Pulm Flashcards

1
Q

What does the heart look like on PA and AP?

A

PA vs AP: heart size-looks larger in AP
Lateral: use the spine and heart for orientation

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2
Q

Describe anatomical structures of the thorax

A
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3
Q

What do the carina and bronchi look like on CXR?

A
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4
Q

Where should the trachea be?

A

Over the vertebrae with the division of the carina.

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5
Q

An aspiration from the right bronchus will end up in what lobe?

A

RLL

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6
Q

What do the lungs look like on chest x-ray?

A

The lung fields: left upper and lower, right upper, middle, lower, and posterior lower lung extends superiorly and inferiorly. Usually need 2 views a AP and PA view.

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7
Q

What is the anatomy of the diaphragms on x-ray?

A

Liver elevates right side just a little. Sometimes we can see gastric bubbles from the stomach
1= right hemidiaphragm-higher because it is right under the heart.
2= left hemidiaphragm-higher at spine

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8
Q

You should be able to define the costophrenic angles and cardiophrenic angles

A

The costophrenic angles are in yellow, green, and blue
The cardiophrenic angles are in red.
Angles should be sharp
If in cardiophrenic-may be sticky like pneumonia. Usually gets rapped in costophrenic fluid collects at lowest portion and we will get a small sign

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9
Q

What does the heart and blood vessel anatomy look like on chest x ray?

A
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10
Q

What parts of the heart can we see on chest x ray?

A
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11
Q

On the PA view, how does heart size look?

A

Heart diameter should be less than ½ of the chest diameter. If they have a bigger heart than this in this view it indicates CHF

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12
Q

What does the aorta and other vessels look like on chest x ray?

A

The aorta has a small area that may be visible on the right side. The aortic knob is on the left and the descending aorta along the thoracic vertebrae. There is also the pulmonary trunk and the aortopulmonary window.

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13
Q
A
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14
Q

What is the aortopulmonary window?

A

In fetal development it is called the ductus arteriosum which will degrade into the ligamentum arteriosum. it helps develop the aortopulmonary window.

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15
Q

If the ductus arteriosum does not close, what happens?

A

It will cause a congenital heart defect

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16
Q

Describe the structures of the aorta, pulmonary trunk, and aortopulmonary window

A

Aorta in red, pulmonary trunk in yellow, and aortopulmonary window in blue

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17
Q

Describe the structure of the pulmonary arteries

A

L/R pulmonary arteries and L/R hilum

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18
Q

Define these vessels (aortic knob, aorta, pulmonary trunk, right pulmonary artery, and left pulmonary artery) in this picture.

A
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19
Q

Define these heart areas: aorta (ascending arch, descending), pulmonary trunk, aortopulmonary window.

A

The circle is the aortopulmonary window

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20
Q

The aortopulmonary window is easier to see on what view of chest x ray?

A

Lateral

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21
Q

What are the 3 lateral spaces and how do they look on a lateral chest x-ray?

A

Retrosternal (red), retrocardiac (yellow), and retrotracheal (blue). Vertebrae become darker inferiorly. Spinal processes are whiter as we go down, there is an infiltrate.

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22
Q

How does the scapula look on chest x-ray and how can it be confusing?

A
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22
Q

When looking at vertebrae on chest x-ray what should we be looking for?

A

Should see brighter white of spinous process in center of trachea and look for alignment of the vertebrae.

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23
Q

What do the clavicles look like on chest x-ray?

A

Medial portions of the clavicles should be aligned and acromioclavicular joints should be equidistant to clavicles. The bottom of the clavicle should line up with the bottom of the acromioclavicular joint

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24
Q

Can you identify these things in the following chest x ray?
Trachea and L/R bronchi
L/R hemidiaphragms and costophrenic angles
heart areas and heart borders
Aortic knob, aorta, aortopulmonary window
L/R pulmonary arteries/hila
Vertebrae and spinous processes
Count the ribs
Clavicles, scapula
Any other organs-liver, stomach

A
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25
Q

Correctly identify an artifact on a chest x-ray.

