Psychopathology P1 Flashcards

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1
Q

Statistical infrequency (def of abnormality)

A

= When an individual shows statistically rare behaviours that fall more than two standard deviations below or above the mean. Rare characteristics like being more depressed or less intelligent than the standard population.
- Used to measure the frequencies of behaviours/traits on a normal distribution curve.

In a normal distribution, most people have a score ranging from 85-115. Only 2% of people has a score below 70. Those individuals are very unusual or ‘abnormal’ .

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2
Q

deviation from social norms (def of abnormality)

A

= Behvaiours that are different from the accepted standards in a society
- norms are specific to the culture we live in (ie homosexuality may be seen as abnormal in certain cultures, but normal in others)
EXAMPLE:
- People with antisocial personality disorder (psychopaths) do not conform to our moral standards as they have an absence of prosocial internal standards associated with failure to conform to lawful and culturally normative ethical behaviour.
- skipping a queue
- invading someone’s personal space

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3
Q

Failure to function adequately (def of abnormality)

A

= Occurs when someone is unable to cope with ordinary demands of day-to-day living (ie unable to maintain basic nutrition and hygiene, cannot hold down a job or maintain relationships)

Rosenhan and Seligman’s additional signs of when someone is failing to function adequately…

1) when a person’s behaviour becomes irrational or dangerous to themselves or others
2) when a person experiences severe personal distress
3) when a person no longer conforms to standard interpersonal rules (ie holding eye contact)

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4
Q

Deviation from ideal mental health (def of abnormality)

A

= Occurs when someone does not meet a set of criteria for good mental health (what makes someone normal> what makes someone abnormal).
- idea behind this to acknowledge what is meant to be psychologically healthy so that we can begin to identify how we can reach this as as a goal/target in therapy.

Jahoda’s 6 criteria:
- we self actualise (strive to reach our potential)
- we can cope with stress (resistant to stress)
- we have environmental mastery (adapt to change in env)
-accurate perception of reality
- we have autonomy (freedom of choice)
-we have positive self attitudes

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5
Q

Deviation from social norms evaluation

A

Pro:
1) Real world application -> Used in clinical practice. For example, it helped the diagnosis of defining characteristics of people with antisocial personality disorder as a failure to conform to culturally normal ethical behaviour. Which is why they are often reckless, aggressive and violate the rights of others. All of these are deviations of social norms which therefore shows it has value in psychiatry.

Cons:
1) Culturally relative -> DFSN vary between different cultures. A person from one culture may label another as abnormal using their standards which may not cohere to that of another culture (ie hearing voices is the nrom in some cultures - as messages from ancestors - but in the UK is seen as abnormal/ homosexuality/ drinking alcohol at 18). Therefore, it is difficult to judge DFSM across different cultures so it is not a universal, stable criterion.
2) Social norms change overtime 0> This means the era in which a behaviour is being displayed, effects our perception of it (i.e homosexuality was illegal in England until the 1970’s and now the social norm has changed. Thus, it is a limitation as it suggests that as social norms change, our definition of abnormality does as well. (never consistent/confusing)

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6
Q

Failure to function adequately evaluation

A

Pro:
1) Sensible threshold for when people need professional help
= Most of us have symptoms of mental disorder to some degree at some point. According to the mental health charity Mind, around 25% of people in the UK will experience a mental health problem in any given year. However, many people press on in the face of fairly severe symptoms. It tends to be at the point that we cease to function adequately that people seek professional help what are noticed and referred for help by others. Therefore, meaning that treatment and services can be targeted to those who need the most.

Con:
1) Too subjective -> Too easy to label nonstandard lifestyles as abnormal. It’s very difficult to say when someone is really failing to function and when they have simply chosen to deviate from the norms. For example, those who favour high risk leisure activities could be classed as irrational and perhaps a danger to themselves. Due to rosenhan and seligman. Therefore, people who make unusual choices are at risk of being labeled abnormal and therefore their freedom may be restricted.
-> Relative to the situation: sometimes valid function adequately is the right response. E.g. Mum dies.

