Pediatric CV and BLT Flashcards

1
Q

Coarctation

A

This condition represents 5-8% of all congenital cardiac lesions. It represents 7% of critically ill infants with heart disease. 3% of neonatal death attributable to congenital heart defects due to CoA in US

Birth incidence may be underestimated because many cases not diagnosed until adulthood

Associated with many other cardiac lesions/ syndromes
60% of infants, 30% of older children have bicuspid aortic valve

Pathophysiology:
Narrowing of the aorta, usually distal to the left subclavian artery and proximal to the ductus arteriosis

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2
Q

when symptoms of CoA may begin

A

Closing of the ductus arteriorsis:

Occurs in the first 2 weeks of life

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3
Q

Closing of the ductus arteriosis with coarctation

A

Closing of the ductus in the presence of obstruction results in

Decreased blood flow distal to the obstruction

  • Brachiofemoral delay
  • -Pulse differentials- less detectable distal to obstruction
    • Blood pressure differentials- lower BP distal to obstruction

Increased pre-coarctation pressure

  • Pulmonary congestion
  • Hepatic congestion
  • Tachypnea
  • Heart failure

Decreased post-coarctation pressure

  • Decreased perfusion
  • —> Cool skin (cutis marmoratum)
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4
Q

coarctation x-ray

A

enlarged heart shadow

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5
Q

Effects of coarctation

A
  • mechanical obstruction causes increased pressures proximal to the coarctation (upper extremities), and decreased pressure distal
  • decreased pressures distal to the coarctation leads to:
    • Hypoperfusion of the kidneys
    • Which increase renin secretion
    • leads to arterial constriction and hypertension proximal to the obstruction
  • Lower extremity circulation is largely dependent on collateral circulation as these children age
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6
Q

Systemic Effects of COA in Older Children and Adults

A

rib notching

the 3 signs: dilation of the aortic arch and subclavian artery, the “tuck” of the coarctation and the post-stenotic dilation of the descending aorta

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7
Q

Osteopathic Management for coarctation of the aorta (peds)

A

Prompt recognition of the problem and need for immediate intervention

  • Asymptomatic infants can be watched closely
  • Those with progressive severity need intervention more rapidly
  • –> NICU care, cardiology evaluation, surgical evaluation

Important to maintain patent ductus arteriosus in symptomatic newborns
— Prostaglandin E2 (PGE2)

Surgical intervention is ultimately the ‘cure’
- Surgery (anastomosis), balloon angioplasty, and/or stent placement

Osteopathic manipulative medicine
- Aimed at enhancing tissue repair after surgery and during long term management

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8
Q

Follow up care

A

Cardiology/primary care: watching for recoarctation
Increase in irritability, decreased feeding, tachypnea, pulse inequalities, perfusion, new/progressing heart murmurs, hypertension

OMM
At growth spurts to augment and support tissues as they move through growth

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9
Q

Osteopathic Management: OMM (coarctation patient)

A

Balance SNS activity

  • Cervical region to affect the cervical ganglia
  • Upper thoracics
  • Rib raising

Diaphragm and ribs

  • To improve gas exchange
  • To improve fluid dynamics

Cranial base, ** Occipitomastoid Suture, OA and cervical spine

    • Balance parasympathetics (vagus)
  • ——- Exits skull in the jugular foramen (occipital and temporal bones)
    • To improve fascial mechanical strain that result from intubation

Lymphatic pumps, thoracic inlet
– Can teach parents to do this at home (after healing)

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10
Q

Balanced Ligamentous Tension (BLT) Introduction

A

Dr. Sutherland used the principle of balanced membranous and ligamentous tension as his approach to treatment in the cranium and the body

Ligaments provide * proprioceptive information that guides the muscle response to positioning

Ligaments * guide the motion of articular mechanisms
His technique includes both direct and indirect methods
The area of strain is brought to the point of balanced membranous/ ligamentous tension
All techniques involve engaging and utilizing the “powers within the patient’s body” from a “point of balanced tension”
Inherent forces: “reset” nervous system, reduce firing of joint receptors and nociceptors, expression of inherent motion (respiration, pulse, PRM)

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11
Q

evaluation and goal of BLT

A

The evaluation of somatic dysfunction should include the impact on the local expression of inherent motion, such as respiration, pulse, and the primary respiratory mechanism (PRM)

This will provide evidence of the physiological impact the somatic dysfunction is having on local physiology

“The goal of treatment is to improve the interchange of all the fluids of the body across all tissue interfaces.” – A. Wales, DO

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12
Q

the point of blaanced membranous/ ligamentous tension

A

defined as “the point in the range of motion of an articulation where the ligaments and membranes are poised between the normal tension present throughout the free range of motion and the increased tension preceding the strain…” – H. Lippincott, DO and R. Lippincott, DO

Although the concept is simple, the depth lies in the precision in which it is applied, and the window into body physiology it affords

  • The advanced technique is the simple technique done really well
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13
Q

A diagragmatic representation of a ligamentous articular strain- BLT INDIRECT approach

A

box with straps attached to the lid, one taut and one slack

Partially closing the lid and slacking both straps (ligaments), a point of balanced ligamentous tension would be achieved. This represents an indirect approach.

