PsychoPathology Flashcards

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1
Q

Defining abnormalities: Outline deviation from social norms

A

Abnormalities= behaviour/characteristics that’s uncommon/unusual (crucial we define abnormalities b4 finding reason for it or even treat it)

DSM( The diagnostic n statistical manual of mental disorders) lists every known disorders and it’s characteristics to help clinicians diagnose mental illnesses. other way of diagnosing abnormalities is:

  • Deviation from social norms- behaviour that doesn’t conform to accepted/expected standards = abnormal
    eg adult eating bogeys, something children do and it’s unhygienic
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2
Q

Defining abnormalities: evaluate deviation from social norms

A
  • Who defines what’s normal? Norms aren’t always universally accepted
    eg bw social classes there’s different rules of behaviour so a patient will b judged subjectively according to person diagnosing them.
    Means deviation from social norms too subjective n susceptible to abuse (norms of dominant grp may -> subordinate grp being labelled mentally ill for not conforming)
    Szasz 1974 claimed concept of mental illness j way society used to excuse non-conformists eg Russia, 50yrs ago, any1 that disagreed w state had risk of being put in mental institute
  • lacks Temporal validity- norms change over time eg homosexuals regarded as mentally ill in 1970s but now it’s normal.
    means deviation from social norms is time-specific, ever-changing and only reflects morals of the time so limits usefulness
  • Cultural relativism- norms differ across cultures
    eg talking to spirits desirable in some cultures n done by high rank ppl but in the west this’d be abnormal.
    Cultural relativism is acknowledged by DSM so ‘culture-bound’ syndromes also been included- shows no universal way of defining norms so there’s no universal standard in defining abnormality using deviation from social norms
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3
Q

defining abnormalities: outline statistical infrequency

A

looking at stats n finding average (mean,median,mode) of likelihood of behaviour occurring. Anything on the extreme ends of a normal distribution curve is considered abnormal
eg
•OCD is a rare disorder to have so anyone w it is abnormal

•in UK, average age to have baby is 25-30 so anything slightly more/less in realms of norm.
A baby at age of 10 or 60 would be abnormal. As age increases or decreases this is increasingly abnormal

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4
Q

Defining abnormalities: evaluate statistical infrequency

A
  • Abnormal stat not always bad
    eg scoring 150 in IQ test would be considered abnormal as not many score this high but is desirable trait not deviant, so we don’t consider these ppl abnormal
  • undesirable trait maybe norm but unhealthy
    eg depression so common now anyone without it could be said to be statistically abnormal
    so, this is extreme n limited way of defining abnormalities for mental health
  • labelling someone that’s statistically infrequent characteristics may cause issues
    eg someone w low IQ isn’t suffering but labelling them abnormal could affect them so label is bad not abnormality
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5
Q

Defining abnormalities: outline failure to function adequately (FFA)

A

abnormal behaviour causing someone distress n inability to cope w everyday life (includes bizarre/unpredictable behaviour-can include behaviour distressing to others too)
eg
•someone w ocd maybe late to school because they’re constantly ironing their clothes for no creases

failing to get outta bed in morning is bad for someone who gotta be at skl so can distress parents so therefore considered abnormal if done often

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6
Q

defining abnormalities: evaluate FFA

A
  • involves judgments about what’s adequate
    eg may think wearing sandals to class fine but parents think ‘inadequate’ footwear.
    suin may vio a person’s moral standards but be acceptable for others - this lack of objectivity makes it hard to use to judge abnormality
  • Cultural relativism where aspects of 1’s social class/ethnicity being judged as inadequate
    eg psychiatrists often white middle class n use their standard n culture to judge others-could explain y so many working class ppl judged as mentally ill asw as a much higher proportion being black than expected
  • Individual differences- 2ppl may’ve same mental illness but cope differently eg 2 OCD p(s) checking locks before leaving 50x. 1 late to work cos of it n other gets up earlier to check so on time.
    Highlights importance of individual differences so nomothetic approach involving tickbox method not appropriate to identify abnormal behaviour- idiographic considering individual differences = better to identify abnormal behaviour
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7
Q

Defining abnormalities: outline deviation from mental health

A

Jahoda proposed 6 key criteria for ideal mental health instead of focussing on criteria for abnormalities:
- pos attitude to self
- personal growth
- resistance to stress
- personal autonomy
- accurate perception of reality
- adapting to env
felt deviating from these means person vulnerable to mental disorders n classified as abnormal
eg student that has exams but can’t motivate themselves to revise

