Psychopathology Flashcards

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1
Q

What is statistical infrequency?

A

Occurs when an individual has a less common characteristic, for example being more depressed than most of the population.

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2
Q

What is normal distibution?

A

Where the majority if people’s scores will cluster around the average and the further above or below that average, the fewer people will attain that score.

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3
Q

Relate statistical infrequency to intellectual disability disorder.

A

The average IQ is set to 100 and 68% of people have a score in the range of 85-115.
Only 2% have a score lower than 70 therefore this would be ‘abnormal’ and so would be able to receive a diagnosis of intellectual disability disorder.

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4
Q

What is deviation from social norms?

A

It concerns behaviour that is different from the accepted standards of behaviour in a community or society.

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5
Q

How are norms specific to the culture we live in?

A

Norms may be different between generations and cultures, so there are relatively few behaviours which could be considered universally abnormal on the basis that they breach social norms.
For example, homosexuality was considered abnormal in the past in our culture but is still viewed as abnormal in some countries, such as Brunei.

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6
Q

Relate deviation from social norms to antisocial personality disorder.

A

A person with antisocial personality disorder is impulsive, aggressive and irresponsible.
According to the DSM-5 one important symptom is ‘absence of prosocial internal standards associated with failure to conform to lawful and culturally normative behaviour’.
Essentially, we make the social judgement that psychopaths are abnormal because they don’t conform to our moral standards.

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7
Q

Does statistical infrequency have real world application?

A

Yes
Is used in clinical practice, both as part of a formal diagnosis and as a way to assess the severity of symptoms.
An example of statistical infrequency used as an assessment tool would be the Beck depression inventory- a score of 30+ is widely interpreted as indicating severe depression.

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8
Q

What are the benefits and problems of labelling someone as abnormal (statistical infrequency)?

A

+ Helps them access support, for example someone with a high BDI may benefit from therapy.
- Someone with a low IQ who can cope with their chosen lifestyle may not benefit with a label due to the social stigma attached.

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9
Q

What is a limitation of statistical infrequency?

A

Infrequent characteristics could be positive as well as negative.
For every person with an IQ below 70 there is someone with an IQ above 130 but not think of someone as abnormal for having a high IQ.
Similarly, we wouldn’t think of someone with a low score on the BDI as abnormal.
This means that although statistical infrequency can form part of assessment and diagnostic procedures, it is never sufficient as the sole basis for defining abnormality.

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10
Q

Does deviation from social norms have real world application?

A

Yes
Is used in clinical practice.
For example, the key defining characteristic of antisocial personality disorder is the failure to conform to culturally normal ethical behaviour.
Signs of the disorder are all deviations from social norms.
Such norms also play a part in the diagnosis of schizotypal personality disorder where the term ‘strange’ is used to characterise the thinking, behaviour and appearance of people with the disorder.

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11
Q

Relate human rights abuses to deviation from social norms.

A

Defining someone as abnormal carries the risk of unfair labelling and leaving them open to human rights abuses.
Historically, a diagnosis such as nymphomania (women’s uncontrollable or excessive sexual desire) have been used to control women.
On the other hand it can be argued that we need to be able to use deviation from social norms to diagnose conditions such as antisocial personality disorder.

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12
Q

Name one limitation of deviation from social norms.

A

The variability between social norms in different cultures and situations.
For example, hearing voices is the norm in some cultures but it would be seen as an abnormality in the UK.
Aggressive and deceitful behaviour in the context of family life is more socially unacceptable than in the context of corporate deal-making.
This means it is hard to judge deviation from social norms across different situations and cultures.

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13
Q

Name the four definitions of abnormality.

A
  1. statistical infrequency
  2. deviation from social norms
  3. failure to function adequately
  4. deviation from ideal mental health.
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14
Q

What is failure to function adequately?

A

Occurs when someone is unable to cope with ordinary demands of day-to-day living.
For example being unable to maintain basic standards of nutrition and hygiene. This can also being unable to hold down a job or maintain relationships.