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14
Q

A diagragmatic representation of a ligamentous articular strain
- BLT DIRECT approach

A

box with straps attached to the lid, one taut and one slack.

If the articulation is carried into the strain, the articulation would be displaced the slacked strap (ligament), and this would also bring this to a point of balanced tension. This is achieved through direct (into the barrier) positioning.

When positioning into the barrier, do not attempt to overcome resistance.
Match the tension which allows the inherent forces to make the correction.

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15
Q

Balanced Ligamentous Tension (BLT)– Exaggeration

A

Exaggeration
Indirect
Most commonly used
Takes advantage of natural recoil in tissue

(boxes going in opposite directions)

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16
Q

Balanced Ligamentous Tension (BLT)- Direct Action

A

Direct Action
Retrace the path of the lesion within range of free motion

(boxes going toward one another)

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17
Q

Balanced Ligamentous Tension (BLT)- Disengagement

A

Disengagement
Used in conjunction with exaggeration and direct action

(boxes going up and down away from each other)

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18
Q

Balanced Ligamentous Tension (BLT) Opposing physiologic motion

A

Opposing physiologic motion
Used where physiologic pattern has been severely violated (trauma)
One part toward physiologic position (direct) and the other away (indirect)

(boxes going in the same direction)

19
Q

Balanced Ligamentous Tension (BLT) Molding

A

used t help reshape

boxes getting mashed into an oval ?

20
Q

Observation in BLT

A

Observe the changes in physiologic phenomena
Observe what happened before (diagnosis)
Observe what’s happening now (treatment)
Observe what will happen (prognosis)

** Balanced membranous/ligamentous tension is a treatment principle which an area of strain is brought to a point of balanced tension to engage and utilize the inherent forces within the patient’s body to make the correction

21
Q

Sternal anatomy

A

Pericardial sac:

  • Fibrous pericardium
  • Bound to central tendon of the diaphragm posteriorly
  • Bound to sternum via sternopericardial ligaments anteriorly (phrenic nerves embedded within)
  • Adherent to mediastinal pleura

Rationale: pericardium and ligaments can be strained from insults due to trauma or pathology of the area

22
Q

BLT of the Sternum

A

Place the heel of one hand on the manubrium and fingers on sternal body. Place the other hand posteriorly on the patient’s thoracic spine.

Sink attention to structure to be treated (sternopericardial ligaments).*

With both hands, induce translation, rotation, and inferior/superior glide until balance is achieved. Hold until release is perceived.

Return to neutral and reassess by inducing motions again.

*For myofascial release, one would pay attention to fascia in the area (sternal, pericardium, etc). Barrier would be followed as it moves and release is palpated.

23
Q

BLT of the Ribs rationale:

A

Balance autonomics: the sympathetic chain ganglia lie just anterior to heads of the rib
Treat thoracic and rib segments that are facilitated due to visceral input
Structural: Connections with sternum, thoracic spine, diaphragm, fascia from above and below

24
Q

BLT of the Thoracic Spine

A

Patient supine, physician seated at side of patient
With index and/or middle finger(s) of cephalad hand, contact the spinous process of a dysfunctional thoracic vertebra. With the index and/or middle finger(s) of caudad hand, contact the spinous process of the thoracic vertebra immediately below.
Motion test : Flexion (SPs together), Extension (SPs apart), Rotation (rotate SP toward/away), and Sidebending (translate SPs toward/away).
Find a point of BLT (either direct or indirect) between the two vertebrae.
Hold until a release is perceived.
Return to neutral and reassess

25
Q

BLT of the Ribs

A

With index fingers of both hands, contact the patient’s rib posteriorly near the costotransverse/costovertebral joint and anteriorly at the costochondral junction. Thumbs contact rib as near to mid-axillary line as possible

Add compression or distraction, and motion test to bring rib toward a point of balanced tension (medial/lateral, inferior/superior, anterior/posterior, inhaled/exhaled).

Hold until a release is perceived.

Return to neutral and reassess.

26
Q

BLT of the 12th Rib- rationale and the 5 arches

A

A Handle for the Diaphragm

Rationale: The 12th rib is a posterior/lateral/inferior attachment of the diaphragm

5 Arches:
Median arcuate ligament (1): formed by left (L1-2) and right (L1-3) crura of diaphragm. Aortic hiatus (aorta, azygous vein, thoracic duct) passes underneath.
Medial arcuate ligament (2): Body of L1/L2 to TP of L1/L2. Forms upper part of psoas major.
Lateral arcuate ligament (2): TP of L1 to tip of 12th rib. Forms upper part of quadratus lumborum.

