periodontal treatment Flashcards

1
Q

what are the 5 phases of treatment planning?

A

1- pain relief and initial exam
2- cause-related therapy
3- re-evaluation
4- definitive tx plan
5- maintainance

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2
Q

what is involved in phase 1?

A
  • always begin with relief of acute pain
  • full exam including history, clinical exam, special investigations, diagnosis
  • formation of tx plan and gaurded prognosis
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3
Q

what is involved with phase 2? INDIVIDUALISED

A

identify and control risk factors and begin active tx

hygiene phase therapy- OHI and smoking cessation

NSPT- instrumentation related- removal of plaque retentive factors, supra/sub ging PMPR, tx of furcations

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4
Q

what is phase 3?

A

re-evaluation in 10-12 weeks:
- look at the tissues response to tx
- look at pt cooperation and motivation
- assess their periodontal status i.e stable, partly responsive or non-responsive, and decide whether they should move to phase 4, phase 5 or palliative care.

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5
Q

what is phase 4?

A

definitive treatment - if patients are non responsive or partly responsive and have good OH- 1st line of treatment would be repeated treatment. If more complex e.g FI or complex root morphology or complex med hx- pts may be referred for chemical adjuncts and periodontal surgery.

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6
Q

what is phase 5- maintenance phase?

A

if patients achieve stability- they are still a periodontitis patient.
- they can move onto maintaince recall system where they will get PMPR as and when required.
- initially may require more frequent appts to demonstrate stability over time and ensure good OH.
- this is an individualised recall system based on things such as susceptibility, risk factors, pocket depths, initial tx required to reach stability.
- if they become unstable which could be due to poorer OH or taking up smoking- can be moved back to phase 4 for further treatment.

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7
Q

what is palliative care?

A
  • when patients are unable to achieve stability
  • patients will be given simple scale and polish every 2-3 months
  • aim is to keep patient comfortable with a functioning dentition
  • must be informed of palliative care
  • if OH improves and potential to achieve stability can be moved off of palliative care.
  • must be informed that teeth of poor prognosis or causing any issues will likely be xla.
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8
Q

what are some reasons that palliative care may be the only option?

A

those who cant and those who wont
- poor OH
- poor compliance
- poor manual dexterity
- immunocompromised
- complex medical history

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9
Q

what are the types of uss?

A

magentostrictive and piezoelectric

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10
Q

how does magentostrictive uss work?

A

electrical current is passed through a coil of wire within the handpeice which causes rapid expansion and contraction of metal stacks within the handpeice causing vibrations
- can use all sides
- tip vibrates in back and forth pattern

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11
Q

how does peizoelectric uss work?

A

electrical energy is used to acitivate crystals within the handpiece which cause vibrations
- can you 2 sides
- tip vibrates in eliptical pattern

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12
Q

what are the modes of action of USS?

A

mechanical- movement of the vibratory working tip breaks deposits from the tooth surface and disrupts the biofilm

cavitational- vibrations of working tip cause the implosion of bubbles which cause shock waves which break deposits from the tooth surface and disrupt the biofilm- implosion of bubbles also leads to release of oxygen which kills GNAB

accoustic mode of action- flushes debris from the pocket

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13
Q

indications of USS

A
  • supragingival pmpr
  • subgingival pmpr
  • stain removal
  • removal of amalgam ledge
  • cavitational effect only for things such as ng
  • pre/post RSD
  • FI/narrow pockets
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14
Q

contraindications of USS

A
  • implants
  • dentine hypersensitivity
  • anxiety
  • ceramic crowns
  • contagious respiratory disease
  • demineralised areas
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15
Q

what is the main aim of RSD?

A

render the root surface biologically compatible with healing

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16
Q

what does RSD do?

A
  • disrupts the biofilm
  • decreases overall no of microorganisms
  • decreases the ratio of GNAB and gram + aerobic bac
  • removes endotoxins and necrotic cementum
  • removes bulk of calculus deposits
  • leave residual aerobic bacteria
17
Q

does all calculus need to be removed ?

A

no- small amounts of calculus remaining can stilll render the root surface biologically compatiable with healing

18
Q

what does PMPR stand for?

A

professional mechanical plaque removal

19
Q

how is RSD carried out?

A
  • discuss with pt and consent and LA
  • consult 6 ppcs before beginning
  • probe areas with cp12
  • pre USS to remove bulk of calculus and disrupt biofilm
  • reprobe
  • use hand instruments to for fine tuning and remove remaining calculus
  • reprobe
  • post USS to remove debris and disrupt biofilm
  • reprobe
  • POIG
20
Q

what should patients be warned of prior to tx?

A
  • gaurded prognosis if risk factors
  • gingival shrinkage
  • post op sensitivity
21
Q

what is calculus?

A

plaque that has been mineralised by the Ca and po4 ions found in saliva

22
Q

what are the properties of calculus?

A
  • sterile
  • crystaline structure
  • porous cant withold bac endotoxins
  • can form supra and subgingivally
  • has a layer of unmineralised plaque on its surface
23
Q

why should calculus be removed?

A
  • impedes OH
  • may give false ppd
  • is a plaque retentive factor
  • has layer of unmineralised plaque on its surface
24
Q

what is a true pocket?

A

one which has been pathologically deepened due to periodontal disease
- has LOA
- JE has migrated apically
- root surface is contaminated

25
Q

what is a false pocket?

A

increased ppd due to inflammation of the gingivae in a coronal direction which gives the appearance of an increased pp- there is no loss of attachment

26
Q

why might rsd fail?

A
  • poor OH
  • uncontrolled diabetes
  • high genetic susceptibility
  • immunocompromised
  • furcation involvement
  • poor operator skills
  • tenacious calculus
  • complex root morphology
  • incorrect initial diagnosis- may be endodontic problem
  • smoker
27
Q

what is full mouth disinfection?

A
28
Q

what can gingival shrinkage cause?

A

dentine hypersensitivity

29
Q

what causes dentine hypersensitivity?

A

exposed dentine tubules

30
Q

how can dentine hypersensitivity be treated?

A
  • ohi diet advice
  • advise patient to use sensitive toothpaste after brushing- rub directly onto exposed dentine but do not mix with diff toothpastes
  • fluoride varnish (sodium fluoride)-
  • desensitising agents such as seal and protect which occlude tubules (fluoride and triclosan)
  • CaOH- adhere poorly to dentine
  • insulative restorations
  • extreme cases= devitalise the tooth
31
Q

what are forms of mechanical plaque control?

A
  • tooth brushing
  • interdental cleaning
  • PMPR
32
Q

what are forms of chemical plaque control?

A
33
Q

what are signs of treatment success?

A
  • comfortable patient
  • reduced ppds
  • reduced bop
  • gingival shrinkage
  • pink and firm gingivae
  • resolved ulceration of pocket
  • reduced gingival inflammation
34
Q

what are signs of treatment failure?

A
  • red and swollen gingivae
  • poor oh
  • no reduction in ppd
  • uncomfortable patient
  • bop
  • ulceration of perio pocket
35
Q

recession v gingival shrinkage

A

recession has many different causes and most commonly affects the l/b/p gingivae and has no ID recession

gingival shrinkage- is the result of successful periodontal tx and cause the full gum to shrink including ID recession.

36
Q

what can cause recession?

A
  • frenum pull
  • excessive scaling
  • ortho appliances
  • chronic minor trauma- t.b
  • factitious injury
37
Q

what can recession lead to?

A
  • hypersensitivity
  • root caries
  • root abrasion