Periapical Tissue and Jaw Infections Flashcards

1
Q

What is the difference between a peridontal abscess and dentoalveolar abscess?

Etiological differences?

A
  • periodontal
    • abscess forms along tooth root
      • polymicrobial
    • from periodontitis
      • will notice gum receding, and reddness
  • dentoalveolar
    • abscess forms at end of tooth root
      • polymicrobial
      • anaerobes very important
    • abscess can extend and show itself in the gums along the tooth
    • caries have gotten thorugh enamel & dentin, down to the root
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2
Q

What is the major symptom for both dentoalveolar/periodontal abscess? How could you determine which tooth is affected?

A
  • symptoms
    • pain in and around affected tooth
    • swelling of face over the abscess site
  • can tapp tooth to see which causes pain – prob the one wiht the abscess
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3
Q

Diagnosis and treatment of dentoalveolar/peridontal abscess?

A
  • Diagnosis
    • refer to dentist
    • radiograph of tooth
  • Therapy
    • remove tooth, root canal, incise adn drain abscess
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4
Q

What is Ludwig’s angina?

Why is it so concerning & where does it most commonly occur?

Predisposing factors?

A
  • cellulitis of sublingual/submylohyoid spaces
  • rapidly fatal without treatment
    • can occur in children wtih out any precipitating cause
    • rare, but most commonly encountered neck space infection
  • predisposing factors
    • dental caries
    • recent dental treatment
    • sickle cell disease
    • compromised immune system
    • trauma
    • tongue piercing
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5
Q

common etiological agents of Ludwig’s angina

A
  • Bacterial isolates vary adn are often mixed
    • streptococcus
    • Bacteriodes
    • Fusobacterium
    • Staphylococcus aureus

If have foul breath odor, more likely to be anaerobes

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6
Q

Symptoms of Ludwig’s angina?

A
  • severely ill/fever
  • severe dysphagia (trouble swallowing)
  • trismus (lock jaw)
  • appear toxic
  • sittign upright
  • drooling
  • dysphonia
  • swelling and erythema of neck under chin
    • floor mouth tender to touch
    • pain on movement of tongue
    • tongue moved up and bacwards
    • acute airway closure can occur at any time
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7
Q

How do you diagnose Ludwig’s angina?

Treatment?

Prevention?

A
  • Diagnose
    • clinically
    • CT scan
    • blood cultures
  • Treatment
    • adequate airway managment
    • antibiotics
      • anaerpbes & Staf aureus
    • Incision and drainage (only if antibiotics fail)
    • nutrition and hydration
  • Prevention
    • good oral hygeine
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8
Q

12 year old male; tooth pain. Most likely diagnosis?

  1. Caries?
  2. Gingivitis?
  3. Periodontal disease?
  4. Dentoalveolar abscess?
  5. Periodontal abscess?
A

Caries

no history of gums bleeding/they aren’t receding/look healthy

also, not usually a complaint of pain if it is gingivitis

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9
Q

12 year old male; tooth pain

Most important virulence factor causign patient’s current condition?

  1. Acidogenicity
  2. Anaerobic growth
  3. Capsule
  4. Collagenases
  5. Hemolysins
A
  1. Acidogenicity

acid produced by the organism is producing the caries

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10
Q

25 year old male; bleedign gums. Most likely diagnosis?

  1. Caries
  2. Gingivitis
  3. Periodontal disease
  4. Alveolar abscess
  5. Periodontal abscess
A
  1. Gingivitis

no lossening of teeth (prob not periodontal disese)

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11
Q

35 year old female; 2 year history of bleeding gums; loose teeth/ Most likely diagnosis?

  1. Dental Caries
  2. Gingivitis only
  3. Acute nectorizin ulcerative gingivitis
  4. Chronic periodontitis
  5. Aggressive periodontitis
A

Chronic periodontitis

Do not have history of bad breath, no history of poor hygeine, only complaining of bleeding – it also has been happenign for 2 years, with aggressive it would have progressed in months

We don’t see any dental caries in the teeth

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12
Q

35 year old female; 2 year history of leedign gums; loose teeth. Most important virulence factor causing the loose teeth in this patient?

  1. Acidogenicity
  2. Anaerobic growth
  3. Capsule
  4. Collagenases
  5. Hemolysins
A
  1. Collagenases

(but also, the hemolysins do cause damage to the red blood cells– increasing inflammation, but not creating loosening of teeth)

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13
Q

50 year old male; swelling of the face and painful tooth (right lower 1st molar). Vitals: temp-99, pulse-85/min, resp-15/min, BP-12/85. No signs of periodontal disease or pain on tongue movement. Most likely diagnosis?

  1. Dental Caries
  2. Gingivitis
  3. Acute necrotizing ulcerative gingivitis
  4. Dentoalveolar abscess
  5. Periodontal abscess
  6. Ludwigs’s angina
A
  1. Dentoalveolar abscess

no signs periodontal disease, so probably not a periodontal abscess

could be ludwig’s but seems to be more on the side of the face than the chin (would also expect a much higer temp & pain on tongue movement)

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14
Q

35 year old male; vitals: temp-104, pulse-120/min, resp-17/min, BP-140/90. Sore throat and drooling, unable to open mouth more than 3 mm. Tongue movement is painful. Most likely diagnosis?

  1. Dental caries
  2. gingivitis
  3. acute necrotizing ulecerative gingivitis
  4. dentoalveolar abscess
  5. periodontal abscess
  6. ludwig’s angina
A

Ludwig’s angina

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15
Q

What is the name for inflammation of the mucosal surfaces of the mouth and tongue?

