production animal Flashcards

1
Q

reasons for a down cow

A

metabolic- low ca, mg or phos
ketosis- rarer but more common with twins

injuries- post calving, L6- may be selayed effect due to soft tissue swellong putting pressure on nerves

severe ilness- mastitis, ut infection, metritis (less than a week post calving- day 3-7)

abdominal bleeding

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2
Q

down cow clinical questions

A

when did she calve?- remember its possible for it just to have injured itself even if it has just calved
herd history of down cow? historicaly? this lactation?
how was the calving?- describe why it was difficult if so, big calf? assistance needed?
stage of lactation?
how long has she been down?
have you given her anything?
any other isses on farm? infection? mastitis?
milk records?- has her yeild dropped recently
whats her diet like? any preventative diets for milk fever
did you see her before she went down? was she normal?- was she missed

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3
Q

treatment for enteritis downer cow

A

tmps- cheapest, lowest withdrawl
NSAIDs

fluids- iv when scouring- hypertoninc fluid MUST be followed with lots of oral fluids
nursing
reasses- lifting, check for mastitis as can occur as shes been laying

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4
Q

antibiotics for CODD

A

longer acting
amoxicillan
penecillan

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5
Q

antibiotics for foot rot

A

oxytetracycline

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6
Q

prevention for foot rot

A

vaccination
culling
foot bathing
avoid trimming

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7
Q

prevention for CODD

A

avoid gathering- FOOT BATHS RISK AND NOT EFFECTIVE
avoid trimming

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8
Q

describe the 5 point plan for herd foot health

A

vaccinate- foot vac
cull- tag each time treated and then once threashold reached cull avoid- biosecurity
treat- antibiotics (Penicillin and oxytetracycline), foot bath
quarentine

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9
Q

dichelobater nodosus

A

causal bacteria of foot rot and scald
survives for 30 dyas in soils
can be vaccinated against

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10
Q

causal agents and key control of digital dermatitis

A

fusobacterium necrophorum
treponemes- spirochetes- commensal on feet and enter through wound and pressure cuased by dirty moist enviroment

key control is footbathing, slurry managment, hygene,
treatment- topical oxytet spray

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11
Q

causal agents and key control of white line disease

A

non infectious
nurtition- trace minerals ( zinc, biotin(made in rumen an high producing cows on concentrates dont produce enough))
sheering forces- handling, flooring, sharp turns
trimming

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12
Q

causal agents and key control of interdigital dermititis

A

foul in the foot

infection
fusobaterium necrophorum

NSAIDs- ketoprophen
systemic antibiotics- oxytet, betomox (amoxixillan best as short withdrawl), not responding- macrolide (in milking cows tyloxine)

control of hygeine
management of slurry
footbath

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13
Q

causal agents and key control of sole ulcers and haemorrage

A

standing times
thin/ fat cows

treat with trimming and blocking, spray and nsaid
salacilic acid contrevercial and not generally evidenced based or on licence
cubical management

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14
Q

causal agents and key control of interdigital growths

A

continued slurry exposure
interdigital dermatitis exposure
gentetics

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15
Q

key causes of lameness in cows

A

solar haemorrage
sole ulcers
digital dermatitis
white line disease
interdigital dermatitis
interdigital growths

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16
Q

ketoprofen

A

short acting but cheap with 1 day withdrawl

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17
Q

rumaphen

A

good middle ground between metacalm and ketoprofen, shorter withdral and cheaper than metacalm but longer lasting than ketoprofen

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18
Q

metacalm

A

more expensive with longer withdrawls than ketoprophen but lasts longer

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19
Q

antibiotics for toxic mastitis

A

injectable not often used- norodene sometimes used for toxic cases in conjunction with antibiotic tubes- tmps: not evidenced unless sings of baceremia seen

flunixin- 24 hour duration and longer milk withdrawl with antiinflamatory effects

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20
Q

What is the legal limit for somatic cell count

A

400,000 bulk tank

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21
Q

what is a normal somatic cell count

A

150,000- every 100,000 over could be 10% of the herd with disease

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22
Q

diagnostics for mastitis

A

CMT
3M Clearanced

milk samoling- technique
-on farm culture vs lab
bacteriology vs ocr

bulk tank diagnostics-t

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23
Q

treatment of mastitis

A

availabe?
antibiotic ocnsiderations?
licensed?
Cure rate?- 50 % average

24
Q

RDA clin exam

A

right sided ping
right side succussion- when absent this is lower on the diagnostic list
low temp
pain signs- sitiin gdown, high hr high rr