A

Breast shadows for an objects, artifacts from clothing, jewelry, pacemakers, wires, or tubes, external or internal hardware from prior surgeries and subcutaneous air. Breasts, pacemakers/defibrillators, sternotomy wires, heart valves, EKG wires and electrodes, metal snaps from a gown, jewelry, hair, hair bands

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25
Q

Accurately count anterior and posterior ribs on a chest x-ray.

A

Ribs-anterior vs posterior
Vertebrae-spinous processes, body, transverse processes
Clavicles, Scapulae, Humeruses
Posterior (dorsal) portion runs horizontalish
Anterior (ventral) portion runs at angle
The 1st rib-smaller and broader and the 2nd rib right under 1st

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25
Q

Can you identify these things on the following lateral chest x-ray?
Trachea
L/R hemidiaphragms and costophrenic angles
Heart areas
Aorta, pulmonary trunk, aortopulmonary window
Lateral spaces
vertebrae

A

This is not a good retrotracheal space and we should be worried mass/abscess right there

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26
Q

What are the indications for a CXR?

A

This is often the initial imaging modality. Indic: SOB, dyspnea on exertion, chest pain (aortic dissection), Hemoptysis (lung mass, cancer, CXR), increased sputum production

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27
Q

List the things that need to be assessed to determine the quality of a chest x-ray.

A

Rotation
Inspiration
Position
Exposure
Left hemidiaphragm should be visible to the spine. Thoracic vertebrae should be barely visible behind the heart. Lung markings should extend to the edges (vessels inside the lungs we’re seeing). CXR has the most quality markers. Make sure there is good exposure.
Look at rotation of spinous processes and these should be midway between the medial ends of the clavicles. Make sure positioning is good and the spinous processes should be equidistant from the medial end of each clavicle. Check degree of inspiration and rib counting- should see 9-10 posterior ribs/ 6-7 anterior ribs

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28
Q

Explain the appearance of a chest x-ray if it is over-exposed versus under-exposed.

A

Make sure there is good exposure.
Too dark means there is over penetration and overexposure. Too bright it is underpenetrated and underexposed.

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29
Q

What is this pathology?

A

tram tracks
Bronchiectasis is often described as “tram tracks” more severe inflammation in the walls of the bronchus. These are pts that we initially think have COPD and it doesn’t get better. Walls of bronchi are thickened, inflamed, and floppy. Seen better on CT. Two parallel white lines= tram tracks

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29
Q

What is this pathology?

A

Steeple sign: Kids croup
A narrowing at the start of the trachea and is characterized by laryngotracheal inflammation.

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30
Q

What is this pathology?

A

Trachieal deviation. Both of these pictures are masses. Masses like lymphoma, sarcoidosis, enlarged mediastinal lymph nodes
Tension Pneumothorax (collapsed lung)
Prior pneumonectomy (where they have taken out a lobe of the lung, now there are pressure differences between the lungs)

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31
Q

What is this pathology?

A

Bronchial occlusion. Left main bronchus occlusion on left and right upper bronchus occlusion with superior lobe occluded on right image. If it is along the lines of the lobes, this gives a clue that it is not an infection, but an occlusion. Often causes collapse of lung tissue distal to obstruction. Examples include the left main bronchus obstruction and the right upper bronchus obstruction. The collapse will appear brighter and if you see whiteness, this is most likely an occlusion of the bronchus. Can happen in single lobes as well like seen in image 2. Follows lines of fissure of the lobes. Can be caused by thick mucus secretions and plugs (bronchoscopy)

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32
Q

What does fungal pneumonia look like on CXR?

A

Often bilateral because we are inhaling fungal spore. scattered denser areas in more diffuse ground glass opacities

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32
Q

What is this pathology?

A

gGroung class opacities. Bottom left is normal. Viral pneumonia-tends to be bilateral and less dense on xray (“ground glass”), Covid, flu

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32
Q

What is this pathology?

A

peribronchial cuffing
Inflammation of walls of smaller bronchi/mucus plugging of small bronchi. Common causes are: bronchiolitis (RSV), cystic fibrosis. Developmental disorders in premature infants. Look like a bright ring with a dark center. We are seeing the ring of the wall inflamed.