What are the two most common infections causes?

A

Stomatitis

causes: HSV 1 & 2; Candida albicans

gingivostomatitis common with HSV

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16
Q

What test could you run to determine if the agent was Candida albicans vs. another species?

A

germ tube test

17
Q

What is the incubation period for oral herpes?

Duration?

What are the symptoms?

A
  • Incubation
    • 2-12 days
  • Duration
    • 2-3 weeks
  • Primary symptoms-
    • fever
    • malaise
    • muscle aches
    • irritable
  • Additional
    • pain/itching/burning at infection site (often first infection will be within the mouth and subsequent will be on the lips)
    • cluster of blisters erupt
    • blister breakdown forms a small, shallow gray ulcer on a red base
    • cervical lymphadenopathy
18
Q

Describe the oral herpes epidemiology

A
  • usually seen in children (60% infected by 15)
  • HSV1 causes about 80 %
    • contact with infected saliva, mucous membranes or skin
  • HSV2 causes abou 20%
    • oral sex
  • Recurrences in the mouth are uncommon
  • Recurrnences are usually sen on the lips and face
  • asymptomatic shedding is infectious
19
Q

Oral herpes pathogenesis

A
  • Primary infection
    • oral sores, fever
      • many more asymptomatic infections
  • Latency – likes nerves (cervical root ganglion)
  • Recurrence
    • during times of stress
    • new sores
20
Q

Oral herpes diagnosis?

Treatmetn?

A
  • Diagnosis
    • Clinical diagnosis
    • culture ofr virus
      • serology
    • Tzanck test positive
      • big syncytia
  • Treatment
    • usually self-limiting
    • if sever- antiviral agents
    • antivirals will NOT cure
21
Q

Whta are the 3 types of candidiasis?

A
  • Pseudomembranous - thrush (common in young infants)
    • acute
  • Erythematous - red (common in people with dentures)
    • acute
    • chronic
  • Hyperplastic (leukoplakia) – white or speckled
    • chronic
    • can result in a cancerous growth
22
Q

The following clinical manifestations indicate waht diagnosis?

  • creamy-white plaques (mucosal cells, neutrophils, yeast cells)
  • hard to remove
  • when removed can leave behind inflamed base that may be painful and bleed
A

Candidiasis

Acute pseudmembranous (thrush)

23
Q

The following clinical manifestations indicate what diagnosis?

  • red areas of varying sizes
  • any part of the oral mucosa
  • if on tongue, fiery red and shiny with depapillation
  • can be acute or chronic
  • problem for denture wearers
A

Erythematous candidiasis

24
Q

The following clinical manifestations indicate what diagnosis?

  • individual lesion of cheek near commisure, at angles of mouth or on the tongue
  • chronic discrete, raised lesions
  • homogenous or speckled areas
  • does not rub off
A

Candidiasis

Hyperplastic (leukoplakia)

can become malignant

25
Q

Candidiasis epidemiology

A
  • very common normal flora inhabitiant (50%)
  • acquired
    • birth canal, from saliva, etc.
  • carriers higher in women than men
  • for disease certain predisposing factors are usually present
    • infants and neonates
    • taking antibiotics
    • taking steroids
    • polyendocrine disorders
    • smokers
    • denture wearers
    • drugs that cause dry mouth
    • patients with immune dysfunction
26
Q

Candidiasis pathogenesis:

Acute pseudomembranous (thrush)

Erythematous candidosis

Hyperplastic (leukoplakia)

A
  • Candida albicans
    • ability to produce biofilms
    • germ tubes
      • helps penetrate tiss & set up shop
  • Acute pseudomembranous
    • overgrowth of fungus
    • pseudomembrane formation - accumulatin of keratin, bacteria, yeast, and necrotic tissue
    • curd-like
    • in infants will ususally also see diaper rash
  • Erythematous candidosis
    • both acute adn chronic
    • inflammation due to overgrowth
    • no pseudomembrane
      • follows pseudomembrane type de novo in AIDS patients
      • prolongued drug prescription
      • usually in denture wearers
  • Hyperplastic (leukoplakia)
    • marked by hyperplasia of parakeratinized layer with candidal hyphae invading at right angles to the layer
    • can become malignant
27
Q

Candidiasis Diagnosis and Treatment

A
  • Swab lesions for culture
  • scrapings can be stained for yeast cell
  • biopsy hyperplasia for histology
  • antifungal agents
  • hyperplastic conditions may require surgery if malignant
28
Q

What is oral hairy leukoplaia?

What is the etiological cause?

A
  • latera borderes of tongue
  • usually in
    • immunocompromised,
    • those with malignant tumors
    • organ transplant recipients
  • Etiology
    • Epstein Barr Virus
29
Q

Which groups of people are most affected by oral hairy leukoplakia?

A
  • IV drug users
  • homosexual males wiht HIV (most common)
30
Q

Oral Hairy Leukoplakia pathogenesis

A
  • Latent infection of Epstein Barr Virus
  • Immune suppressin
  • EBV replicates in oral mucosa
  • Results in benign thickening of mucosa and hyperproliferation of epithelial cells
31
Q

Oral Hairy Luekopplakia Dianosis and Treatment

A
  • Diagnosis
    • made clinically
    • scraping look for Cowdry type A inclusions (cidophilic material of droplet-like masses surrounded by clear halos within nucle)
  • Treatment
    • Rarely treated unless patient has HIV
    • anti-retroviral therapy
    • If treating leukoplakia
      • podophyllum resin and antiviral agent