PT4 - Health Flashcards

1
Q

How do you recognise health?

A
  • social & economic environment
  • physical environment
  • persons individual characteristics and behaviours e.g. skin, hair, nails, demeanour
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2
Q

What is health?

A
  • being able to do what you need/want to do (agency)
  • the context of people’s lives determines their health
  • health is not always within someone’s control e.g. wealth
  • ability to adapt to internal (mental/emotional, chemical, physical) and external environment
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3
Q

What are the 3 health categories?

A
  • psychological (mental/emotional/social)
  • chemical (disease processes e.g. diabetes)
  • physical (NMS e.g. dysfunction/tissue damage/OA)
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4
Q

List the 3 type of patient

A
  • simple - largely biomechanical, simple story, often young
  • complex - mixed picture, yellow flags (social and psychological barriers), often older
  • complicated - comorbidities (chemical), multiple flags (yellow - social, orange - psychological), chronic, shared management
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5
Q

What determines health?

A
  • income + social status
  • education
  • physical environment
  • social support network (families, friends, community)
  • genetics
  • personal behaviour
  • health diseases
  • gender - types of disease/life expectancy
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6
Q

What are personal health determinants?

A
  • nutirtion
  • activity
  • smoking
  • drinking
  • coping skills/habits - locus of control
  • previous experiences and outcomes - expectations
  • approach to life’s stresses/challenges - opportunities/threats
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7
Q

How do health determinants and personal behaviour explain adaptation?

A

+ health domains (chemical, physical, psychological)

+ health determinants (income, status, education, physical environment, support, genetics, behaviour, disease, gender)

+ personal behaviour (nutrition, activity, smoking, drinking, skills/habits, experiences, outlook)

=> influence individual adaptability and responsiveness to challenge (domain theory + BPS model) ==> opportunity for change

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8
Q

What is dysfunction?

A
  • NMS functional loss
  • acute traume => weakness, reduced/increased ROM
  • chronic loss of function
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9
Q

What is disease?

A
  • pathophysiological change => symptoms
  • subclinical (elevated BP)
  • clinically present (w or w/o diagnosis)
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10
Q

What is maladaption?

A
  • failure of body to function in normal fashion due to dysfunction or disease
  • lack of adaptation => function loss
  • loss of agency
  • can involve BPS
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11
Q

What is the Big Picture?

A
  • everything that contributes to a person in their ecology, agency, lifestyle and personal health determinants
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12
Q

John or Jayden - ideas for prognosis, who might have the more favourable prognosis and what factors influence your decision?

John & Jayden present with R medial knee pain of 3/12 duration after a mild valgus knee strain injury on a ski trip. They are both healthy with no comorbidities

John is 30 YO computer programmer who does little exercise and is a little overweight.

Jayden is a 30 YO gym instructor who trains 3 hours on 5/7 and has a good BMI

A

Fiona Hendry Balloon Model of Stress

John => small balloon, runs more at minimal loading, tissues are not used to load, more likely to get an injury, felt the issue more, less force, easier to treat, low grade exercises should have a big impact on him

Jayden => big balloon runs closer to ‘maximal’ loading all the time, more significant to cause injury to his tissues as they are more robust, should have probably healed by now. Need to change how they are using their body or reduce forces on his body to see change

Cross training expands the balloon for the person

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13
Q

What happens as a person ages?

A
  • kyphosis due to spine becoming more osseous
  • thorax gets arthritis first
  • centre of gravity moves forwards
  • more weight on toes
  • more likely to catch toes on curb stones => PT reflexes are slow + joint Proprioception diminished => get PT to look forwards
  • suggest walking heel first with older PT => heel pushed back centre of gravity
  • agency very important => being able to self-care
  • average life expectancy in care home = females: 3 years, males: 6 months
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14
Q

Where are forces most likely to be felt in bodies?

A
  • back
  • neck
  • knees
  • shoulders
    -hips
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15
Q

What happens in the shoulders as we age?

A
  • forces travel in straight lines
  • rotator cuffs work in straight lines
  • shoulder slump => forces felt more as harder to move through joint of rounded shoulders => compromising GH + complicated by thorax in kyphosis
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16
Q

How do muscles work @ a microscopic level?