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15
Q

When is someone failing to function adequately?

A

Rosenhan and Seligman (1989) proposed additional signs that can be used to determine when someone is not coping.

  • When a person experiences severe distress
  • No longer conforming to standard interpersonal rules such as maintaining eye contact and respecting personal space.
  • When a person’s behaviour becomes irrational or dangerous to themselves or others.
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16
Q

Relate the failure to function adequately with intellectual disability disorder.

A

To have a diagnosis for intellectual disability disorder, an individual must be failing to function adequately.

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17
Q

What is deviation from ideal mental health?

A

Occurs when someone does not meet a set of criteria for good mental health.

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18
Q

What does ideal mental health look like?

A

Marie Jahoda (1958) suggested we are in good mental health if we meet the following criteria-
- No symptoms or distress
- We are rational and can perceive ourselves accurately
- We self-actualise
- Cope with stress
- Realistic view of the world
- Good self-esteem and lack guilt
- Independent of other people
- Successfully work, love and enjoy our leisure.
There is some overlap between deviation from ideal mental health and failure to function adequately.

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19
Q

Explain how the failure to function adequately can be normal.

A

There are some circumstances where most of us fail to cope for a period of time. It may be unfair to give someone a label that may cause them future problems just because they react to difficult circumstances.
However, the failure to function is no less real just because the cause is clear.

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20
Q

Name one strength of the failure to function adequately.

A

It represents a sensible threshold for when people need professional help.
The criteria can mean that treatment and services can be targeted to those who need them most.

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21
Q

Name one weakness of the failure to function adequately.

A

It is easy to label non-standard lifestyle choices as abnormal.
Not having a job may seem like failing to function but for some may choose it as a alternative lifestyle.
Therefore people who make unusual choices are at risk of being labelled abnormal and their freedom of choice may be restricted.

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22
Q

Name one strength of deviation from ideal mental health.

A

It is highly comprehensive.
This means that ideal mental health provides a checklist against which we can assess ourselves and others and discuss psychological issues with a range of professionals.

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23
Q

Name one weakness of deviation from ideal mental health.

A

It may be culture bound as that the elements of Jahoda’s list are not equally applicable across a range of cultures.
For example within western Europe there is variation between countries for example independence is high in Germany but low in Italy.
This means it is hard to apply the concept of ideal mental health from one culture to another.

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24
Q

Relate extremely high standards with deviation from ideal mental health.

A

Very few people attain all of Jahoda’s criteria for mental health and not many will achieve all of them at the same time or keep them up for long.
However having such a comprehensive set of criteria for mental health to work towards might be of practical value to someone wanting to understand and improve their mental health.

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25
Q

What is a phobia?

A

An irrational fear of an object or situation.

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26
Q

What are the three behavioural characteristics of phobias?

A

Panic
Avoidance
Endurance (e.g keeping an eye on a spider instead of leaving the room)

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27
Q

What are three emotional characteristics of phobias?

A

Anxiety
Fear
An ‘unreasonable’ emotional response

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28
Q

What are three cognitive characteristics of phobias?

A

Selective attention to the phobic stimulus
Irrational beliefs
Cognitive distortions (e.g an arachnophobe may see a spider as ‘aggressive and scary’)

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29
Q

What are three behavioural characteristics of depression?

A

Aggression and self harm
Disruption to sleep and eating behaviour
Reduced activity levels.

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30
Q

What are three emotional characteristics of depression?

A

Lowered mood
Anger
Lowered self-esteem.

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31
Q

What are three cognitive characteristics of depression?

A

Poor concentration
Absolutist thinking
Attending to and dwelling on the negative.

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32
Q

What are three behavioural characteristics of OCD?

A

Compulsions are repetitive
Compulsions reduce anxiety
Avoidance.

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33
Q

What are three emotional characteristics of OCD?

A

Anxiety and distress
Accompanying depression
Guilt and disgust.

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34
Q

What are three cognitive characteristics of OCD?

