Pulpal, Periapical, and Periodontal Disease Flashcards
Abscess
- Confined pocket of puss
- collects in tissues, organs or spaces inside the body
Cyst
- Closed sac
- distinct membrane
- develops abnormally in cavity or structure of body
Granuloma
- Mass or nodule of chronically inflamed tissue
- associated w/infective process
Primary Dentin
- Dentin formed before crown completion
Secondary Dentin
- After primary dentin
- leads to smaller pulp chambers and canals
- Deposition not uniform
- associated w/calcification related disease
- arthritis
- gout
- kidney. stones
- gall stones
Causes of Secondary Dentin
- Early formation associated with progeria (accelerated aging)
- Significant traumatic injury
- early obliteration of pulp chamber and canal =calcific metamorphosis
- leads to yellow discoloration of crown
- Physiologic secondary dentin
- result of aging
- functional forces of occlusion promote deposition
- begin in apical areas and spread coronally
What is the timeline for pulpal and periodical disease?
- Caries, Trauma, Periodontitis
- Death of Dental Pulp
- Apical Bone inflammation
- Dental Granulation formation
- Stimulation of epithelial rest of Malassez
- Epithelial Proliferation
- Periapical Cyst formation
How does Dentin show up radiographically?
Secondary & Tertiary Dentin
- accelerated closure of pulp chamber and canal
- obliterated pulpal space
how long do Alterations in dentin take after trauma?
As early as 3 months
What separates primary and secondary dentin?
- line of demarcation
- bending of tubules
Tertiary Dentin
- localized to pulpal end of odontoblastic processes
Calcific Metamorphosis
- hard tissue deposited in root canal space
- yellow discoloration of clinical crown
Calcific Metamorphosis
- hard tissue deposited in root canal space
- yellow discoloration of clinical crown
Pulpal Calcifications
- prevalence increases with age
- associated w/chronic pulpitis
Types of Pulpal Calcifications?
- Denticles
- Pulp Stones
- Diffuse Linear Calcifications
Pulpitis
- Inflammation of the Pulp
What does pulpits cause?
- increased fluid pressure
- adjacent to affected dentin
- increases interstitial pressure
- →causes increased flow of fluid back into capillaries of adjacent uninflamed tissue and increased drainage
What are the causes of Pulpitis:
- Mechanical Damage
- Thermal Injury
- Chemical Irritation
- Bacterial effects
Chronic Hyperplastic Pulpitis
- aka Pulp polyp
- Teeth w/large chamber
- molars
- Mostly children
- Pink to red nodule
- in pulp chamber
- inflammatory hyperplasia
Periapical Granuloma
- Aka Apical Periodontitis
- inflamed granulation tissue at apex of non-vital tooth
- Formation of apical inflammatory lesion:
- Early stages:
- neutrophils predominate
- release prostaglandins→activate osteoclasts
- radiographic alterations absent
- acute apical periodontitis
- neutrophils predominate
- Chronic Inflammatory cells dominate host response
- stimulate fibroblasts and microvasculature
- Early stages:
- Phoenix Abscess
Acute Apical Periodontitis
- constant dull, throbbing pain
- pain on biting and percussion
- periodical granuloma-asymptomatic
- represent 75% of apical inflammatory lesions
- 50% fail to respond to conservative endo
Periapical Cyst
- aka Radicular Cyst
- True epithelium-lined cyst in response to inflammation
- Source of epithelium
- Rest of malassez
- No Symptoms
- Radiograph:
- loss of lamina dura
- Root Resorption-Common
- Cyst Development-Common
What are the variants of Periapical Cysts
- Periapical Pocket cyst
- Periapical True cysts
- Lateral Radicular Cyst
- Residual Periapical Cyst
Periapical Pocket Cysts
- Incomplete epithelial lining
- apical portion of tooth extends into cyst lumen
Periapical True Cysts
- Complete Epithelium-lined
- bag like structure
- adjacent to tooth apex
Lateral Radicular Cyst
- appear along lateral aspect of root
- mimic radiograph of lateral periodontal cyst
Residual Periapical Cyst
- Inflammatory cyst
- post extraction
Periapical Abscess
- Acute Apical Periodontitis
- earliest stage of all forms of periodical inflammatory disease
- symptomatic and asymptomatic presentations
- Radiograph:
- Thickened PDL
- ill defined radiolucency
- or both
- Parulis (gum boil)
- Dental abscesses drain via cutaneous sinus
- perforate buccally-thinner bone
- Drain through lingual cortical plate
- Maxillary Lateral incisors
- palatal roots of maxillary molars
- mandibular 2nd and 3rd molars\
- Can spread through bloodstream=systemic symptoms
- fever
- lymphadenopathy
- malaise
Cellulitis
- Abscess that doesn’t drain through surface of skin or into oral cavity
- spread diffusely through fascial planes of soft tissue
- Types:
- Ludwigs Angina
- Cavernous Sinus Thrombosis
Ludwig’s Angina
- aggressive and rapid spreading cellulitis
- involves:
- sublingual
- submandibular
- submental spaces
- Acute infection from lower molar teeth-70%
- extend to lateral pharyngeal space→retropharyngeal space
- spread to mediastinum
- serious consequences
- massive swelling of neck-→clavicle
- Woody Tongue
- sublingual space
- compromised airway
- Bull neck
- submandibular space
- Restricted neck movement
Cavernous Sinus Thrombosis
- edematous periorbital enlargement
- involve:
- eyelids
- conjunctiva
- involve:
- Involve Canine space
- Protrusion and fixation eyeball
- proptosis, chemises, ptosis