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8
Q

Defining abnormalities: evaluate deviation from mental health

A
  • Refreshing- focus on pos behaviour over neg. definition takes holistic view bcz focuses on diff aspects of person’s experiences affecting mental health
    May’ve pos effect on patients to use pos checklist over neg-may feel better mentally to c they satisfy so many pos criteria and if things lacking, may feel motivation to work on them
    when seeking neg behaviour, danger psychiatrists may c behaviour that’s not there (observer bias). highlighting neg behaviour to patient may make em feel worse n run mental health further so his explanation maybe useful in defining abnormalities
  • unrealistic criteria- judging ppl on these would render every1 abnormal. standards too high n anyone judging themselves/others risk creating anxiety in person as they’d feel insufficient
    Doesn’t consider neg emotions from an event eg death of loved one where neg emotions normal so enviromental triggers must be considered
  • cultural relativism- criteria based on individualistic culture not collectivist where personal sacrifice/working to common good maybe more important
    means someone in collectivist that don’t possess personal autonomy would be abnormal so definitions can’t be universally applied
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9
Q

Phobia traits

A

Phobia= type of anxiety disorder
eg Johnny Depp- coulrophobia (clowns)
• specific phobia- objects, situs, animals
• Agoraphobia- open space/public/not being home
• Social phobia- social situs bcz of possibility can be judged

  • behavioural characteristics: avoidance (lot of effort to avoid fear eg claustrophobic ppl avoid lifts)
    some phobias interferes w daily life. may freeze/faint bcz of wrong activation of fight/flight response
  • Cog characteristics: irrational thinking
    eg fear of planes bcz think crash n die. resistant to rational thoughts eg stats show how unlikely crash doesn’t make em feel better
  • Emotional characteristics: panic/anxiety. fear is excessive n persistent
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10
Q

outline the behaviourist approach to phobias

A

Behaviourists believe phobias r learnt same way behaviours r learnt.
Mowrer 1947 proposed two-process model:

1) classical conditioning: learned by association. claims phobia learnt w a stimulus being associated w bad experience
eg person has no fear of dogs (neutral stimulus)
SUPPORT: Watson n Rayner 1920:
Lil Albert-11month old w no phobias.
Given white rat (neutral stimulus) to play w and whilst playing w it, metal bar was struck loudly-cried.
Noise= unconditioned stimulus
Fear= unconditioned response
this was repeated few times.
When given white rat, cried even w no noise
Rat= conditioned stimulus
fear= conditioned response
fear extended to all white fluffy objects even Santa’s beard
Behaviourists believe this how all phobias learnt

2) operant conditioning: phobia maintained thru this-This is where behaviour is learnt by rewards and reinforcement.
Neg reinforcement= unpleasant consequence avoided
eg child brushes teeth to not go dentist. Avoiding dentist is the neg reinforcer which encourages behaviour of stimulus.
In the case of phobias, p(s) find way to avoid phobic stimulus to decrease chance of experiencing fear and is therefore neg reinforcer
Neg reinforcement is achieved via avoidance so the phobias maintained

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11
Q

evaluate the behaviourist approach to phobias

A
  • Sue et al 1934 found ppl w agaraphobia recall sudden panic attacks in public
    suggests classical conditioning can explain phobias
  • But, this not always case as many ppl w phobias recall no traumatic event where classical conditioning occurred so there must be other explanations too
    If phobias learnt we’d expect more ppl to be afraid of common things eg cars cause more damage than suin like spiders which r v unlikely to harm, but this isn’t the case
  • implies phobias can b unlearnt leading to practical applications in treatment eg systematic desensitisation (gradual exposure to phobia paired w relaxation to remove fear and associate it w positivity
    or flooding (p flooded w feared stimulus till they realise there’s no harm in it)
    both have been used n effective w p(s) so supports learning theory
  • enviromental reductionism and ignores cog factors that maybe involved in developing and maintaining phobia- irrational thoughts
    eg someone scared of lifts thinks they’ll get trapped n suffocate to death
    support for cog view is from CBT- v effective in dealing w phobias
    Engels 1993 found ppl w agaraphobia were helped more w CBT than j taking behavioural therapy so it weakens 2-process model explanation
    Would be wise to acknowledge some phobias r learnt n some r cog/evo
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12
Q

outline methods of treating phobias: Systematic desensitisation (SD)

A

Behaviourists believe if phobias are learnt they can be unlearnt

Systematic desensitisation
developed by Wolpe

SD= form of classical conditioning-counter conditioning used where phobias removed by associating fear w relaxation response
p(s) gradually exposed to whilst experiencing relaxation bcz of idea that they’re incompatible (reciprocal inhibiton) and so the fear’s dispelled.