A

Obsessive thoughts
Cognitive coping strategies
Insight into excessive anxiety (aware).

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35
Q

What approach has been used to explain phobias?

A

Behavioural

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36
Q

Who proposed the two-process model?

A

Mowrer (1960)

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37
Q

What is the two-process model?

A

States that phobias are aquired by classical conditioning and then maintained due to operant conditioning.

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38
Q

What study is used to show acquisition of phobias by classical conditioning?

A

Watson and Rayner (1920)- Little Albert

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39
Q

Watson and Rayner (1920)

A

Created a phobias in a 9 month old child called ‘Little Albert’.
Albert at the start had no anxiety and played with the white rat.
However, after a while the experiementers made a loud noise with a iron bar close to his ear when the rat was presented.
The noise is a UCS which creates a UCR of fear.
When the rat (NS) and noise (UCS) are encountered together at the same time, the rat is associated with fear.
The rat is now the (CS) and the fear is the (CR).
This fear generalised to similar objects such as a non-white rabbit.

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40
Q

Maintainance by operant conditioning.

A

Operant conditioning take place when our behaviour is reinforced (rewarded) or punished.
Both positive and negative reinforcement increase the frequency of behaviours.
In the case of negative reinforcement and individual avoids a situation that is unpleasant and such behaviour results in a desirable consequence, which means behaviour will be repeated.
Mowrer suggested avoiding the phobic stimuluas means we successfully avoid the anxiety that would have been experienced- this reduction in fear reinforces the avoidance behaviour and so the phobia is maintained.

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41
Q

Real-world application of the two process model.

A

It has been used for eposure therapies such as systematic desensitisation.
The idea that phobias are maintained by avoidance, explains why it is important that people with a phobias benefit from being exposed to the phobic stimulus.
Once the avoidance behaviour is prevented it ceases to be reinforced by the experience of anxiety reduction and so avoidance declines.
In behavioural terms, the phobias is the avoidance behaviour so when this avoidance is prevented the phobia is cured.

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42
Q

A limitation of the two-process model.

A

Does not account for cognitive aspects of phobias.
In the case of phobias the key behaviour is avoidance of the phobic stimulus.
However, phobias are not simply avoidance responses- they also have a significant cognitive component.
For example many people have irrational beliefs about the phobic stimulus.
The two-process model explains avoidance behaviour but does not offer an adequate explaination for phobic cognitions.

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43
Q

A strength of the two-process model.

A

The two-process model is evidence for a link between experiences and phobias.
Little Albert is evidence of this.
De Jongh et al. (2006) found 73% of people with a fear of dental treatment had experinced a traumatic experience, mostly involving dentistry. This can be compared to a control group of people with low dental anxiety where only 21% had experienced a traumatic event.
This confirms the association between stimulus and UCR does lead to the development of fear.

44
Q

Counterpoint to phobias and traumatic experiences.

A

Not all phobias appear after following a bad experience.
Some common phobias such as snakes occur in populations where very few people would have had any experience of snakes, let alone traumatic experiences.
This means the associaed between phobias and frightening experiences is not as strong as expected if behavioural theories provided a complete explanation.

45
Q

What is systematic desensitisation?

A

A behavioural therapy designhed to gradually reduce phobic anxiety through the principle of classical conditioning. If a person can learn to relax in the presence of the phobic stimulus they will be cured.
Essentially a new response (relaxation) to the phobic stimulus is learned and this learning of a different response is called counterconditioning.

46
Q

What three processes are involved in systematic desensitisation?

A

The anxiety heirachy- a list of situations related to the phobic stimulus that provoke anxiety arranged in order from least to most frightening.
Relaxation- therapist teaches client to relax as deeply as possible. It is impossible to be afraid and relaxed at the same time, so one emotion prevents the other- this is called reciprocal inhibition.
Exposure- the client is exposed to the phobic stimulus in a relaxed state, moving up the anxiety heirachy over session. Treatment is successful when the client can stay relaxed in situations high on the anxiety heirachy.