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33
Q

What is this pathology?

A

Bacterial pneumonia- tends to be lobar, unilateral, consolidation, wont follow lines of the lobes. Can often see a consolidation of dense area of fluid/inflammation
‘lobar’= confined to a lobe

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33
Q

What is this pathology?

A

Aspiration pneumonia -more likely in lower lobes and more common to end up in the right than left.

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34
Q

What is this pathology?

A

costophrenic angle blunting
PLEURAL EFFUSION. Blunting of the costophrenic angles (bilateral vs unilateral)
Fluids work with gravity and therefore collect at the lowest point. Supposed to be able to see costophrenic angles.
Bilateral: heart problems, backup of fluid. heart failure, liver cirrhosis, nephrotic syndrome, and pancreatitis. Usually non-pulmonary reasons
Unilateral: masses, lung issues-pneumonia and cancer. Usually pulmonary causes.
Bilateral on the left. Unilateral on the right.
Effusions can take up the whole lung, we would just see a little white around the encasing of the lungs.

35
Q

What is this pathology?

A

Cavitary lesion in the right lung is fluid filled. cancers can cause this. We can figure out sometimes based on sx and CT, but we will most likely need to biopsy it.

36
Q

What is cavitary lesion?

A

Walled off area in the lung caused by an air or fluid level imbalance. Common causes include: TB, fungus like aspergillosis, infection, and malignancy

37
Q

What is this pathology?

A

Cavitary lesion in the left lung inferior lobe that is not fluid filled.

38
Q

Describe how to determine if an infection is more likely bacterial vs viral vs fungal on a chest CT.

A

Bacterial: lobar, segmented, localized
Viral: diffuse or peribronchial ground glass opacities, bilateral or diffuse
Fungal: diffuse or focal nodules, cavitary lesions, or ground glass opacities

38
Q

Describe how to determine if an infection is more likely bacterial vs viral vs fungal on a chest x-ray.

A

Bacterial: lobar, unilateral, consolidation, may include air bronchograms, costophrenic angle blunting
Viral: tends to be bilateral and less dense on xray (“ground glass”), Covid, flu, hyperinflation, thickening of bronchiole walls
Fungal: bilateral, inhaling fungal spore. scattered denser areas in more diffuse ground glass opacities

39
Q

Compare and contrast the advantages and disadvantages of a CT chest versus a chest x-ray.

A

Advantages to chest CT: No superimposed structures, high definition of structures, better visualization of nodular opacities, can image vasculature with contrast, high resolution, thin slices, 3D reconstruction often available.
Disadvantages to chest CT: Radiation exposure increased, not portable and there is risk of contrast with allergies
CXR: accessible, cost effective, less radiation, quick

39
Q

What are the indications for a CTA chest?

A

IV contrast can be used to enhance vasculature. Indications: SUSPECTED PE, suspected dissection/rupture, evaluate aneurysm, evaluate masses.

40
Q

What are the indications for a V/Q scan?

A

Often used to look for PE when CTA can’t be done (contrast allergy or kidney disease). Suspected PE and are unable to do contrast

41
Q

Describe how to interpret V/Q scan results.

A

Ventilation scan: nuclear med radioactive aerosol (that doesn’t cross the alveolar membrane, does not get into bloodstream) breathed in/out and images are taken
Perfusion scan: radiolabeled albumin (albumin preferentially goes to the lungs. very low dose, not worrisome after) injected into venous system, aggregates in pulmonary capillaries and images taken.
Compare the two. Images scanned for mismatch-area with ventilation but not perfusion indicates PE (specific for pulmonary embolism and reported as low, intermediate, or high probability).

42
Q

What is the gold standard for visualization of pulmonary arteries?

A

Pulmonary angiography and fluoroscopy x-ray
Dye injected into pulmonary arteries individually and images taken. Minimally invasive-right heart catheterization. Gold standard for blood clots, but rarely done

43
Q

What is this pathology?

A

This would be going to the left lung. Will usually go into either the radial artery or femoral artery. There are risks for perforation.
They can dissolve the clot by dripping TPA on it or they can go in and physically pull the clot out.