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7
Q

Deviation from ideal mental health evaluation

A

Pro:
1) Positive/ Optimistic approach -> Focuses unhealthy behaviour over unhealthy behaviour. This is because it focuses on how people strive to become normal, rather than focusing on problems and maladaptive behaviour. Therefore, allowing people who are struggling with their mental health to acknowledge what is meant by ‘normal people’ and gives them a clear and coherent goal to work towards.

Con:
1) Cannot be universally applied due to Western/Individualistic bias -> Some of Jahoda’s criteria are firmly located in the context of Western Europe and America. For example, self-actualization would probably be dismissed as self-indulgent in much of the world. Collectivist countries such as China are less likely to focus on each individual’s ability to reach self-actualization whereas countries like England, which are western/individualistic countries are likely to prioritize the individuals needs over what society needs. Therefore, it is difficult to apply this approach from one culture to another, suggesting it lacks replicability?

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8
Q

Statistical Infrequency evaluation

A

Pro:
1) Real world application (objective) -> Used in clinical practice as a diagnosis to assess severity of an individual symptoms. For example a diagnosis of intellectual disability requires an IQ or below 70. Due to its quantitative and objective nature, it is easier to diagnose if researchers can visibly see when an individual’s IQ deviates from the mean (infrequent). Therefore, useful diagnostic and assessment process.

Con:
1) Infrequent characteristics can be positive> negative -> For every person with an IQ below 70, there is a person with an IQ above 130. Yet we would not think someone as abnormal for having a high IQ. Similarly, we would not think of someone with a very low depression score on a BDI as abnormal. These examples show that being unusual or at the end of a psychological spectrum does not make someone abnormal. Therefore, statistical infrequency is not sufficient for the sole purpose of defining abnormality and maybe other definitions for abnormality are more appropriate.

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9
Q

maladaptive

A

difficulty adjusting to new environments or situations

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10
Q

phobia def

A

An irrational fear of an object or situation

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11
Q

Cognitive characteristics of phobias

A

=Refers to the process of ‘knowing’, including thinking, reasoning, remembering and believing
1) selective attention -> An individual sufferer who is presented with the phobic stimulus finds it difficult to divert their attention from the stimulus (eg an arachnophobe will find it difficult to concentrate if there is a spider in the room). Can be a strength as it gives us the best chance of reacting quickly to a threat, but a phobia is not useful as the fear is irrational.

2) Catastrophic thinking -> The person may hold irrational beliefs such as thinking spiders are deadly and that all of them will kill you. OR the person may have cognitive distortions which is when their perceptions of a phobia may be inaccurate or unrealistic (eg spiders to an arachnophobe may look like hairy monster-aliens)

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12
Q

Emotional characteristics of phobias

A

= Related to a persons feelings or moods
1) anxiety -> High levels of anxiety are produced by the worries surrounding the presence or anticipation of phobic object. This involves an unpleasant state of high arousal, preventing the person from relaxing which makes it difficult to experience any positive emotions (can be long term).

2) fear -> Is the immediate and extremely unpleasant response when we encounter or think of a phobic stimulus. This fear is much greater than is normal and is disproportionate to any threat imposed. (emotional responses are unreasonable)

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13
Q

Behavioural characteristics of phobias

A

= Ways in which people act (physically)
1) panic -> In the presence of a phobia an individual displays panic behaviour such as signs of ‘flight’ (running away, crying, screaming) or freezing. Children react differently, they throw tantrums or cling/freeze.

2) avoidance -> The person consciously goes to a lot of effort to avoid places they may encounter their phobia. For example, fear of public toilets may lead to the person having limited time spent away from their home.

