physiological adaptations to resistance training Flashcards

1
Q

neural adaptations with any new task….

A
  • early increases in strength (newbie gains)
  • coordination
  • learning increases activation of prime mover muscles
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2
Q

recognizing changes from RT

A
  • depends on movement literacy, skills that transfer across a person
  • high movement = easier
  • low movement = harder
  • general training age
  • after a plateau, gains start to come from physiological adaptations
  • cross-over effect : gains/strength travel when working on one muscle
    ex. greater cross-over = higher hypertrophy
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3
Q

firing frequency

A
  • RT allows motor units to maintain consistent firing intervals at lower rates = maintaining forceful contractions for longer periods of time
  • higher threshold MU requiring more neural input) are able to fire longer before firing intermittently or shutting off
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4
Q

specifically to velocity

A
  • nature to which exercise is performed
  • change = training in that manor
  • MAX RFD (exp > heavy resistance training (HRT))
  • Peak force (HRT > EXP)
  • MU activation (EPX > HRT at early peak onset)
  • SAID principle
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5
Q

increased synchronization

A
  • synergists and antagonists
  • untrained
  • strong and rapid movements
  • highly precise
  • all these increase antagonist activation
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6
Q

hormone response to RT

A
  • most influential to alter hormone concentrations
  • optimal programing to change these hormones
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7
Q

optimal programming includes …

A
  • MU recruitments
  • exercise selection
  • intensity, volume, rest
  • nutritional intake
  • need to recover in order to capitalize on stress!
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8
Q

testosterone

A
  • increases protein synthesis + inhibits protein breakdown rates
  • RT increased total test. in males, conflicting results in females
  • differences in training age, physiological ceiling, volume intensity (> 80% 1RM)
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9
Q

testosterone response to RT

A
  • increases hypertrophy
  • free testosterone in RT vs. endurance training
  • certain training factors impact differently
  • sequencing of exercise
  • be careful of diminishing returns !
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10
Q

sequencing of exercise

A
  • multijoint over single joint exercises INCREASES testosterone
  • 80-85% elicits highest response
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11
Q

yerkes-dodson law

A
  • relationship between pressure and performance
  • 4 sets, 6-8 reps, 80-85% intensity
  • sweet spot at the top of the bell-curve
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12
Q

testosterone response in females

A
  • limited concentrations -> minimal response to RT
  • possible chronic adaptations with correct programming
  • low volume circuit training ( increases at 12 weeks/ decrease baseline at 24 weeks)
  • periodized high volume multi set programming increases @ 12 weeks / doubly increases at 24 weeks
  • may be a result of growth hormone in females
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13
Q

human growth hormone

A
  • stimulates uptake of amino acids to make muscles
  • secreted by pituitary gland
  • RT causes secretion of GH isoforms with extended half lives = sustained action on target muscle tissue including skeletal muscle
  • upregulation of IGF-1
  • temporary elevation of GH aprox. 30 min part RT
    -> impaceted by exercise selection, intensity, volume, and rest
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14
Q

cortisol

A
  • “stress hormone” (stress is stress is stress)
  • detrimental to muscle development b/c it reduces protein synthesis
  • acute response increases with high intensity/volume, volume being the biggest driver, and rest periods decreasing the acute response
  • buffered by CHO (carbs) (reduces cortisol possibly)
  • when following a training program there is a less acute response to cortisol b/c the body becomes used to it
    ex. crossfit = bad to buffer cortisol and recover
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15
Q

Insulin-like growth factor (IGF-1)

A
  • no optimal prescription
  • provided main anabolic response for whole body
  • deceases proteolysis (atrophy)
  • hypertrophy increases the rate of protein synthesis
  • RT enhances acute response, IGF-1 is released during mechanical stress as it “kick-starts” hypertrophy at a muscular cellular level
  • chronic levels affected by long term RT
  • resting levels = trained > untrained males
  • small/no change over 4-12 weeks
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16
Q

why is henneman’s size principle important for hypertrophy ?

A
  • type I muscle fibers are more oxidative and have a VERY HIGH training status in most people because they are used constantly
  • type II are used infrequently and have a lower training state and response to training
17
Q

primary mechanisms that effect muscle growth

A
  1. mechanical tension
  2. muscle damage
  3. metabolic stress
18
Q

3 keys of mechanical tension

A
  1. nature of the tension (active/passive)
  2. role of external resistance
  3. effects of fatigue
19
Q

nature of the tension

A
  • active is preferred
  • passive includes myofibrils and muscle segments
20
Q

role of external resistance

A
  • extent of MU firing frequency is influenced by % external resistance
  • larger resistance = slower contraction velocities = more actin-myosin cross-bridges = increase in tension that each fiber provides
  • fatiguing muscle types as exercise progresses
  • time / tension = giving muscle fibers time to be exposed to elicit cellular responses
21
Q

effects of fatigue

A
  • increase in fatigue = decrease in contraction velocity
  • working muscle fibers = unable to produce required force to do the work — higher threshold MU required
  • slower speeds = more cross-bridging = more mechanical tension on individual fibers
  • increase motor recruitment = greater stim of muscle fibers = increase hypertrophy response
22
Q

muscle damage

A
  • can occur to various components of contractile tissue (inflammatory response )
  • eccentric = increase in muscle fiber length
  • concentric = increase in fiber diameter
  • need to have a balance and alternation!
23
Q

metabolic stress

A
  • anerobic glycolysis for ATP = build up of lactate, hydrogen ions (acid), and phosphates
  • muscle ischemia
  • fatiguing sets that produce metabolic stress are directly linked to mechanical tension –> more high threshold MU needed to be recruited
24
Q

muscle ischemia

A
  • restriction of blood flow causing a shortage of O2 can increase metabolites