Peripheral Arterial and Venous Disease Flashcards

1
Q

Outline deep veins

A

Found in the muscle

Flow aided by muscle contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Outline superficial veins

A

Found in the subcutaneous fat

Superficial veins always flow into deep veins by perforating veins, not the other way round

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is the purpose of fascia around a compartment?

A

When a muscle contracts it pulls the facia inward creating a -ve pressure

This aids blood flow in the veins

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the deep veins in the leg?

A
  • IVC
  • Common iliac vein
  • External iliac vein
  • Femoral vein
  • Popliteal
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the superficial veins?

A

Long saphenous – drains into the femoral vein, anterior to the medial malleolus

Short saphenous – drains into the popliteal vein

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the one constant vein in the body?

A

Long saphenous vein is ALWAYS anterior to the medial malleolus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What direction does blood flow in the aorta?

A

Nearly all the blood travels towards the periphery

Very small amount of blood is refracted back due to bifurcations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Does Doppler measure flow or velocity?

A

Velocity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Why is the velocity high in the common iliac than the aorta?

A

Same amount of blood flowing through a vessel that has no branches

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the def of a varicose vein?

A

Tortuous twisted or lengthened vein

Vein wall is inherently weak, leads to dilation and separation of valve cusps = become incompetent

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the symptoms of varicose veins?

A

Heaviness, tension, aching, itching

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the complications of varicose veins?

A

Haemorrhage, thrombophlebitis (thrombosis prod inflam including pain)

Oedema, skin pigmentation (ferrous oxide), varicose eczema, lipodrmatosclerosis(hardened fat)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What causes venous ulceration?

A

Calf muscle failure venous hypertension venous ulcers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What causes calf muscle pump failure?

A
  • Failure calf contraction = immobility, obesity
  • Deep vein incompetence
  • Volume overload
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is a retrograde circuit?

A

Superficial vein incompetence leading to ulceration

Valve incompetence setting up an incorrect circuit of blood flow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is the pathophysiology of arterial vs venous thrombosis?

A

A = changes in lining of vessel wall

V = changes in blood flow

17
Q

Why does stasis lead to venous thrombosis?

A

Normally stasis plus another ‘provocateur’ = oral pill, dehydration, cancer

18
Q

What is natural collateral circulation?

A

Particularly common across joints – otherwise when flexed joint of flow would take place

Devel in response to stenosis

When there is acute ischaemia collateral circulation has no time to devel (need to reverse in 6 hours, hyperkalemia)

19
Q

How does DVT present?

A

Calor, dolor, functio laesa, rubor, tumor

Pain, cannot walk, swelling, calf tenderness, distended warm veins

20
Q

Why is surgery associated with DVT?

A

Immobility prior and post surgery

Surgery is trauma = body’s response includes a prothrombic state

21
Q

What is acute limb ischaemia?

A

Impaired blood supply over period of minutes

No collateral blood devel

Commonest cause = embolism

Symp/signs = pain, paralysis, paraethesia, pallor, perishing cold, pulseless

22
Q

What is fixed mottling?

A

Non-blanching skin

Thrombosed Hb

23
Q

Outline chronic peripheral arterial disease?

A

Intermittent claudication = pain in muscles of lower limb elicited by walking commonly due to diseased superficial femoral artery

24
Q

What are the arteries of the lower limbs?

A

External iliac artery

Common femoral artery (branching off – profunda femoris artery)

Superficial femoral artery

Popliteal artery

Divides into – anterior tibial artery, peroneal artery, posterior tibial artery

25
Q

What is the mid-inguinal point?

A

Half way between ASIS and pubic synthesis

Femoral pulse felt here

26
Q

What places can pulses be felt?

A

Femoral – mid-inguinal point

Popliteal – deep in popliteal fossa

Dorsalis pedal – lateral to extensor hallucis longus tendon

Posterior tibial – behind medial malleolus

27
Q

What is critical ischaemia?

A

Rest pain = pain in foot at night when lying

Ulceration/gangrene

28
Q

What is ischaemia?

A

Insufficient blood supply to cell, lack of O2, pH drops

Reversible

29
Q

What are the diff types of ischaemic damage to the heart?

A

Unstable angina
Stable angina
MI

30
Q

Outline referred pain

A

Visceral and somatic afferents synapse in similar place in spinal cord

Brain cant tell if pain is from skin or heart

31
Q

What beats are irregularly irregular?

A

Ventricular/atrial ectopics

AF

32
Q

What beats are regularly irregular?

A

2nd degree heart block

33
Q

What is the cause of a bounding pulse?

A

Lowing of the diastolic pressure = widens pulse pressure = bounding

34
Q

Why does raising the arm exaggerate a bounding pulse?

A

Increased pulse pressure as blood falls away more profoundly

35
Q

Why is S2 normally split on inspiration?

A

On inspiration the chest wall moves out and the diaphragm flattens = -ve pressure = air rushes into the lungs and more blood drawn into the right side of the heart

= pulmonary valve closes after aortic valve = split second heart sound

36
Q

Why are right-sided murmurs increased during inspiration?

A

-ve pressure = more blood drawn into the right side of the heart

Blood flowing though tricuspid and pulmonary valves increased = hear R-sided murmur better

37
Q

Why are left-sided murmurs increased during expiration?

A

+ve pressure = more blood pushed out of the heart

More blood flowing through the mitral and aortic valve = hear L-sided murmurs better