3) endurance -> Alternative response from avoidance. This occurs when the person chooses to remain in the presence of a phobic stimulus (eg arachnophobe may stay in the room with the spider to keep an eye on it rather than leaving).

PEA

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14
Q

DSM-5 ( categories of phobias)

A

Diagnostic and Statistical manual of Mental disorders (5th ed)
= used to categories phobias
1) specific phobia -> phobia of an object, or situation (injections)
2) social phobia -> phobia of social situations such as public speaking or public toilets
3) agoraphobia -> phobia of being outside or in a public place

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15
Q

Behavioural explanation of phobias + examples

A

The Dual Process Model (by Hobart Mowrer)
= Said that phobias are created by…
1) initiated by classical conditioning (associations)
2) maintained by operant conditioning (negative reinforcement)

E.G-> Dentophobia (fear of dentists) is initiated by a negative previous experience.
Before C-> US (pain) = UR (fear)
NS (dentist) = no response
During C-> US+NS are paired together via associations which = UCR (fear)
After C-> NS becomes the CS(dentist) = CR (fear)

Dentophobia is maintained by operant conditioning.
- The person may avoid going to the dentist of fear of being put through pain. This means the negative consequence (pain) is avoided meaning this behaviour of avoidance is more likely to occur.
- Therefore, by using negative reinforcement the phobia of the dentist is maintained.

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16
Q

Evaluations of the behavioural explanation of phobias

A

PROS:
1) Little Albert
-> classical conditioning could create a fear of rats for Little Albert. By using the UCS(bang) which originally gave the UCR of crying, was paired with the NS (rat) during conditioning. This resulted in the baby being fearful of rats due to the negative association made between the UCS + NS .Therefore, this supports the idea that classical conditioning is effective as a behavioural explanation for phobias - giving it value.

2) Practical application: Treatment
-> If it is true that classical conditioning is an affective approach into the behavioural explanation for phobias, it could be used as a treatment. For example, if it is the association made between something negative (the UCS) and the NS, to create a phobia. We could use something positive and associate this with a fear to produce a treatment for phobias. Therefore, suggesting that the behavioural explanation for phobias has practical application to real life, giving it high replicability.

CON:
1) Irrational thinking> negative experience/association
-> REDUCTIONIST as only considers the role of learning processes, meaning this approach ignores the role of cognition (thinking) in the formation of phobias, Cognitive psychologists suggest that phobias may develop as a result of irrational thinking, not through a bad experience. (Ie arachnophobia -> mostly through irrational thoughts, catastrophic thinking>neg event). Therefore, the behavioural explanation cannot provide a complete explanation for the development of phobias.

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17
Q

Two ways of treating phobias

A
  • systematic desensitisation
  • flooding
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18
Q

Systematic Desensitisation (treating phobias)

A

= A behavioural therapy designed to reduce an unwanted stimulus (such as anxiety).
-It involves drawing up a hierarchy of anxiety-provoking situations related to a persons phobic stimulus, teaching the person to relax and exposing them to phobic situations. The person works their way through the hierarchy whilst maintaining relaxation.

The process:
1) Anxiety hierarchy
= A list of situations related to the phobic stimulus that provoke anxiety, arranged in order from least to most frightening.
2) Relaxation
= Uses reciprocal inhibition which is the idea that a person cannot be afraid and relaxed at the same time, so one emotion prevents the other.
- relaxation reached via breathing exercises, imagery techniques, meditation etc
3) Exposure
= Person is exposed to phobic stimulus in a relaxed state (starts at bottom of hierarchy, working their way up)

…Treatment is SUCCESSFULL when client can stay relaxed in situations high on the anxiety hierarchy.

