Peds - Exam 3 - Neuro Flashcards

0
Q

Levels of consciousness in descending order

A

Full consciousness
Confusion—impaired decision making
Disorientation—to time and place
Lethargy—sluggish speech
Obtundation—arouses with stimulation
Stupor—responds only to vigorous and repeated stimulation
Coma—no motor or verbal response to noxious stimuli
Persistent vegetative state—permanently lost function of cerebral cortex

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1
Q

Increased ICP - Clinical Manifestations in Children

A

Headache
Vomiting—with or without nausea (projectile)
Seizures
Diplopia, blurred vision

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2
Q

Increased ICP - Clinical manifestations in infants

A
Irritability, poor feeding
High-pitched cry, difficult to soothe
Fontanels—tense, bulging
Cranial sutures—separated
Eyes—setting-sun sign (eyes bilaterally deviate downward)
Scalp veins—distended
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3
Q

Late signs of increasing ICP

A

Decreased LOC
Decreased motor response to command
Decreased sensory response to painful stimuli – really late sign
Alterations in pupil size and reactivity
Papilledema – swelling to optic disc
Decerebrate or decorticate posturing
Cheyne-Stokes respirations – periods of fast breaths and apnea – look at video posted with lecture

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4
Q

Seizure vs. Epilepsy

A

Seizure
A single seizure not generally classified as epileptic
Single seizure not generally treated with long-term Rx
Will resolve once medical condition is resolved

Epilepsy
Condition characterized by two or more unprovoked seizures
Multiple causes by a variety of pathologic problems in the brain

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5
Q

Behavioral signs of increasing ICP

A

Irritability, restlessness
Drowsiness***, indifference, decrease in physical activity and motor skills
Complaint of fatigue, somnolence
Inability to follow commands, memory loss
Weight loss

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6
Q

What to note when assessing pupil size

A

Medications can affect - important to know what medications that patient is on
Size of pupils, whether they are reactive - large pupils may be normal if they are reactive

Bilateral fixed pupils for longer than 5 mins usually indicates brainstem damage

One pupil dilated and fixed –
Bleed or lesion on same side of brain
Neurosurgical emergency - should be reported immediately!

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7
Q

Doll’s Head Maneuver

A

ONLY perform this after a spinal cord injury is ruled out!

  • Rotate child’s head quickly from one side to the other
  • Positive - brainstem intact, eyes move in opposite direction of head (normal reaction)
  • Negative - eyes are remaining in fixed position no matter where head is being moved, brainstem dysfunction or damage to CN III (oculomotor)
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8
Q

Caloric test

A

Used to assess brain death in child

** Never going to be performed on an awake/conscious child
Very painful
Irrigate auditory canal over 20 seconds with ice cold water

HOB elevated 30 degrees

Normal response—eyes move toward the side of stimulation.
Abnormal response—no response

Very precise procedure, physician likely performing, nurse assisting

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9
Q

Decorticate/Decerebrate Positioning

A

Posturing may only be present with painful stimuli

Neither type of posturing is good

Ideal response – patient would withdraw

Decorticate – flexion – bringing it into the CORE
- Occurs with severe dysfunction of cerebral cortex or lesions above the brainstem

Decerebate – extension of extremities
- Occurs with dysfunction at level of midbrain or damage to brainstem

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10
Q

CT or MRI considerations

A

Most kids will need to be sedated for them - not because it’s painful
But just can be really hard for the child to lay still, especially for an MRI
Nurse or parent may be present in room (behind window) to comfort child, if facility permits

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11
Q

Signs of pain in comatose patient

A
  • Tachycardia
  • Increased respiration
  • Increased BP
  • Sats drop

(Pain can also increase ICP, so try not to do things to increase pain)

