Physics Flashcards
Over or underestimation of TDCO: Injectate bolus volume is greater than the programmed volume
Underestimation
Over or underestimation of TDCO: Large volume of fluid is administered during a CO reading
Underestimation
Over or underestimation of TDCO: Injectate solution’s actual temperature is colder than the preprogrammed Ti
Underestimation
Over or underestimation of TDCO: Self-measuring Ti probe is warmer than the actual injectate temperature
Underestimation
Over or underestimation of TDCO: Injectate bolus volume is less than the programmed volume
Overestimation
Over or underestimation of TDCO: Injectate solution’s actual temperature is warmer than the preprogrammed Ti
Overestimation
Over or underestimation of TDCO: Self-measuring Ti probe is colder than the actual injectate temperature
Overestimation
In order for a surgical fire to occur, all three components of the “fire triad” must be present. What are these 3 things?
hese include a fuel (e.g. surgical prep solution, surgical drapes, sponges, endotracheal tube, oxygen tubing), an oxidizer (e.g. oxygen, nitrous oxide), and an ignition source (e.g. lasers, electrocautery, drills, fiber optic light source).
Most popular ignition source for fires?
Electrocautery unit Lasers have the capability of generating the most heat, but the near-universal use of electrocautery makes it the most common ignition source.
Risk factors for OR fires:
Risk factors for operating room fires include monitored anesthesia care with an open oxygen delivery system, outpatient surgery, head/neck/upper chest surgeries (85% of all surgical fire claims), and older patient age.
SvO2 and sepsis-late vs early.
Peripheral tissues are unable to extract and utilize oxygen effectively in late sepsis. Since oxygen utilization decreases, more oxygen stays in the blood and SvO2 is increased. In early sepsis, oxygen demand may be too large for oxygen supply so there is potential for decreased SvO2. However, the microvascular shunting aspect of sepsis explains why there is reduced oxygen extraction seen with distributive shock.
As a whole, I would say that sepsis increases SvO2, but if they differentiate between early vs late, early has the potential for decreased SvO2.
Myocardial infarction and SvO2?
Myocardial infarction results in damage to cardiac cells resulting in both increased O2 demand and decreased cardiac output.
How does the SvO2 thing work? Normal SvO2 level?
When O2 demand increases or O2 delivery decreases, the body compensates by increasing cardiac output and/or increasing O2 extraction. An SvO2 less than 75% indicates that more O2 is being extracted from the blood by peripheral tissues before it returns to the right heart.
How often do pacemakers need to be evaluated?
Once every 3-12 mos
Electrocautery does what to a pacemaker?
It inhibits it, so whatever the underlying rhythm/rate is, the electrocautery will bring that out.
Asynchronous mode will ensure
proper pacing un-inhibited by electrocautery.
T/F: For patients that are not pacemaker dependent, placing a magnet could be detrimental.
For patients that are not pacemaker dependent, placing a magnet could be detrimental.
Why should you consider serum electrolytes with a pacemaker?
In addition, checking serum electrolytes (especially serum potassium) should occur because alterations can make capturing of the pacemaker easier (hyperkalemia) or more difficult (hypokalemia).
Positioning of pacemakers: 1-5
For position 3, what does D mean?
1: Chambers Paced
2: Chambers sensed
3: Response to sensing
4: Programmability
5: Multi-site pacing
For position 3, D means that it can both inhibit and trigger a response.
In aortic stenosis, are you relying on the atrial kick?
YEs please!
Why do EMI and pacing not get along? If you have to use EMI, which one is better? If you can’t use the better one, then what?
EMI may result in oversensing by the pacer causing inappropriate inhibition on the pacing function. If electrocautery is required, a bipolar cautery device should be used if possible else the grounding pad should be placed as far away from the pacemaker as possible without the electrical pathway going through the pacemaker. If feasible, bursts should be limited to less than 10 seconds.
Let’s say that you can’t avoid monopolar EMI in a pt with a pacemaker, what could you do? What happens if you place a magnet over an AICD?
Reprogramming of the device may be the best way to avoid intraoperative problems if monopolar electrocautery is planned. Most clinicians will place the patient into an asynchronous mode (AOO, VOO, DOO), this causes the pacemaker to fire at a fixed preset rate, independent of the patient’s underlying cardiac activity. The rate will need to be higher than the patients underlying rhythm to try to ensure that an R-on-T phenomenon does not occur, protecting the patient from arrhythmias.
This is crucial for patients that are not pacemaker dependent. (to prevent the R on T thing) Setting the patient in asynchronous mode can result in competition with the patient’s intrinsic rhythm and may result in the development of a malignant arrhythmia, thus appropriate means to treat this should be available (namely a defibrillator and appropriate antiarrhythmic and blood pressure support medications). In addition, if the patient has an automated implantable cardioverter defibrillator (AICD), this needs to be deactivated if electrocautery is planned under certain circumstances. Tachyarrhythmia therapies, such as overdrive pacing, also need to be disabled. This may also be completed with a magnet in modern devices. Of note, a magnet does not affect pacemaker function for modern AICDs. Rather, magnets inhibit defibrillation in those devices.
Placing a magnet and going into asynchronous mode is not without risk-explain.
Placing a magnet is not without risk since asynchronous pacing may trigger ventricular asynchrony in patients with hypoxia, electrolyte imbalances, and myocardial ischemia.
T/F:Whenever minute ventilation (calculated as tidal volume * respiratory rate) exceeds fresh gas flow, rebreathing of exhaled gases will occur, leading to a lower FiO2, compared to the dialed O2 concentration.
True, this question was saying that you want to prevent rebreathing of gases, and so basically,When using deep breathing, minute ventilation can easily exceed the fresh gas flow delivered by the anesthesia machine. For example, if one assumes a 70 kg patient with normal pulmonary function has a ~5 L vital capacity, breathing four vital capacity breaths every 30 seconds would mean a minute ventilation of 40 L/min. Even if the patient does not achieve vital capacity in each breath, deep breathing can exceed the 15 L/min delivered by the anesthesia machine, leading to rebreathing and lowering FiO2.