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19
Q

Flooding (treating phobias)

A

= A behavioural therapy in which a person with a phobia is exposed to an extreme form of a phobic stimulus in order to reduce anxiety triggered by that stimulus.
-takes place across a small number of long (2-3 hour) therapy sessions
-breaks the association with fear
- a person cannot have high levels of anxiety for a long duration

  • Different to systematic desensitisation as you go straight to the top of the hierarchy of fear; exposure is immediate and massive (i.e an arachnophobe may be exposed to holding a tarantula instead of gradually increasing the intensity).
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20
Q

Systematic Desensitisation evaluation

A

PRO:
1) ethical
- Increases the feeling of self-control. The risk of dependence on the therapist or of perceiving improvements as being external to the patient are minimised in this technique. Therefore, due to the gradual increase in intensity, no harm to participant is activated as the therapist allows it in a time where the individual feels in control to handle.
2) evidence of effectiveness
- Lisa Gilroy followed 42 people who had SD for arachnophobia in three 45 min sessions. She found that at both 3 and 33 months, the SD group were less fearful>control group who were treated with relaxation without exposure. Therefore, showing SD is likely to be helpful for people with phobias, giving the treatment value.

CON:
1) does not work for all phobias
- Some phobias are as a result of unconscious issues rather than due to a negative experience. Therefore, lacks value as it cannot be universalised to all phobias.

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21
Q

Flooding evaluation

A

PRO:
1) cost- effective
- Clinically effective and not expensive. Can work in as little as one session opposed to say ten SD sessions to achieve the same result. Therefore, more people can be treated at the same cost with flooding>SD.

2) Ost: Supports Flooding as he found it often delivers rapid and immediate improvements, particularly when patients are encouraged to continue self-directed, exposure to feed objects and situations outside of their sessions.

CON:
1) traumatic
- Highly unpleasant experience as provokes tremendous amounts of anxiety to a phobic stimulus. This raises ethical concerns as psychologists are knowingly causing distress to clients as a treatment. No protection from harm. Also no right to withdraw .Therefore, people may avoid using this treatment as due to its distressing nature, may lead to high rates of attrition (dropping out).

22
Q

Characteristics of depression

A

Behavioural (ways in which ppl act) ->
-self harm
-disruption to sleep (insomnia/hypersomnia)
-disruption to eating behaviour (increase or decrease in appetite which leads to weight gain or loss)

Cognitive (refers to the process of ‘knowing’ inc thinking, reasoning, remembering and believing) ->
-negative thinking (inclined to pay attention to the negative aspects of life)
-absolute thinking (aka ‘black and white thinking’ or ‘polarised thoughts’, thinking a situation is all bad)

Emotional (related to a persons feelings/mood) ->
-low mood (lethargic, sadness which is persistent)
-low self esteem (which could lead to self hatred)

23
Q

Phobia vs Depression

A

phobia is an anxiety disorder whereas depression is a mood disorder (a mental disorder charcaterised by low mood and low energy levels)

24
Q

Beck’s Negative Triad (cognitive approach to explain depression)

A

=Depression can be caused by distortions of thinking (ie negative thoughts and irrational beliefs) without full awareness​.

Negative self schemas -> Ideas that an individual has about themselves which they interpret all information in a negative way.

The triad:
1)Negative views about the world = “the world is a cold place”. Creates the impression that there is no hope anywhere.
———->
2)Negative views about future = “the economy will never get better”. Reduces hope and enhances depression.
———->
3)Negative views about self(neg self schema) = “I am worthless”. Which enhances depressive feelings by confirming existing emotions of low self esteem.
——>(repeats)

25
Q

Ellis’ ABC model (cognitive approach to explaining depression)

A

= explains how individuals respond to negative events - and that if the response is negative - can lead to depression
- Ellis claimed that good mental health is the result of rational thinking, thus depression is a result of irrational thinking.

A- activating event (negative events that could trigger irrational beliefs e.g failing an exam)
B- belief (how the individual interprets the event eg maladaptive ‘faulty’ response or adaptive ‘good’ response)
C-consequence (the feelings and behaviours as a result from these beliefs eg giving up a course/developing depression)

26
Q

The cognitive approach to treating depression

A

CBT : Aims to identify , challenge and change these irrational and dysfunctional thought processes in order to treat the individual’s depressive symptoms.