47
Q

Effectiveness of systematic desensitisation.

A

Gilroy et al. (2003) followed up 42 people who had SD for spider phobia in three 45 minute sessions.
At both 3 and 33 months, the SD group were less fearful than a control group treated by relaxation without exposure.
In a recent review Wechsler et al. (2019) concluded that SD is effective for specific phobia, social phobia and agoraphobia.
SD is overally likely to be useful treating people with phobias.

48
Q

People with learning disabilities and systematic desensitisation.

A

Some people requiring treatment for phobias also have a learning disability. However, the main alternatives to SD are not suitable.
People with learning disabilities often struggle with cognitive therapies that require a high level of rational thought.
People with learning disabilities may feel confused and distressed by flooding and so SD is the most appropriate treatment.

49
Q

What is flooding?

A

Flooding involves exposure to the phobic stimulus but this time is an immediate exposure to a frightening situation.
Flooding sessions are longer than SD sessions, with one session lasting two to three hours.
Sometimes only one long sessions is needed to cure a phobia.

50
Q

How does flooding work?

A

Flooding stops phobic responses very quickly.
This may be because, without the option of avoidance behaviour, the client learns the phobic stimulus is harmless
In classical conditioning terms this is called extinction- the conditioned stimulus will no longer produce the conditioned response.

51
Q

What are the ethical safeguards of flooding?

A

Clients must give fully informed consent to the procedure so that they are fully prepared before the flooding session.
A client would normally be given the choice of systematic desensitisation or flooding.

52
Q

What is a strength of flooding?

A

It is highly cost effective and also clinically effective.
Flooding can work in one session as opposed to SD which could take ten sessions to achieve the same result.
Even allowing for a longer session make flooding more cost-effective.
This means that more people can be treated at the same cost with flooding than with SD or other therapies.

53
Q

What is a limitation of flooding?

A

It is a highly unpleasant experience.
Schumacher et al. (2015) found that participants and therapists rated flooding as significantly more stressful than SD.
This raises the ethical issue for psychologists of knowingly causing stress to their client, however this is not a serious issue as the client gives informed consent.
The traumatic nature means that attrition (droupout) rates are high than SD.
This may suggest that therapists may avoid using this treatment.

54
Q

What is the relationship between symptom substitution and flooding?

A

Flooding may only mask symptoms and not address the underlying causes of phobias.
Persons (1986) reported the case of a woman with a phobia of death who was treated using flooding. Her fear of death declined but her fear of being criticised got worse.
However, the only evidence for symptom substitution comes in the form of case studies and so is hard to generalise.

55
Q

What approach is used to explain depression?

A

Cognitive

56
Q

What three parts did Beck (1967) suggest to explain why some people are more vulnerable to depression?

A

Faulty information processing
Negative self-schema
The negative triad.

57
Q

What is faulty information processing?

A

This is when depressed people attend to the negative aspects of a situation and ignore the positives.
People tend to blow small problems out of propotion and thing in ‘black-and-white’ terms.

58
Q

What is negative self-schema?

A

A schema is a ‘package’ of ideas and information developed through experience.
A self-schema is the package of information people have about themselves.
People use schema to interpret the world, so if someone has a negative self-schema they interpret all information about themselves in a negative way.

59
Q

What is the negative triad?

A

Beck suggested that a person develops a dysfunctional view of themselves because of three types of negative thinking that occur automatically, regardless of the reality of what is happening at the time.
These three elements are called the negative triad.

60
Q

What are the three components of the negative triad?

A

Negative view of the world
Negative view of the future
Negative view of the self- enhance depressive feelings because they confirm the existing emotions of low self-esteem.

61
Q

What is some research support of Beck’s negative triad?

A

‘Cognitive vulnerability’ refers to ways of thinking that may predispose a person to becoming depressed.
Clark and Beck (1999) concluded that not only were these more common in people with depression but they preceded the depression.
A recent study by Cohen et al. (2019) tracked the development of 473 adolescents, regularly measuring cognitive vulnerability- it found that showing cognitive vulnerability predicted later depression.

