Perio 2 Test 1 Flashcards
Perio health:
1-3 Probe depths
No history of Aloss
No clinical inflammation
Gingival recession can also be known as:
Incidental attachment loss
Stillman’s method of brushing
Vs
Modified stillman
Modified has a rolling stroke incisally
Best way to clean interproximal spaces
Toothbrush and proxy brush
6 abrasives found in toothpastes
Ca-phosphates Ca-pyrophosphates Ca-carbonate Na-bicarbonate Hydrated silica Alumina
Fluoride in toothpastes can be found in what 3 forms
NaF
Monofluoro-Phosphate
Stabilized Stannous Fluoride
The bulk of the ingredients in toothpaste is _ such as _
Humectants
Glycerine
Sorbitol
Xylitol
Propylene glycol
_ is clinically effective in reducing plaque and gingival inflammation compared with a fluoride toothpaste
Triclosan/copolymer toothpaste
3 active ingredients of antimicrobial rinses and how they work
EO: broad spectrum activity against G+ and G- bacteria
- disrupts cell wall
- reduces endotoxin levels and pathogenicity
Cetylpridinium chloride: ruptures bact. Cell membrane, may alter bact. Metabolismi
Chlorhexidine: causes bact. Cell membrane to leak, binds salivary mucins reducing pellicle, binds bacteria
Preventing periodontitis is done by preventing _ which is done through control of _ and _
Gingivitis
Plaque accumulation and gingival inflammation
What is specific about pts with history of perio disease
Present with Aloss, ging. Recession, Furcation inv.
And
High risk of developing clinical Aloss
Study designs from top down
Systematic reviews and meta-analyses Randomized controlled double blind Cohort Case control Case series Case reports Opinions, ideas, editorials Animal research Test tube research
SRCCCCOAT
Efficacy vs. effectiveness
Efficacy = ideal circumstances
Effectiveness = usual circumstances
6 risk factors for perio
Bad oral hygiene History of perio Aloss Furcation inv Smoking Diabetes
BAD FHS
2 local factors that play a role in gingival recession
Biotype of gingiva
Bone dehiscence
3 main goals of perio therapy
Remove bacteria from tooth
Shift pathogenic microbes to healthy ones
Decrease inflammation and probing depth
Where can bacteria remain even after mechanical therapy
Root surfaces not accessed
Gingival epithelial cells and conn tiss
Dentinal tubules
Supragingival plaque/other infected sites
When are systemic antibiotics indicated
Aggressive perio Perio with secondary systemic involvement Some types of chronic perio Severe perio abscess NUG
3 host modulators agents
Bisphosphonates
NSAIDs
Low dose tetracyclines
What do bisphosphonates do?
Indicated for:
Side effects:
Incapacitated osteoclasts, reducing bone resorption
For: Paget’s disease, hypercalcemia, osteoporosis, metastatic bone diseases
SE: osteomalacia, allergic rxns
NSAIDs do what in cells?
Side effects
Effect on perio
Inhibit biosynthesis and release of prostaglandins in cells
SE: ulcers, allergic rxns, GI and renal toxicity
Perio: reduce inflammation but effects on attachment levels are modest
_ is a low dose of doxycycline which is a _
Cellular effect
How
Periostat, low dose tetracycline
Inhibits tissue destructive enzymes, concentrates in GCF and uses cementum as reservoir
Local delivery devices of perio antimicrobials
Fibers
Strips
Films
Injectables
3 ways to deliver antimicrobials subgingivally
Chlorhexidine - gelatin matrix film/chip
Doxycycline - flowable PLA gel
Minocycline - PLA/PLGA powder
T/F local antimicrobial delivery is indicated for localized aggressive chronic perio
FALSE. Slight to moderate chronic perio
T/F local antimicrobial delivery is adjunctive therapy
TRUE not primary