  • p learns relaxation techniques from therapist eg breathing exercises
  • desensitisation/anxiety hierarchy made by p n therapist
    eg imagine phobia then situ gets harder to handle till ready to face phobia
    eg imagine in same room as spider then reaching for it but not touching then holding it
  • once hierarchicy agreed, p works thru it using relaxation techniques so now new association of phobia is w relaxation. Once each stage is mastered, phobia should be gone
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13
Q

evaluate treating phobias: SD

A
  • SUPPORT:
    McGrath et al 1998 found 75% p responded well to it esp w vivo technique (exposed to acc object) rather than vitro (imagining)
    Capatons et al 1998 reported p(s) w fear of flying less scared after SD compared to control grp that didn’t have
    so research supports effectiveness on p(s) w phobias
  • SUPPORT:
    less therapy/self-administer
    v important as therapy demand increased since COVID so burdens eco and NHS
    so any therapy that can be self-administered valuable
    Al Khubaisy et al 1992 compared p(s) that went thru SD self-administered n w therapist n found same success rates
    suggests SD has important role in treating phobias
  • AGAINST:
    Ohman et al 1975 says SD not effective w p(s) w phobias of evolutionary survival components eg darkness
    since it don’t originate from traumatic event suggests SD would b less effective as learning theory suggests it had to be learnt from classical conditioning to begin w
    so evo phobias maybe better to have other treatment eg CBT
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14
Q

Outline treatments of phobias: Flooding

A

1 v long sesh w extreme exposure of phobia paired w relaxation-urged by therapist till anxiety disappears
eg p w fear of clowns taken to room w them till they’re know they’re harmless n phobias extinguished
As they can’t avoid, neg reinforcement can’t occur so phobia not maintained

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15
Q

evaluate treatments of phobias: Flooding

A
  • SUPPORT:
    good for eco as 1 or few sesh needed eg CBT strains finances
    Flooding more effective on those who complete it
    Choy et al 2007 found SD n flooding effective but flooding more
    better for p as phobia removed faster
  • AGAINST:
    can b v traumatic n hella p don’t finish treatment
    treatment can b seen as unethical tho consents likely to b obtained-p gotta be fully prepd for exposure bcz possible they’ll undergo psych harm
    Wolpe 1969 recalled p hospitalised due to extreme anxiety undergone shows it’s not suitable for every1 or all phobias
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16
Q

traits of depression

A

Depression is a mood disorder that has various types of severities

  • behavioural: fatigue/restless, insomnia/excessive, appetite over/under
  • Cog: neg beliefs/self-concept, suicidal, slow thought process eg concentration/decision-making
  • emotional: sad, hopeless, loss of interest in hobbies

DSM requires 5 symptoms including sadness n loss of interest to diagnose someone depressed

17
Q

Cog approach to depression: Beck’s negative triad

A
  • Beck’s Neg triad
    vulnerable if way of thinking is neg
    ie negative schema of world

schema = package of info that develops thru their expression n is used to judge world around em
eg over critical parents

Neg schema = everything interpreted neg w no regard to pos outcome.
This cog bias maintains neg triad n keeps em depressed:
• Neg view of themselves eg i’m ugly
• Neg view of world eg must succeed to be good person
• Neg view of future eg never gonna be successful

18
Q

Cog approach to depression: Ellis’s ABC model

A

belief that depression stems from irrational thoughts

Activating event: disorder begins here when experience suin
eg failed exam leads to belief

Belief: unhealthy/unrealistic expectations what should happen
eg need to pass or im failure

Consequences: cos of A&B when things don’t happen as expected, depression stems from it- consequence is exaggerated n dire