Procedure:
1 -> IDENTIFY irrational/neg thoughts
= Open discussion between client and therapist to identify the clients specific negative thoughts (e.g “I will never have a successful relationship”) and their activating event (“…because all my past relationships ended with me getting hurt”).

2-> CHALLENGE and CHANGE irrational/neg thoughts
By…
a) logical disputing -> therapist questions the logic
b) empirical disputing -> therapist seeks evidence for belief
c) pragmatic disputing -> therapist questions the practical value of their thinking
= This is done to achieve COGNITIVE RECONSTRUCTING- which is a change to the way the person perceives and interprets the world.

-> Then set homework to apply to real life. For example, small achievable goals and practicing positive affirmations.

27
Q

Evaluation of Becks Cognitive Triad (cog approach to explain dep)

A

Pros:
1) Supporting research of cognitive vulnerabilities within depression
= Beck concluded that cognitive vulnerabilities (fault information processing, triad and neg self-schemas) were not only common in depressive patients but also preceded (comes before) depression.
- Cohen’s research which tracked the development of 473 adolescents, measuring their CV. Found that CV predicted later depression. Therefore, association between CV and depression. GOOD PREDICTIVE VALIDITY -> can prevent in future

Con:
1) Only a partial explanation
= It explains the association between cognitive vulnerabilities and depression, but is less useful in explaining extreme anger, hallucinations and delusions with depression.

28
Q

Evaluation of Ellis’ ABC model (cog approach to explain dep)

A

Pro:
1) Real world application (treatment)
= Uses Ellis’ REBT (rational emotive behavioural therapy) by challenging depressive patients irrational thinking. which are making them unhappy, to alter their cognitive thinking. David’s research supports REBT. Therefore has value.

Cons:
1) Partial explanation
= Only explains reactive depression, not endogenous depression.
Reactive: depression caused by a life event
Endogenous: depression caused by biology or genetics.

2) Places blame on individual
= Suggests the depressive patient has complete control and free ill on their cognitive vulnerability. Only they can change their thinking. If they are already in a low mood with low self esteem, it may lead to them giving up on trying to get better as it feels far too big of a task for them alone to conquer. Therefore, the approach could be considered insensitive and harsh on the individual.

29
Q

Evaluations of CBT (pros)

A

1) Hollon found..
-> Depressed patients relapse within 12 months …​
CBT : 40%​
Drugs: 45% ​
Placebo: 80%
Therefore, people with depression are likely to have their symptoms reduced if they engage in CBT, increasing their likeliness of increase in mental wellbeing - suggesting CBT should be offered as a successful treatment.

2) Long term effectiveness
-> result in cognitive reconstruction which is LT​
-> in contrast to drug therapy (relief of symptoms whilst on medication)​
-> less likely to suffer in future using CBT (more positive mindset)

30
Q

Evaluation of CBT (cons)

A

1) Only focuses on cognitions
= This is a problem because the negative cognitions seen in depression may be a result of other factors such as environmental factors (difficult relationships/traumatic events/biology). This means for some people CBT may be ineffective as it would not address these environmental or biological factors. Therefore, perhaps more interactionist approaches may be more effective for treating depression (whereby patient receives a range of different therapies , drug treatment and CBT, to aid their condition).

2) Depressive client require a lot of motivation and commitment
= Require to attend a number of sessions, work hard in them and complete tasks set by therapist to use in their real life. This lengthy process is very effortful which is a problem as one of the key symptoms of depression is a lack of motivation/concentration - meaning they may find it difficult to complete the techniques at a standard to be effective. Furthermore, this may lead them to view the treatment as ‘unsuccessful’ which may strengthen their negative feelings. Therefore, it is essential that patients are encouraged to complete the full course of CBT.