44
Q

Describe the appearance of the following on an ultrasound:
pleural effusion

A

US done at the bedside. Blood or fluid is black. Guiding the needle for thoracentesis.
Pleural effusion- dark area between lung and liver/diaphragm

45
Q

Describe the appearance of the following on an ultrasound:
pneumothorax

A

Normal pleural line- bright white where movement of pleura is noted in normal lung-sliding and PTX air space if noted, no sliding of pleura
The visceral and parietal pleura slide against each other. Pneumothorax will not slide, looks like a barcode.

46
Q

Describe the appearance of the following on an ultrasound:
pneumonia

A

Areas of consolidation appear more heterogenous and bronchograms are noted as bright

47
Q

Describe the Allen test and how to interpret it.

A

To assess adequate blood flow of the hand. We would hold/occlude both the radial and ulnar arteries and we will ask the patient to make a fist and then release and we will release pressure form the ulnar artery. If the hand gets its red color back, the test is positive, and this means there is good blood flow through the ulnar artery. If it is negative, it will look like the picture on the right. Right side picture is a negative allen test
Positive means good blood flow and this means we are good to proceed

48
Q

What are the indications for an ABG?

A

They are used to assess O2 status and acid/base balance, from the arterial side. They are typically analyzed using blood drawn from radial artery (respiratory therapists)

49
Q

What are the contraindications for an ABG?

A

These are absolute. Poor blood flow to the hand determined by the allen test. Known vascular disease in the extremity, AV fistula for dialysis in the extremity (because there is a device that causes connection between the venous and arterial systems), expected need for dialysis in near future (don’t want to mess up vasculature). Relative contraindications: bleeding disorder or on a blood thinner, low platelet count.

50
Q

What are the components of an ABG?

A

PaO2, SaO2, PaCO2, pH, HCO3-

51
Q

What are the normal ranges of PaO2?

A

Partial pressure of oxygen (mmHg)- not the same as O2 saturation and normal 80-100 mmHg with 75-100 mmHg

52
Q

What are the normal ranges for SaO2?

A

Finger monitor and O2 saturation of hemoglobin and Normal 94-100%

53
Q

What are the normal ranges for pH?

A

Normal 7.35-7.45
<7.35 acidosis
>7.45 alkalosis
For ABG analysis anything less than 7.40 is acidic and anything above is alkalytic. Anything outside the normal ranges indicates that the blood is acidic or alkalotic.

54
Q

What are the normal ranges for PaCO2?

A

Partial pressure of CO2 (mmHg) Normal 35-45 mmHg
>45=acidosis
< 35 alkalosis
For ABG analysis, determine if acidosis or alkalosis on 40
Anything outside the normal ranges indicates that the blood is acidic or alkalotic.

55
Q

What are the normal ranges for HCO3-?

A

Bicarbonate level
Normal 22-26 mEq/L <22= acidosis and >26 alkalosis

56
Q

What two systems respond to pH changes in the blood?

A

respiratory and renal systems

57
Q

What does the renal system do in response to change in the blood pH?

A

Regulate HCO3-
Longer term response -> 3-5 days to fully adapt
H+ + HCO3- <-> H2CO3 <-> CO2 +H2O

58
Q

What does the repsiratory system do in response to change in the blood pH?

A

Regulates level of CO2 by increasing/decreasing rate of respiration. Change in level of CO2 will cause new equilibrium for this equation: H+ + HCO3- <-> H2CO3 <-> CO2 +H2O. Lungs= quick response to pH changes-> minutes

59
Q

How does the respiratory system respond to acidosis?

A

Increased respiration rate= more CO2 exhaled= decreased CO2 in blood. Equilibrium shifts to right, reducing H+. Pt will be breathing fast (RR is faster, tachypnic- they are either hypoxic or acidotic)

60
Q

How does the renal system respond to acidosis?

A

Reabsorb more HCO3- and excrete more H+
Body is acidic, urine is acidic, longer term response- 3-5 days to fully adapt

61
Q

How does the respiratory system respond to alkalosis?

A

Decreased respiration rate= less CO2 exhaled= increased CO2 in blood. Equilibrium shifts to left, increasing H+

62
Q

How does the renal system respond to alkalosis?