A
  • shorten on contraction (using ATP)
  • actively get food (ATP) + stretching => DOMS
  • overworking MM => tightness
  • compresses arteries and veins as thinner walls => occludes drainage => toxins can’t drain away
  • increase in metabolites
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17
Q

What happens when you cross fibre stretch a MM?

A
  • drain metabolites out
  • increased heat due to improved blood flow
  • increase water + nutrition to replenish myotendons
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18
Q

What do myotendons do?

A
  • transmit large forces from MM to tendon
  • dissipitate forces (spring)
  • weak link => common location for MM strain
  • strain can be prevented by heavy eccentric exercise
  • e.g. biceps femoris, biceps brachii, quadratus femoris
  • interdigitations of MTJ shorten with age -> less contact area for force transmission => increase risk of injury
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19
Q

What are the grading of MTJ injuries? (Myotendonous Junction)

A
  • 1: Mild strain => interstitial edema + fluid/hemorrhage around MTJ
  • 2: Moderate strain => intramuscular hematoma + perifascial fluid/hemorrhage
  • 3: severe strain: MTJ tear + laxity
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20
Q

How long do MM, tendons, myotendons, cartilage and bone respond to training?

A
  • MM: 2-3 weeks
  • tendons + myotendons: 4-7 weeks
  • cartilage + bones: 2-3 months recovery
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21
Q

What is the little picture?

A
  • local areas causing symptoms + dysfunction
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22
Q

What concepts should you explore in the little picture?

A
  • force transmission
  • knowledge of structure/function relationships
  • developmental + aging changes on structure/function
  • adaptability
  • compensatory demand
  • knowledge of effects of trauma/dysfunction/pathology
  • normal/abnormal - function/loss of function
23
Q

What it’s the role of forces in NMS dysfunction?

A
  • NMS dysfunction => failure to absorb or dissipitate forces effectively
  • e.g. sudden force causing clear tissue damage OR low grade repetitive forces on a particular area (OA due to altered gait mechanics => repetitive stress/overuse elsewhere)
24
Q

What is the role of bone in force transmission?

A
  • connective tissue
  • trabeculae to absorb forces and transmit forces to tissue to dissipate
  • responds to demand/flex/bend
  • loss of resilience as blood supply diminishes with age (femoral neck shaft angle)
  • brittle with age
25
Q

What is the role of articular cartilage in force transmission?

A
  • allow close joint approximation for effective force transference
  • designed for compression
  • regular compression drive fluid health/metabolic turnover/nutrition
26
Q

What is the role of ligaments?

A
  • maintain joint congruity
  • force transference during locomotion
  • variable importance relative to joint e.g. GH vs hip
27
Q

What is the role of fascia?

A
  • transmission of forces to where they can be dissipated + managed
  • exercising => MM tightening => more difficult to dissipate forces
  • connecting
  • MM embedding
  • Tensegrity
28
Q

What should a normal spine be able to do?

A
  • remove lordosis + kyphosis in spine
29
Q

What should happen when you side bend:

A
  • should use hips + SIJ
30
Q

What should happen when you move the csp?

A
  • rotation should translate down to thorax
  • shoulders should not move up too quickly
31
Q

What is the lumbar-sacral joint designed for?

A
  • rotation
32
Q

What happens to the tsp in >40 yo?

A
  • ossifies
  • responds well to Axx
33
Q

What transmits forces around our bodies?

A
  • fascia => while walking engaging with myofascia
34
Q

What are the origins and insertions of the iliolumbar ligament?

A
  • TP L5
  • Iliac Crest
35
Q

What is the significance of the iliolumbar ligament?

A
  • when walking legs swings forwards + bringing TP in spine towards ilium
  • opposite shoulder rotates
  • tension is built by ligaments
  • in children iliofemoral ligament tight to start with => need posterior chain MM to be able to sit
  • core MM health + MM capacity of ligaments
  • as child starts to stand => iliofemoral ligament kicks in
  • child has wide base due to lack of proprioception
  • iliofemoral ligament stretched out as standing
  • SIJ => plane joint, non-congruent (smooth) as needs to be able to glide
36
Q

What happens in children under 10 years old to the iliofemoral ligament?

A
  • vestibular system is one balance sensation fully formed - proprioceptive + vision is impaired
  • iliofemoral ligament is tight => not able to sit as posterior chain MM needed + core MM + mm capacity of ligaments
  • as stand iliofemoral ligament stretched (between 1-2.5 YO)
37
Q

What type of joint is the SIJ?