31
Q

Cognitive characteristics of OCD

A

(thinking)
1) obsessive thoughts -> thoughts which are recurrent and intrusive relating to a particular situation, which might be perceived as inappropriate/forbidden.

2) insight into excessive behaviour -> individuals are aware that their thoughts and behaviours are irrational and excessive.

32
Q

Behavioural characteristics of OCD

A

(acts)
1) compulsions -> The need to repeat behaviours over and over again to reduce anxiety.

2) avoidance -> Staying away from the item/situation that triggers their feared response to help reduce their anxiety . This can result in them not being able to lead a normal life.

33
Q

Emotional characteristics of OCD

A

(feelings)
1) anxiety and distress -> overpowering feeling of anxiety resulting from the obsessions and the requirement to fulfil compulsions.

2) guilt and disgust: awareness their behaviour is excessive can result in negative emotions of shame (obsessions concerning germs can result in disgust).

34
Q

OCD

A

= Obsessive Compulsive Disorder
- A condition characterised by obsessions and/or compulsive behaviour

obsessions -> cognitive (ie recurring images, thoughts etc)
compulsive -> behavioural (ie repetitive handwashing)

35
Q

DSM-5 categories of depression (4)

A
  • major depressive disorder = severe but often short-term
  • persistent depressive disorder = long-term or recurring depression
  • disruptive mood dysregulation disorder = childhood temper tantrums
  • premenstrual dysphoric disorder = disruption to mood prior to and/or during menstruation

MyPetDiedPeacefully

36
Q

DSM-5 categories of OCD (3)

A
  • Trichotillomania = compulsive hair pulling
  • Hoarding disorder = the compulsive gathering of possessions and the inability to part with anything
  • Excoriation disorder = compulsive skin picking

THE

37
Q

What are the two biological explanations for OCD

A

The genetic explanation
The neural explanation

38
Q

The genetic explanation of OCD

A

= OCD is a result of different gene combinations (polygenic condition ) past down through relatives suffering with OCD.
Research evidence = family studies indicate that a higher percentage of first degree relatives, from sufferers of OCD, also have OCD (approx 10%) —-> compared to a prevalence rate of 2% amongst the general population. This indicates that some individuals are genetically prone to developing OCD.

  • OCD is a polygenic condition (a disorder cause by a combination of genes) and is aetiologically heterogenous (a disorder caused by a variety of different gene permutations).

Genes which create vulnerability for OCD

1) SERT gene -> Responsible for regulating the transmission of serotonin in the brain. Individuals with versions of this gene that have a lower/ poorer transmission have reduced serotonin which is more likely to cause OCD.

2) COMT gene -> Responsible for producing enzymes which regulate the function of dopamine in the brain. The mutated version of COMT produce fewer enzymes, increasing dopamine, meaning the individual is more likely to develop OCD.

38
Q

Client genes (GE of OCD) def and ex

A

= Genes which create vulnerability for OCD

1) SERT gene -> Responsible for regulating the transmission of serotonin in the brain. Individuals with versions of this gene that have a lower/ poorer transmission have reduced serotonin which is more likely to cause OCD.

2) COMT gene -> Responsible for producing enzymes which regulate the function of dopamine in the brain. The mutated version of COMT produce fewer enzymes, increasing dopamine, meaning the individual is more likely to develop OCD.

39
Q

Evaluations of the Genetic explanation of OCD

A

Pro:
1) Research support : Nestadt
- Research that compared monozygotic twins (MZ) and dizygotic twins (DZ) for concordance rates of OCD. Found that MZ had a 68% cr > 31% cr for DZ twins. Thus, there is a genetic basis to OCD as the identical twins (which share 100% of genotype) were both more likely to suffer from OCD.

