questions 3 Flashcards
Definition of APH
Any bleeding
>20 wks
>before onset of labour
fetal fibronectin
what is it
when do we test
indications
glycoprotein found at maternal-fetal membrane
normally found at low levels- higher levels may indicate increased risk of preterm labour
used bewteen 26-34wks
Indications
- intact membranes
- cervix <3cm
- only slight PV bleeding
- no cervical cerclage
- no sex / pelvic exam in last 24hrs
not appropriate if woman is asymptomatic (even if she is considered high risk)
IOL contraindications
any maternal/fetal condition not to birth vaginally
malpresentation - transverse/ oblique
classical CS
Cord prolapse
Active herpes
placenta praevia
absolute cephalopelvic disproportion
invasive carcinoma of cervix
baby is severely compromised
relative CI’s
- FHR is non reassuring
- breech
- polyhydramnios
- not engaged
- severe hypertension
- maternal heart disease
cord presentation:
- cord is inbetween leading part of fetus, and internal os AND membranes are intact
- diagnosis may be identified via VE and colour doppler studies
- high risk of cord prolapse, if membranes break
occult cord
- cord is alongside presenting part
- not identified via VE
Cord prolapse- overt
-Membranes ruptured +- cord descends past fetal presenting part, through cervix into vagina
- diagnosis
* -visual (seen in introitus)
* palpation in vagina
* Abnormal FHR
- high risk of fetal hypoxia
cord prolapse- occult
-membranes ruptured
-cord is trapped between presenting part and pelvis
diagnosis
* FHR change
* rarely seen / felt
cord presentation / prolapse
risk factors
general
- multiparous
- small baby (preterm / SGA)
- abnormalities
- malpresentation (breech, transverse/ oblique /unstable)
- second twin
- polyhydramnios
- unengaged presenting part
- abnormal placenta (e.g. low lying)
intervention related
ARM
ECV / Vaginal manipulation of baby after ARM
Referral for cord presentation + prolapse
Emergency
cord prolapse - mgmt
1) change woman’s position (exaggerated SIMs)
2) call for help
3) confirm FHR viability
4) minimise compression bewteen fetal head and pelvis
* digital pressure via VE
* bladder fillling
5) Expedite birth (CS unless birth imminent)
give oxygen + IV line
consider tocolytics
*avoid handling cord / exposing cord to cool air to avoid vasospam of cord vessels
Insulin mechanism
insulin unlocks glucose transporters (passive transport)
for women already with diabetes and on insulin, what changes to diet can they expect
may become more restrictive
what is HbA1c, when do you refer
universal sreening <20wks
looks at average glucose in blood over last 4-6wks
<40 “ NORMAL”–> polycose at 24-28 wks
41-50–> “prediabetic” clinic + OGTT at 24-28wks
>50- ->”probable diabetes”
polycose test-
indication / normal results
for women with normal HbA1c
1hr 50g glucose
<7.8 NORMAL
>7.8 -11.0 -> do OGTT
»11.0–> refer to clinic
OGTT
indication / what is it/diagnostic results
only for high risk women
- women with high polycost tet results
- hbA1c >40
diagnostic
- > 5.1 (fasting)
- >8.5 post prandil
why do women with GDM have more UTI’s
glucose in urine ‘feeds’ bacteria
hyperemesis gravidarum
* when is it most commonly occuring
* what is it
* what are causes
* mgmt
* risks if not treated
when
- usually from 6-20 wks
- what
persistent / vomiting - fluid + electrolyte depletion
- ketonuria
- nutritional deficiency
- rapid weight loss
causes
- multifaceted -
- `hcG- (higher risk for women with more hcg (e.g. molar + multiples)
psychological
gastric abnormalities (delayed emptying, reduced acid)
vestibular disorders
ANS changes
liver enzyme
high thyroxine
- no evidence that its sociioeconomic, racial, ethnic
- management
rest
avoid stimulating factors
antimetics (ginger, b6, metoclopramide, ondansetron if necessary)
IV fluids
electrolyte replacement
peripheral catheter/ nasal gastric tube
long term risks
- renal
- neurological - (thiamine deficiency–> wernicke’s encephalopathy- emergency)
- liver
definitions + referrals
- chronic hypertension vs gestational hypertension
-
chronic
>140 / 90 (>2 readings (At least 4hrs apart))
>confirmed <20wks
>referral- CONSULT <16wks
gestational hypertension
>140/90
>20 wks gestation
>no features of PE
>BP returns to normal within 3mths of birth
>referral- CONSULT
Mgmt of thin mec
no referral req
assess for fetal distress
more common in post dates / preterm
put on a pad and monitor ongoing loss
post partum-if resus required / APGAR <9 at 5mins- 4hourly obs for 24hrs
referral guidelines for delay in 2nd stage
Consult
-primip- pushing for 2hrs with no progress
- multip- pushing for 1hr with no progress
Mendelson’s syndrome
aspiration pneumonia that can occur in CS- hence avoid eating before surgery
- OMEPRAZOLE given to reduce acidity in stomach
Placenta praevia- types
type 1 - placenta is mostly in upper segment
type 2- partially in lower segment (minor)
type 3- partially covers internal os
Type 4- completely covers internal os (major)
when can women with placenta praevia birth vaginally
if inferior edge of placenta is >2cm from internal os
signs of possible placenta praevia
- painless / unprovoked vaginal bleeding >20wks
- high presenting part
- abnormal lie
- (irrespective of previous imaging results)
TV USS is safe with placenta praevia
What is AN mmgt of placenta praevia if no bleeding / placenta reaches edge
- explain that is bleeding / contractions start- go to hospital immediately
- check for anemia
- avoid sex / digital exams/ speculum (unless we are sure about placenta location)
recheck placenta location at 32 wks
>32wks- transfer
uterine hypertonus / hypersystole- definition
contractions >2min duration OR <60sec break in between
FHR normal
uterine tachysystole- definition
> 5 contractions :10mins
FHR normal