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12
Q

Drug therapies for pain in comatose child

A

Opioids – morphine or dilaudid
Fentanyl (opioid) + midazolam (versed – short-acting, causes amnesia) + vecuronium (paralytic, neuromuscular blocker, frequently used in pediatric pts who are intubated)
Acetaminophen and codeine – comfort measures

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13
Q

Comatose child - respiratory management

A
  • Airway management is primary concern
  • Cerebral hypoxia lasting more than 4 minutes will likely cause irreversible brain damage
  • CO2 causes vasodilation, increased cerebral blood flow, and increased ICP
  • May have minimal gag and cough reflexes
  • Risk of aspiration of secretions

*** periodic or irregular breathing is an ominous sign – usually indicates brain stem is affected – often see prior to death with head injury

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14
Q

Indications for ICP monitoring

A

Glasgow Coma Scale score 8 with respiratory assistance
Deteriorating neurologic condition
Subjective judgment

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15
Q

Subarachnoid bolt

A
  • Most frequently seen
  • Scary looking for parents
  • Place bolt directly to the ventricles – monitors ICP
  • Held intact by dressings on the outside
  • Need to be VERY careful around it – only neurosurgeon should touch it
  • Nurses don’t even touch the dressing to assess the site
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16
Q

Nursing care for child with increased ICP

A

Patient positioning

  • HOB 15-30 degrees
  • Head should be maintained midline ideally
  • Avoid frequently turning head

Avoid doing things that increase ICP

  • Pain
  • Too much stimuli
  • Avoid doing things that will make a child cry
  • ROM exercises still done but GENTLY

Suctioning issues

  • Usually poorly tolerated
  • May need to do because of loss of gag reflex –> will have to suction
  • Really GENTLE suction & hyperoxygenate beforehand

Nutrition and Hydration

  • Avoid overhydration – strict I&O – to avoid cerebral edema
  • IV fluids and TPN and lipids or gastric feedings
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17
Q

Comatose patient - nursing care needs

A
  • Elimination – will always have foley initially, need to record BMs (may require stool softeners as needed)

Hygienic care – skin integrity and good mouth care

  • Emollients to lips – often very dry
  • Eye drops/ointments
  • Position and exercise

Stimulation

  • Hearing is last sense to go – may be capable of hearing/sensing – still going to explain what you are doing
  • Encourage family to talk with patient
  • Soft music

Family support

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18
Q

Concussions

A
    • transient and reversible
  • Results from trauma to the head
  • Instantaneous loss of awareness and responsiveness lasting for minutes to hours – DONT always have to lose consciousness for it to be concussion
  • Can have nausea and vomiting afterwards
  • Generally followed by amnesia and confusion

Post concussion syndrome

  • Headache, dizziness, fatigue, irritability, issues with concentration or memory impairment
  • Usually develop the day of injury and last up to 3 months (typically resolve by 3 months)

Behavioral changes, memory, concentration issues are normal/expected

What to report ASAP or bring in right away:
- Vomiting
- Suddenly report blurred vision, or any LOC
(Generally later signs of ICP)

Worry about repeated concussion, repeated injuries
A lot of sudden death in sports is related to multiple injuries

Rule of thumb
3 head injuries during contact sports and you’re done – no more contact sports

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19
Q

Contusion and laceration

A

Terms used to describe visible bruising and tearing of cerebral tissue
Coup—bruising at the point of impact
Contrecoup—bruising at a site far removed from the point of impact (brain hits the back of the skull)

KNOW with any head injury
Assess ABCs first
THEN stabilize head and neck

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20
Q

Reye Syndrome (RS)

A
  • A disorder defined as metabolic encephalopathy associated with other characteristic organ involvement
  • Characterized by fever, profoundly impaired consciousness, and disordered hepatic function
  • Etiology is not well understood
  • Most cases follow a common viral illness (usually influenza or varicella)
  • Potential association between aspirin therapy for fever and development of RS

** If you think a child has varicella or influenza, educate parents do NOT give any aspirin or aspirin-containing products (a lot of cold preps can also contain aspirin)