Con:
1) Too reductionist
= MZ are 100% genetically the same so if one twin has OCD, so should the other. But the research support showed concordance rates of 68% not 100% for MZ twins. This suggests that there must be an interaction between nature and nurture, so environmental factors must also be considered - not just genetic ones - when explaining the causes of OCD.

40
Q

Neural explanation for OCD (role of neurotransmitters)

A
  • Neurotransmitters relay information from 1 neuron to another. The neuron transmitters linked to OCD include…

1) Serotonin -> This regulates mood. So, when serotonin levels decrease it can cause OCD. This is because that can affect mood related information being transmitted appropriately and as a result increases levels of anxiety and impulsive behaviour.

2) Dopamine -> This regulates many aspects of behaviour (ie reward, motivation etc). Increased dopamine levels can cause OCD (especially compulsive behaviour) as it may result in experiencing a ‘rewarding ‘ sensation to a sequence of performing ritual behaviour and therefore continues to repeat them.

41
Q

Neural explanation of OCD (role of abnormal brain circuits)

A

THE WORRY CIRCUIT
Neurotypical brain:
- OFC (orbitofrontal cortex) sends worry signals to caudate nucleus which is responsible for interpreting ‘worry signals’
- sent to thalamus which in turn sends it back to OFC.
= When a person completes the behaviour which addresses the worry, the OFC ‘switches off’ the worry signals.

OCD:
- The caudate nucleus is damaged/ dysfunctional and so fails to suppress minor worries signals and therefore the thalamus is alerted too often.
- Heightened activity in OFC meaning it sends out abnormally high numbers of worry signals and because they cannot ‘switch off’ the worry signals received from the thalamus ,when they complete the behaviour to address the worry, it will be insufficient to reduce the worry impulse. Therefore, they will need to continue with that particular behaviour, resulting in repetitive compulsive actions.

42
Q

Evaluations of the neural explanation of OCD

A

Pros:
1) Practical application
= By identifying the potential neurotransmitters (dopamine and serotonin) involved in the development of OCD it is possible to design drugs that can influence the levels of neurotransmitters, to address the symptoms of OCD.
-For example, Selective Serotonin Reuptake Inhibitors (SSRI’s) have been used to increase serotonin levels in OCD sufferers, with good success rates. Due to the increase in serotonin levels, it lessens obsessive thoughts, due to the improved transmission of mood related information. This strengthens the claim that OCD has a neural (biochemical) basis and also shows how this neural explanation has been beneficial to OCD sufferers.

2) Reasearch support ; Coccaro
= PET scans taken one a patient is actively experiencing their symptoms (ie a person with a germ obsession holding a dirty cloth) have shown higher levels of activity in the OFC. This supports the ‘worry circuit’ as it empirically and scientifically shows activation of this area in relation to the dirty cloth. Therefore, showing strong support for the claims that neuroanatomical differences, such as an increased activity in OFC, could be a potential cause of OCD.

Con:
1) Neural factors identified retrospectively
= Identified after the individual has received a diagnosis of OCD. Therefore, it is hard to establish cause and effect from whether these neural differences were the cause of OCD [IE may have been present before diagnosis] or have come about as a consequence of experiencing OCD. As prospective evidence is rare, establishing whether these neural differences are casual is very difficult to determine. Therefore, undermining the knowledge/ understanding about the causes of OCD.

43
Q

The biological treatment of OCD (main drug treatment)

A

SSRIs (selective serotonin reuptake inhibitors)
= They work by blocking the reuptake transporter of serotonin on the presynaptic Axon terminal.
- people with OCD -> have less serotonin released into synapse and a quick reabsorption.
1) As a consequence, serotonin which has been released into the synapse cannot be reabsorbed, therefore remaining longer in the synapse.
2) This means the serotonin has an increased opportunity to bind to its receptor sites on the postsynaptic terminal, thereby resulting in an increased serotonin function; reducing symptoms of OCD.