62
Q

What is the real-world application of Beck’s negative triad?

A

Screening and treatments.
Cohen et al. (2019) concluded that assessing cognitive vulnerability allows psychologists to screen young people, identifying those most at risk of developing depression in the future and monitoring them.
Understanding cognitive vulnerability can also be applied in CBT. These therapies work by altering the kind of cognitions that make people more vulnerable, and make them more resilient.

63
Q

What is a limitation for Beck’s negative triad?

A

Whilst Beck’s suggestion of cognitive vulnerabilities provides at least a partial explanation for depression, there are some aspects of depression that are not particularly well explained by cognitive explanations.
For example, some people feel extreme anger and some experience hallucinations.

64
Q

What did Ellis define irrational thoughts as?

A

Illogical or unrealistic thoughts, but as any thoughts that interfere with us being happy and free from pain.

65
Q

What did Ellis (1962) use to explain how irrational thoughts affect our behaviour and emotional state?

A

The ABC model.

66
Q

What did Ellis say good mental health is the result of?

A

Rational thinking.

67
Q

What are the three components of the ABC model?

A

Activating event- Negative events trigger irrational beliefs.
Beliefs- Ellis identified a range of irrational beliefs for example utopianism which is the belief that life is always meant to be fair.
Consequences- when an activating event triggers irrational beliefs there are emotional and behavioural consequences.

68
Q

What is the real-world application of the ABC model?

A

Ellis’s approach to cognitive therapy is called Rational Emotive Behaviour Therapy (REBT).
The idea of REBT is that by vigorously arguing with a depressed person, the therapist can alter the irrational beliefs that are making them unhappy.
There is some evidence to support the idea that REBT can both change negative beliefs and relieve the symptoms of depression (David et al. 2018).

69
Q

What is a limitation of Ellis’s ABC model?

A

It only explains reactive depression and not endogenous depression.
Reactive depression is triggered by life events (activating events).
However, many cases of depression are not tracable to life events and it is not obvious what has led the person to depression- this is called endogenous depression.
Ellis’s model therefore is only a partial explanation.

70
Q

What are ethical issues associated with the ABC model?

A

It located the responsibility of depression purely with the depressed person.
Critics say this is effectively blaming the depressed person, which would be unfair.
On the other hand, provided it is used sensitively, the application of the ABC model in REBT does appear to make at least some depressed people achieve more resilience and feel better.

71
Q

What is Cognitive behavioural therapy?

A

A method for treating mental disorders based on both cognitive and behavioural techniques.
For the cognitive viewpoint the therapy aims to deal with thinking, such as challenging negative thoughts.
The therapy also includes behavioural techniques such as behavioural activation.

72
Q

What is Beck’s cognitive therapy?

A

Cognitive therapy is the application of Beck’s cognitive theory of depression. The idea behind cognitive therapy is to identify automatic thoughts about the world, the self and the future- this is the negative triad.
One these thoughts are idendtified, the must be challenged and this is the central component of the therapy.
Clients may be set homework to test the reality of their negative beliefs.

73
Q

What is Ellis’s rational emotive behaviour therapy?

A

REBT extends the ABC model to ABCDE, where the D stands for dispute and E stands for effect.
The central technique of REBT is to identify and dispute irrational thoughts.
A REBT therapist challenges irrational beliefs through arguing and the intended effect is to change the irrational belief and so break the link between negative life events and depression.

74
Q

Name two types of disputing Ellis identified.

A

Empirical argument- disputing whether there is actual evidence to support the negative belief.
Logical argument- disputing whether the negative thought logically follows from the facts.

75
Q

What is behavioural activation?

A

As individuals become depressed, they tend to increasingly avoid difficult situations and become isolated, which maintains and worsens symptoms.
The goal of behavioural activation is to work with depressed individuals to gradually decrease their avoidance and isolation, and inrease their engagement in activities which have been shown to improve mood.