Diagnostic evaluation
Definitive diagnosis by liver biopsy

Majority of children recover rapidly – if intervention is done early
1/3 of patients can die or have long-term neuro deficits

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21
Q

Reye Syndrome (RS) Patho

A

Cerebral edema
Fatty liver changes – changes of AST, ALT
Neurologic changes

22
Q

Reye Syndrome Therapeutic Management

A

Therapy is supportive
Prevent dehydration
Monitor for increased ICP
Monitor liver function

23
Q

Reye Syndrome - Onset

A
  • Onset – pt with profuse vomiting
  • Typically following varicella or influenza infection
  • Neuro changes:
    Personality changes or seizures
24
Bacterial Meningitis by age group
Occurs in epidemic form, predominantly in school-age children and college students because of close contact or living conditions ≥1 month and 3 months and <19 years
25
Meningitis - Signs and Symptoms
Brudzinski's sign + - Passively elevate the head, knees would also flex toward abdomen Kernigs sign + Passively elevate the leg and try to extend it at the knee, there would be resistance Most definitive test - LP done to rule out meningitis Onset can be abrupt or develop over 1-2 day - High fever (103-104 typically) - Headache - Rash - Neck pain, nuchal rigidity - Vomiting - Then deteriorating neuro changes - Many times present with seizure or will have seizure within 48 hours Infants - Fever or hypothermia - Poor feeding - Irritability - Restlessness - Bulging tense fontanel - High pitched cry
26
Meningitis - Treatment and nursing considerations
- IV antibiotics STAT - Maintain strict iso precautions, maintain adequate hydration, manage sepsis and septic shock as needed - Decrease stimuli – quiet dark room, to help keep them comfortable - Manage pain - Encourage vaccines (Hib, Menectra)
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Nonbacterial Meningitis (Aseptic Meningitis)
Causative agents are principally viruses Frequently associated with other diseases - Measles, mumps, herpes, leukemia Onset abrupt or gradual - Manifestations—headache, fever, malaise - Symptoms usually milder Diagnosis and treatment - Might start abx even before the results of LP - Also manage symptoms – maintain hydration, manage pain Prognosis - Prognosis good, self-limiting - Would work it up as we would bacterial meningitis
28
Seizures
- Seizure is a SYMPTOM, rather than a disease, caused by malfunctions of brain’s electrical system - Determined by site of origin - Most common neurologic dysfunction in children (mostly under 2 years of age)
29
Seizure vs. Epilepsy
Seizure - A single seizure not generally classified as epileptic - Single seizure not generally treated with long-term Rx - Will resolve once medical condition is resolved (e.g., meningitis) Epilepsy - Condition characterized by two or more unprovoked seizures - Multiple causes by a variety of pathologic problems in the brain
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Seizures - S/S
Change in LOC Involuntary movements Posturing Changes in perception, behaviors, or sensations
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Seizures - Major causes
Birth injuries (anoxia) or congenital defects of CNS Acute infections in late infancy and early childhood In children older than 3 years, usually is idiopathic (no known cause)
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What to know about seizure medications
- Monotherapy - gradually increase dose until seizure controlled or signs of toxicity - Polypharmacy if uncontrolled with just one - Monitor therapeutic levels, increase dose as child grows - Monitor side effects - Avoid abrupt discontinuation - needs to gradually dose reduce to take child off med Are they going to have to be on medication life-long? - Can come off when seizure-free for 2 years and normal EEG - Not going to d/c during puberty or times when pt suffers frequent illnesses - Recurrence can occur within first year and they may have to go back on meds
34
When seizure happens
- Side lying - Maintain patent airway - No suctioning during - ready for after (many secretions) - No tongue blade - Put pillow under their head - Do not restrain - Time seizure Home mgmt: - CPR training for fam members - How to give rectal diazepam More than 5 minute seizure at home/school: CALL 911! First time having seizure, regardless of duration, CALL 911!