25
what is the whithers pinch test
common diagnostic test for traumatic reticuloperitonitis is the withers pinch test, in a normal cow if you pinch their withers the cow should dip and perform a shrug like action. In a cow with tyre wire they will be reluctant to perform the dip, or will grunt whilst dipping; indicating pain.
26
abdominal catastrophie
non specific abdominal issue treatment: conservative medical managment: coffee fluids roll buscopan- keep eye on withdrawl NSAID- ketoprophen has short withdrawl but if giving buscopan just give metacalm review in a couple of hours- be sensible about time of day! look at schedual and give time to do other treatment if needed- never let sun go down on RDA. if not better go to ex-lap surgery- rules out emergency slaughter ex lap
27
differentials for right sided ping
caecal torsion?- often result in zero faecal output dilated caecum- not as painfull as some other differentials small intestinal issue spiral colon torsion RDA- offten acoumpanied by pos secussion gas in the euterus- woud be felt on rectal peritonitis- can produce ping!!!- bubble wrap feeling on rectal- this is important as in this senario you CANNOT send for slaughter
28
pnemonia in sheep
diagnostics-- swab histopathology from lungs in any dead baermans flotation test faecal egg count wheelbarrow test ultrasound post mortem diffferentials- bacterial pnemonia- pasturelosis, manhemia haemolitica clostridial disease viral pneumonia lungworm Ovine pulmonary adenocarcinoma (OPA) fluke- aub acute faciolisis - especially if grazed with or ater cows as cows create big burden on pasture texel throat- Laryngeal chondritis bolus gun injury treatment for non specific cause- oxytetracicline- good penetration, cat d, cheap, la prep available, broad spectrum (clear anitbiotics clear the airways) vaccinate- heptivac + but not the sick animals give to non pyrexic members of herd and recovered ones later multivit injection?- price? time? supportive rehydration check back in tomorrow euthanasia?- cant be casualty slaughter as pyrexia captivebolt most efficient
29
Group 1 antihelmenthic
1-BZ (white) Benzimidazoles broad spectrum effective against trichostrongyles, haemonchus, nemotodirus(esspecially effective) and mature fluke at higher doses
30
group 2 anthelmintics
2-LM (yellow) levamizole broad spectrum effective against trichostrongyles, haemonchus, nemotodirus
31
group 3 anthemintics
3-ML- (clear) macrocyclic lactones broad specrum effective against trichostrongyles, haemonchus, nemotodirus
32
groups 4 anthelmentics
4-AD-(orange) amino-acetonitrite derivatives broad spectrum
33
group 5 anthemintics
5-SI-(purple) spironoindoles
34
on farm parasite prevention
breed own replecments-closed herd quarentine incoming stock healthy livestock- bvd vaccination lungworm vaccine nutrition natural antiparisitics- herbal leys trees- willow= cobalt, salacilic acid, tanin longer sward lenght- ryegrass- revents eggs from clinbing to top of feed mixed graxing- different parasites regional warnings/ forecasts breedingrest perios youngstock paddocks multipliers
35
sheep mastitis predisposers
teat end damage- large lambs on ewes orf- jagged pasture a predisposer to this thistles and gorse chapped from cold staph,aureus from ewews skin manhemia heamolytica from lambs mout strep dysgalactica- ?
36
treatment for sheep mastitis
tubes can damage teats systemic antibiotics more common- theoretically amoxicillan and oxytetracicline in very early stages resistance in manheimias and pasturellas for oxytet long acting macrolides best - draxin (Tulathromycin) has a nice 16 day withdrawl and lasts for a long time- wont save qaurter NSAIDS- to treat toxic shock that is often a factor often not seen in early stages as udder harder to monitor in sheep
37
prevention of mastitis in sheep
investigation of teat damage adequate ewe nutrition orf prevention vax and thistles decreased exposure to incliment weather often difficult ot control- strep. dysgalctiae taxels predisposed- management? predisposition?
38
joint ill in lambs
massive welfare problem cs- sudden onset lameness, pain heat and swellin in multiple joints, reduced feeding, ill thrift as young as 5 day but often 2-4 weeks high lamb losses in bad outbreaks naval BIGGEST route of infection- strict naval hygene- iodene and spirit ambing shed and pen hygene also important
39
joint ill pathogens
strep. dysgalctiae e.coli staph. aureus other streps t.pyogenes naval or oral rout haemotogenous spread route of intorduction hard to pinpoint
40
joint ill treatment
