Paediatric Orthopaedics Flashcards

1
Q

Pain medication in children

A

Codeine and tramadol are not used in children as they are unpredicatble in their metabolism

Aspirin is not used in children younger than 16 yo due to Reye’s syndrome

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2
Q

Differentials for a limp

A

0 - 4 years:

  • Transient synovitis
  • Septic arthritis
  • Developmental dysplasia of the hip

5 - 10 years

  • Perthe’s disease
  • Transient synovitis

10 - 16 yo

  • Juvenile idiopathic arthritis
  • Slipper upper femoral epiphysis
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3
Q

Presentation of hip pain

A
Limp 
Not bearing weight on affected side 
Delayed walking 
Pain 
Swollen and tender joint
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4
Q

Red flags for hip pain

A
Children under 3 yo 
Fever 
Waking at night with pain 
Weight loss and anorexia 
Night sweats
Fatigue 
Persistent pain 
Stiffness in the morning 
Swollen or red joint
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5
Q

When to urgently refer a limping child

A
Children under 3 yo 
Child > 9 yo with a restricted or painful hip 
Unable to weight bear 
Neurovascular compromise 
Severe pain 
Suspicion of abuse
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6
Q

Investigations for hip pain

A
Bloods - CRP, FBC
Xray - fracture, SUFE
USS - effusion 
Joint aspiration - sepsis 
MRI - osteomyelitis
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7
Q

Causative organisms for septic arthritis

A
Staphylococcus aureus - most common 
Neiserris gonorrhoea 
Streptococcus pyogenes 
Haemophilus influenzae 
E. Coli
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8
Q

Management of septic arthritis

A

Antibiotics dependent on joint aspirate culture for 3 - 6 weeks

Surgicsl drainage and washout

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9
Q

Transient synovitis pathophysiology

A

Inflammation of the synovial membrane associated with a recent viral upper respiratory tract infection

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10
Q

What age range does transients synovitis typically affect

A

3 - 10 yo

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11
Q

Presentation of transient synovitis

A
  • after viral URTI - coryzal symptoms
  • limp
  • refusal to weight bear
  • joint pain
  • low fever
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12
Q

Management of transient synovitis

A

Exclude septic arthritis
Symptomatic relief - analgesia

Can be managed in primary care if 3 - 9yo and limp present within 48 hours whilst systemically well

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13
Q

Prognosis of transient synovitis

A

Significant improvement after 24 - 48 hours

Symptoms fully resolve within 1 - 2 weeks without any lasting problems

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14
Q

Perthes disease pathophysiology

A

Avascular necrosis of the femoral head

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15
Q

Who does perthes disease typically affect?

A

Children aged 4 - 12 yo

Commonly between 5 - 8 yo boys

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16
Q

Complication of perthes disease

A

Early osteoarthritis of the the hip

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17
Q

Presentation of perthes disease

A

Pain in the hip or groin
Limp
Restricted hip movements
Referred pain to the knee

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18
Q

Investigations for perthes disease

A

Bloods - normal
Technetium bone scan
MRI scan

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19
Q

Management of Perthes disease

A

Under 6 yo mild to moderate disease - supportive:

  • bed rest and analgesia
  • traction
  • crutches
  • physiotherapy
  • regular xrays - monitoring

Severe disease > 6yo - surgery

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20
Q

Slipper upper femoral epiphysis pathophysiology

A

Head of the femur is dispaced along the growth plate

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21
Q

Who does SUFE typically affect

A

Obese boys aged 8 - 15yo

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22
Q

Presentation of SUFE

A
  • hip, groin, thigh or knee pain
  • restricted hip movement - inability to internally rotate
  • painful limp
23
Q

Investigations for SUFE

A

Bloods - usually normal
Technetium bone scan
CT scan
MRI scan

24
Q

Management of SUFE

A

Surgery - cannulated screws

25
Pathophysiology of osteomyelitis
Infection of the bone or bone marrow
26
Causative organism of osteomyelitis
Staphylococcus aureus
27
Risk factors for osteomyelitis
- open bone fracture - orthopaedic surgery - immunocompromised - sickle cell anaemia - salmonella - HIV - TB
28
Presentation of osteomyelitis
``` Painful limp Refusal to weight bear Pain Swelling Tenderness May have fever if acute ```
29
Investigations for osteomyelitis
``` Joint xray - may be normal MRI - gold standard Bone scan Bloods - FBC, CRP Blood culture Bone marrow aspiration or bone biopsy ```
30
Management of osteomyelitis
Antibiotics - flucloxacillin | Surgical drainage and debridement
31
Features of osteosarcoma
- typically affects 10 - 20 yo | - commonly affects the femur, tibia and humerus
32
Presentation of osteosarcoma
Persistent bone pain Particularly worse at night Bone swelling - palpable mass Restricted joint movements
33
Investigations for osteosarcoma
- urgent direct access xray within 48 hours of unexplained bone pain or swelling - bloods - ALP - Staging CT scan - PET or bone scan - Bone biopsy
34
Signs of osteosarcoma on xray
Sun burst appearance - fluffy due to periosteal reaction
35
Management of osteosarcoma
Surgical resection Adjuvant chemotherapy MDT
36
Developmental hip dysplasia pathophysiology
Structural abnormality of the hips causing instability and an increased tendency to sublux or dislocate as well as an abnormal gait and weakness
37
Risk factors for developmental hip dysplasia
- Family history - breech presentation - multiple pregancy
38
DDH screening
USS scan of hip if risk factors present | Neonatal exam - ortalani and barlow tests
39
Signs of DDH
Different leg lengths Restricted hip abduction either unilateral or bilateral Difference in knee level when hips are flexed Clunking during the Ortolani and Barlow test
40
Ortolani test
Dislocate anteriorly whilst hips and knees are flexed by abducting
41
Barlow test
Whilst hips are adducted and flexed at 90 degrees. Gentle downwards pressure to see if hip dislocates posteriorly
42
Investigation for DDH
USS of hip
43
Management of DDH
Pavlik harness for 6 - 8 weeks - < 6 months yo Surgery if > 6 months with hip spica cast
44
Pathophysiology of rickets
Defective bone mineralisation causing soft bones due to a vitamin D or calcium deficiency Calcium deficiency stimulates parathyroid hormone which stimulates bone resorption
45
Presentation of rickets
``` Lethargy Bone pain Swollen wrists Bone deformity Poor growth Dental problems Pathological fractures ```
46
Bone deformities in rickets
Bowing of the legs - valgus deformity Knock knees - varus deformity Rachitic rosary - lumps along the chest where ribs expand at the costochondral junctions Craniotabes - soft skull, frontal bossing and delayed closure of the sutures Delayed teeth
47
Risk factors for rickets
``` Darker skin Low exposure to sun light - muslim women Live in colder climates Indoors CKD ```
48
Investigations of rickets
Bloods - vitamin D, calcium, PTH, ALP, phosphate, FBC, CRP, LFTs, U+Es, TFTs Anti TTG - coeliacs (malabsorption) Autoimmune and rheumatoid tests Xray
49
Management of rickets
Vitamin D and calcium supplements
50
Osgood schlatters disease pathophysiology
Inflammation of the tibial tuberosity where the patella ligament inserts due to multiple small avulsion fractures
51
Who typically gets Osgood schlatters disease
10 - 15 year old boys
52
Presentation of Osgood Schlatters disease
Normally unilateral anterior knee pain Pain exacerbated by activity Visible lump below the knee - hard and non tender (initially may be tender)
53
Managament of Osgood schlatters
- reduce physical activity - RICE - NSAIDs Stretching and physiotherapy