Paediatric knee Flashcards

Osteochondral dessicans osgood schlatters disease sliding- larsen- johnansen

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1
Q

What is osteochondritis dissecans?

A
  • A pathologic lesion affecting the articular cartilage and subchondral bone w a variable clinical patterns
  • Juvenile form
    • occurs 10-15 while physis open
  • Adult form ( skeletal mature)
  • location
    • knee
      • _posterolateral aspect of Medial femoral condyle 7_0%
    • capitellum of humerus
    • talus
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2
Q

What is aetiology of osteochondritis dissecans?

A
  • Hereditary
  • Traumatic
  • vascular
    • cause of adult form
  • pathoanatomical cascade
    • softening of overlying casrtilage w intact artcular surface
    • early articular cartilage separation
    • partial detachment of lesion
    • osteochondral separation w loose bodies
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3
Q

What is the prognosis of osteochondritis dissecans juvenile and adult form?

A
  • Juvenile
    • younger age correlates with better prognosis
    • opend distal femoral physes best predictor of non op mx
  • location
    • **lesions in lateral femoral condyle & patella = **poorer prognosis
  • appearance
    • synovial fluid behind lesion on MRI = poor prognosis
  • Adult
  • worse prognosis
  • usually symptomatic & -> DJD if left untx
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4
Q

What is the classification of osteochondritis dissecans?

A
  • Clanton
  • Type 1= depressed osteochondral fx
  • type 2= fragment attached by osseous bridge
  • type 3= detached non-displaced fragment
  • type 4= displaced fracture
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5
Q

What is the presentation of a pt with osteochondritis dissecans?

A

Symptoms

  • Pain
    • Activity related, vague and poorely localised
  • Recurrent effusions

O/E

  • Localised tenderness
  • stiffness
  • swelling
  • Wilson’ test
    • Pain on internal rotation during extension of the knee between 90o and 30o then relief w tibial external rotation
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6
Q

What is seen on imaging of osteochondritis dissecans?

A

Xray

  • lytic area in medial femoral condyle

MRI

  • Size of lesion
  • signal intensity surrounding the lesion
  • presence of loose bodies
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7
Q

What is the tx of osteochondritis dissecans?

A

Non operative

  • restricted weight bearing & bracing
    • stable lesions in children open physis
    • 50-75% will heal without fragmentation

​Operative

  • Diagnostic arthroscopy
  • Microfracture
    • ​stable lesion
    • tap awl to depth of 1-1.5cm below articular depth
    • -> formation of fibrocartilage tissue
    • improves outcomes in skeletaly immature pts
    • NWB 4-6 wks with CPM
  • Fixation of unstable lesion >2cm
    • 85% healing rates in juvenile
    • cannulated screw/herbert screw/ k wire
  • **Chondral resurfacing **
    • Lesions >2cmx2cm
    • osteochondral grafting
    • arthroscopy if lesion < 3cm, arthrotomy if >3cm
    • allograft plugs
    • Osteochondral autograft transferal system = OATS/ mosacioplasty
  • Knee arthroplasty
    • pts > 60yrs
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8
Q

What is Osgood schlatter’s disease?

A
  • Osteochondrosis or traction apophysitis of tibial tubercle
  • >boys
  • 12-15y boys and girls 8-12 yrs
  • location
    • bilateral 20-30%
  • Risk factors
    • jumpers or sprinters
  • Pathophysiolgy
    • stress from extensor mechanism
  • Prognosis
    • self limiting but doesn’t reolve until growth has halted
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9
Q

Describe the anatomy of the tibial tubercle?

A
  • A secondary ossification centre
  • <11 yrs tubercle is cartilaginous
  • age 11-14 apophysis forms
  • age14-18 apophysis fuses with tibial epiphysis
  • >18 yrs epiphysis and apophysis is fused to rest of tibia
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10
Q

What is seen on imaging the tibial tubercle?

A

Xray

  • Irregularity and fragmentation

MRI

  • soft tissue swelling
  • thickened oedema of inferior patella tendon
  • fragmentation and irregularity of ossification centre
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11
Q

What is the presentation of Osgood shlatter’s disease?

A
  • Pain on anterior aspect of knee
  • exacerbated by knee

O/E

  • Enlarged tibial tubercle
  • tenderness over tubercle
  • pain on resisted knee extension
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12
Q

What is the tx of Osgood shlatter’s disease?

A

Non operative

  • Nsaids, rest, ice, activity modification, strapping/sleeves to decrease tension on the apophysitis and quad strengthening
    • first line of tx
    • 90% have complete resolution
  • ​cast immobilisation 6 wks
    • ​severe symptoms not responding to above
    • can -> quads wasting

Operative

  • Osscile excision
    • refractory cases 10%
    • in skeletally mature pts with ongoing symptoms
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13
Q

What is Sinding- Larson-Johansson syndrome?

A
  • Overuse injury causing anterior knee pain at the inferior pole of patella
  • more common in adolescence
  • location
    • patellar tendon insertion at inferior pole of patella
  • pathphysiology
    • chronic injury
    • similar pathogenesis to Osgood-schlatter’s disease
    • overuse causes traction apophysitis
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14
Q

What is the classification of Sinding- Larson-Johansson syndrome?

A
  • Blazina
  • Stage 1- pain occurs after activity
  • Stage 2- pain present while preforming activity and persists after activity
  • Stage 3- pain affecting/limiting function during activity
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15
Q

What is the presentation of Sinding- Larson-Johansson syndrome?

A
  • Insidious onset of pain on anterior aspect of knee after or during activity

O/E

  • tenderness over inferior patella
  • swelling
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16
Q

What is seen on imaging of Sinding- Larson-Johansson syndrome?

A
  • Xray
    • Normal
    • spur to inferior pole
  • MRI
    • Inflammation best seen on T2 sagitals
    • bony spurs on t1
17
Q

What is the tx of Sinding- Larson-Johansson syndrome?

A

Non operative

  • activity modification, nsaids, physio
    • mainstay of tx
    • usually self limiting process

Operative

  • Debridement of damaged tissue/stimulation of healing response
    • ​if refractiory to consx tx