35
Absence Seizures
Formerly called petit mal or lapses Sudden onset Brief loss of consciousness, a blank stare, and automatisms (motor changes - lip smacking or eye twitching) Most common between ages 5 and 12 years Often stop spontaneously in the teenage years Sudden onset of 20+ events per day No warning/no aura Duration 5 to 10 seconds Motor—lip smacking, twitching of eyes, face, slight hand movements May drop object; child rarely falls No incontinence Often misdiagnosed with inattention or ADHD –Inattention/daydreaming ADD/ADHD School issues-- missing bits of information throughout day
36
Generalized Seizures
AKA tonic­clonic seizure Formerly known as grand mal Usually occurs without warning Consists of two distinct phases Tonic Eye rolling and loss of consciousness Tonic muscle contractions Clonic Intense jerking movements Average duration 30-50 seconds May have incontinence, caution w/ airway concerns
37
Partial seizures
Further subdivided into three parts Simple partial seizures No alteration of consciousness Clinical manifestations related to the area of cerebral involvement May consist of motor, sensory, autonomic, or psychic symptoms. Complex partial seizures Complex symptoms and impairment of consciousness. Simple or complex seizures secondarily generalize Simple or complex partial seizures that evolve into generalized seizures, usually a tonic–clonic event
38
Atonic seizure
Sudden momentary loss of muscle tone Onset usually ages 2 to 5 years May or may not have loss of consciousness Sudden fall to ground, often on face – no motor ability to catch themselves Patient will wear helmet to protect themselves if it happens frequently Less severe—head droops forward several times
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Myoclonic seizure
Sudden brief contractions of muscle group May be single or repetitive No loss of consciousness Often occur when falling asleep May be nonspecific symptom in many CNS disorders Nonspecific symptom of other disease May be mistaken as exaggerated startle reflex in infant If you can flex head forward, then NOT a seizure
40
Febrile seizure
Transient disorder of childhood Affect approximately 3% of children Usually occur between ages 6 months and 3 years Rare after age 5 Twice as frequent in males 95% to 98% of children with febrile seizures do not have epilepsy or neurologic damage – usually happens with that rapid temp change ``` Management Avoid tepid baths—usually ineffective Vigorous use of antipyretics Protect child from injury during seizure Call 911 if seizure lasts more than 5 minutes ``` Cause? Usually in temperatures higher than 101.8° F Seizure occurs when temperature is rising, not after Seizure usually over before the child arrives in the emergency department
41
Status Epilepticus
Definition—seizure lasting more than 30 minutes or series of seizures without regaining premorbid LOC Treatment Maintain airway Establish IV access ASAP – can be difficult w/patient actively seizing Medications to stop it
42
Neural Tube Defect prevention
Treatment aimed at prevention Supplementation—0.4 mg/day folic acid If history of NTD—4 mg/day folic acid 1998 FDA fortifies cereal grains with folic acid Begin at preconception Ideally 3 months prior Women of child bearing age should be supplementing with folic acid
43
NTD diagnosis
Elevated α-fetoprotein in amniotic fluid at 16 to 18 weeks Chorionic villus sampling but not until 10 weeks of gestation Uterine ultrasound Why do we want to know? Family to be prepared – especially if they are considering termination If giving birth, will be delivered via c-section
44
Spina Bifida (SB) Types
Spina bifida occulta Not visible externally Spina bifida cystica Visible defect Saclike protrusion
45
Myomeningocele
Neural tube fails to close May be anywhere along the spinal column Lumbar and lumbosacral areas most common May be diagnosed prenatally or at birth Sac contains meninges, spinal fluid, and nerves Varying and serious degrees of neurologic deficit Clinically, the term “myelomeningocele” is interchangeable with “spina bifida” The sac May be fine membrane Prone to leakage of CSF; easily ruptured May be covered with dura, meninges, or skin Degree Location and magnitude of defect determine nature and extent of impairment Not necessarily uniform on both sides of defect Initial management Prevent infection Assessment of neurologic and associated anomalies (hydrocephalus) Positioning – prone position / parents won't be able to hold Early closure within 12 to 72 hours after birth – surgery to repair
