Psychopathology Flashcards

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1
Q

OCD – Biological explanations (Genetic)

A
  • OCD is 5 times more likely to develop in first degree relatives with the disorder than those that don’t.
  • Twin studies (Billet et al 1998) show a concordance rate of 57% for monozygotic but only 22% for dizygotic twins.
  • Don’t explain the differences between obsessions and compulsions.
  • Too simplistic there is a gene for OCD due to it potentially being a learnt behaviour (Two Process Model).
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2
Q

OCD – Biological explanations (Neural)

A
  • Brain imaging of people with OCD shows hyperactivity in the orbitofrontal cortex which plays a role in learning and decision making compared to those without the disorder.
  • Max et al (1995) A young adolescent developed OCD after traumatic brain injury and damage in the frontal and temporal lobe was discovered (linked to development of OCD)
  • Correlational relationships don’t imply causation.
  • Scientific evidence
  • Small sample can’t be generalised
  • Individual differences
  • Aylward et al (1996) patients with OCD showed no structural or functional differences in caudate nucleus
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3
Q

OCD – Biological explanations (Drug Therapies)

A
  • Anti-anxiety drugs (anxiolytics) like Valium increase levels of GABA which inhibits neurons and helps people calm down.
  • Anti-depressants like sertraline help boost mood and reduce tendency to worry.
  • Soomro et al (2008) reviewed 17 studies and SSRIs proved to be effective in the short term (3 months)
  • Side effects of drug therapy (mild)
  • Take 4-6 weeks to take effect.
  • Large sample
  • Practical applications
  • Treats symptoms not the case
  • Koran et al (2007) CBT should be used first to provide long-term effects.
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4
Q

Phobias – Behavioural explanations (Little Albert - Watson and Raynor 1920) (P&F)

A
  • Classical conditioning causes the development of phobias.
  • Watson argued that environmental factors are significant to the development of behaviour which was contradictory to Darwin’s evolutionary theory.
  • Little Albert was classically conditioned to fear rats following the association to a frightening loud noise.
  • The loud noise was the unconditioned stimulus that caused an unconditioned response of fear.
  • The white rat was the neutral stimulus but became the conditioned stimulus to cause little Albert fear.
  • This fear was associated to other stimuli eg. Fur coat and father’s Christmas mask.
  • Pavlov added conditioned associations can fade if not reinforced (extinction) and this conditioned association can be linked to wider stimuli (generalised).
  • Albert was withdrawn from experiment before the conditioned responce could be reversed.
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5
Q

Phobias – Behavioural explanations (Little Albert - Watson and Raynor 1920) Evaluation

A
  • No control subject/no objective measurement of fear responce
  • Low external validity (Albert’s enviroment isn’t representative leading to a unique response)
  • Lack of consent
  • Lack of protection from psychological harm
  • Reductionist limiting it to stimulus responce
  • ignores mental processes (cognitive)
  • Tomarken et al (1989) presented snake and neutral images to phobic and non-phobic participants, Phobics overestimated the number of snake images.
  • Seligman (1970) Biological preparedness developed due to evolution (adaptive to past)
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