A
  • non-congruent plane joint
  • smooth as needs to be very mobile for walking
38
Q

What is a primitive reflex?

A
  • involuntary motor responses originating in the brainstem present after birth in early child development that facilitate survival

E.g. upwards curl of toes (Babinski reflex) < 9 months old once walking this changes to downward turning of toes in adults

39
Q

How many primitive reflexes are we born with?

A
  • over 200
  • before 38 weeks, no sucking reflexes, these muscles must be inhibited for MM to override control of body
40
Q

At what age does adaptive balance kick in?

A
  • 6 YO => able to do star jumps
41
Q

Why is fascia good for force transmission?

A
  • invested in tiny muscles => allows for force transmission
42
Q

What do bones do for our bodies?

A
  • provide us with shape
  • can be osteoporotic, however, activity maintains myofascia
43
Q

What is the most common joint for OA?

A
  • knee => cartilage breaks off
  • vulnerable due to q angle in hips, especially medially
44
Q

What is the role of lifespan factions on dysfunction?

A
  • congenital anomalies => usually well compensated => reduce adaptability
  • increase risk factors if force transmission affected e.g. sacralisation (lumbar and sacrum become fused, possibly due to ossification of lumbo-sacral ligament calcification)
  • human development - acquired changes => can be well compensated, but reduce adaptability e.g. idiopathic scoliosis, PSLE (progressive systemic lupus Erythematosus), limb fractures, postural changes, co-morbidities
45
Q

What is the impact of emotional stress on NMS?

A
  • cortisol => long + short term
  • increased MM contraction => reduced fluid flow in MM + reduce ROM in joints
  • MM tissue reduced elasticity
  • ST => influence on short MM/fluid flow + increase serotonin + dopamine; may reduce insulin + cortisol lowering HR + BP
  • breathing rate + type => upper rib breathing => increase respiratory rate + increased risk of COPD/asthma attacks + constriction of nasal passages
  • other hormones => adrenaline + noradrenaline => increased blood pressure/HR changes
  • increased inflammation
  • glucocorticoids (+ cortisol) regulate immune system + inflammation
  • chronicle stress => impaired communication between immune system + HPA axis (hyperthalamic-pituitary-adrenal)
  • linked to development of physical + mental health conditions => chronic fatigue, metabolic disorders (diabetes, obesity), depression and immune disorders
46
Q

What is the impact of physical stress on NMS?

A
  • nerves - 3 days (neural response)
  • MM - 4 weeks
  • Tendons, myotendons - 8 weeks
  • ligaments - 10 weeks
  • Articular cartilage - 18 weeks
  • Bone - 18 weeks
47
Q

What are the features of an Acute NMS presentation?

A
  • sudden onset => may or may not be trauma
  • pain
  • loss of function
  • may include inflammation/tissue damage => may not be obvious
48
Q

Questions to ask regarding trauma related onset:

A
  • does onset correlate well to S+S?
  • what does the speed of onset tell us?
  • what does the current duration tell us?
  • what factors are affecting/delaying recovery?
  • Is this a simple, complex or complicated presentation?
49
Q

Questions to ask with an acute onset NMS non-trauma related onset:

A
  • does Hx correlate well to S+S?
  • is this due to acute failure of compensation for changes/tissue damage elsewhere?
  • is this an acute presentation of a chronic maladaption?
  • what factors are affecting/delaying recovery?
  • is this simple, complex or complicated presentation?
50
Q

How do chronic patients present with NMS complaint?

A
  • slow/intermittent onset
  • gradually worsening
  • no clear story
  • occasionally inappropriate beliefs re: story
  • is this a chronic failure of compensation for changes/tissue damage elsewhere?
  • what factors are affecting/delaying recovery?
  • is this a simple, complex or complicated presentation?
51
Q

What is the trendelenberg testing?

A
  • hip abduction
  • rare to get nerve impingement
  • tendons and ligaments take load after MM fatigue
  • slow to respond to TT and repair => other MM recruited to help
  • what is the competence of the lower back?
  • testing abduction integrity + force transmission
52
Q

Why is the joint capsule important?

A
  • maintaining joint health
  • if you do surgery, then alpha-motor reception decreases
53
Q

What 3 things do we need to do for PTs?

A
  • educate PTs
  • TTT to assist in health
  • provide Exx