44
Q

drug treatments in OCD

A

= regulate abnormal levels of neurotransmitters (usually serotonin) in the brain

  • SSRIs
  • Benzodiazepines
    -SNRIs
45
Q

The biological treatments of OCD (alternative drug treatment - BZ)

A

=Alternative drug treatments are given when the individuals symptoms remain unresponsive to SSRIs.

1) Benzodiazepines (anti-anxiety med)
- Increases the effect of GABA.

GABA: an inhibitory neurotransmitter (increases neg charge, less likely to fire) - telling the neuron to stop ‘stressing’

In neurotypical people:
- GABA binds to recpetor site causing increase flow of chloride ions into neuron.
- Chloride ions (neg charge) make it difficult for receiving neurons to be stimulated by further neurotransmitters.
=Therefore, the neurotransmitter is slowed down making the person feel more relaxed.

OCD patient using BZ:
- BZ binds to a receptor site and enhances the effect of GABA -> more chloride ions flood the neuron making it even more negatively charged and less likely to fire.
- Therefore, any anxiety, which is experienced as a result of obsessive thoughts, is reduced.

more GABA = less anxiety
less anxiety = less obsessions
less obsessions = less compulsions
—–> reduced symptoms of OCD

46
Q

The biological treatment of OCD (alternative: SNRIs)

A

1) SNRIs (serotonin noradrenaline reuptake inhibitors)
= Increases the levels of serotonin as well as another different transmitter- noradrenaline which regulates our flight or fight responses e.g increases blood pressure
- Second line of defence for people who don’t respond to SSRIs

47
Q

PROS of the biological treatments of OCD (drug t)

A

Pros:
1) Research evidence
= Soomro conducted a meta-analysis of 17 studies that compared SSRIs to placebos. He found that SSRIs were 70% more effective than the placebo as those taking the drug had a decline in symptoms. The other 30% responded to alternative drug treatments (BZ, tricyclics etc). Therefore, the use of drugs to address biochemical imbalances have been successful and improved the well-being of sufferers from OCD. Thus, validating the claim that drug treatments are an appropriate and successful treatment to offer for OCD patients.

2) Cost-effective and easily accessible
= Because drugs like SSRI’s are readily available and much cheaper for public health services like the NHS compared to psychological therapies (such as CBT). Drug treatments don’t require any effort from patients and so it doesn’t disrupt their life, as they can take the drug until their symptoms decline whilst maintaining their everyday life.

48
Q

CONS of the biological treatments of OCD (drug t)

A

Cons:
1) Soomro’s research was most effective alongside CBT
= This indicates drugs used in combination with psychological therapies make improvements of symptoms more significant. These findings suggest that drug treatment shouldn’t be used in isolation, but instead incorporating a range of different treatments as an interactionalist approach, will be the the best course of therapy for OCD patients.

2) Side-effects
= Including indigestion, loss of sex drive, tremors, weight gain, disruption to blood pressures and more. These can affect the patient’s compliancy to take the drug, which can lead to a return of their symptoms. It could also lead to a reduction in their well-being and some of the side effects may be as bad as living with OCD. Therefore, the ‘costs’ of drug treatments must be carefully weighed against the ‘benefits’ they offer (in terms of symptom reduction) when deciding whether to offer this form of treatment to a patient.

49
Q

Support of cog approach to explaining depression

A

Koster (2005)
presented Ps with either a positive, negative or neutral word on a screen.
-After the word disappeared a square
appeared (and then rapidly disappeared) and participants had to indicate (by pressing a button) which area of the screen the square had appeared in.
-Koster found Ps who had depressive symptoms took longer (thancontrols) to indicate the area of the screen the square had appeared in after viewing a negative word (compared to positive or neutral words).

This suggests that individuals with Depression are more focused andaffected by negative cognitive experiences than controls. Both of these pieces of evidence support the claims that Depression has a cognitive basis and validates the claims that a person’s perceptions and interpretations
of their environmental stimuli is what causes the Depression.