A

Excrete more HCO3- and reabsorb more H+
longer term response- 3-5 days to fully adapt

63
Q

What are the four major acid base disorders?

A

Respiratory acidosis, respiratory alkalosis, Metabolic acidosis, metabolic alkalosis

64
Q

What is respiratory acidosis?

A

Increased pCO2 leads to decreased pH. rate of ventilation of CO2 is decreased (COPD, asthma attacks, pneumonia, narcotics- RR drops significantly 4-6 per minute and they die from build up of CO2)

65
Q

What causes respiratory acidosis?

A

Decreased ventilation:
Decreased movement of air to the alveoli (Decreased respiratory rate- strokes. Obstruction to flow- bronchial occlusion, mass)
Decreased gas exchange in the alveoli (Increased fluid-pneumonia, pneumonitis. Decreased surface area- emphysema, pulmonary fibrosis).
Decreased perfusion: think PE

66
Q

What are some sx of respiratory acidosis?

A

HA, tachycardia, CNS depression, cardiac arrhythmias, lethargic, confused

67
Q

What are some compensatory mechanisms of respiratory acidosis?

A

Kidneys renin, HCO3- and excrete H+, takes 3-5 days

68
Q

What is the therapy for respiratory acidosis?

A

Artificial ventilation, measures to improve ventilation. CPAP, BiPAP

69
Q

What are some common causes of repsiratory acidosis?

A

Central: opiate overdose, stroke, status epilepticus, decreased rate of breathing. Perfusion abnormalities: PE, cardiac arrest, decreased perfusion. Airway abnormalities: obstruction, asthma, COPD, anaphylaxis, decreased flow. Neuromuscular: spinal cord, injury, Guillian-barre syndrome, myasthenia gravis, MS, diaphragm paralysis, decreased flow and rate. Alveolar issues: pneumonia, emphysema, and effusion

70
Q

What are some sx of respiratory alkalosis?

A

Lightheadedness, CNS irritability (muscle spasms, alt mental status), cardiac arrhythmias

70
Q

What causes respiratory alkalosis?

A

HYPERVENTILATION. Anxiety, fear, stress, CNS disease, drug use (salicylates (aspirin OD starts this way), pregnancy (diaphragm can’t go down as far), sepsis, liver disease, hypoxemia, low O2 (PE, anything that causes this)

70
Q

What are some compensatory mechanisms for respiratory alkalosis?

A

Kidneys excrete HCO3- and retain H+

71
Q

What is respiratory alkalosis?

A

Decrease pCO2 leads to increased pH. Blowing off more CO2. rate of ventilation of CO2 is increased (anxiety, fever, PE)

71
Q

What are some therapies for respiratory alkalosis?

A

Anti-anxiety measure, O2 if due to hypoxia, CO2 rebreathing

71
Q

What are some common causes of respiratory alkalosis?

A

Central: anxiety, head trauma, brain tumors, salicylates OD, fever, pain, pregnancy
Peripheral: hyperventilation due to low O2 state, such as PE, pulmonary edema, altitude
Iatrogenic: mechanical ventilation (intubation)
Normal for pregnant females to have respiratory alkalosis- stimulated by progesterone
Salicylate (aspirin) poisoning: respiratory alkalosis- metabolic acidosis
rebreathing= paper bag

72
Q

What is metabolic acidosis?

A

Decreased HCO3- leads to decreased pH. either- increased production of an acid (lactic acid from sepsis, ketoacids) OR increased loss of bicarbonate (usually secreted by pancreas and reabsorbed in the large intestine, if not reabsorbed, can be caused by diarrhea)

73
Q

What is metabolic alkalosis?

A

Increased HCO3- leads to increased pH. either: increased bicarbonate (tums) OR increased loss of acid (vomiting)

73
Q

What are some sx of metabolic acidosis?

A

HA, CNS depression, altered mental status

73
Q

What are some causes of metabolic acidosis?

