Pregnancy Flashcards

1
Q

Changes in the centre of gravity

A

As your baby grows as pregnancy progresses, there is more weight in front of the centre of gravity.
– meaning the posterior chain has to work harder, putting it under more stress, to maintain an upright standing position
– this puts the facts, discs, ligaments and muscle under more pressure

The abdomen will have to expand a lot, putting pressure and stretch through the fascial structures like the lines alba and the semilunaris.

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2
Q

Changes in spinal curves and how the body adapts during different trimesters

A

12 weeks
– thoracic spine kyphosis increases
—- due to increased anterior weight (breasts become bigger)
– flexion affects discs

– reduced lumbar spine lordosis
—- due to a posterior pelvic tilt that is caused by a shortening of the rectus abdominalis
– affects discs more due to a flatted lordosis, so the spine is slightly more flexed

– compromised TOS
– increased sacral counternutation

– baby is the size of a grapefruit
—- uterus rises above the pelvic rim

Anterior musculature:
– rectus abdomens shortening, which may cause the posterior rotation of the pelvis
– anterior hip muscles need to be stretched

24 weeks
– CSP lordosis increases

– expanding uterus and displaced viscera produce pressure underneath as it elevates up
—– so the TSP needs to extend

– TSP
—- kyphosis increases due to increased anterior weight
—– lower ribs are displaced and need to expand laterally
—– distal sternum elevates
—– TOS may be present due to the viscera being pushed up and there is 1st rib dysfunction

– pelvis still rotated posteriorly, just starting to rotate anteriorly
– so lumbar lordosis still reduced, however anterior rotation of the pelvis will cause an increased lumbar lordosis again

30 weeks
– 80% of women develop a lordotic posture
—- approximating the facets causing facet irritation

– there will be altered head carriage (may be thrown back or forward), to help with posture

– increased kyphosis TSP
—- due to enlarged breasts, belly having more weight
—- this can play a part in the increased lumbar lordosis as well
—- strain at the CT junction
—- thoracic inlet compromisation

– deep lumbar lordosis
—- anterior pelvic rotation and increased TSP kyphosis plays a part in this
—– stretch through the rectus abdominals (60% of cases rectus diastases occurs)
—- there is pubis strain in 89% of cases in 2nd or 3rd trimesters

Pelvis:
– anterior rotation
– increased sacral nutation
– coccyx needs to flex

Hips:
– widened pelvis, leads to lateral rotation of the hips
– leading to an altered orientation of the knee and feet
– leading to a waddling gait

40 weeks
– pelvis rotated posteriorly, so lumbar spine flattened

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3
Q

Fluid dynamics

A

Pregnancy can affect venous return

How supine position affects the inferior vena cava:
– the positioning of the foetus tends to obstruct venous return, this is due to the uterus compressing the inferior vena cava, reducing sometimes more than 80% of blood flow in the inferior vena cava
– the pregnancy uterus can also partially block the aorta leading to a reduction of about 30% in blood flow through the aorta

Pregnancy is the leading cause of varicose veins:
– women blood during pregnancy usually becomes hypercoagulable (increased tendency to stick together and form clots)
– vein wall tension can decrease leading to the stagnation of blood and this paired with hypercoagulability can predispose women to varicose veins

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4
Q

Internal support- ligaments of the uterus

A

Form a sling and support the uterus and bladder

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5
Q

Hypermobility vs instability, the role of relaxin

A

Hypermobility- where someone has a larger range of motion in a joint than normal.

– pts that are pregnant are hyper mobile, can be very difficult to treat. Hypermobile patients should avoid doing yoga.

Instability- where tissues, such as muscles, ligaments and bones weaken. Once they are weak, they no longer hold the bones of the joint in proper place.

Role of relaxin:
– hormone only produced during pregnancy.
– its role is to increase flexibility of the ligaments during pregnancy to help open up the pelvis during labour.
—- it alters the properties of cartilage and tendons by activating collagenase
– 89% of pubic synthesis disorders occur in the 2nd and 3rd trimesters
– peak production at 12 weeks

– relaxin can cause ligament laxity within the body, this may cause instability at certain joints. For example, if there is ligament laxity within the spine it can cause instability at segmental levels of the facets, or the vertebrae

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6
Q

How neurology can be affected by pregnancy

A

Myalgia parasthetica, due to its position it can be caused by compression on the lateral femoral cutaneous nerve

Sciatic irritation (maybe due to tight muscles due to a tight posterior chain) may cause the sciatic nerve to be compressed (piriformis syndrome)

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7
Q

Spinal curves

A

Increased TSP kyphosis (due to anterior weight)
– posterior stretching of the discs, leading to an increased likelihood of herniation
– rib head facets are displaced leading to joint surface irritation

Increased CSP lordosis (due to increased TSP kyphosis, so the head orientates itself upwards)
– approximates the facets, leading to an increased likelihood of facet dysfunction

Decreased LSP lordosis (12 weeks, due to posterior pelvic tilt)
– puts facet capsules on stretch, also puts more pressure on discs, leading to an increased likelihood of herniation

Increased LSP lordosis (30 weeks, due to anterior pelvic tilt):
– approximates the facets, leading to an increased likelihood of lumbar facet dysfunction

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8
Q

ligaments

A

Relaxin causing ligamentous laxity, which can lead to instability at a vertebral level.

Leads to increased range of movement at joints and increased joint irritation.

May also lead to pubic symphysis problems

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9
Q

Muscles

A

Muscles of the posterior chain have to work harder due to increased anterior centre of gravity.

Increased muscle tone due to ligament instability

Both lead to increased muscle fatigue and chronic pain

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10
Q

Vascularity

A

Decreased venous return due to pressure on the inferior vena cava due to positioning of the foetus
– worse in the supine position

Reduced blood pressure

Increased likelihood of heamorhoids

Increased chance of varicose veins

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11
Q

Neurology

A

Myalgia parasthetica due to increased pressure on pelvic bowl, which puts pressure on the lateral femoral cutaneous nerve (L2, L3).

Sciatica due to increased tone and tightness of posterior chain muscles
– especially the piriformis muscle, illiciting sciatic symptoms

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12
Q

Relaxin

A

Hormone that is only released during pregnancy, intends to increase ligament flexibility in order to open up the pelvis to make it easier for the mother and baby

Increases ligament flexibility by changing the properties of ligaments by releasing collegenase.

Can cause increased ROM and lead to ligament instability at a vertebral level

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13
Q

Ligaments of the pelvic floor

A

Pubocervical ligament

Inguinal ligament

Transverse cervical ligament

Uterosacral ligament

Retrovaginal septum

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14
Q

Hypermobility and pregnancy

A

They may be difficult to treat

They may have restrictions in the thoracic spine, with increased tension in the ES muscles to try and stabilise the hyper mobile segments

Hypermobile patients should avoid doing yoga- as they are already mobile enough

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15
Q

Hypermobility vs instability

A

Relaxin can cause instability of the joints

Hypermobility- having unusual or abnormal ROM at a joint

Instability- When tissues around the joint weaken meaning they cant hold the joint in place properly so they weaken

Hypermobility:
– multiple joints involved
– visceral symptoms
– chronic pain for 3 months
– recurring episodes of pain in the same area
– exercise can improve or exacerbate pain

Instability:
– single joint involved
– pain may resolve itself in days
– no visceral symptoms

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