76
Q

What is some evidence for effectiveness of CBT?

A

March et al. (2007) compared CBT to antidepressant drugs and also to a combination of both treatments when treating 327 depressed adolescents. After 36 weeks, 81% of the CBT group, 81% of the antidepressants group and 86% of the CBT + antidepressants group were significantly improved.
CBT is usually a fairly brief therapy requiring 6-12 sessions so is cost-effective whilst also effective for the patient.

77
Q

What is a limitation of CBT as a treatment for depression?

A

There is a lack of effectiveness for severe cases and clients with learning disabilities.
It may be hard for people with severe depression to motivate themselves to engage with the cognitive work of CBT.
Sturmey (2005) suggests that, in general, any form of psychotherapy is not suitable for people with learning difficulties, and this includes CBT.
HOWEVER
Lewis and Lewis (2016) concluded CBT was as effective as behavioural therapies and antidepressants for severe depression.
Taylor et al. (2008) concluded that, when used appropriately, CBT is effective for people with learning disabilities.

78
Q

What are relapse rates like for depression?

A

Recent studies show that long-term outcomes are not as good as had been assumed.
Ali et al. (2017) assessed depression in 439 clients every month for 12 months following a course of CBT. 42% of the clients relapsed into depression six months of ending treatment and 53% relapsed within a year.
This means CBT may have to be repeated periodically.

79
Q

Yrondi et al. (2015)- client preference of depression treatment.

A

They found that depressed people rated CBT as their least preferred psychologivcal therapy.

80
Q

What approach is used to explain OCD?

A

Biological

81
Q

What two biological explanations are there for OCD?

A

Genetic and Neural

82
Q

Lewis (1936)

A

37% of his OCD patients had parents with OCD and 21% had siblings with OCD.
This suggests the GENETIC VULNERABILITY to OCD is passed on from one generation to another.

83
Q

What are candidate genes in terms of OCD?

A

Candidate genes are genes which create vulnerability to OCD.
Some of these genes are involved in regulating the development of serotonin.
For example the gene 5HT1-D beta is implicated in the transport of serotonin across synapses.

84
Q

Is OCD polygenic?

A

Yes- this means OCD is not caused by one single gene but by a combination of genetic variations that together increase vulnerability.
Taylor (2013) found evidence of up to 230 different genes that my be involved in OCD.

85
Q

Genes and different types of OCD.

A

One group of genes may cause OCD in one person but a different group of genes may cause the disorder in another person- aetiologically heterogeneous.
There is some evidence to suggest that different types of OCD may be the result of particular genetic variations.

86
Q

Research support of genetic explanations of OCD.

A

TWIN STUDIES

Nestadt et al. (2010) reviewed twin studies and found of 68% of monozygotic twins shared OCD as opposed to 31% of dizygotic twins.

Marini and Stebnicki (2012) found that a person with a family member diagnosed with OCD is around four times as likely to develop it as someone without.

87
Q

What have animal studies showed us about candidate genes and OCD?

A

It has proved difficult to find candidate genes but there is evidence from animal studies showing that particular genes are associated with repetitive behaviours in other species, for example mice (Ahmari 2016).
However, although mice and humans share most genes, the human mind and brain are much more complex, so it may not be possible to generalise.

88
Q

What is a limitation of the genetic explanation of OCD?

A

Environmental risk factors .
OCD does not appear to solely be caused by genetics and environmental risk factors can also tigger or increase the risk of developing OCD.
Cromer et al. (2007) found that over half the OCD clients in their sample had experienced a traumatic event in their past.
OCD was also found to be more severe in people with one or more traumas.

89
Q

What is the role of serotonin in terms of neural explanations of OCD?

A

Some cases of OCD may be explained by a reduction in the functioning of the serotonin in the brain.
If a person has low levels of serotonin then normal transmission of mood-relevant information does not take place and a person may experience low moods.