46
Hydrocephalus
Commonly associated with myelomeningocele Hydrocephalus develops in 80% to 85% of children with SB May not be apparent at birth May appear after primary closure of defect Results from disturbances in dynamics of CSF Enlarged lateral and third ventricle Management Head Circumferences daily Fontanel tension – increased ICP? (tense bulging fontanel?) Serial ultrasounds—ventricle size CT or MRI for initial diagnosis – enlarged ventricles?
47
EVD—External ventricular drainage
Drains CSF intermittently or continuously Closed, sterile system with transparent pliable tubing, a collection bag, and possibly a drip chamber between the parts Hung on IV pole at bed-side Placed at the level of the child’s external ear HOB at 20- to 30-degree elevation **** If lower or raise HOB, close system (for short periods of time) / but generally maintain position
48
VP Shunt Complications
Infection Signs/symptoms Shunt malfunction (Fever and inflammation of tract Abdominal pain Malfunction- Emergency! can happen from exudate, kinking, plugging, displaced by child's growth spurt) Chances greatest in 1-2 mos after placement Signs/symptoms Increased ICP Worsening neurologic status/altered LOC
49
Types of CP
Spastic Most common clinical type Initially presents with hypotonia Hypertonicity and poor control of posture, balance, and coordinated motion Athetoid/dyskinetic Slow, wormlike movements of extremities, trunk, face, tongue Drooling, speech problems Lead to jerky involuntary movements worsen w/ emotional stress Ataxic rapid repetitive movements; wide gait, unable to hold onto objects Mixed/dystonic combination or spastic and athetosis
50
Goals of therapy for CP
* *Goals of therapy for children with CP are early recognition and promotion of an optimum developmental course to allows these children to attain their potential within the limits of their dysfunction. There is no cure for CP and therapy is symptomatic and preventive. - orthotics used on feet to prevent or reduce deformity and control alignment - customized devices to help with mobility - surgery reserved for kids who do not respond to conservative measures or if spasticity is causing progressive deformities (correct contractures, stabilize joint, bone alignment, tendon-lengthening) - Meds: to control spasticity and pain - technical aides: toys used to improve functioning. Computerized toys to enhance hand-eye coordination or to aide with speech difficulties
51
CP and IQ
**Children with CP have a wide range of intelligence, and 50-60% have normal IQ. A large percentage of children with CP do not have mental impairment. Can be difficult to assess intelligence d/t motor and sensory deficits. Many individuals who have severely limiting physical impairment have the least amount of intellectual compromise.
52
Autism Diagnostic Criteria
- Children with ASD have core deficits in social interaction, communication and behavior. Impaired social interaction is one of the hallmarks- may include early abnormal eye contact, failure to mile, failure to orient to name, lack of imitation, lack of interactive play, and lack of gesture use, such as waving or pointing. - communication impairment may be absent to delayed speech or abnormal speech like grunting or humming - autism regression= when the ASD child develops normally and then regresses suddenly. - any kid who doesn’t babble or gesture by 12 mo, a single word by 16 mo, and 2-word phrases by 2 years should have immediate hearing and language eval - behavior impairments range from mild-severe - repetitive impulsive spinning, twirling, rocking, flapping hands, head nodding are all common with ASD
53
Autism Nursing considerations
When inpatient should provide decreased stimulation and place child in private room when possible. Each child with ASD are unique and their care should be individualized. Communication should be at the child’s developmental level, brief, and concrete. Wide variation in individual client response to treatment efforts No cure for autism Most promising results seem to be through highly structured routines and intensive behavior modification programs