27
Q

BLT of the 12th Rib

A

Utilizes principles of disengagement & direct method
Diagnosis: Inhalation/exhalation of the right or left hemi-diaphragm
Patient is supine, with the physician seated on the side of dysfunction
1. With one hand, contact the 12th rib lateral the costovertebral joint. Support the contact with the other hand.
2. Gently apply distraction in the direction toward the tip of the rib, lining up vector with tension perceived (inferolateral). Practice bringing attention to arches of the diaphragm.
3. Fine-tune to achieve point of balanced tension. Wait until release is perceived (e.g. more motion with respiration).
4. Return to neutral and reassess.

28
Q

Heart Murmurs

A

Common in asymptomatic, otherwise healthy children
May be sole finding in children with structural heart disease
Prevelance of Congenital heart disease (CHD): 4-50 / 1,000 live births
Infants: feeding difficulties may be first sign of CHF (present in 1/3 of those with CHD)
Most common symptoms: Dyspnea, Nausea/ Vomiting, Fatigue, Cough
Rarely a presenting symptom: Chest Pain

29
Q

Heart Murmurs: Patient’s History

A

Findings suggesting structural heart disease in children with heart murmurs:

  • Family history
  • —- CHD, sudden cardiac death/ hypertrophic cardiomyopathy, SIDS

Patient history
—– aneuploidy (Trisomy 21, Turner syndrome), connective tissue disorder (Marfans), inborn error of metabolism, congenital defects of other organ symptoms, frequent respiratory infections, Kawasaki disease, rheumatic fever

Prenatal/perinatal history of in utero exposure to
—– alcohol/ toxins/ SSRIs/ teratogenic medications, intrauterine infection, maternal diabetes mellitus, preterm delivery

30
Q

Heart Murmurs: Physical Exam

A

Compare vital signs to age-established norms
General: appearance, activity level, color, respiratory effort
Neck: prominent vessels, abnormal pulsations and bruits
Chest: abnormalities of sternum, cardiac impulses or thrills
Lungs: abnormal breath sounds (crackles= pulmonary congestion), wheezing
Abdominal exam: liver location, liver size, ascites
Enlarged liver and ascites = CHF
Peripheral pulses: rate, rhythm, volume, character, cap. refill
Weak/absent femoral pulses = Coarctatcion of the aorta

31
Q

cardiac exam

A

Auscultate over tricuspid, pulmonary, mitral, and aortic areas with bell and diaphragm while patient is supine, seated, standing
Innocent murmurs: Sensitive (changes with child’s position or respiration), Short duration, Single (no clicks or gallops), Small, Soft, Sweet (not harsh sounding), Systolic)

Red flags:
Holostyolic murmur, Grade 3 or higher, Harsh, Abnormal S2, Max intensity at upper left sternal border, Systolic click, Diastolic murmur, Increased murmur with standing

32
Q

Heart Murmurs: Physical Exam Findings

A

Abnormal growth

  • Feeding difficulties could be a sign of cardiac disease in newborns and infants
  • Genetic disorders may increase risk of delayed growth and CHD

Abnormal vital signs

  • Arrythmia, tachycardia, hypoxia, and tachypnea (structural heart disease)
  • Blood pressure discrepancy between upper and lower limbs (coarctation of the aorta)

Adventitial breath sounds
- Wheezing may be associated with cardiac asthma; rales may be associated with pulmonary congestion secondary to CHF

Chest contour maldevelopment of the sternum
- Defective segmentation of the sternum may occur in children with CHD

Dysmorphic features
- Certain genetic or congenital conditions increase risk of CHD

33
Q

Abnormal S2

A

Wide-split fixed S2 with atrial septal defects; abnormal S2 in other types of CHD

34
Q

Capillary refill

A

Normal <2-3 seconds, delay may indicate poor perfusion secondary to diminished cardiac output

35
Q

Displaced point of maximal impulse; precordial impulses (heaves, lifts, thrills)

A

Possible structural abnormality or ventricular enlargement

36
Q

Edema

A

CHF

37
Q

Left-sided precordial bulge

A

Cardiac enlargement

38
Q

S3 or S4

A

Structural heart disease; (S3 can be normal but disappears when pt is upright)

39
Q

Substernal heave

A

Right ventricular hypertension

40
Q

Systolic ejection click

A

Semilunar valvular stenosis

41
Q

Weak or absent femoral pulses

A

Coartaction of the aorta

42
Q

Ascites

A

CHF

43
Q

Hepatomegaly

A

CHF

44
Q

Location of liver signals abdominal situs

A

High rate of CHD with abdominal situs