19
Q

Evaluate cog approach to depression

A
  • IRL application- used as basis for CBT where all aspects of depression can b challenged n discussed eg ABC model, irrational thoughts. CBT proven to b v successful in treating depression n other mental illnesses
  • Irrational thoughts maybe rational
    Alloy n Abramson 1979 found depressed realists c things as they’re- they gave accurate estimates of disasters than control grp. Said depressed ppl sadder but wiser so concept of irrational thinking challenged
  • can’t est cause n effect- irrational thought cause depression or opposite?
    ppl w maladaptive cog processes likelier to have mental disorders but can’t say for certain what caused what
  • reductionist- no account for other credible explanations of depression
    eg bio approach- low serotonin been linked w depression n drug therapies help, so weakens cog explanation
    humanistic approach beneficial cos diff approaches considered together in explaining depression
20
Q

Outline the cog approach to treating depression

A
  • CBT common for depression/other mental illnesses:

aims to identify, challenge, change irrational thoughts bcz it sees that as root w idea that thoughts /behaviour r linked. by treating thghts, depression goes.
initial assessment identifies main issue that needs dealing w then plan made n goals est’d to give direction to therapy

  • Ellis developed form of CBT:
    Rational Emotive Behavioural Therapy (REBT)
    n extends his model to ABCDE
    D= distinguish thoughts- therapist works on challenging thoughts to show it’s unreal
    E= effect-
    (once thoughts gone should be happy/less anxious)
    so then depressions treated
  • Part of Distinguishing thoughts in REBT:
    logical disputing- p encouraged to logically follow thoughts to c they make no sense
    empirical disputing= thoughts can’t be proven to b true n r inconsistent w reality
    pragmatic disputing= useless to focus on bad beliefs by seeing they of no use
    P encouraged to let go of em n replace w pos beliefs
21
Q

evaluate the cog approach to treating depression

A
  • SUPPORT: Hella research support for CBT
    March et al 2007 compares effects of CBT for treating 327 teens w depression vs antidepressants instead.
    After 36w found, 81% improvement in both. shows it’s effective as drugs but w no side effects n addiction
    Found CBT taken w drugs = 86% improvement suggesting best to have cog n bio treatment n that CBT not only used for depression

AGAINST:

  • CBT dependant on p accepting n challenging irrational thoughts.
    Severe depressed p can’t motivate themselves to engage w therapy so CBT not helpful bcz symptom of depression is lack of focus.
    best to initialise w antidepressants n CBT after when motivated.
    shows CBT not useful alone so not universally useful for all depression p(s) therefore is a limitation in this regard
  • CBT doesn’t address irrational envs.
    P may exist in situations like domestic abuse, challenging work situs that produce stress so changing thoughts may not solve depression
  • CBT focus on present/future n p may wanna talk of past trauma they believe led to situ to make sense of disorder. for p that feel past crucial in shaping/challenging current thoughts CBT won’t be satisfactory treatment
    suggests idiographic approach needed for treating depression or individual circumstances n cause of irrational thghts maybe key to treatment n nomothetic that tries change way of thinking without regard to env/past not effective for some p
22
Q

Traits of OCD

A

Compulsive disorder
80% have obsessions n compulsions
20% only have obsessions
eg contamination= fear of germs so they clean excessively (compulsion)
eg Beckham lining up cans of soft drinks

  • behavioural: compulsions carried out to decrease anxiety created from obsessions. repetitive behaviour and avoidance of thing triggering OCD
    eg public places due to hygiene
  • Cog: obsessions intrusive recurring thoughts
    uncontrollable thoughts->anxiety
    aware irrational but powerless to control
  • emotional: anxiety/distress caused by obsessions/compulsions feeling shame, guilt, embarrassed bcz know compulsions excessive
    low-self mood/worth
23
Q

Bio approach to OCD: Genetic explanation

A

argues OCD genetically inherited-supported by studies showing likelihood higher if parents had

GENETIC EXPLANATION: genes made of chromosomes consisting of DNA which r inherited

  • Louis 1936 found 37% of his p(s) had parents w it n 21% had siblings w- supports idea of genetic cause
  • Twin studies useful:
    monozygotic twins (MZ) share 100% same genetic makeup so if suin caused by genes concordance rates should be high bw twins
    dizygotic twins (DZ) share 50% of genetic makeup so lower concordance rate expected

Nestadt et al 2010 retrieved twin studies. Found:
68% concordance rate w MZ
31% w DZ
strong support of OCD having genetic origin

  • Researchers say certain genes create vulnerability for OCD- could be polygenic (caused by several genes)
    Taylor 2013 found previous studies showed up to 230 diff genes could be involved in OCD
24
Q

Evaluate the genetic explanation for OCD

A

AGAINST:

  • twin studies show link bw OCD n genes but concordance rates never 100% meaning other factors must be involved eg env
    agreed genes make vulnerable but env factors eg traumatic events can trigger condition more easily (diathesis stress model)
    support for env factor:
    Cromer et al 2007 found 1/2 of his OCD p(s) sample experienced traumatic event n those who experienced more trauma likelier to have OCD
  • not much irl application
    tho there’s link bw genes n OCD can’t isolate genes responsible bcz of multitude of genes involved so has no predictive value n can’t help treat it effectively. perhaps w further research genetic explanation will be more useful to psychiatrists for OCD p(s)
25
Q

outline the neural explanation for OCD

A

focus on how brain structures n neurotransmitters can contribute to OCD.

