PSY2004 SEMESTER 1 - WEEK 3 Flashcards

1
Q

define neuroplasticity

A

capacity of nervous system to modify its organisation to altered demands and environments

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2
Q

what is Baltes model

A

cultural/environmental factors become less important to biological factors in terms of explaining age-related changes in cognition

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3
Q

outline common cause hypothesis

A

age-related decline in cognitive and sensorimotor function is due to deterioration of common neurological processes (domain-general mechanism)

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4
Q

how does sensorimotor ability indicate biological integrity of brain as a strong predictor for cognitive declines

A

all age differences in WM, EM explained by sensorimtor function
because of fluctuation of sensorimotor abilities increaes with age, this neurological deterioration and its correlation with cognitive ability increases with age

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5
Q

what did Lindenberger & Baltes show regarding sensorimotor functioning and age-impact on intelligence

A

controlling for sensorimotor functioning (vision, hearing, balance, gait) reduced age-intelligence relation (reasoning, knowledge, etc) to null

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6
Q

what is the sensory deprivation theory

A

declining sensory acuity creates communication and mobility difficulties, increasing likelihood of social withdrawal and disengagement from intellectually stimulating activities which could have knock-on effects on cognitive ability over extended period

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7
Q

what is cognitive load on sensory performance theory (opposite to sensory deprivation theory)

A

this causal direction works in reverse
deficit cog ability adversely affects sensorimotor function
eg; deficits sustained attention diminishes capacity to detect auditory/visual stimuli
relationship may be bidirectional even if Lindenberger & Baltes presented evidence for other direction
increase social withdrawal, reduced participation in mentally stimulating activity

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8
Q

difference between longitudinal and cross-sectional studies on sensory, cognitive decline presentations

A

longitudinal shows more modest associations between rate of sensory and cognitive decline, provide support for role of domain-specific factors
cross-sectional accentuates this relationship

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9
Q

give a broad overview of relationship between age and sleep

A

negative relationship is seen between age and sleep (older, less time asleep)

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10
Q

what is WASO

A

waking up after sleep onset

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11
Q

what do older people do in sleep

A

sleep less, awaken more, increased WASO, decreased SWS and activity in PFC, less spindle density, unchanged REM but decreased phasic REM

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12
Q

what was Rasch (2007) study into sleep and memory games

A

play memory games before sleep, presented with odour (roses) during learning
memory improved when representing odour during SWS vs control. no difference in other experiment not presenting odour
evidence for SWS in LTM consols

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13
Q

explain hippocampal-neocortical dialogue during sleep

A

reactivations associated with sharp wave ripples driven by SWS oscillations, syncrhronising hippocampal memory reactivations with sleep spindle
SWS= repeated activation of recently learned info in hippocampus, helps consolidation

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14
Q

apply hippocampal-neocortical dialogue to older people learning

A

less SWS, so less activation in hippocampus to consolidate newly learned info
change to HPA axis, increased evening cortisol level, when low cortisol level needed for memory consolidation in sleep
high cortisol means impacts high number of cortisol receptors in hippocampus, impairing transfer into LTM storage

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15
Q

compare rate of forgetting after sleep between OA, YA

A

rate of forgetting greatly reduced in YA, but OA shows no difference between those who slept and those awake

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16
Q

apply morningness-eveningness questionnaire to OA

A

YA prefer TOD afternoon/evening
OA peak arousal is in morning

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17
Q

explain rough relationships for exercise and cognitive abilities

A

modest but significant correlations between exercise and cog ability independent of other factors, but adding education to model substantially diminished effect

18
Q

what are issues with correlational studies of physical exercise impacting on cognitive abilities

A

inherent assumption physical activity will cause improvement to cognitive ability but not clear if that is direction (those with high cognitive ability chooses to do more physical etc), does not disambiguate direction of physical-cognitive relationship

19
Q

how can interventions for physical activity be created

A

define a control, and treatment group
eg; cardiovascular fitness, resistance, both
better to use passive control group eg, stretching

20
Q

name 4 different aspect of cognitive task

A
  1. executive task
  2. controlled task
  3. spatial task
  4. speed task
21
Q

name characteristics of cognitive executive task

A

planning, inhibition, scheduling mental procedure eg: Flanker task

22
Q

give example of cognitive controlled tasks

A

choice reaction time

23
Q

name example of cognitive spatial task

A

transform or remember visuospatial info, Benton task

24
Q

give example of cognitive speed task

A

simple reaction times

25
Q

explain results of meta-analysis of physical exercises impact for executive, controlled, spatial, speed cognition

A

impacted executive most, then controlled and spatial, then speed

26
Q

what brain plastic changes occur with aerobic exercise

A

increased vol in gray and white matter in OA
(anterior cingulate cortex, supplementary motor cortex, left superior temporal gyrus, right inferior frontal gyrus, anterior white matter)

27
Q

what part of brain shows best effect from physical exercise

A

areas suffereing effects of age, increased hippocampal volume by 2%, associated improvements in spatial memory due to increased blood flow, O2 delivery so prompt structural, funcitonal change

28
Q

what impact can regular physical exercise have on mental health

A

self esteem, self concept, coping with stress, positive social interaction, feelings of control and competency

29
Q

what health issues can physical activity reduce

A

cardiovascular disease, stroke, hypertension, type 2 diabetes, causing dementia

30
Q

apply ageing in real world to workplace

A

more workstations space, more time, louder isntruction and visual aid, longer break, less need of fine motor skills, increased technology, less heavy lifting, awareness training

31
Q

what is hippcampus involved in, and what can increased gray matter cause

A

spatial nav and memory, training causes less loss and improved acquisitions of new language

32
Q

how can reorganisation of somatosensory and auditory cortex help in congenitally blind

A

enhance sensorimotor and auditory skills, and compensatory behaviour

33
Q

whats maladaptive neuroplasticity

A

reorganisation in frontal and hippocampal regions = addiction, phantom limb and tinnitus

34
Q

name issues of cross-sectional research for physical activities

A

difficult for ineference for causal relationships
third variable problem= education, SES, health conscious lifestyle
objective and self-reported studies
ignores non-physical leisure time

35
Q

what was Richards middle age study show

A

exercise age 36-43 slower memory decline than age 43-53
continuous exercise needed in maintaining cognitive capacities
gray matter volume in frontal larger for exercise x2 per week

36
Q

what was a positive correlation age 18 found

A

between cardiovas fitness and intelligence, associated with higher academics

37
Q

what is selective improvement hypothesis in exercise

A

effects of 6 month aerobic exercise training compared to stretching
increased performance on exec control tasks, and counteract hippoc vol loss

38
Q

why is VO2 beneficial for cognitive abilities

A

increased blood flows, nutrition supply, neurogenesis, synaptogenesis

39
Q

why is location of exercising important

A

sensory stimulation

40
Q
A