long course of penecillan- procaine penecillan for a week best but betaox/ trimox alos works. long acting common so offlicense injection daily has been done at least 7 days NSAIDs or steriod- steriod preference?? corticosteriod on day 1 and 2-3: o.5ml raidexon caught early is far better success
41
strep.dysgalctiae
big joint ill and mastatis pathogen survives for long time in DRY conditions- so even seemingly clean shed can hold infection moving sheep a solution? try to reduce load in shed- high output commonly struggle- reducong stocking density good solution
42
watery mouth
e.coli cs- watery mouth, fluid in belly, lethargy, scour e.coli ingested before colostrum, in conjunction with poor or too little colostrum or in large quntites twins, triplets and quads more prone investigation- look at shed: clenliness? mastitis? hand hygene? COLOSTRUM- igg levels can be used, total protiens from less than a week old lambs more practical. nutrition and food acess important factor to quality poor pem hygene and nutrition two big factors prelambing bloods can be doen- 2-4 weeks before lambing. blood sample ewews to look at total protene and urea (indication of short term protiene) and bhps to check protiens and energy for milk prodution
43
reproductive managment of ewes
Flushing – Increasing nutrition with forage or concentrates before breeding, increases ovulation rate in ewes Intravaginal sponges impregnated with progestogens (medroxyprogesterone acetate or fluorgestone acetate) have been the most widely used agents for control of ovulation but are not available for clinical use in the US. Progestogen treatment is administered for 10–14 days in sheep CIDR ovis also use progesterone and are currently more availcable- T-shaped nylon insert molded with silicone rubber skin containing progesterone that's released at a controlled rate into the bloodstream after insertion- 12 days removal causes rapid drop in progesterone and therefore ovulation, put tups in straight away PMSG melatonin implants- regulin: behind the ear as this part is not going into the food chain teasers light managment
44
average cost of mastitis per case
£250
45
on average per year how many cases of mastitis per 100 cows is there
35
46
sudden death in cattle
anthrax hardwear disease clostridial disease- black leg, pulpy kidney lightning strike tetnus Botulism plant toxcicity- yew, acorns, bracken, rhododendron, ragwort ruminal acidosis caudal vena cava syndrome copper or lead poisoning haemorrage thyamine defficancy heart attack trauma fluke- black disease fog fever- l-tryptophan staggers- hypomagnesaemia colic- intestinal torsion halocure overdose electrocution iatrogenic- bolus gun injury
47
sudden death with hypomagnesaemia in cows
at grass evidence of paddling vitrous humour to test mg levels
48
sudden death with colic in cows
can be very quick ping palpable per rectum scant faeces torsed cas=ecum rda teisting and causing toxicity small intentinal torsions or interssuseption mesenteric torsion
49
sudden death with poisoning in cows
lead- car battery- blind if alive- found in blood and kidneys zcrorn creosote bracken yew rhodedendrin fresh autumn grass- fog fever
50
sudden death from ruminal acidosis in cows
change of diet? acess to grain? rumen ph <5 at pm
51
lightning strike/ stray voltage in cows
often multiple animals near tree can travek in wet ground instant- grass in mouth singe marks in midline and medial hindlimbs petechiation of dermis often clained for insurance
52
haemorrhage as a cause of sudden death in cows
could be caudal vena cava syndrome- bilateral epistaxsi milk vein-abcessate vaginal/ uterine artery
53
blackleg as a cause of sudden death in cattle
ingested clostridia deposited in muscles and react to trauma or execive excersise- young strong beef animas rapid autolysis usually best anias in herd acute onset, febrile crepetiuus, swolen limbs alos in myocardium and diaphram
54
clostridial disease as a cause of suden death in cows
black disease- migration of immature liver fluke malignant oedema- often im injection activating latent spores tetanus- lockjaw, ears back, hindlin=mb stifness, raised tailhead. castration and isiopathic due to gut overgrowth botulism- flaccid paralysis in 4-7, tounge most obvious to begin with
55
total protien as a measure of colostrum imunity
albumin is made during pregnacy but globulin is obtained by maternal antibodies low total protien is a crude reflection of globulin levels ASSUMING albumin is normal and the animal is not dehydrated cheap and quick method constent measures of 35-40 flags problems
56
causes of scour
e.coli- irst few days and ofen more septicemia crypto- mostly uner 3 weeks rota virus corona virus
57