among respiration, phonation, and resonation necessary for efficient voice production (including increased musculoskeletal tension) or abusive voice behaviors (yelling, throat clearing, etc)

inappropriate voice production can cause nodules, polyps, reinke’s edema, cysts, contact ulcers, and granulomas

18
Q

neurogenic voice disoders

A

caused by a neurologic disease/disorder (motor neuron disease)

19
Q

neoplastic voice disorders

A

vocal fold masses that arise from disease processes
- patients may develop dysfunctional voice production to compensate for lesions
- most common malignant tumore is carcinoma; leukoplakia and dysplasia are precancerous
- benign lesions: papillomas and amyloid masses

20
Q

vocal nodules

basic cause and primary impact on the voice

A

structural lesion causing dysphonia

functional

21
Q

vocal polyps

basic cause and primary impact on the voice

A

unilateral lesion; the mass can be large (when it volves the whole vocal fold it is considered reinke’s space edema or polypoid degeneration)
- occasionally a reactive nodule forms on other fold
- can reulst from a single even of shouting or can be caused by smoking

functional

22
Q

vocal fold cysts

basic cause and primary impact on the voice

A

unilateral lesions (may be reactive nodule/swelling); epidermoid or mucous retention
- long-standing (can be asymptomatic)

functional

23
Q

contact ulcers and granulomas

basic cause and primary impact on the voice

A

at the posterior commissure & vocal processes
- result from ‘aggressive’ speaking styles that cause increased pressure and irritation at the vocal process
- also caused by intubation and gastroesophageal reflux

functional

24
Q

muscle tension dysphonia

basic cause and primary impact on the voice

A

comes from the imbalance of extrinsic and intrinsic laryngeal muscles used to produce voice–larynx can be virtually immobile in neck
- supraglottic hyperfunction and glottal ‘under closure’
- can be primary (result of psychological stressors) or secondary (result of organic disease)

functional

25
Q

psychogenic voice disorders

basic cause and primary impact on the voice

A

associated with psychological disorders
- conversion aphonia and dysphonia; mutational falsetto

functional

26
Q

upper motor neuron voice disorder

basic cause and primary impact on the voice

A

cortical and subortical strokes, pseudobulbar/supracuclear palsy, some presentations of ALS and multiple sclerosis
- voice characteristics: roughness, with a spastic and strain/strangled component
- can have phonatory apraxia (can’t initiate phonation or other nonspeech laryngeal behavior)
- tremorous quality on sustained phonation

neurogenic

27
Q

lower motor neuron voice disorder

basic cause and primary impact on the voice

A

result from damage or disorders in the brainstem and peripheral nerves
- dysphonia may be absent or minimal if affect fold is paretic and there is good compensation from the intact fold
- voice characteristics: breathiness, roughness, diplophonia
- inspiratory stridor can signal possiblity of bilateral recurrent laryngeal nerve damage (along with higher than normal habitual pitch)

neurogenic

28
Q

extrapyramidal voice disorder

basic cause and primary impact on the voice

A

incules parkinsons disease and huntinton’s chorea; also can cause essential voice tremor
- hypokinetic dyarthria (pd) or hyperkinetic dysarthria (hc)

neurogenic

29
Q

cerebellar voice disorder

A

damage to cerebellum by degenerative process or stroke
- voice: rough voice with fluctuations in loudness and pitch

neurogenic

30
Q

spasmodic dysphonia

basic cause and primary impact on the voice

A

uncertain etiology (not a disease or associated with a spcific one)
- excess of normal movement
- adductor, abductor, & chronic constriction
- tremor at high sustained pitch

neurogenic

31
Q

myoneural junction disorder

basic cause and primary impact on the voice

A

disturbances of myoneural junction or muscle
- includes myasthenia gravis, polymyositis, and oculopharyngeal dystrophy
- symptoms related to site of damage
- mg: progressive hypophonia with use

neurogenic

32
Q

paradoxical vocal fold motion

what is it and how is it diagnosed

A

vocal fold approximation during inhalation and abduction during exhalation
- this ‘contradictory’ movement pattern causes inspiratory stridor and a feeling of constriction at the throat

diagnosed by exclusion
- fold movement during quiet breathing is normal
- differential diagnoses include asthma, structural airway obstruction, and focal dystonia; pulmonary function testing, gerd, decrased laryngeal sensation
- then thoroughly asses the conditions and trigger
- perform nasal endoscopy after and attach is elicited