90
Q

What is the influence of decision-making systems in terms of OCD?

A

Some cases of OCD are associated with impared decision making.
This in turn may be associated with abnormal functioning of the lateral of the frontal lobes of the brain. The frontal lobes are the front part of the brain that are responsible for decision making.
There is also evidence to suggest that an area called the left parahippocampal gyrus functions abnormally in OCD.

91
Q

What is some research support of neural explanations of OCD?

A

Antidepressants that work purely on serotonin are effective in reducing OCD symptoms.
Some disorders such as Parkinson’s disease, which is a biological disorder, produce OCD symptoms so we can assume that biological processes underlie OCD.

92
Q

What is a limitation of neural explanations of OCD?

A

The serotonin-OCD link may not be unique to OCD.
Many people with OCD also experience clinical depression (co-morbidity).
This depression probably involves disruption to the action of serotonin so it could be assumed that serotonin activity is disrupted in many people with OCD because they are depressed as well.

93
Q

What biological treatment is used to treat OCD?

A

Drug therapy

94
Q

What are drugs often used alongside and why?

A

CBT- drugs can reduce a person’s emotional symptoms, such as feeling anxious or depressed, meaning people can engage more effectively with CBT.

95
Q

What is an SSRI?

A

Selective Serotonin Reuptake Inhibitor, which is an antidepressant.

96
Q

How do SSRIs work?

A

Serotonin is released by certain neurons in the brain.
It is released by the presynaptic neuron and travels across a synapse.
The neurotransmitter chemically conveys the signal from the presynaptic neuron to the postsynaptic neuron and then it is reabsorbed by the presynaptic neuron where it is broken down and reused.
SSRIs increase levels of serotonin in the synapse by preventing the reabsorption and breakdown, and thus continue to stimulate the postsynaptic neuron.

97
Q

Name an example of a SSRI.

A

Fluoxetine.

98
Q

Name two alternatives to SSRIs.

A

Tricyclics

SNRIs

99
Q

How long does it take for SSRIs to have an impact on symptoms?

A

Three to four months of daily use.

100
Q

What are tricyclics?

A

An older type of antidepressant.
An example would be clomipramine.
These act on various systems including the serotonin system where it has the same effect as SSRIs.
Clomipramine has severe side-effects compared to SSRIs so it is generally kept in reserve for people who do not respond to SSRIs.

101
Q

What are SNRIs?

A

Serotonin-noradrenaline reuptake inhibitors.
Have been used more recently to treat OCD.
These are a different class of antidepressant drugs and are a second line of defence for people who don’t respond to SSRIs.
SNRIs increase the levels of serotonin as well as noradrenaline.

102
Q

What is a limitation of drug treatments for OCD?

A

They have potentially serious side effects.
Some people experience temporary side effects with SSRIs such as blurred vision, but for some side effects can be long lasting.
The tricyclic clomipramine has more common serious side effects such as 1 in 10 experience weight gain and 1 in 100 experience heart-related problems.

103
Q

What is some evidence of drug treatment for OCD being effective?

A

Soomro et al. (2009) reviewed 17 studies that compared SSRIs to placebos to treatment of OCD. All 17 studies showed significantly better outcomes for SSRIs than for the placebo conditions.
Typically symptoms reduce around 70% for people using SSRIs.
The remaining 30% can be helped through psychological therapies or alternative drugs.

104
Q

What is some evidence that drug treatments may not be the most effective treatments for OCD?

A

Skapinakis et al. (2016) carried out a systematic review of outcome studies and concluded that cognitive and behavioural (exposure) therapies were more effective than SSRIs in the treatment of OCD.
This means drugs may not be the optimum treatment for OCD.

105
Q

What is a strength of drug treatments for OCD?

A

They are cost-effective and non-disruptive to people’s lives.
Drugs are cheaper compared to psychological treatments.
Using drugs to treat OCD is good for public health systems like the NHS.
SSRIs unlike psychological therapies are non-disruptive to people’s lives.