  • Argued serotonin lvls contribute to it as OCD p(s) found to have low lvls
    SUPPORT:
    Piggot et al 1990 found p(s) that took SSRI (antidepressant that raises serotonin) reported OCD symptoms reduced n experienced obsessions n compulsions less
  • Brain structure examined:
    Frontal lobe= responsible for logical thinking and decision making
    caudate nucleus job to suppress ‘worry signals’- failure causes overstimulation bcz brain focuses on minority thoughts that needs to be dismissed
    Brain scans show caudate nucleus damaged in OCD p(s) bcz when symptoms r active (obsessions) the frontal lobe overactive.
    Causes ‘worry circuits’ where their obsessions r constantly activated.
    serotonin plays key role in caudate nucleus- could explain y serotonin lvls cause OCD
26
Q

Evaluate the neural explanation to OCD

A
  • cause n effect can’t be est’d. whilst there’s a link bw serotonin n OCD, maybe the OCD caused serotonin imbalance n not the other way round.
    Serotonin is associated w depression n many OCD p have that (COMORBIDITY) so maybe serotonin lvl cos of depression n not OCD
    so this weakens bio explanation for OCD
  • ** REDUCTIONIST**- assumes all OCD from innate factors so reduces it to gene/neurotransmitter cause which’s too simplistic

eg behaviourist idea 2 process model is plausible.
obsessions start when stimulus paired w suin neg via classical conditioning n maintained via operant conditioning by the compulsions thru neg reinforcement since compulsions reduce anxiety
Support:
Albucher et al 1998 found behavioural treatments improved symptoms of 60-90% of p(s) so bio approach reductionist and wider range of approaches should be considered

27
Q

bio treatments for OCD

A

treatment focused on neurotransmitter imbalances as low serotonin believed to be cause of OCD- drug therapies used to deal w imbalance

  • low serotonin associated w depression. drugs for depression used for OCD.
    SSRI (selective serotonin re-uptake inhibitors) include drugs like Prozac
  • serotonin released into synapses, targets a receptor cell and is reabsorbed by nerve that sent msg
    as serotonin low in OCD p(s), drug aims to limit re-uptake of serotonin so more remains in the cell. so in effect drug increases serotonin lvls to deal w deficiency in which then reduces anxiety n regulates p mood
  • Benzodiazepines (BZs) aims to slow down activity in nervous system
    this increases lvls of GABA (neurotransmitter that tells brain stop firing) n in effect slows down nervous system so has calming effect on body n consequently reduces anxiety n OCD symptoms
28
Q

evaluate bio treatments for OCD

A

SUPPORT:
- Soomro et al 2008 reviewed 17 studies of OCD p(s) found SSRIs reduced symptoms after 3month compared to when placebo drug given suggests bio treatment effective in dealing w OCD

AGAINST:
- Side effects common w bio treatments like most drugs solve problem cause another
SSRIs insomnia, headaches n more
BZs: aggressiveness, memory impairment, addictive so can only be used short term
Q’able if drugs ethical if causing more problems than fixing
Drugs better for short term eco cos cheap n ez to administer than therapy eg CBT
if causes more problems than long run strains finances as longer term care needed by p(s) to tackle conditions caused from side effects

  • inhibit symptoms but rarely treats disorder
    if drug stopped then relapse rates high so effects short term n other treatments needed for long term care.
    nomothetic giving all p same treatment unable to deal w cause as varies w individuals

could combine cog/behavioural ideas as OCD involves recurring thoughts so cog factors can tackle but CBT needs focus n suffering p may not bother so drug maybe convenient n relief from symptoms so then CBT can be taken so therefore long term benefit

  • bio approach has important role in treating p(s) but perhaps treating symptoms whilst working on long term solution they other therapies would be ideal for em