A

Acid production or too much HCO3- lost (diarrhea)
Acid production-poisoning (methanol poisoning), abnormal metabolism (ketoacidosis, lactic acidosis), shock/low perfusion.
Loss of base (HCO3-)- diarrhea, pancreatic fistula- losing bicarb from the colon

74
Q

What are some therapies for metabolic acidosis?

A

Correct underlying cause (ketoacidosis, diarrhea, etc.). Improve tissue oxygenation (lactic acid). Consider giving NaHCO3 (sodium bicarbonate-fluid) if pH < 7.0. Can prescribe bicarb but DON’T DO IT ON YOUR OWN. very dangerous because it is typically IV

74
Q

What are some compensatory mechanisms for metabolic acidosis?

A

Lungs excrete more CO2 by increasing rate of respiration (Kussmaul breathing)

75
Q

What are some causes of metabolic alkalosis?

A

Base accumulation-excessive antacid intake (tums), blood transfusion. Loss of acid-vomiting, gastric suction (NG tube), diuretic use (trigger kidneys to excrete out acid)

76
Q

What are some sx of metabolic alkalosis?

A

CNS irritability, eventual CNS depression

77
Q

What are the ions involved in the anion gap?

A

Amount of cations (+) has to equal the amount of anions (-).
Major cations: Na+, K+
Major anions: Cl-, HCO3-, phosphate (PO4-)

77
Q

What are some compensatory mechanisms of metabolic alkalosis?

A

Lungs retain CO2 (decrease rate of respiration)

77
Q

What are some therapies for metabolic acidosis?

A

Reverse underlying cause (antacids, low Cl-, low K+, vomiting). Almost never give acid (HCl)

78
Q

What are some causes of metabolic acidosis?

A

Caused by administration of base or removal of H+ causes increased HCO3- concentration in extracellular fluid. Volume depletion releases aldosterone and causes reabsorption of Na and HCO3-. Most common cause= vomiting or diuretic use. Vomiting -> chloride leaves body and is replaced by bicarbonate

78
Q

Explain how to tell if a respiratory acidosis is acute or chronic

A

Acute: (no compensation, kidneys have not compensated yet) CNS depression-drugs (opiod OD), acute stroke, etc. Acute airway obstruction (aspirations), severe pneumonia or pulmonary edema
Chronic: (compensation-kidneys have a chance to work) Chronic lung disease (COPD, bronchiectasis, pulmonary fibrosis), Chronic neuromuscular disorders (muscular dystrophy, ALS), Chronic respiratory center depression-central hypoventilation and obesity

79
Q

What are we measuring in the anion gap?

A

Easier to measure Na, K, Cl, HCO3-. Hard to measure total PO4- -> most is bound to organic molecules
The difference between measured anions and measured cations. Normal anion gap can be calculated from values on BMP.

80
Q

Describe what an anion gap is, and how to calculate it.

A

Anion gap= Na - (Cl+ CO2) = 8-16 meq/L when we dont use potassium
When the gap is over 20: this is concerning for an extra acid present

81
Q

List possible causes of a high anion gap metabolic acidosis.

A

(HAGMA) Anion Gap > 20
Ketoacidosis (diabetic/alcholic/starvation), renal failure, lactic acidosis, rhabdomyolysis (crush injury or trauma), toxins (methanol, ethylene glycol-antifreeze, paraldehyde, salicylates)

82
Q

What is the mnumonic to remember some of the causes of a high anion gap?

A

Retention of 1 or more unmeasured anions. Weak base from acid accumulation. An acidosis going on
M: Methanol/metformin (lactic acidosis, very uncommon but deadly and this is a common med for diabetics)
U: uremia (high BUN)
D: Diabetic ketoacidosis
P: paraldehyde/ phenformin
I: iron/isoniazid (TB)
L: lactate
E: ethylene glycol (antifreeze OD)
S: salicylates (aspirin)

SCUMPILED is similar but includes cyanide

83
Q

List possible causes of a normal anion gap metabolic acidosis.

A

(NAGMA)
GI bicarbonate loss (diarrhea/urethral diversions)
Renal bicarbonate loss (renal tubular acidosis, early renal failure, carbonic anhydrase inhibitors, aldosterone inhibitors)
HCl administration
Post Hypocapnia